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      <title>Serotonin: depression and SSRIs by </title>
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      <description>Regulating serotonin levels in depression patients</description>
      <language>en-us</language>
      <pubDate>2016-11-03 18:10:40 UTC</pubDate>
      <lastBuildDate>2025-10-26 03:53:06 UTC</lastBuildDate>
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         <title>The &quot;Feel good&quot; chemical</title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135200359</link>
         <description><![CDATA[<div>Serotonin is a neurotransmitter usually referred to as the feel good chemical. </div>]]></description>
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         <pubDate>2016-11-03 18:33:24 UTC</pubDate>
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         <title>how does 5-HT link with depression?</title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135218105</link>
         <description><![CDATA[<div> Depression which is also known as major depressive disorder (MDD) is characterised by prolong helplessness or discouragement about the future. <br>The serotonin (5-HT) hypothesis of depression proposes that it might be caused by the decrease of serotonin neurotransmission..<strong> Coppen A.(1967)</strong> published an article:<strong> The biochemistry of affective disorders </strong>in which he says<strong> "</strong> the weight of evidence, although it is by no means conclusive, suggests that biochemical changes are most important in the aetiology of affective disorders." He backed up this hypothesis by a series of studies on brain monoamines and proposed that certain drugs (MAO) manipulate neurotransmission of 5-HT. </div>]]></description>
         <enclosure url="https://www.ncbi.nlm.nih.gov/pubmed/4169954/" />
         <pubDate>2016-11-03 19:21:25 UTC</pubDate>
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         <title>SSRIs</title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135233416</link>
         <description><![CDATA[<div>Most medication work on synaptic level, by increasing or decreasing the amount of available neurotransmitter in the synapse, this means that they can make it more or less likely that a message will be delivered to the next cell. Fluoxetine (Prozac) is a good example of an SSRI, which blocks the re-uptake of serotonin in the synaptic cleft thereby increasing the availability of serotonin in the synapse.</div>]]></description>
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         <pubDate>2016-11-03 20:22:23 UTC</pubDate>
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         <title>Genetics</title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135245559</link>
         <description><![CDATA[<div>A Study article suggesting the possibilities that genes passed on from parents may also have a say in whether you're more or less likely to suffer from depression. <strong>Caspi A. et al -Influence on life stress on depression.</strong> The article suggests that people that have either one or two copies of short allele 5-HT promoter polymorphism exhibited more depressive symptoms in relation to those people showed homozygous for the long allele. </div>]]></description>
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         <pubDate>2016-11-03 21:22:03 UTC</pubDate>
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         <title>Current issues with anti-depressants </title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135247711</link>
         <description><![CDATA[<div>In recent news, People that have been prescribed SSRIs have said that the many side effects of these drugs are too extreme to be ignored. Side effects of SSRIs include dependence on anti depressants when introduced at high doses.</div>]]></description>
         <enclosure url="http://www.bbc.co.uk/news/health-37682355" />
         <pubDate>2016-11-03 21:35:01 UTC</pubDate>
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         <title></title>
         <author>solomongeorge1996</author>
         <link>https://padlet.com/solomongeorge1996/elrkp34m0dig/wish/135250935</link>
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         <pubDate>2016-11-03 21:55:32 UTC</pubDate>
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