https://padlet.com/shankarmunusamy1/bb en-us 2016-11-03 08:07:36 UTC 2016-11-03 08:12:55 UTC hello@padlet.com https://padlet.com/shankarmunusamy1/bb/wish/134999519
Q1) bronchspasm , hypoglyccemia , hypotension, decrease pulse rate, CNS toxicity 

Q2) excessive beta-adrenergic blockade

Q3) Because there are B-adrenoreceptors in the brain CNS 

Q4) ECG , Electrolyte ( BUN, ABG, creatinin ,  Glucose ) 

Q5) -Maintain open airway and assess ventilation 
 - Tx coma and seizure, hypothermia ,  
- Tx Bradycaria w/ glucogan 
- Tx bronchospasm w/ nebulizer bronchodialator

Q6 ) 
a) Glucagon
b) epinehrin 
c) high dose of insulin + glucose therapy 
 a,b,c -> sx

* activated charcol orally 

]]> 2016-11-03 08:18:24 UTC https://padlet.com/shankarmunusamy1/bb/wish/134999519 https://padlet.com/shankarmunusamy1/bb/wish/134999814 1)hypotension ,bradycardia, AV block, asystole, cardiogenic shock 
CNS (confusion ,coma, respiratory arrest, bronchospasm ,hyperkalemia, hypoglycemia
2)
-decrease myocardial conductivity and conduction
-if lipophilic =go to the CNS =cause seizure/coma
-partial B agonist=can lead to tackycardia and HTN
-satolol=ventricular fibrillation ,tpd
QT prolongation B1,B2,alpha agonists may leads to vasodilation and hypotension
3)
lipid solubility of some agents and penetrate CNS causing seizure and coma
4) vital signs ,ECG ,at least 6hrs after infection
5)
-stabilize pt by maintain open airway-assws

]]> 2016-11-03 08:20:36 UTC https://padlet.com/shankarmunusamy1/bb/wish/134999814 https://padlet.com/shankarmunusamy1/bb/wish/135001686 .]]> 2016-11-03 08:35:23 UTC https://padlet.com/shankarmunusamy1/bb/wish/135001686