The Sympathetic also known as the Adrenergic, fight, flight, fright and the Parasympathetic, the rest and digest, AKA, Cholinergic.

The Sympathetic- the adrenal medulla releases epinephrine (adrenaline) and some norepinephrine. 

The Parasympathetic- releases (ACH) Acetylcholine.

Sympathetic releases Epi from the organ adrenal medulla, norepinephrine is released from  postganglionic NT, ACH is released at preganglionic nerve fibers.

Parasympathetic- ACH released from postganglionic and from ganglia.

Sympathetic Adrenergic receptors:

Alpha-1 located on the glands, muscles, eyes, most arteries and veins. Alpha-2 located at nerve endings.

Beta-1, located on the heart.

Beta-2, located in smooth muscle, uterus, coronary and skeletal muscle, blood vessels.

Parasympathetic (cholinergic) -Alpha-2 located in smooth muscle ( negative feedback)

The adrenergic nerve ending takes up the amino acid tyrosine and through a series of enzymatic forms dihydroxyphenylalanine (DOPA). Then the adrenal medulla releases the hormone epi, which travels in the blood also stimulates the adrenergic NE binds to adrenergic receptors and produces the effects that are associated with sympathetic. 

In the parasympathetic-the choline is taken up into the nerve ending and with acytyl-CoA forms ACH. ACH is then stored in the vesicles and when stimulated is released into the synaptic cleft to stimulate muscarinic receptors on the internal organs and glands, Nn on autonomic ganglia and Nm on skeletal muscle. ACH is inactivated enzymatically by acetylcholinesterase.

The drug classification of the ANS under each classification, identify which receptors are interacted with then include the action that occurs. 
Alpha-1 Adrenergic Agonists, ephedrine, phenylephrine (Neo-Synephrine)-contraction of smooth muscles, blood vessel constriction, vasoconstriction of nasal passages, decongestant/  Adrenergic Agonist, epinephrine (Adrenalin)Stimulate all alpha and beta receptors and exception, stimulate the cardiac, bronchodilator.
Alpha-1 Adrenergic Blocker/ prazosin (Minipress)- terazosin (Hytrin)cause relaxation of the muscle, vasodilation of blood vessels, decrease b/p, relaxes smooth muscle of uterus and inc. urine flow also included doxazosin (Cardura).
     Beta-1 Adrenergic Agonist/ dobutamine-stimulates d-1 receptors, stimulates beta-1 in the heart, increases force contraction and cardiac output, stimulates alpha-1 receptors causing vasoconstriction.
  Beta-2 Adrenergic Agonist/ formoterol (Foradil)/ albuterol (Proventil/Ventolin)/ terbutaline (Brethine)-stimulates beta-2 receptors, relaxation of the smooth muscle of the lungs leads to bronchodilation, relaxation of smooth muscle in the uterus.
Beta Blocker/ Nonselective/ nadolol (Corgard),propranolol (Inderol), timolol (Blocadren)-blocks beta-1 and beta-2 receptors, decreases force of contraction, neg. inotropic,decrease h/r, decrease conduction, decrease in cardiac output aslo decrease b/p, can cause bronchoconstriction.
Selective / atenolol (Tenormin),metoprolol (Lopressor)- beta-1 receptor is blocked, dec. force of contraction, dec. cardiac output, decrease heart rate, dec. in cardiac output and decrease in b/p.
Direct Acting Cholinergic Agonist/ acetylcholine, pilocarpine (Pilocar),  bethanechol (Urecholine)-they act upon the neurotransmitter ACH.
Indirect Acting Cholinergic Agents/ neostigmine (Prostigmin)- increase the availabIlity of ACH at teh recpetors.
  Anticholinergic/ atropine, dicyclomine (Bentyl)-cause mental loss, block the action of ACH, urinary inc.,overactive bladder, and certain types of poisoning.
Skeletal Muscle Relaxants/ Peripherally Acting - slows down CNS functions
  Depolarizing Agent/succinylcholine-bind and activate teh ACH receptor causes muscle contraction.
Non-depolarizing Agent/ rocuronium bromide (Zemuron)-bind directly to nicotinic receptors on the postsynaptic membrane and blocks ACH, so the motor endplate cannot depolarize.
Direct Acting/ dantrolene (Dantrium)-bind to cholinergic receptors, activating them.
Centrally Acting/ cyclobenzaprine (Flexeril), carisoprodol (Soma),methocarbamol (Robaxi) orphenadrine citrate (Norflex), chlorzoxazone  (Parafon Forte DSC), metaxalone (Skelaxin)-mimic ACH, contracts smooth muscles, dilates blood vessels. 
Respiratory Drugs/cause lung disease.   Mast Cell Stabilizers
cromolyn Sodium (Intal® / Nasalcrom®)-causes low b/p, itching, hives,breathing issues
Bronchodilators/ Sympathomimetics “B-2 agonist”  (Short and long acting)/ albuterol (Proventil®/Ventolin®) formoterol   (Foradil®)                                                                          terbutaline (Brethine®)                                                               salmeterol (Serevent )- they bind to muscarinic receptors and block action of ACH, 
Xanthine Derivatives/ theophylline (Theo-Dur) aminophylline (Paladrol)-they inhibit tissue of phosphodiesterase increase cellular AMP, mild  diuresis.
  Cysteinyl Leukotriene Receptor Antagonist/  montelukast       (Singulair®)-receptor antagonist, blocks the action of leukotriene D4 in the lungs.
Anticholinergic/  ipratropium bromide (Atrovent®)-inhibits vagally-mediated reflexes by antagonizing the action of ACH.
Combination Bronchodilator/ ipratropium bromide and albuterol sulfate (Combivent®)-stimulation of B-2 by agonist causes bronchodilation by activation of the B2- AR found on the surface of the smooth muscle
Antihistamines/ Generation I (sedating)/ brompheniramine (Dimetapp®) chlorpheniramine (Chlor-Trimeton®) clemastine (Tavist®) diphenhydramine (Benadryl®) hydroxyzine (Atarax®) promethazine (Phenergan®) azelastine (Astelin® or Astepro®)- easily cross the bbb into the cns and antagonize h-1 receptors leading to a different therapeutic effect.
Generation II (non-sedating) loratidine (Claritin®) fexofenadine (Allegra® cetirizine (Zyrtec®)- blocks the action of histamine.
Corticosteroid (asthma or allergic rhinitis), budesonide (Rhinocort® or Pulmicort®),mometasone (Nasonex® or Asmanex®) fluticasone (Flonase® or Flovent®) flunisolide (Nasalide®), triamcinolone (Nasacort®/Azmacort®)-serve as a receptor complex, can stimulate or inhibit the synthesis of specific proteins.
Antitussives/  Narcotic hydrocodone (Hycodan®) codeine/guaifenesin (Robitussin AC®)-inhibit the region for coughing reflex arc.
Nonnarcotic/ dextromethorphan/guaifenesin (Robitussin DM®) benzonatate  (Tessalon Perles®)- desentising the tissues of the lungs and pleura  
Decongestants/ oxymetazoline (Afrin®)/ pheniramine (Dristan®) phenylephrine (Neo-Synephrine®)pseudoephedrine (Sudafed®)- activation of postjunctional alpha-aldrenergic receptors.
Expectorants/ guaifenesin (Robitussin®, Mucinex®, Organidin®) -reduce the viscosity of tenacious secretions by irritating the gastric vagal.
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