<?xml version="1.0"?>
<rss version="2.0">
   <channel>
      <title>My fancy padlet by briannon mcneice</title>
      <link>https://padlet.com/briannonmcneice21/o3tvcug9vmkbfpxm</link>
      <description></description>
      <language>en-us</language>
      <pubDate>2022-11-07 17:40:37 UTC</pubDate>
      <lastBuildDate>2022-11-07 18:24:56 UTC</lastBuildDate>
      <webMaster>hello@padlet.com</webMaster>
      <image>
         <url></url>
      </image>
      <item>
         <title>Diabetes </title>
         <author>briannonmcneice21</author>
         <link>https://padlet.com/briannonmcneice21/o3tvcug9vmkbfpxm/wish/2373620616</link>
         <description><![CDATA[<div>Autosomal Recessive; mutation of <strong>MODY2, glucokinase (GCK)</strong><br><mark>Etiology</mark>: A group of diseases that result in too much sugar in the blood (high blood glucose). In all cases, sugar builds up in the bloodstream. This is because the pancreas doesn't produce enough insulin. Both type 1 and type 2 diabetes may be caused by a combination of genetic or environmental factors.<br><mark>Pathology</mark>: The pathophysiology of diabetes involves plasm concentrations of glucose signaling the central nervous system to mobilize energy reserves. It is based on cerebral blood flow and tissue integrity, arterial plasma glucose, the speed that plasma glucose concentrations fall, and other available metabolic fuels.<br><mark>Phenotype</mark>: Most phenotypes include the concept of 'abnormal glucose', typically defined as random glucose greater than or equal to 200 mg/dl, fasting glucose greater than or equal to 126 mg/dl, or a hemoglobin A1c (HbA1c) laboratory test result of 6.5% or more.</div>]]></description>
         <enclosure url="" />
         <pubDate>2022-11-07 18:24:29 UTC</pubDate>
         <guid>https://padlet.com/briannonmcneice21/o3tvcug9vmkbfpxm/wish/2373620616</guid>
      </item>
   </channel>
</rss>
