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      <title>My phenomenal wall by Lalaine Biboso</title>
      <link>https://padlet.com/lalaine65biboso/biotech</link>
      <description>Made with swagger</description>
      <language>en-us</language>
      <pubDate>2018-01-29 03:37:41 UTC</pubDate>
      <lastBuildDate>2025-09-24 17:15:22 UTC</lastBuildDate>
      <webMaster>hello@padlet.com</webMaster>
      <image>
         <url></url>
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      <item>
         <title>Vergara,Mercado,Cadiz</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187634</link>
         <description><![CDATA[<div>1) No</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:53:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187634</guid>
      </item>
      <item>
         <title>Buladaco, Cerado,  Flores</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187645</link>
         <description><![CDATA[<div>No</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:53:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187645</guid>
      </item>
      <item>
         <title>Arcilla,Arduo,Olar</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187705</link>
         <description><![CDATA[<div>Answer:Yes<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:54:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187705</guid>
      </item>
      <item>
         <title>Krystle Ann Paguray &amp; Princess Kyla Endaya</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187718</link>
         <description><![CDATA[<div>Yes</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:54:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187718</guid>
      </item>
      <item>
         <title></title>
         <author>jhonbertfacurib27</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187782</link>
         <description><![CDATA[<div>Ej Carl Banlasan<br>Jefferson Llamera<br>Patrick James Lagang<br><br><br>What if the organisms is dead? Is the cells still alive?<br>Yes<br><br>Yehey<br><br><br>PS. We borrowed Jhonbert's phone and account</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:55:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187782</guid>
      </item>
      <item>
         <title>Baluso, mato, kano</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187789</link>
         <description><![CDATA[<div>No</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:55:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187789</guid>
      </item>
      <item>
         <title>Fajardo</title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187923</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:58:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187923</guid>
      </item>
      <item>
         <title>Fernandez, galo, pertimos</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187924</link>
         <description><![CDATA[<div>Answer:no</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:58:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187924</guid>
      </item>
      <item>
         <title>Fajardo,Nikko</title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187927</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:58:43 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187927</guid>
      </item>
      <item>
         <title></title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187933</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:58:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187933</guid>
      </item>
      <item>
         <title>Jhonbert Facurib &amp; Ramer Kent. Fagutao </title>
         <author>kentfagutao</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268187967</link>
         <description><![CDATA[<div>What if the organism is dead? Is the cell still alive?&nbsp;<br>Yes<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 05:59:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268187967</guid>
      </item>
      <item>
         <title></title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188010</link>
         <description><![CDATA[<div>Fajardo, Nikko G.<br>Sabelita, Francine P.<br>Valdez, Leana N.<br>1. NO</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:00:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188010</guid>
      </item>
      <item>
         <title></title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188025</link>
         <description><![CDATA[<div>Fajardo, Nikko G.<br>Sabelita, Francine P.<br>Valdez, Leana N.<br>1. NO </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:00:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188025</guid>
      </item>
      <item>
         <title></title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188035</link>
         <description><![CDATA[<div>Fajardo, Nikko G.<br>Sabelita, Francine P.<br>Valdez, Leana N.<br>1. No&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:00:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188035</guid>
      </item>
      <item>
         <title>Mahinay,magallano,CUBETA</title>
         <author>gclikerz2134</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188140</link>
         <description><![CDATA[<div>NO</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:02:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188140</guid>
      </item>
      <item>
         <title>Yes delsreyes and coscos and monngi</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188244</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:04:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188244</guid>
      </item>
      <item>
         <title>hihihihi</title>
         <author>luckygrande05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268188331</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 06:05:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268188331</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL VOCABULARY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268193570</link>
         <description><![CDATA[<div>Names:<br>Alberto,Achilles Alexis R.<br>Berame,Bernadette Sue B.<br><br><strong>If you hear a new vocabulary, just think of a HOUSE</strong>.<br><br><strong>Ribosomes</strong>:<br>it serves as the kitchen of the house because it is where the food are processed and made of.<br><strong>Lysosomes</strong>:<br>it is just like the comfort room of a house. it is where waste of a human throw out.<br><strong>Centrioles</strong>:<br>is the bedroom of the house it is where you can produce your own kind. <br><strong>Endoplasmic Reticulum</strong>:<br>it is like the living room of the house. It is where visitors are entertained and served properly.<br><strong>Golgi Bodies</strong>:<br>The dressing room of the house. it is where you change your clothes or where you wrap yourself with clothes.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 07:10:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268193570</guid>
      </item>
      <item>
         <title>Plant and Animal Cell y</title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268193734</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 07:11:54 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268193734</guid>
      </item>
      <item>
         <title>Plant and Animal Cell Vocabulary</title>
         <author>cimagalamary</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268193858</link>
         <description><![CDATA[<div>Bantes, Jannah H.<br>Hamajo, Mika C.<br><br>When you hear the new vocabulary, just think of a "CASTLE."<br><br>Endoplasmic Reticulum (smooth and rough)- hallways that transport cellular material.<br>Ribosome- workers who carry around everything.<br>Golgi Body- warehouse of the castle that stores certain packages.<br>Lysosome- a maid who cleans up things and breaks down cell materials.<br>Centrioles- supervisors that organize things within the castle.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 07:13:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268193858</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL VOCABULARY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268211584</link>
         <description><![CDATA[<div>Lorania,Chorena grace<br>Sangatanan, Shanlee<br><br>GREEK MYTHOLOGY AS CELL ORGANELLS<br><br><strong>Nucleus:</strong><br><strong>Zues </strong>is th king of all gods so as the nucleus because they command the others what they're supposed to do<br><br><strong>Centrioles:</strong><br><strong>Hephaestus </strong>is the blacksmith and since metal can be used as a tool for dividing, we thought that he and centrioles do the same thing(building divisions).<br><strong>Lysosomes</strong>:<br>the <strong>Underworld </strong>could also be a lysosome if only the place is an organelle because it is where dead souls are sent.<br><br><strong>Endoplasmic reticulum:<br>Hermes </strong>is the messenger of the family and so as the E.R. and they could make a good tandem.<br><br><strong>Ribosomes</strong>:<br>we know all about the greek mythology is not true and losing our belief would make the whole thing vanish, so <strong>belief </strong>serves as the ribosomes because without it, everything loses energy and its life...</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 10:16:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268211584</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL VOCABULARY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268211897</link>
         <description><![CDATA[<div>Names:<br>Belicena, Michael Francis P.<br>Cagungao, Angela Lester<br><br><strong>if you hear a new vocabulary, just think of a HOUSE.<br><br>Lysosome:<br></strong>it like a trash can of the house.It where garbage or waste thrown.<br><strong>Centrioles:<br></strong>a centrioles can be a clothing rack because it organize cell division in a cell.<br><strong>Vacoule<br></strong>this part of the cell can be a storage room.A house and a vacoule store materials needed for the structure they are in.<br><strong>Mitochondria<br></strong>a mitochondria can be compare as the electricity in a house.Appliances in home need energy to do work.<br><strong>Endoplasmic Reticulum<br></strong>can be a hallway of a house. A rough ER can be compared like a hallway with different rooms branching on it.The smooth ER can be a hallway in a house that only leads from point A to point B.<br><strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 10:19:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268211897</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268214161</link>
         <description><![CDATA[ ]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 10:51:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268214161</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL VOCABULARY</title>
         <author>christiansalas68</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268215136</link>
         <description><![CDATA[<div>Christian Ian Charles R. Salas<br>Alvin Jay A. Deleverio<br>Chrys Jan S. Benjamin<br><br>When you hear the new vocabulary just think of a mall.<br><br><strong>Endoplasmic Reticulum</strong>:<br>&nbsp; &nbsp; Its just the workers that delivers foods (nutrients) to the supermaket.<br><strong>Lysosomes</strong>:<br>&nbsp; &nbsp; Its just the area were do the workers throw the garbage of the mall. (Garbage disposal of cell)<br><strong>Mitochondria:<br>&nbsp; &nbsp;</strong>Mitochondria can be compared as the generator or the electricity of the mall or the cell ,because it gives energy so all parts of it will function well.<br><strong>Ribosomes:</strong><br>&nbsp; &nbsp; Can be the workers that delivers things to any parts of mall or the cell.<br><strong>Golgi Bodies:</strong><br>&nbsp; &nbsp;Its like the warehouse of the mall or the cell, where they stores things such us can goods, clothes , and more.<br><strong>Centrioles:</strong><br>&nbsp; Centrioles can be the managers of every department in the mall, because they organize the divisions of the mall or the cell.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 11:02:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268215136</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268218113</link>
         <description><![CDATA[<div>NAMES: <br>NATIVIDAD, DENIEL DAVE<br>JUANICO, DJ CHRISTIAN<br>ENOJALES, DESIRE FAITH<br><br>ANALOGY: KINGDOM<br>If you hear a new vocabulary, just think of a KINGDOM.<br><br>Ribosomes - Slave<br> It follows instruction of the nucleus the king of the kingdom to make protein together with Endoplasmic Reticulum.<br><br>Centrioles- Commander<br>It is the commander when the soldier of the cell acquire a territory to form another kingdom.<br><br>Endoplasmic Reticulum- Transporter<br>It transport the protein that ribosomes make, It will deliver to Golgi Apparatus.<br><br>Golgi Apparatus- Market<br>After the Endoplasmic Reticulum delivers the protein the Golgi Apparatus package it into small membrane sacs called vesicles.<br><br>Lysosomes- Soldiers<br>It is the soldier of the cell that  eliminates the foreign bodies that infiltrate the cell</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 11:48:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268218113</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL VOCABULARY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268218189</link>
         <description><![CDATA[<div>Cabiles, Zyrreg Zhyller<br>Urdaneta,&nbsp; Caryl Jean<br>Cutamora,&nbsp; Mizpah Gabrielle<br><br>Analogy: COMPUTER<br>If you hear the word "cell" think of a computer<br><br>LYSOSOME: RECYCLE BIN<br>Its like a recycle bin,&nbsp; they store all unecessary files or substance that are not needed by the cell.<br><br>CENTRIOLES: MOUSE<br>They direct the files on where to copy or d</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 11:49:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268218189</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268219596</link>
         <description><![CDATA[<div>Tan, JB Earl G.&nbsp;<br>Emata, Simone Anne C.&nbsp;<br>Figuracion, Kathleen Reanne&nbsp;<br><br><strong>Analogy:&nbsp;</strong>Movie Theater<br>If you hear the word "cell" think of a&nbsp; Movie Theater.&nbsp;<br><br><strong>Nucleus: Movie Player&nbsp;</strong><br>The movie player is just like the nucleus since it is the one who controls the movie just like the nucleus in the cell.&nbsp;<br><br><strong>Cell Membrane: Ticket Checker<br></strong>Just like a ticket checker the cell membrane he allows people to go inside.&nbsp;<br><br><strong>Lysosomes: Trash Can&nbsp;<br></strong>People throw there food in here, which is like the lysosomes that digest the food.&nbsp;<br><br><strong>Endoplasmic Reticulum: Food Makers<br></strong>Just like the ER it is responsible for giving nutrients to people.&nbsp;<br><br><strong>Mitochondria: Food&nbsp;<br></strong>It is like the mitochondria because it produces in order for our bodies to work.&nbsp;<br><br><strong>Vacuole: The one who delivers the food to the movie theater<br></strong>It also responsible for giving nutrients to people. Just like the ER <br><br><strong>Ribosomes: Manager of the Movie House <br></strong>It is like the manager because a ribosomes in the cell that gives instructions. <br><br><strong>Golgi Bodies: Food Packages in Movie House </strong><br>It packages all the nutrients. <strong><br>&nbsp;<br>A cell is like a movie theater! </strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:05:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268219596</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268219936</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:10:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268219936</guid>
      </item>
      <item>
         <title>Plant and Animal Cell Vocabulary</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268219981</link>
         <description><![CDATA[<div>Dejillo,Lata,Lapating (8-Aristotle)<br>If you hear the word cell, think about a <strong>CELL PHONE<br><br>CELL MEMBRANE</strong>- Phone Cover<br>•It covers the phone to protect it from harm.<br><strong>MITOCHONDRIA - </strong>Charger<br>•It gives the battery an energy which makes the phone work.<br><strong>VACUOLE- </strong>SD card<br>•It is the storage part of the cellphone.<br><strong>CELL WALL- </strong>Phone Case<br>•It covers and give protection to the cover of the battery.<br><strong>&nbsp;CHLOROPLAST- </strong>LCD<br>•It gives color to the screen.<br><strong>RIBOSOMES- </strong>speakers<br>•It is like dots.<br><strong>CENTRIOLES- </strong>Playstore<br>•It produces new&nbsp;aplications.&nbsp;<br>&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:11:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268219981</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL ANALOGY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268221392</link>
         <description><![CDATA[<div>ANALOGY: COMPUTER<br><br>Ribosome: PC Repairer<br>They are the PC repairer,&nbsp; they repairer and make new&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:26:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268221392</guid>
      </item>
      <item>
         <title>Plant And Animal Cell Analogy</title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268224349</link>
         <description><![CDATA[<div>Suarez,Alyanna Shin D.<br>Sarmiento,Nadine <br>(8-Aristotle)<br><br><strong>Analogy: Types of Jobs<br><br>Ribosomes</strong>: Ribosome is like a chef because this is the one who makes proteins and this is needed for directing chemical processes.<br><br><strong>Endoplasmic Reticulum</strong>: This is the delivery men because it transport various proteins and carry it to the Golgi Apparatus.<br><br><strong>Lysosomes</strong>: Lysosome are the garbage collectors, as the garbage disposal of the cell, lysosome also breaks down left overs cellular wastes.<br><br><strong>Golgi Bodies</strong> : These are the manufactures because golgi bodies  are processing proteins and sent throughout the cell.<br><br><strong>Leukoplasts</strong> : These are the store owners because it stores or this is the storage of various materials.<br><br><strong><br></strong><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:53:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268224349</guid>
      </item>
      <item>
         <title>The Plant and Animal cell (Own Vocabulary)</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268224440</link>
         <description><![CDATA[<div>&nbsp;Members:<br>Jaesar Justin P. Malunes<br>Shanlee S. Sangatanan<br>Zofia Amina S. Seromines&nbsp;<br><br>If you hear the word "cell",&nbsp;<br>think of a MALL....<br><br></div><ul><li><strong>Centrioles</strong>- It is the stalls/rented space because it is the division around the mall just like the centrioles in the cell.</li></ul><div><br></div><ul><li><strong>Endoplasmic Reticulum (ER) </strong>- It is like an elevator because it transport lipids around the cell and creation of lysosomes.</li></ul><div><br></div><ul><li><strong>Lysosome</strong>- It is&nbsp; the "grocery store" or "supermarket" because it stores and digest food from Golgi apparatus.</li></ul><div><br></div><ul><li><strong>Golgi bodies/Golgi apparatus </strong>- it functions as a manufacturing and packaging system just like "counters" in the mall it packages the grocery items.</li></ul><div><br></div><ul><li><strong>Ribosomes</strong>- It is the "foods in the food court" because like ribosomes, they contain proteins. The food you eat contains protein as well.</li></ul><div><br></div><ul><li><strong>Vesicle</strong> - It is used as the mall cart that are used to transport materials and also at the same time can be a storage in metabolism and enzyme in cells.</li></ul><div><br><br><strong>The cells are like malls they need their parts to work&nbsp; as one to be the better one.......</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-22 12:54:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268224440</guid>
      </item>
      <item>
         <title>PLANT AND ANIMAL CELL ANALOGY</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268273111</link>
         <description><![CDATA[<div>(8- Aristotle)&nbsp;<br>Cabiles,&nbsp; Zyrreg Zhyller<br>Urdaneta, Caryl Jean&nbsp;<br>Cutamora, Mizpah Gabrielle<br><br>ANALOGY: COMPUTER<br>If you hear the word cell think of a computer<br><br>Ribosomes: PC Repairer<br>They repairer the PC and make new solution to Salve a new damage.&nbsp;<br><br>Lysosome: Recycle bin<br>They store all unecessary files or deleted files from the software to be erased.&nbsp;<br><br>Centrioles: Mouse<br>They direct the path for the files to be copied or to be used by the PC.<br><br>Smooth Endoplasmic Reticulum: Network Connection<br>They are the paths for connection of transfering files to the PC or from the PC to the internet.&nbsp;<br><br>GOLGI Bodies: File Manager<br>They package the files to be used and store to the PC or to be used by the internet.&nbsp;<br><br>Rough Endoplasmic Reticulum: Update Systems<br>They make new updates or revises to the PC for the PC to have new info about the new files to be used.&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 03:19:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268273111</guid>
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      <item>
         <title>Plant and Animal Cell </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268279674</link>
         <description><![CDATA[<div>Patrick James Lagang<br>Jefferson Llamera<br>EJ Carl Banlasan<br>(8-Mendel) <br><br>Answers:<br>2. What is the meaning of specialized cell/job? <br>-modified by their shape, size, color, etc according to their purpose or it has their own job specialized for a specific&nbsp;organelle<br><br>3. What are cell membranes made of? <br>-phospholipids and proteins<br><br>4. Why is the cytoplasm describe as turtle's blood and other liquids? <br>-Because cytoplasm is a gel like fluid that keeps the organelles stay in place just like the turtle's blood and other liquids, it helps the organs stay in their places. <br><br>5. Why does the cell wall makes the cell strong and rigid? <br>-Cell wall provides structure and protection to the cell due to cellulose fibers. <br><br>6. What is the meaning of turgid<br>-it meana swollen or bombastic<br><br><strong>ANALOGY:</strong><br>If yo<strong> </strong>u hear the word Cell, just think of a RESTAURANT<br><br>NUCLEUS = MANAGERS<br>controls what is happening inside the cell or the restaurant<br><br>CELL MEMBRANE = DOORS<br>controls the in and out of anything<br><br>CYTOPLASM = FLOORS<br>keep everything inside stay in their places<br><br>VACUOLE = FOOD STORAGE/REFRIGERATOR&nbsp;<br>store food until organelles or costumers need it<br><br>GOLGI BODY = DELIVERYMEN&nbsp;<br>they're both at packaging and transportation<br><br>NUCLEAR MEMBRANE = GUARDS<br>protects the manager to be unharmed<br><br>CHOLOROPLAST = KITCHEN<br>this is where they produce food<br><br>ENDOPLASMIC RETICULUM = WAITERS<br>delivers food to different parts or costumers</div>]]></description>
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         <pubDate>2018-06-23 07:41:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268279674</guid>
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      <item>
         <title>Animal and Plant cell(Own Vocabulary)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268283443</link>
         <description><![CDATA[<div><strong>(Howard Godwin Delos Reyes<br></strong>Pette Bernard<br>Wendell Mejares)The ones you scolded maam<br><br>If tou heard the word cell just think <br>Of a zoo.<br><br><strong>Cell Wall:</strong><br>=<strong><em>The Gates</em></strong> around the zoo are like the cell wall because they are a stiff, rigid structure that protect the cell.<br><strong>Cell Membrane:<br></strong>=<strong><em>The Entrances of the Zoo</em></strong> is like a cell membrane as it selectively permeable. It only lets visitors enter the zoo at certain time, while restricting them at other.<br><strong>Nucleus</strong>:<br>=<strong><em>The Lead Officer of the Zoo</em></strong> is like the nucleus of a cell. It directs all operations concerning thw zoo, including how it works and what happens within it.<br><strong>Chloroplast</strong>:<br>=<strong><em>The Zoos Ticket Purchasing Booth </em></strong>is like th cells chloroplast. Money in the forms of pesos are taken exchange for the ticket of the zoo.money and tickets are also stored there. Waiting to be given away or used for other purposes. The money changer is like the energy. The sustance that allows the zoo to run itself.<br><strong>Vacuole</strong>:<br>=<strong><em>The Cages that Contain Animals and the Buildings </em></strong>that stores the food for the animals are like a cells vacuole. Both the vacuole and these cages occupy more than 50% the area of their location.<br><strong>Endoplasmic reticullum</strong>:<br>=<strong><em>The Pathway,</em></strong> that wind through a zoo, connecting various areas, are like tubes and channels that connect organelles in a cell.<br><strong>Golgi Bodies</strong>: <br>=<strong><em>The Zookeepers</em></strong> of a zoo are like tha golgi bodies of a cell. They package the waste and food of the zoos animal.<br><strong>Ribosomes</strong>:<br>=Dotting the pathways of the zoo, <strong><em>They are Smaller attractions, like rides, food booths , and souvenir </em></strong>shops generate food and amusement for zoo visitors.<br><strong>Mitochondrion</strong>:<br>=The mitochondria are <strong><em>Attractions Within the Zoo</em></strong> that collects ticketor passes in exchange fpr entrance fpr exibits,stores, and animals ride. Like the catabolic mitochondrion ,these attractions break down the value of tickets for various forms of amusement.<br><br><br><br><br>2)Specialized Job:<br>Has its own job or its only job or the best job for a specific organelle of a cell.<br>3)Cell membrane is made up of composed of a bilipid laye of protein and carbohydrates.its fluid like.<br>4)Cytoplasm is called the body of the cell and is constantly streaming because it contain almost blood and water. And water is above 50% of rhe cells bodyw which Made it the body of the cell.<br>5)Because it protects the cell making it more upgraded inside.(sorry maam i dont know the answer)</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 09:28:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268283443</guid>
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      <item>
         <title>Plant and Animal Cell</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268285589</link>
         <description><![CDATA[<div><strong>Krystle Ann Z. Paguray<br>Princess Kyla Endaya<br>Angelle Kate Y.&nbsp;Brocoy<br>(8- Mendel)<br><br>I. Answer the Questions<br>Q2. </strong>Cells become specialized to perform different functions.<br><strong>Q3. What are the components of cell membrane?<br></strong>The components of Cell membrane are:</div><ul><li>PHOSPHOLIPIDS</li><li>CHOLESTEROL</li><li>PROTEINS</li><li>CARBOHYDRATES</li></ul><div><strong>Q4. Why is the cytoplasm described as the turtle's blood and other liquids?<br></strong>The cytoplasm is described as the turtle's blood and other liquids because blood contains necessary substances such as the nutrients and oxygen while cytoplasm contains all of the organelles.<br><strong>Q5. Why does cell wall makes the cell strong and rigid?<br></strong>Cell walls makes the cell strong and rigid due to the cellulose fibers inside the walls.<br><strong>Q6. What is turgid?<br></strong>Turgid means swollen &amp; distended or congested.<br><strong>Q7. What's the difference between plant cell and Animal Cell?<br></strong>Plant cells can be larger than animal cells. Plant and Animal Cells have different structures to perform the conversion of energy; Animal cells use mitochondria while Plant Cells use Chloroplast for photosynthesis.<br><br><strong>II. Vocabulary (OWN)<br></strong>When describing the functions of the parts of the cell just think of</div><div><strong>&nbsp;A PERSON'S ROLE IN SOCIETY (JOB)</strong></div><ul><li><strong>CENTRIOLES</strong></li></ul><div><strong>-</strong>The TEACHER because they control the flow of students in the hallways, just like how CENTRIOLES control the microtubules in the cytoplasm during cell division.</div><ul><li><strong>RIBOSOMES</strong></li></ul><div><strong>- </strong>The CHEF because they are cooking the meals like how RIBOSOMES produce proteins in the cell.</div><ul><li><strong>ENDOPLASMIC RETICULUM</strong></li></ul><div><strong>-&nbsp; </strong>The TRANSPORT WORKER because they transport the packages just like how ENDOPLASMIC RETICULUM transports the material produced by the cell.</div><ul><li><strong>NUCLEOLUS</strong></li></ul><div><strong>- </strong>The MANAGER OF A COMPANY because they give orders to the workers just like how NUCLEOLUS gives order to the RIBOSOMES.</div><ul><li><strong>GOLGI BODIES</strong></li></ul><div><strong>- </strong>The DRIVER because like GOLGI BODIES they collect passengers and send them to the proper location.</div><ul><li><strong>LYSOSOME</strong></li></ul><div>- The JANITOR because LYSOSOME cleans the cell. The main purpose of lysosome is to digest excess or worn out organelles, it's just like cleaning.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 10:43:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268285589</guid>
      </item>
      <item>
         <title>Plant and Animal Cell</title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268292832</link>
         <description><![CDATA[<div><strong>Kristine Joy B. Palmaera<br>Julianne B. Agorilla<br>(8- Mendel)</strong><br><br><strong>I. Answer the given questions.</strong><br><strong>Q2.</strong> <strong>Specialized cells</strong> make up tissues, tissues make up organs, and organs make up the systems that work together to make up our bodies.  <strong>Cells</strong> become <strong>specialized</strong> in order to perform different functions.<br><strong>Q3. The components of cell membrane are:<br></strong>- Lipids<br>- Phospholipids<br>- Carbohydrates<br>- Proteins<br><strong>Q4. Cytoplasm</strong> consists of all of the contents outside of the nucleus and enclosed within the cell membrane of a cell. It is clear in color and has a gel-like appearance. Cytoplasm is composed mainly of<strong> water</strong> but also contains enzymes, salts, organelles, and various organic molecules.<br><strong>Q5. Cell walls</strong> provide the structure and protection of the plant <strong>cell </strong>due to the cellulose fibers inside the <strong>walls</strong>. All of the <strong>cell</strong> organelles <strong>are</strong> contained within these <strong>walls</strong>.<br><strong>Q6. Turgid </strong>means swollen, distended or congestes in dictionary but turgid's definition in biology is plant cell fully inflated with water.<br><strong>Q7.</strong> The key difference between <strong>plant</strong> and <strong>animal cells</strong> lies in the structural differences. <strong>Plant cells</strong> are rectangular wheres <strong>animal cells</strong> are round and <strong>plant cells</strong> contain chloroplasts, a cell wall, and vacuoles while<strong> animal cells</strong> do not.<br><br><strong>II. Make your own vocabulary.<br></strong>If you hear the word "cell" just think of an <strong>AMUSEMENT PARK</strong>.<br><br><strong>- Centrioles</strong> is to help in cell division but only in animal cell. They are like the lines of the ride themselves. These two relates because  they both help divide up certain areas of their systems. The lines divide up the people waiting for the ride and centrioles contribute in cell division. <br><strong>-  Endoplasmic Reticulum (ER) </strong>is the way of transportation for proteins to the Golgi Bodies. In an amusement park, this relates to the walkways for citizens and segways for workers. They are similar because both are used as the primary sources of transporting in their systems.<br><strong>- Golgi Bodies</strong> primarily modifies proteins (packages) transported from the ER but can also transport lipids through cells.This is similar to the gift shop workers in amusement parks because they wrap and package gifts for the costumers.<br><strong>- Lysosome </strong>are known as "suicide sac" in a cell, they mainly break down cellular debris. They are similar to the garbage cans and decomposers of amusement parks. They relate because they both get rid of waste for their system. <br><strong>- Nucleulos' </strong>primary function is to make ribosomes for the cell. The ribosomes of an amusement park is the rides, so the nucleulos would be the power circuit to all the rides in the park. <br><strong>- Vacuole</strong> isolates unwanted substances that can harm or cause any threat to the cell. They then export these unwanted substances from the cell. They are similar to janitors or people in charge of cleaning the amusement park. These two are similar because they both get rid of the unwanted things for their systems.<br><strong>- Ribosomes' </strong>primary function is to make proteins. These proteins drive the cell. The whole idea of amusements parks is to have fun, so the protein of an amusement park are the rides because they are the main thing that provides fun for everyone. <br><strong>- Chloroplast </strong>are the site of photosynthesis in the cell and it also consist of a double membrane, it relates to the solar panels of an amusement park because both things take energy from the sun and use it to provide energy for their systems. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 14:11:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268292832</guid>
      </item>
      <item>
         <title>Plant and Animal Cell(Own Vocabulary)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268294899</link>
         <description><![CDATA[<div>Jasmin Imelda P. Silva<br>Francis Albert L. Calonia<br>Kenny Macjewr T. Sulilap<br>(8-Aristotle)<br><br>Analogy:<strong>Daily Life in School</strong><br>If you hear the word "CELL",just think of a <strong>DAILY ACTIVITY IN SCHOOL</strong><br><br><strong>Endoplasmic Reticulum</strong>=<br>You're going to school.<br><br><strong>Lysosome</strong>=<br>Every teacher taught us the right thing we do. That's why students must throw their bad attitudes and intake what did teachers taught in order that the grade wouldn't be affected.<br><br><strong>Golgi Apparatus</strong>=<br>Sharing your knowledge and ideas to your classmates.<br><br><strong>Centrioles</strong>=<br>The students are divided into different class sections.<br><br><strong>Ribosomes</strong>=<br>The teacher is producing knowledge in order to digest it by the student.<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 15:01:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268294899</guid>
      </item>
      <item>
         <title>Plant and Animal Cell</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268295419</link>
         <description><![CDATA[<div><strong>Leana Jan Riz N. Valdez<br>Francine Nicole Sabelita<br>Nikko Fajardo<br>8 - Mendel<br>Answers:<br></strong>2. Specific Function or when the cell is multitasking. It is called a specialized&nbsp;cells.<br>3. Phospholipids, embedded proteins, carbohydrates and lipids<br>4. Cytoplasm is described as the turtle's blood and other liquids because cytoplasm is the watery gel inside a cell it is goop and holds the organelles<br>5. It makes the cell strong and rigid because it is the main protection barrier that surrounds a plant cell.&nbsp; It is built mostly of cellulose fibers which provide the structure and support of the cell wall.&nbsp; They are thick and are able to bond together to provide the plants structure.<br>6. Turgid means swollen or bombastic.<br>7.The difference of the plant and animal cell is their shape and structure because the plant cell has cell wall and chloroplasts while animal cell has centrioles <br><strong>Vocabulary:<br>If You Hear The Word Cell We Can Compare It In School. <br>Endoplasmic Reticulum </strong>can be compared to the hallways of the school. This is because hallways transport students and staff around the school.<br><strong>Golgi Bodies </strong>is like a teacher because, they help the students (endoplasmic reticulum) get their work done.<br><strong>Centrioles</strong> are the pipes of Cell School because they look similar and help divide up the main water supply to all the classrooms.<br><strong>Lysosome</strong> can be compared to the recycle bin of the School. A recycle bin is something that stores various unwanted files, and which the computer has discarded considering the files to be useless, or dangerous.<br><strong>Ribosomes</strong> workers and teachers are like ribosomes because they are assembled by the principal and they produce knowledge for students to learn; like proteins.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 15:13:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268295419</guid>
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         <title></title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268295595</link>
         <description><![CDATA[s]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 15:18:05 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268295595</guid>
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         <title></title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268295726</link>
         <description><![CDATA[are]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 15:21:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268295726</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268313006</link>
         <description><![CDATA[<div><strong>Plant and Animal Cells<br><br>Baluso, James Lian David<br>Kano, Safwan<br>Mato, Lacman<br>(8-Mendel)<br><br>I. Answer the Given Questions.<br>2. Specialized Cells.<br></strong>When the cell is multitasking or doing more than one job it is called a specialized cells.<br><br><strong>3.What are the components of Cell Membrane?<br></strong>The components of Cell Membrane are phospolids, proteins, carbohydrate, and colesterol.<br><br><strong>4.Why is the cytoplasm described as the turtle's blood and other liquids?<br></strong>Cytoplasm described as the turtle's blood and other liquids because cytoplasm is a basic component of turtle's blood and cytoplasm contains all organelles and other liquids such as blood contains proteins, enzymes, glucose, and fats.<strong><br><br>5.Why does Cell Wall makes the Cell strong and rigid?<br></strong>Cell wall makes the cell strong and rigid so that it could support, protect, and keep the shape of the cell.<br><br><strong>6.What is turgid?<br></strong>Turgid means swollen or corgested, when a cell takes up water by osmosis and starts to turgid or swollen the cell become turgid cell.<strong><br><br>7.Compare and contrast Plant cells and Animal cells.<br></strong>Plant cell is larger than animal cell. Plant cells have chloroplasts, cell wall, and vacoules. And animal cells doesn't have that parts. Plant cell have a fixed shape and animal cells have irregular shape.<br><br><strong>II. Own Vocabulary</strong><br>If you hear cell just imagine a <br><strong>&nbsp; &nbsp;</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-23 23:56:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268313006</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268313508</link>
         <description><![CDATA[<div><strong>Plant and Animal Cells<br><br>Baluso, James Lian David<br>Kano, Safwan<br>Mato, Lacman<br>(8-Mendel)<br><br>I. Answer the Given Questions.<br>2. Specialized Cells.<br></strong>When the cell is multitasking or doing more than one job it is called a specialized cells.<br><br><strong>3.What are the components of Cell Membrane?<br></strong>The components of Cell Membrane are phospolids, proteins, carbohydrate, and colesterol.<br><br><strong>4.Why is the cytoplasm described as the turtle's blood and other liquids?<br></strong>Cytoplasm described as the turtle's blood and other liquids because cytoplasm is a basic component of turtle's blood and cytoplasm contains all organelles and other liquids such as blood contains proteins, enzymes, glucose, and fats.<strong><br><br>5.Why does Cell Wall makes the Cell strong and rigid?<br></strong>Cell wall makes the cell strong and rigid so that it could support, protect, and keep the shape of the cell.<br><br><strong>6.What is turgid?<br></strong>Turgid means swollen or corgested, when a cell takes up water by osmosis and starts to turgid or swollen the cell become turgid cell.<strong><br><br>7.Compare and contrast Plant cells and Animal cells.<br></strong>Plant cell is larger than animal cell. Plant cells have chloroplasts, cell wall, and vacoules. And animal cells doesn't have that parts. Plant cell have a fixed shape and animal cells have irregular shape.<br><br><strong>II. Own Vocabulary</strong><br>If you hear the word "cell", just think of a school.<br><br><strong>Ribosomes:Teacher</strong><br>Ribosomes is like a teacher because teacher gave us knowledge and ribosomes gave/make proteins for the cell.<br><br><strong>Lysosomes:Janitors</strong><br>Lysosomes is like a janitor in the school. Janitors clean the school to get rid of the unwanted substances, just like what lysosomes do.<br><br><strong>Golgi</strong> <strong>Body:Hallways</strong><br>Golgi bodies is like the hallways in the school, it helps students where to go and golgi body helps proteins go throughout the cell.<br><br><strong>Smooth</strong> <strong>Endoplasmic</strong> <strong>Reticulum:Bag/Lockers</strong><br>Smooth ER is like a bag or a locker because smooth ER serves as storage in a cell and bag or lockers serves as storage in the school.<br><br><strong>Rough</strong> <strong>Endoplasmic</strong> <strong>Reticulum:School</strong> <strong>Bus</strong><br>Rough ER is like a school bus because it transport student and rough ER transport proteins.<br><br><strong>Centrioles:School</strong> <strong>Coordinator</strong><br>It is like a school coordinator because they have the same job to do for the Coordinator he/she organized an event or ativities and for the Centrioles it organized cell division.<br><strong>&nbsp; &nbsp;</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-24 00:17:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268313508</guid>
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      <item>
         <title>Cell</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268314563</link>
         <description><![CDATA[<div>Miase, Bea Clarisse A.<br>Evangelista, Christian Louie<br>Tariao, Darryl<br><br><strong>Analogy: Human Body parts<br></strong>If you think of the word "cell", just think of our human body parts<br><br>Ribosomes: Bone marrow (creates blood like amino acids for the body to use, just like ribosomes, which creates proteins for the cell)<br><br>Smooth endoplasmic reticulum: Liver (Takes out all the toxins out of the cell)<br><br>Rough endoplasmic reticulum: Blood vessels (makes molecules for the cell and distributes those molecules)<br><br>Lysosome: Kidneys (breaks down nutients into forms which the cell can absorb and use)<br><br>Golgi Apparatus: Intestines (Sorts and packages proteins and other substances in the cell for storage/distribution to other cell)<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-24 01:14:50 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268314563</guid>
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      <item>
         <title>1. Answer:</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268315673</link>
         <description><![CDATA[<div>The cell membrane itself is composed of phospholipids bilayer, where as other molecules that are attached to the membrane can include proteins, glycolipids, and cholesterol <br><br>Vocabulary:<br>chloroplasts: leaves of the plant<br><br>cell wall- like the back (shell) of the crab.<br><br>Vacuoles- where the crabs store their food<br><br>nuclear membrane- the skin of the crab<br><br>nucleus- the brain of the crab<br><br>cell membrane- the carapace of the crab<br><br>chromosomes- the eyes of the crab<br><br>cytoplasm- crabs blood and other liquuds<br><br>mitochondria- the foods that the crab ate <br><br>2.Because it is the gel-like substance enclosed within the cell membrane. <br>3 why does the cell wall makes the cell strong and ligid<br>Answer: it is strong and ligid to protect the shapes and the contents of the cell. <br>4.what is turgid? <br>Answer: turgid means <br>swollen and distended or congested.<br>5. What is difference between plant cells and animal cells<br>Answer: they differ in their shapes.the shape of the plant is rectangular while the animal cell is circle shaped. They also differ in their parts <br>the plant cells has plastids and large vacuole. Animal cells has cetrosome, lysosome, small vacuoles, cilia and flagella. <br>Fernandez,nicole D. <br>Pertimos, Ashley Marie <br>swollen and distended or congested.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-24 02:01:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268315673</guid>
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         <title>Members:Arduo,Arcilla,Olar (grade 8 </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268316187</link>
         <description><![CDATA[<div>Q2:Some cells in multicellular organisms are modified to carry out a particular function,such as transporting a certain substance or executing a specific task. It is called specialized cells.<br>Q3:Phospholipids,cholesterol,proteins,carbohydrate<br>Q4:Cytoplasm is described as turtle's blood because it builds new substances from materials taken into the cell and other liquids such as blood that delivers necessary substances such as nutrients and oxygen to the cells.<br>Q5:Cell walls gives supports to plant's growth.It makes the cell strong and rigid because it is made up of cellulose,as non living substance.<br>Q6:Turgid means swollen or bombastic.Turgid can be used in a figurative sense to describe things that are overblown.<br>Q7:Plant cell is bigger than the animal cell.They are both eukaryotic cells.Plant cells have cell wall,large vacuole,chloroplasts while animal cells has no cell wall,no vacuole and it has centrioles.They have both lysosome,mitochondrion,golgi apparatus,roung and smooth reticulum,nucleus,cell membrane,cytoplasm,ribosomes,nucleolus.<br><br>Own Vocabulary<br>If you hear the word "cell" just think of a school<br><strong>golgi apparatus</strong>:parking lot<br>Where the teachers and students can go to different places.This is how the golgi apparatus packages and ships molecules to different places.<br><strong>nucleolus</strong>:principal's office<br>The nucleolus of a cell is compared to a principal because he assembles the teachers and students like the nucleolus assembles ribosomes<br><strong>ribosomes</strong>:teachers<br>Teachers are like ribosomes because they are assembled by the principal and they produce knowledge to students to learn just like proteins.<br><strong>rough endoplasmic reticulum</strong>:teacher's lounge<br>The teacher's lounge is like the RER because it contains like it contains ribosomes and teachers can modify lessons they are teaching in there.<br><strong>lysosomes</strong>:janitors<br>Janitors are compared to the lysosomes because they can clean things from the school just like lysosomes can clean the material from a cell.<br><strong>centrioles</strong>:stairs<br>The centrioles of the cell are compared to the stairs because it seperated the ways of which can students go up and down.<br><br><br><br><br><br><br></div>]]></description>
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         <pubDate>2018-06-24 02:21:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268316187</guid>
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         <title>I. Answer the Given Questions.2. Specialized Cells.When the cell is multitasking or doing more than one job it is called a specialized cells.3.What are the components of Cell Membrane?The components of Cell Membrane are phospolids, proteins, carbohydrate, and colesterol.4.Why is the cytoplasm described as the turtle&#39;s blood and other liquids?Cytoplasm described as the turtle&#39;s blood and other liquids because cytoplasm is a basic component of turtle&#39;s blood and cytoplasm contains all organelles and other liquids such as blood contains proteins, enzymes, glucose, and fats.5.Why does Cell Wall makes the Cell strong and rigid?Cell wall makes the cell strong and rigid so that it could support, protect, and keep the shape of the cell.6.What is turgid?Turgid means swollen or corgested, when a cell takes up water by osmosis and starts to turgid or swollen the cell become turgid cell.7.Compare and contrast Plant cells and Animal cells.Plant cell is larger than animal cell. Plant cells have chloroplasts, cell wall, and vacoules. And animal cells doesn&#39;t have that parts. Plant cell have a fixed shape and animal cells have irregular shape.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268318412</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-06-24 03:35:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268318412</guid>
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         <title>Jhonbert Facurib &amp; Ramer Kent Fagutao</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268318413</link>
         <description><![CDATA[<div><strong>I. Answer the Given Questions.<br>2. Specialized Cells.<br></strong>When the cell is multitasking or doing more than one job it is called a specialized cells.<br><br><strong>3.What are the components of Cell Membrane?<br></strong>The components of Cell Membrane are phospolids, proteins, carbohydrate, and colesterol.<br><br><strong>4.Why is the cytoplasm described as the turtle's blood and other liquids?<br></strong>Cytoplasm described as the turtle's blood and other liquids because cytoplasm is a basic component of turtle's blood and cytoplasm contains all organelles and other liquids such as blood contains proteins, enzymes, glucose, and fats.<strong><br><br>5.Why does Cell Wall makes the Cell strong and rigid?<br></strong>Cell wall makes the cell strong and rigid so that it could support, protect, and keep the shape of the cell.<br><br><strong>6.What is turgid?<br></strong>Turgid means swollen or corgested, when a cell takes up water by osmosis and starts to turgid or swollen the cell become turgid cell.<strong><br><br></strong><a href="http://7.compare/"><strong>7.Compare</strong></a><strong> and contrast Plant cells and Animal cells.<br></strong>Plant cell is larger than animal cell. Plant cells have chloroplasts, cell wall, and vacoules. And animal cells doesn't have that parts. Plant cell have a fixed shape and animal cells have irregular shape.<br><br><strong>II. Own Vocabulary<br></strong>CELL = RESTAURANT Cells are made up of parts called organelles that help the cell function properly. Restaurants also require several parts in order to function properly.</div><ol><li><a href="https://image.slidesharecdn.com/cellanalogy-130902124751-phpapp02/95/cell-analogy-example-2-638.jpg?cb=1378126109">&nbsp;</a>CELL MEMBRANE = THE RESTAURANT DOORS The Cell Membrane controls what comes in and out of the cell. The Restaurant Doors let people in and out of the restaurant.</li><li>CYTOPLASM = RESTAURANT FLOOR The Cytoplasm is a jelly-like substance that keeps all the other organelles in their proper place. The Restaurant Floor holds the tables, chairs, counters, and kitchen equipment in their proper place.</li><li>NUCLEUS = RESTAURANT MANAGER The Cell Nucleus controls what happens inside of the cell. Restaurant managers control what happens inside of their restaurant.</li><li><a href="https://image.slidesharecdn.com/cellanalogy-130902124751-phpapp02/95/cell-analogy-example-5-638.jpg?cb=1378126109">&nbsp;</a>RIBOSOMES = HAMBURGER COOK Ribosomes make proteins for the cell. The Hamburgers are made from protein as well.</li><li>MITOCHONDRIA = BURGER DRAWERS The mitochondria store energy obtained from food until other organelles need it. The burger drawers store food until it is needed for a customer’s order.</li><li>THE ENDOPLASMIC RETICULUM = RESTAURANT KITCHEN The ER produces various substances used within the cell and throughout the body, such as protein (rough ER) and fats (smooth ER). The Restaurant Kitchen also produces several different products that can be used inside the restaurant or taken elsewhere through the drive-thru.</li><li>GOLGI BODIES/VESICLES = FRONT COUNTER The Golgi Bodies sort and transport substances in vesicles that are used within, or discarded outside of, the cell. Front Counter employees also sort products into bags which are used inside or taken out of the restaurant. Golgi Body Vesicles</li></ol><div><br><br><br><br></div>]]></description>
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         <pubDate>2018-06-24 03:35:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268318413</guid>
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         <title>cell</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268319849</link>
         <description><![CDATA[<div>members:<br>Buladaco, lei marie<br>Cerado, Allyza joy<br>Flores, Julijah <br><br><strong>I. Answer the Given Questions.<br>Q2. Specialized Cells.<br>are modified to carry out a particular function. such as transporting a certain substance or executing a specific task.<br>&nbsp;</strong><br><strong>Q3. What are the components of Cell Membrane?<br></strong>The components of Cell Membrane are phospolids, proteins, carbohydrate, and colesterol.<br><br><strong>Q4. Why is the cytoplasm described as the turtle's blood and other liquids?<br></strong>Cytoplasm described as the turtle's blood and other liquids because cytoplasm is a basic component of turtle's blood and cytoplasm contains all organelles and other liquids such as blood contains proteins, enzymes, glucose, and fats.<strong><br><br>Q5.Why does Cell Wall makes the Cell strong and rigid?<br></strong>Cell wall makes the cell strong and rigid so that it could support, protect, and keep the shape of the cell.<br><br><strong>Q6.What is turgid?<br></strong>Turgid means swollen or corgested.<strong><br></strong><br></div><div><strong>Q7. Compare and contrast Plant cells and Animal cells.</strong></div><div>Plant cell is larger than animal cell. Plant cells have chloroplasts, cell wall, and vacoules. And animal cells doesn't have that parts. Plant cell have a fixed shape and animal cells have irregular shape.<br><br><strong>II. Own Vocabulary<br></strong><br>When you hear the word cell just think of a<strong> "amusement park"!<br></strong><br><strong>1. Gate</strong><em> (Cell Membrane)</em><br>&nbsp; &nbsp; -it allows people to go inside the park.<br><strong><br>2. The Manager</strong> <em>(Nucleus)</em><br>&nbsp; &nbsp;- he controls what happens inside the park<br><br><strong>3. Trash Can</strong><em> (Lysosome)</em><br>&nbsp; &nbsp; - it is where people throw their food.<br><br><strong>4. Food Court</strong> <em>(Mitocondria)</em><br>&nbsp; &nbsp; - where the food is stored<br><br><strong>5. Path Way</strong> <em>(Endoplasmic Reticulum)</em><br>&nbsp; &nbsp;-it is where the people walk on like a hallway.<br><br><strong>6. Staff</strong> <em>(Ribosomes)</em><br>&nbsp; &nbsp;- they follow the instructions of the manager (nucleus)<br><br><strong>7.&nbsp; Food Pack</strong> <em>(Golgi bodies)</em><br>&nbsp; &nbsp;- it store food <em>(proteins) </em><br><br>&nbsp; &nbsp;<br><br></div>]]></description>
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         <pubDate>2018-06-24 04:30:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268319849</guid>
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         <title>For aristotle and mendel.</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268358165</link>
         <description><![CDATA[<div>You make an experiment to show the function of cell membrane.&nbsp;<br>A cell membrane is permeable to some liquids specially water. And this is what we call osmosis. Make a simple experiment that can be done at home about osmosis because our next topic is cellular transport. </div>]]></description>
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         <pubDate>2018-06-24 19:55:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268358165</guid>
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         <title>Mendel pls share to aristotle the url. Everytime you submit pls indicate your section.</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268358476</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-06-24 20:00:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268358476</guid>
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         <title>Aristotle and mendel</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268358889</link>
         <description><![CDATA[<div>Here is your time table.<br>Monday &amp; tuesday researching/preparing for the activity by group. Just ok if there is a duplication. But possible you must have your own. You can have same procedure but different materials. <br>Tuesday during our biotech time submit in my padlet account.<br>Wednesday open your padlet for my correction.<br>Thursday perform<br>Friday submit write ups</div>]]></description>
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         <pubDate>2018-06-24 20:08:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268358889</guid>
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         <title>Elias Cadiz, Aira Vergara, Grejie Mercado</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268371006</link>
         <description><![CDATA[<div>Q2. specialized cells<br>Answer:<br>Specialized cells perform specialized functions in multicellar organisms. Groups of speciized cells cooperate to form a tissue, such as a muscle. Different tissues are in turn grouped together to form larger function units, called organs.&nbsp;<br><br>Q3.What are the componentsof Cell Membrane?&nbsp;<br>Answer:The principal components of the plasma membrane are lipids, proteins, and carbohydrate groupsthat are attached to some of the lipids and proteins, carbohydrate, and colesterol.&nbsp;<br><br>Q4. What is the cytoplasm describe as the turtle's blood and other liquids?<br>Cytoplasm described as the turtle blood and other liquids because cytoplasm is the basic component of turtles blood and other cytoplasm contains all organelles and other liquids such as blood contains protein,enzymes, glucose, and fats.&nbsp;<br><br>Q5. Why does Cell wall makes the cell strong and rigid?&nbsp;<br>Answer: in order to protect and support the cell.&nbsp;<br><br>Q6. What is turgid?&nbsp;<br>Answer: Swollen or corgested.<br><br>Q7. Compare and contrast plant cells and animal cells<br>Answer: Plant cell have chloroplasts, cell wall, and vacoules while animals cells doesnt have that parts.&nbsp;<br><br>&nbsp;II-vocabulary&nbsp;<strong>OFFICE <br></strong>1.walls-(Cell membrane) -it protects the people from strangers outside<br>2.Boss-(nucleus)-the boss control the organization <br>3.Canteen-(mitochondria) -where food is store <br>4.garbage can-(lysosome)-where waste throw <br>5.hall way-(Endoplasmic Reticulum) -It is where the people walk on like a hallway<br>6.Employee-(Ribosomes) -they follow the instructions of the boss(nucleus)<br>7.Food pack-(Golgi Body) -it store&nbsp;food(proteins<strong><br></strong><br></div>]]></description>
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         <pubDate>2018-06-24 23:44:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268371006</guid>
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         <title>Biotechnology Assignment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268402879</link>
         <description><![CDATA[<div>Cabiles, Zyrreg Zhyller A.<br>Cutamora, Mizpah Gabrielle S.<br>Urdaneta, Caryl Jean P.<br>8- Aristotle<br><br>1. What are the components of the following:</div><ul><li>Cell membrane - lipids(phospholipids, cholesterol) proteins, carbohydrates</li><li>Mitochondria - outer membrane, porin, intermembrane space, intracristal space, peripheral space, lamella, inner membrane, matrix, cristae, mitochondrial dna, matrix granule, ribosome, atp synthase.</li><li>Chloroplast - envelope, stroma, thylakoids, grana, photosystems, peripheral reticulum</li><li>Cell wall - cellulose, hemicellulose, pectin in plant cells while bacterial cell walls are composed of peptidoglycon.</li></ul><div>2. Why is it strong and rigid?<br>It is stong and rigid because of the cell wall. Cell wall gives the plant its actual shape and it is the protective layer outside the cell membrane that also provides support for the cell structure. Cell wall is needed so that the plant can grow to it's great height and not fall over the wind.<br>3. Explain how cells maintain it's life.<br>The cell maintains energy by the active transport mechanisms, they expend energy to maintain right concentrations of molecules and ions. ATP is used to make energy for the cell because cells spend much energy they get in metabolism to make sure their active transport processes are working. They also maintain health by the organelles who repairs the cell.</div>]]></description>
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         <pubDate>2018-06-25 03:47:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268402879</guid>
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         <title>Jasmin Imelda P. SilvaFrancis Albert L. CaloniaKenny Macjewr T. Sulilap(8-Aristotle)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268402946</link>
         <description><![CDATA[<div><strong>1.What is the composition of cell wall?-why plants stands so strong and rigid?</strong><br>Answer: Cell walls provide the structure and protection of the plant cell due to the cellulose fibers inside the walls.&nbsp; All of the <a href="https://kidsbiology.com/biology-basics/cell-organelles/"><strong>cell organelles</strong></a> are contained within these walls. And while these walls provide a strong structure, they are also permeable. That's why they are rigid. <br><br><strong>2.Components:<br><br>*CELL MEMBRANE</strong></div><ul><li>A <strong>phospholipid</strong> is a lipid made of glycerol, two fatty acid tails, and a phosphate-linked head group.&nbsp;</li><li><strong>Cholesterol</strong>, another lipid composed of four fused carbon rings, is found alongside phospholipids in the core of the membrane.</li><li>Membrane proteins may extend partway into the plasma membrane.</li><li>Carbohydrate groups are present only on the outer surface of the plasma membrane and are attached to proteins, forming <strong>glycoproteins</strong>, or lipids.&nbsp;</li></ul><div><strong>MITOCHONDRIA<br></strong>&nbsp;The <strong>outer membrane</strong> covers the organelle and contains it like a skin. The <strong>inner membrane</strong> folds over many times and creates layered structures called <strong>cristae</strong>. The fluid contained in the mitochondria is called the <strong>matrix</strong>. <br><strong>CHLOROPLAST<br></strong>&nbsp;We'll hit the high points for the structure of a chloroplast. Two membranes contain and protect the inner parts of the chloroplast. They are appropriately named the <strong>outer</strong> and <strong>inner membranes</strong>.One <strong>thylakoid stack</strong> is called a <strong>granum</strong>. <br><br><strong>3.HOW CELL MAINTAIN ITS LIFE?<br></strong>ENERGY-cell require a constant supply of energy to generate and maintain biological order that keeps them alive.<br>HEALTH-Everything is connected to everything else.As you eat,cells are also eating.The food you eat is also  the food your cells consume.Cells will be healthy if the organism they're living is also healthy.</div>]]></description>
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         <pubDate>2018-06-25 03:48:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268402946</guid>
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         <title>Biotechnology Assignment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268404116</link>
         <description><![CDATA[<div>Juanico, DJ Christian&nbsp;<br>Natividad, Deniel Dave&nbsp;<br>Enojales, Desire Faith<br>(8- Aristotle)<br><br>1. What are the components of the following:<br>&nbsp; &nbsp; &nbsp; &nbsp;- Cell membrane: Lipids/ phospholipid, Cholesterol, Carbohydrate.<br>&nbsp; &nbsp; &nbsp; &nbsp; - Mithocondria: Outer membrane that covers the organelle, Inner membrane that folds over many times to and create a cristae, and the fluid is called matrix.<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; - Chloroplast: Inner membrane thhat sorrounds the strana and grana, Chlorophyll that captures light from the sun, thykaloid that acts like a skeleton of the chloroplast.<br><br>2. Why plant stand strong and rigid? What is the composition of the cell wall?<br>&nbsp; &nbsp; &nbsp; &nbsp;- Cell wall have cellulose fiber that provide that plant cell its actual shape and gives of protection inside the walls. Cell wall is needed to not fall over from wind and stand strong while the cell wall gives off this protection it is also permeable.<br>3.) How does the cell maintain its life?<br>maintaining your internal environment and what I mean be that is a humans "normal" homeostasis is 98.6 as to where an animal may be different. It is very very important to maintain your homeostasis. If your body is not functioning properly to maintain the right body temperature you could die. Think about it, if your body doesn't do what it is supposed to do then your body temp can drop drastically if you are cold or raise if you are hot<br><br>&nbsp; &nbsp; &nbsp; &nbsp;&nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp;<br>&nbsp; &nbsp; &nbsp;&nbsp;<br>&nbsp; &nbsp; &nbsp;&nbsp;</div>]]></description>
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         <pubDate>2018-06-25 04:03:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268404116</guid>
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         <title>Answer the following questions:</title>
         <author>cimagalamary</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268404510</link>
         <description><![CDATA[<div>Bantes, Jannah h. </div>]]></description>
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         <pubDate>2018-06-25 04:08:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268404510</guid>
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         <title>Answer the following questions:</title>
         <author>cimagalamary</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268404512</link>
         <description><![CDATA[<div>Bantes, Jannah H.<br>Hamajo, Mika C.<br>8-Aristotle<br><br>1. What is the composition of Cell Wall? <br>-Middle Lamella<br>-Primary Cell Wall<br>-Plasma Membrane<br>-Pectin<br>-Cellulose Microfibril<br>-Hemicellulose<br>-Soluble Protein<br>*Why plant cells are strong rigid?<br>-Plant cells are strong rigid because they have a strong rigid cell wall on the outside of the cell membrane. It'll prevent the cell from bursting. This pressure that is put on the cell walls is known as TUGOR PRESSURE. In fact, most healthy plants are in a constant state tugor pressure, and this is what gives plant their rigid shape.<br><br>2. Components of the following:<br>*Cell Membrane<br>-Phospholipid Bilayer<br>-Cholesterol<br>-Glycocalyx<br>-Proteins (Peripheral Protein &amp; Integral Protein)<br>*Mitochondria<br>-Inner and Outer Membrane<br>-Ribosome<br>-Mitochondrial DNA<br>-Cristae<br>-Matrix<br>-Intermembrane Space<br>*Chloroplast<br>-Inner and Outer Membrane<br>-Stroma Lamellae<br>-Stroma<br>-Thylakoid<br>-Intermembrane Space<br>-Granum/Grana<br><br>3. Explain how cell maintains it's life<br>-Cell maintains it's life by these organelles working together. They are like the organs in a human body and they help the cells stay alive. Each organelle has it's own specific function to help the cell survive. All of the cell's organelles must work together to keep the cell healthy.</div>]]></description>
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         <pubDate>2018-06-25 04:08:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268404512</guid>
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         <title>BIOTECHNOLOGY ASSIGNMENTAnswer the following questions:</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268404882</link>
         <description><![CDATA[<div><br>Members:<br>Alberto, Achilles Alexis R.<br>Berame, Bernadette Sue B.<br><br><strong>1. What are the composition of Cell Wall?<br></strong>- In the primary plant cell, the major carbohydrates are cellulose,hemicelloluse and pectin.<br><strong>-</strong>The secondary cell wall contains cellulose, xylan, lignin, and one type of hemicellulose.<strong><br>-</strong>Plant cell wall also contain enzymes such as hydrolases, esterases, peroxidase and transglysolases<br><strong>-</strong>Plant cell wall composed of three layers: The middle lamella, The primary cell wall and the secondary cell wall.<br><strong><br>Why plant cells are strong and rigid?<br>- </strong>Plant cells have strong rigid cell wall on the outside cell membrane. This stops the cell bursting when it absorbs water by osmosis. The increase in pressure makes the cell rigid. This is useful as plants do not have skeleton. If plant cells lose too much water by osmosis they become less rigid and eventually the cell membrane shrinks away from the cell wall.<br><br><strong>2.) What are the components of Cell Membrane?<br></strong>-The principal components of the plasma membrane are lipids (phospolipids and cholesterol) proteins, and carbohydrate groups that are attached to some of the lipids and proteins.<br><br><strong>What are the components of Mitochondria?</strong><br>-Mitochondria are made of two membranes. The outer membrane covers the organelles and contains it like a skin. The inner membrane folds over many times and creates layered structures called cristae. The fluid contained in the mitochondria is called the matrix.<br><br><strong>What are the components Chloroplast?<br></strong>-Two membranes contain and protect the inner parts of the chloroplast. They are appropriately named the inner and outer membranes. The inner membrane sorrounds the stroma and the grana ( stacks of thykaloids). One of thykaloid stack is called a granum.<br><br><strong>3.)Explain how cell maintain it's life, energy and health?<br></strong>-The cell maintain it's life, health and energy if all the organelles will work and function properly, but once the cell is destroyed or damaged it will never work properly and there is no life, health and energy in the cell.</div>]]></description>
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         <pubDate>2018-06-25 04:12:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268404882</guid>
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         <title></title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268405736</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-06-25 04:19:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268405736</guid>
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         <title>Suarez, Alyanna Shin,\                                     Sarmiento, Nadine 8-Aristotle</title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268405792</link>
         <description><![CDATA[<div>1.) What is the composition of thr Cell Wall? Why plant cells are strong and rigid?<br>In the primary plant cell wall, the major carbohydrates are cellulose, hemicellulose and pectin.<br>The cell wall is tough, usually flexible but sometimes rigid layer that surrounds some types of cells. In plants and algae, the cell wall is made of long molecules of cellulose, pectine, and hemicellulose. The cell wall has channels which let some proteins and keeps others out.<br><br>2.)Components: Cell Membrane, mitochondria, Chloroplast<br>The principal components of the plasma membrane are lipids,, proteins, and carbohydrates groups that are attached to some of the lipids and proteins. A phispholipid is a lipid made of glycerol, two fatty acid tails, and phosphate-linked head group<br><br>The most prominent riles of mitochondria are to produce the energy currency of the cell, ATP, through respiration, and to regulate cellular metabolism. The central set of reactions involved in ATP production are collectively known as the citric acid cycle, or the Krebs cycle.<br><br>Two membranes contain and protect the inner parts of the chloroplast. They are appropriately named the outer and inner membranes. The inner membrane surrounds the stroma and the grana. One thykaloid stack is called a granum.<br><br>3.) How does the cell maintain its life?<br><br></div>]]></description>
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         <pubDate>2018-06-25 04:20:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268405792</guid>
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         <title>Suarez, Alyanna Shin, Sarmiento, Nadine  </title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268405794</link>
         <description><![CDATA[<div>1</div>]]></description>
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         <pubDate>2018-06-25 04:20:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268405794</guid>
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      <item>
         <title>Answer the following questions:</title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268406170</link>
         <description><![CDATA[<div>Figuracion, Reanne Kathleen R. <br>Tan, JB Earl G. <br>Emata, Simone Anne C. <br>(8-Aristotle)<br>1. What is the composition of cell wall? Why plants cell are strong rigids? <br>Composition of cell walls varies between species and may depend on cell type and developmental stage. <br><br>LAND PLANTS: <br>• polysaccharides cellulose <br>• hemicellulose <br>• pectin <br><br>ALGAE: <br>• glycoproteins <br>• polysaccharides (carrageenan and agar) <br><br>BACTERIA:<br>• peptidoglycon <br><br>Plant cells have strong rigid cell wall on the outside of the cell membrane. This stops the cell bursting when it absorbs water by osmosis. The increase in pressure makes the cell rigid. This is useful as plants do not have skeleton. <br><br>2. Components of the mitochondria <br>    Components of the cell membrane<br><br>MITOCHONDRIA: <br>• outer mitochondrial membrane<br>• intermembrane space (between outer and   inner membrane) <br>• inner mitochondrial membrane<br>• cristae (formed by infoldings of the inner membrane) <br>• matrix (space with the inner membrane) <br><br>CELL MEMBRANE<br>• lipids (phospholids and cholesterol) <br>• proteins<br>• carbohydrate groups that are attached to some of the lipids and proteins<br><br>3. How does cell maintain life? <br>The cell's immediate source of energy is a high-energy compound know as Adenosine Triphosphate (ATP), which may be derived from food.</div>]]></description>
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         <pubDate>2018-06-25 04:25:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268406170</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268406633</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-25 04:29:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268406633</guid>
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      <item>
         <title>3 questions </title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268406853</link>
         <description><![CDATA[<div><br><strong>1.What is the composition of cell wall?&nbsp;<br></strong><br></div>]]></description>
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         <pubDate>2018-06-25 04:31:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268406853</guid>
      </item>
      <item>
         <title>Biotechnology Assignment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268406928</link>
         <description><![CDATA[<div>Members:<br>Miase, Bea<br>Tariao, Darryl<br>Evangelista, Christian<br><br>1. What is the composition of Cell Wall?<br>&nbsp; Composition. In the primary (growing) plant cell wall, the major carbohydrates are <strong>cellulose</strong>,<strong>hemicellulose</strong> and <strong>pectin</strong>. The<strong>cellulose microfibrils</strong> are linked via<strong>hemicellulosic</strong> tethers to form the<strong>cellulose-hemicellulose network</strong>, which is embedded in the <strong>pectin</strong>matrix.<br><br>2. </div>]]></description>
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         <pubDate>2018-06-25 04:32:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268406928</guid>
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         <title>1</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268407614</link>
         <description><![CDATA[<div>Miase&nbsp;<br>Evangelista<br>Tariao<br><br>1.&nbsp;<br>*Why plant cell are strong and rigid?<br>Because of cell organelles. The cell walls in a plant are strong and adhere to each other, which helps to support the en</div>]]></description>
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         <pubDate>2018-06-25 04:40:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268407614</guid>
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      <item>
         <title>Bio</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268409029</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-25 04:56:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268409029</guid>
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         <title>BioTechnology Assignment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268409041</link>
         <description><![CDATA[<div>Lata, Ronan Raphael C.<br>Lapating, Angelie Mae M.<br>Dejillo, Lanz S.<br>Gr. 8-Aristotle<br><br>Questions:<br><br>1.) What is the composition of cell wall?<br>Cell Wall is composed of Cellulose,&nbsp; Hemicellulose, Pectin, Lingnin, Cutin, and Chitin.<br>&nbsp;Why is the cell strong and rigid?<br>Plant cells have strong rigid cell wall on the outside of the cell membrane. This stops the cell bursting when it absorbs water by osmosis. The increase in pressure makes the cell rigid.<br><br>2.) Parts of the:<br><br>Chloroplast&nbsp;<br>The chloroplast contains two membranes, the inner and outer membrane. It also has stroma, thylakoid, and grana.<br><br>Mitochondrion<br>The mitochondria as the outer mitochondrial membrane, the intermembrane space which is a space which is a space between the inner and outer membrane, the inner mitochondrial membrane, the cristae (formed by infoldings of the inner membrane),and the matrix (space within the inner membrane)<br><br>Cell Membrane<br>Phisopholipids form the basic structure of a cell membrane called the lipid bilayer. Scattered in the lipid bilayer are the cholesterol molecules, which help to keep the membrane fluid consistent. It also has membrane proteins that are important for transporting substances across the cell membrane<br><br>3.) Explain how cell maintain its life.<br>Life of a cell depend on its organelles. If the organelles will function well, the cell will function too, but if it has lacking organelles it will not work.<br><br><br><br></div>]]></description>
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         <pubDate>2018-06-25 04:57:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268409041</guid>
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      <item>
         <title></title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268409068</link>
         <description><![CDATA[<div>ae</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-25 04:57:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268409068</guid>
      </item>
      <item>
         <title>Suarez,Alyanna Shin</title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268409169</link>
         <description><![CDATA[<div>Sarmiento, Nadine<br><br></div>]]></description>
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         <pubDate>2018-06-25 04:59:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268409169</guid>
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         <title></title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268413491</link>
         <description><![CDATA[<div>S</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-25 05:59:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268413491</guid>
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      <item>
         <title></title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268413495</link>
         <description><![CDATA[<div>Suarez,Alyanna Shin<br>Sarmiento,Nadine<br>(8-Aristotle)<br><br>3.How cell maintains its life?<br>Answer: Cell Life Functions:Cell make up all living things and must carry out certain functions of living cells include transportation of molecules,</div>]]></description>
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         <pubDate>2018-06-25 05:59:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268413495</guid>
      </item>
      <item>
         <title>Biotechnology Assignment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268423580</link>
         <description><![CDATA[<div>Christian Ian Charles Salas<br>Alvin Jay Deleverio<br>Chrys jan S. Benjamin<br>(8-Aristotle)<br>1.) What is the copmposition of cell wall?<br>&nbsp; &nbsp; &nbsp; -cellulose<br>&nbsp; &nbsp; &nbsp;-Hemicelluose<br>&nbsp; &nbsp; &nbsp;-Pectin<br>&nbsp; &nbsp; &nbsp;-Cutin<br>&nbsp; &nbsp; -Enzyme<br>&nbsp; &nbsp; &nbsp;- Lignin'<br>&nbsp; &nbsp; &nbsp;-polysaccharide<br>Why plants cells are strong and&nbsp; rigid?<br>&nbsp; &nbsp;- Plant cells are strong rigid cell wall on the outside of the membrane.This stops the cell bursting when it absorb water by osmosis. The increase in pressure makes the cell rigid.<br>&nbsp; &nbsp;&nbsp;<br>2.) Components of:<br><br>Chloroplast-Stroma, thykaloid, Grana<br>&nbsp;<br>Mitochondria-Matrix, Cristae, Ribosome, Inner and outer mitochondria.<br><br>Cell membrane- Lipids, protiens, carbhohydrates, phospholipids, cholesterol.<br><br>3.) How do cells maintain its life?<br>      - &nbsp;&nbsp;</div>]]></description>
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         <pubDate>2018-06-25 07:45:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268423580</guid>
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      <item>
         <title>assignment in biotechnology 06/25/189</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268444275</link>
         <description><![CDATA[<div>Sangatanan, Shanlee<br>Lorania, Chorena<br><br>*what is the composition of the Cell Wall?<br>-Middle lamella<br>- Primary CLU<br>-Secondary CLU<br><br>*Why plants are strong and rigid?<br>&nbsp;<strong>Plant cells</strong> have a <strong>strong rigid cell</strong> wall on the outside of the <strong>cell</strong> membrane. This stops the <strong>cell</strong> bursting when it absorbs water by osmosis. The increase in pressure makes the <strong>cell rigid</strong>.&nbsp;<br><br>*what are the components of:<br>~mitochondria?<br>-outer mitochondrial membrane<br>-the inter membrane spore<br>the inner mitochondrial membrane<br>-cristae spore<br>-the matrix<br>~cell membrane?<br>-lipids (phopolipids &amp; cholesterol)<br>-proteins<br>-carbohydrates<br>~Chloroplast?<br>-outer membrane<br>-inner membrane<br>-stromal lamellae<br>-thylakoid<br>-stroma<br>-starch/sugar<br><br>*explain how cell organelles maintain the life, energy and health of the cell.<br> Cells have many structures inside of them called organelles. These organelles are like the organs in a human and they help the cell stay alive. Each organelle has it’s own specific function to help the cell survive. The nucleus of a eukaryotic cell directs the cell’s activities and stores DNA. Eukaryotes also have a golgi apparatus that packages and distributes proteins. Mitochondria are the power house of the cell and provide the cell with energy. Both plant and animal cells have mitochondria. Lysosomes are like the stomach of the cell. They contain enzymes that digest the cell’s used parts. All of the cell’s organelles must work together to keep the cell healthy&nbsp;</div>]]></description>
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         <pubDate>2018-06-25 10:20:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268444275</guid>
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      <item>
         <title>BIOTECHNOLOGY ASSIGNMENT</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268444631</link>
         <description><![CDATA[<div>Belicena, Michael Francis P.<br>Cagungao, Angela Lester<br><br>Questions:<br>1.What is the composition of cell wall?<br>&nbsp;The composition of cell walls varies between species and may depend on cell type and developmental stage. The primary cell wall of <a href="https://en.wikipedia.org/wiki/Embryophyte">land plants</a> is composed of the polysaccharides <a href="https://en.wikipedia.org/wiki/Cellulose">cellulose</a>, <a href="https://en.wikipedia.org/wiki/Hemicellulose">hemicelluloses</a> and <a href="https://en.wikipedia.org/wiki/Pectin">pectin</a>. Often, other polymers such as <a href="https://en.wikipedia.org/wiki/Lignin">lignin</a>, <a href="https://en.wikipedia.org/wiki/Suberin">suberin</a> or <a href="https://en.wikipedia.org/wiki/Cutin">cutin</a> are anchored to or embedded in plant cell walls. <br>-Why plant cell is rigid and strong?<br> <strong>Plant cells</strong> have a <strong>strong rigid cell</strong> wall on the outside of the <strong>cell</strong> membrane. This stops the <strong>cell</strong> bursting when it absorbs water by osmosis. The increase in pressure makes the <strong>cell rigid</strong>. <br>2.Composition of the ff.<br>Mitochondrion- The outer and inner membranes and crests may be considered as fluid and com molecular structures. The matrix is gel like and contains a variety of soluble proteins. Coe other smaller molecules are also obtained from the matrix. Several ribosomes and a circ DNA are situated in it. Besides, it contains enzymes for citric acid cycle, lipid and amino metabolism, protein synthesis and respiratory chain. <br>chloroplast- <strong>Chloroplasts</strong> contain proteins, lipids, and chlorophyll, carotenoids, RNA and DNA. Starch granules or osmiophillic droplets are also present in it. Proteins form a part of<strong>chloroplast</strong> membrane and lamellae, and are also present in the matrix in the form of enzymes.&nbsp;<br>Cell Membrane-Phospolipids,cholesterol,integral protein,periperal protien,carbohydrates.<br>3.Explain how cell mantain its life?<br>Cell maintain its life by the help of the organelles present on&nbsp; it. This organelles help the cell to survive.<br><br></div>]]></description>
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         <pubDate>2018-06-25 10:24:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268444631</guid>
      </item>
      <item>
         <title>Assignment in Biotechnology</title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268451051</link>
         <description><![CDATA[<div>Miase, Bea Clarisse A.<br>Evangelista, Christian Louie<br>Tariao, Darryl<br><br>1.<strong> Why are plant cells strong and rigid?</strong><br>-Because of the cell organelles. The cell walls in a plant are strong and rigid/flexible and adhere to each other, which helps to support the entire plant. All cells are surrounded by a cell wall/cell membrane that is rigid/flexible and interacts with the environment.<br><br>1.2 <strong>What are the composition of cell wall?<br></strong>-Middle Lamella, primary cell wall, plasma membrane, Cellulose Microfibrils, Cross-linking Glycan, and Pectin.<br><br>2. <strong>Components of chloroplasts</strong><br>-Outer Membrane, Inner Membrane, Stromal Lamellae, Thylakoid, Stroma, Starch or sugar<br><br>2.2. <strong>Parts of mitochondria</strong><br>-ATP Synthase patricles, Matrix, Cristae, Ribosome, Inter membrane space, Granules, DNA, Outer Membrane, Inner Membrane<br><br>2.3. <strong>Components/Parts of cell mambrane</strong><br>-Glycoprotein (Protein with carbohydrate attached), Protein Channel, Peripheral membrane protein, Integral Membrane protein, cytoskeletal filaments, Cholesterol, Glycolipid: liquid with carbohydrate attached, phospholipid Bilayer<br><br>3. <strong>Explain how cell maintain its life (energy and health)</strong><br>-An understanding of the workings of the cell enables an appreciation of both the capabilities and the limitations of living organisms whether animal, plant, fungus or microorganism. The convergence of cytology, genetics and biochemistry makes cell biology one of the most rapidly evolving disciplines in contemporary biology.&nbsp;</div>]]></description>
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         <pubDate>2018-06-25 11:24:47 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268451051</guid>
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      <item>
         <title>PERMEABILITY OF SPONGE (Osmosis Experiment)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268545697</link>
         <description><![CDATA[<div><br><strong>Members</strong>:<br>Berame, Bermadette Sue B.<br>Alberto, Achilles Alexis R.<br><br>Our experiment is about the permeability of the sponge or the ability of the sponge to allow water pass through iti, just like the cell membrane it is permeable (allowing water or gases to pass through it).<br><br><strong>l. Materials</strong>:<br>2 Sponge <br>1/2 cup of water<br>Basin<br>1/2 cup of salt<br><strong>Procedure:<br></strong>1. Find a place where you can perform this activity.<br>2.Prepare all the materials.<br>3. Prepare the basin and the sponge. While holding the sponge. Pour the 1/2 cup of water to the sponge.<br>4.Observe the changes and record it.<br>5.On the secondsponge put the 1/2 cup of salt.<br>6.Observe the changes and record it.<br><br></div>]]></description>
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         <pubDate>2018-06-26 03:43:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268545697</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268546347</link>
         <description><![CDATA[<div>Cabiles, Zyrreg Zhyller A.<br>Cutamora, Mizpah Gabrielle S.<br>Urdaneta, Caryl Jean P.<br>8—Aristotle<br><strong>I. Materials<br></strong>•Cloth<br>•Basin<br>•Water<br>•Coin<br><strong>II. Procedures<br></strong>1. Cover the basin with the cloth. Make sure you already concealed every opening.<br>2. Drop the&nbsp;coin in the cloth. Observe what happens.<br>3. Pour the water in the cloth. Observe what happens.</div>]]></description>
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         <pubDate>2018-06-26 03:51:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268546347</guid>
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      <item>
         <title>Walking Water (Osmosis Experiment) </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268546602</link>
         <description><![CDATA[<div>Members:<br>Nativadad, Deniel Dave<br>Enojales, Desire Faith<br>Juanico, Dj Christian<br>8-Aristotle<br><br>I. Materials:<br>- Plastic Cups<br>- Paper Towel<br>- Food Coloring<br>- Ketchup<br>- Water<br>II. Procedures:<br>1. Prepare all the materials needed in the experiment.<br>2. Prepare four plastic cups, two plastic cups for set A and two plastic cups for set B.<br>3. In set A fill one plastic cup with water and food coloring.&nbsp; Connect the plastic cup with water and the empty plastic cup with paper towel.<br>4. In set B fill one plastic cup with water and ketchup. Connect the plastic cup with ketchup and the empty plastic cup with paper towel<br>5. Wait overnight and observe the changes on the two sets.<br><br></div>]]></description>
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         <pubDate>2018-06-26 03:53:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268546602</guid>
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      <item>
         <title>OSMOSIS EXPERIMENT </title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268548632</link>
         <description><![CDATA[<div>Members: <br>Tan, JB Earl G<br>Emata, Simone Anne C. <br>Figuracion, Reanne Kathleen B. <br>(8 - Aristotle) <br><br><strong>I. Materials<br></strong>1. floral foam (Oasis) <br>2. water <br><strong>II. Procedure:<br></strong>Step 1: Prepare the materials needed in the experiment <br>Step 2: Pour water in the floral foam. <br>Step 3. Observe what happens to the floral foam and water. <br><br></div>]]></description>
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         <pubDate>2018-06-26 04:16:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268548632</guid>
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      <item>
         <title>OSMOSIS EXPERIMENT</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268548786</link>
         <description><![CDATA[<div>Members: <br>Bantes, Jannah H.<br>Hamajo, Mika C.<br>(8-Aristotle)<br><br>I. Materials<br>*Onion<br>*Salt<br>*Water<br><br>II. Procedures<br>1. Weigh the onion.<br>2. Fill a cup with water and add one tablespoon of salt.<br>3.Submerge the onion in the salt water.<br>4. Soak the onion for at least 8 hours.<br>5. Remove and rinse the onion.<br>6. Record the weight of onion.<br>7. Notice the differences between the onion before and after the experiment.<br>8. Record your result.</div>]]></description>
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         <pubDate>2018-06-26 04:18:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268548786</guid>
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      <item>
         <title>OSMOSIS EXPERIMENT</title>
         <author>alyannashinsuarez</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268549376</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:24:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268549376</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268550557</link>
         <description><![CDATA[<div>Members:&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<br>Suarez, Alyanna Shin<br>Sarmiento, Nadine A.&nbsp;<br>8-Aristotle<br><br>DISSOLVING EGG SHELLS WITH VINEGAR<br><br>I. MATERIALS:&nbsp;<br>* 3 eggs<br>* 3 containers<br>* 3 butter knives<br>* vinegar (3 cups)<br>* Tap water (2 cups)<br>* Cane vinegar (3 cups)<br>* Tongs<br>* Measuring cups and spoons<br>* Masking tape and marker<br><br>II. Procedure:<br>1.)Weigh each egg and record it. Place one egg in each glass, and pour in the limited amount of liquids in each glass. When bubbles start to form around the egg, put a butter knife in the glass to keep it from floating.<br>2.) Put the three glasses in the refrigerator and leave it for 24 hours.<br>3.) Pour out all of the liquids. And replace it all. Then, leave it in the refrigerator for 24 hours again. Repeat this process until the shells are fully dissolved and only the membrane remains. This should take about two to three days.<br>4.) Remove the eggs using the tongs. And rinse with tap water in the sink.<br>5.) Put the shell-less eggs aside for a moment on a plate.<br>6.) Prepare three different sugar-water solutions and label it. Put each shell-less eggs on each sugar solutions. put them in the refrigerator.<br>7.) Remove the glasses from the refrigerator. weigh the eggs and see the difference of the weights from before and after.<br><br></div>]]></description>
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         <pubDate>2018-06-26 04:35:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268550557</guid>
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      <item>
         <title>Biotechnoly</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551328</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:44:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551328</guid>
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      <item>
         <title>BioTechnoly</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551343</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:44:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551343</guid>
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      <item>
         <title>BioTechnolo</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551349</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:45:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551349</guid>
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      <item>
         <title>BioTechnology</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551356</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:45:05 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551356</guid>
      </item>
      <item>
         <title>BioTechnology</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551368</link>
         <description><![CDATA[<div>Mater</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:45:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551368</guid>
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      <item>
         <title>BioTechnology</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551382</link>
         <description><![CDATA[<div>Osmosis Experiment<br><br>Lapating, Angelie Mae M.<br>Dejillo, Lanz S.<br>Lata, Ronan Raphael C.<br><br>Materials:<br>Strainer<br>Basin<br>Water<br>Marble<br>Paper Clip<br><br>Procedures:<br>1. Prepare all the materials.<br>2. Hold the strainer on top of the basin.<br>3. Try to put different materials (Water, Marble, Paper Clip) on the strainer.<br>4. Determine the material which can and can't pass on the strainer.</div>]]></description>
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         <pubDate>2018-06-26 04:45:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551382</guid>
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      <item>
         <title>Os</title>
         <author>ajdeleverio</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551744</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:51:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551744</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author>ajdeleverio</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551745</link>
         <description><![CDATA[<div>Members:(Aristotle)<br>Alvin Jay Deleverio<br>Christian Ian Charles Salas<br>Chrys Jan Benjamin<br><br>I. Materials<br>&nbsp;8 potato slices<br>&nbsp;4 cups<br>&nbsp;4 concentrations of salt dissolved in 100ml of water<br><br>II. Procedure<br><br>1.Gather materials necessary for the experiment.<br>2.Using strands of potatoes and different concentrations of NaCl solution (salt), students design experiments to experiments to explore this osmosis in raw potatoes.<br>3.Explain that this closely models what happens to cells are n their throats when gargling with salt water.<br>4.Use controls in the experiment and collect and organize the data in meaningful ways</div>]]></description>
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         <pubDate>2018-06-26 04:51:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551745</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268551923</link>
         <description><![CDATA[<div><strong>Calonia,Francis Albert L.<br>Silva,Jasmin Imelda P.<br>Sulilap,Kenny Macjewr T.<br>(8-Aristotle)<br><br>PERMEABILITY OF CLOTH<br><br>I.Objectives:<br></strong>1. To classify the permeability of the cloth.<br>2.To know if other substance can pass through.<br><br><strong>II.Materials:</strong><br>2 pieces of cloth <br>3 cups of water(full,partially full and 40 mL)<br>2 food coloring<br>3 empty paper cups<br><br><strong>III.Procedures:</strong><br>1. Prepare the materials.<br>2. Put a pinch of food coloring in one of each paper cups filled with designated amount of water.<br>3. Fold the tissue one half lengthwise and put it on the cups.<br>4.Observe.<br>5.Make a conclusion. <br><br><br><strong>IV. Observations<br><br></strong>1.The absorbency of unsaturated water with tissue is greater than the super saturated one.<br>2.The process will be quicker if both of the substance  is unsaturated.<br>3.In almost every six minutes , an inch will be increased to the absorbency of the tissue in the unsaturated solution.<br><br><strong>V.Conclusion<br></strong>Therefore we conclude that,the permeability will be quicker if both of the substance is unsaturated.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:53:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268551923</guid>
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      <item>
         <title>3 Questions</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268552137</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:56:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268552137</guid>
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      <item>
         <title>3 Questions</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268552138</link>
         <description><![CDATA[<div><br>Members</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:56:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268552138</guid>
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      <item>
         <title>3 Questions</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268552139</link>
         <description><![CDATA[<div><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:56:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268552139</guid>
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      <item>
         <title>3 </title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268552140</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:56:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268552140</guid>
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      <item>
         <title>3 Questions</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268552142</link>
         <description><![CDATA[<div><br>Members:<br>Malunes, Jesse Justin P.<br>Sangatanan, Shandee S.&nbsp;<br>Seromines, Zofia Amina S.&nbsp;<br><br>1.What is the composition of the cell wall?&nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<br>&nbsp; &nbsp; Answer: The composition of cell wall are:<br>&nbsp; &nbsp; &nbsp;Middle lamella – outer cell wall layer that contains polysaccharides called pectins, Pectin aid in cell add hension by helping the cell wall of adjacent cells to bind to one another.&nbsp;<br>Primary CW – Layer formed between the middle lamella and plasma membrane in growing plant cells.&nbsp; It is primarily composed of cellulose microfibril contained within a gel-like matrix of nekicellulose fibers and pectin polysoccharides.&nbsp;<br>Secondary CW – Layer formed between the primary cell wall and plasma membrane in some plant cells.&nbsp; Once the primary&nbsp; cell wall has stopped dividing and growing, it may thicken to form a secondary wall<br><br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;-Why plants cells are strong rigid?&nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp;Answer- Plant cell have strong rigid cell wall on the outside of the cell membrane. This stops the Cell bursting when it absorbs water by osmosis. The Increase in pressure makes the cell rigid. This is useful as plants for not have a skeleton.&nbsp;<br><br><br>2. Components:Cell membrane&nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; Mitochondria&nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;Chloroplast<br><br>Cell membrane – Lipid(phosolipids &amp; cholesterol) &nbsp;<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; - Proteins<br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; - Carbohydrate group that are attached to some of the lipids and proteins<br>Mitochondria – Outer membrane<br>-	Inner membrane<br>-	Stormal lamellae<br>-	Thykaloid<br>-	Stroma<br>3.Explain how cell maintain its life<br>Answer: The cell is dynamic system of intersecting molecules that define life<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 04:56:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268552142</guid>
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      <item>
         <title>Fernandez</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268556031</link>
         <description><![CDATA[<div>Pertimos<br>Procedures<br>1 Place the peeled potato on the tile and using the knife, cut both ends of the potato to make it flat.</div><ol><li>Use the knife to make a cavity at the centre of the potato from one of the flat sides almost up to the bottom.</li><li>Pour distilled water into the Petri dish until it is half full.</li><li>Now, place the potato in the Petri dish.</li><li>Fill half the cavity made in the potato with 20% sugar solution.</li><li>Mark the level of sugar solution in the cavity using a pin.</li><li>The potato now functions as an osmometer.</li><li>Leave the osmometer undisturbed for about two hours.</li><li>Mark the rise in the level of the sugar solution in the cavity with another pin</li></ol><div>Materials:<br>Large potato without peel<br>Knife<br>Petri dish<br>Distilled water<br>Tile<br>Pins<br>20% sugat solutions.&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 05:49:05 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268556031</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268556201</link>
         <description><![CDATA[<div>Members:<br>Frajardo<br>Sabelita<br>Valdez<br><br><strong>Materials:<br></strong>Apple<br>Salt<br>Water<strong><br> </strong>A couple of drinking glasses.<br><br></div><div><strong>Procedure:<br></strong><br></div><ul><li>Fill two glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up an&nbsp;apple into French fry-like pieces</li><li>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.</li><li>Take a guess about how you think these slices might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it</li><li>Remove the&nbsp; pieces onto a plate and make your final observations</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 05:53:01 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268556201</guid>
      </item>
      <item>
         <title></title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268556318</link>
         <description><![CDATA[<div>Members<br>Buladaco<br>Flores<br>Cerado<br><strong>Materials:</strong></div><div>-A potato,<br>- salt, water (if you have distilled water, that kind is best)<br>-couple of drinking glasses.</div><div><br><strong>Procedure:<br></strong><br></div><ul><li>Fill two glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up a potato into French fry-like pieces</li><li>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.</li><li>Take a guess about how you think these slices might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it</li><li>Remove the  pieces onto a plate and make your final observations</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 05:55:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268556318</guid>
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      <item>
         <title>Mercado, Vergara, Cadiz</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268556460</link>
         <description><![CDATA[<div>Osmosis Ex</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 05:58:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268556460</guid>
      </item>
      <item>
         <title></title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268556551</link>
         <description><![CDATA[<div>Members<br>Buladaco<br>Flores<br>Cerado<br><strong>Materials:</strong></div><div>-A potato,<br>- salt, water (if you have distilled water, that kind is best)<br>-couple of drinking glasses.</div><div><br><strong>Procedure:<br></strong><br></div><ul><li>Fill two glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up a potato into French fry-like pieces</li><li>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.</li><li>Take a guess about how you think these slices might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it</li><li>Remove the  pieces onto a plate and make your final observations</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 06:00:05 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268556551</guid>
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      <item>
         <title></title>
         <author>kentfagutao</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268569406</link>
         <description><![CDATA[<div><strong>Osmosis Experiment</strong><br><br><strong>Members; </strong><br>Fagutao, Ramer Kent G. <br>Facurib, Jhonbert U. <br><br><strong>Materials:</strong><br><br>A turnip, salt, distilled water, a couple of drinking glasses.<br><br><strong>Procedure:</strong><br><br>1. Fill two glasses with distilled water.&nbsp;<br>2. In one of the glasses add 2-3 tablespoons of salt, and stir it in.&nbsp;<br>3. Slice up a turnip into French fry-like pieces.&nbsp;<br>4. Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.<br>5. Take a guess about how you think these slices might change by putting them into the different types of water.&nbsp;<br>6.Dunk the pieces in the water, and then let them sit overnight in it.&nbsp;<br>7. Remove the pieces onto a plate and make your final observations.&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 08:12:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268569406</guid>
      </item>
      <item>
         <title>Osmosis Procedure</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268570766</link>
         <description><![CDATA[<div><strong>Krystle Ann Z. Paguray<br>Princess Kyla M. Endaya<br>Angelle Kate Y. Brocoy</strong><br>(8-Mendel)<br><br><strong>I</strong>. <strong>Materials</strong></div><ul><li>Potato</li><li>Distilled Water</li><li>Salt</li><li>Drinking Glasses</li></ul><div><strong>II. Procedure</strong></div><ul><li>Fill two glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up a potato into French fry-like pieces</li><li>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.</li><li>Take a guess about how you think these slices might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it</li><li>Remove the&nbsp; pieces onto a plate and make your final observations</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 08:22:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268570766</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268583083</link>
         <description><![CDATA[<div>Belicena, Michael Francis P.<br>Cagungao, Angela Lester<br><br>Materials:</div><ul><li>6 slices of potato(French Fries cut)</li><li>water</li><li>Drinking Glasses</li><li>Salt</li></ul><div>Procedure:</div><ul><li>Fill the two Glasses With water.</li><li>In the first glass, add 1-2 tablespoon of salt.</li><li>Observe first the characteristics of the normal potato slices.</li><li>Divide the potato slices into 2, then dunk them in to the two types of water.Leave it them overnight.</li><li>Remove the pieces onto a plate and then observe.</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 10:15:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268583083</guid>
      </item>
      <item>
         <title>OSMOSIS EXPERIMENT </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268585430</link>
         <description><![CDATA[<div>Gabrielle Tyresse C. Galo&nbsp;<br>(8-Mendel)<br><br><strong>Materials:<br></strong>A potato, salt, water (if you have distilled water, that kind is best), a couple of drinking glasses.<br><br></div><div><strong>Procedure:<br></strong><br></div><ul><li>Fill two glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up a potato into French fry-like pieces</li><li>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.</li><li>Take a guess about how you think these slices might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it</li><li>Remove the&nbsp; pieces onto a plate and make your final observations</li></ul><div><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 10:37:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268585430</guid>
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         <title>Members:Mejares, Wendell Angelo Pette Bernard CoscosHoward Godwin Delos ReyesMaterials:A potato•salt•water (if you have distilled water, that kind is best)•couple of drinking glassesProcedure:Fill two glasses with waterIn one of the glasses add 2-3 tablespoons of salt, and stir it inSlice up a potato into French fry-like piecesMake your observations on these pieces: pay attention to color, how flexible it is, smell, etc.Take a guess about how you think these slices might change by putting them into the different types of waterDunk the pieces in the water, and then let them sit overnight in itRemove the  pieces onto a plate and make your final observations</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268586490</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 10:48:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268586490</guid>
      </item>
      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268586677</link>
         <description><![CDATA[<div>Members:<br>-Patrick James Lagang<br>-Jefferson Llamera<br>-EJ Carl&nbsp;Banlasan<br><br><strong>Materials:</strong><br>-potatoes; (2 raw ones and 1 boiled) <br>-tray<br>-potato peeler<br>-spoon<br>-knife<br>-tray<br>-water<br>-salt<br><br><strong>Procedure:</strong><br>1. Peel all the potatoes&nbsp;<br>2. Make a hole/cavity in the middle of all potatoes<br>3. Put or fill with salt the raw one and the boiled one, dont fill the other raw potato<br>4. Place all 3 potatoes in the tray ang fill the tray with water<br>5. Observe any changes happened on the 3 potatoes</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 10:50:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268586677</guid>
      </item>
      <item>
         <title>MERXADO, VERGARA, CADIZ</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268589653</link>
         <description><![CDATA[<div><strong>OSMOSIS EXPERIMENT&nbsp;<br><br>MATERIALS&nbsp;<br></strong>-A potato<br>-salt<br>-water( if have distilled water that kind is best)&nbsp;<br>-couple of drinking glasses<br><br><strong>PROCEDURE&nbsp;</strong></div><ul><li>Fill 2 glasses with water</li><li>In one of the glasses add 2-3 tablespoons of salt, and stir it in</li><li>Slice up a potato into french fry like pieces</li><li>Make your observation on these piece pay attention to color, how flexible it os, smell, etc.&nbsp;</li><li>Take a quess about how you think these slice might change by putting them into the different types of water</li><li>Dunk the pieces in the water, and then let them sit overnight in it&nbsp;</li><li>Remove the piece onto a plate and make your final observations</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 11:23:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268589653</guid>
      </item>
      <item>
         <title>Aristotle and mendel. You may conduct youractivity</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268590971</link>
         <description><![CDATA[<div>I. Title<br>II. Objectives:<br>III. Materials<br>IV. Observation<br>In here answer the questions if any in your activity.<br>Write your observation.<br>V. Conclusion</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 11:39:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268590971</guid>
      </item>
      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268591326</link>
         <description><![CDATA[<div>Baluso, James Lian David V.<br>(Individual, because my groupmates didn't help me)<br><br>Materials:<br>      One apple<br>      two small bowls<br>      knife<br>      tablespoon<br>      salt<br>      water<br>Procedure:<br>1. Fill the two bowls with same amount of water. And put one tablespoon of salt in one of the water.<br>2. Slice the apple into two, and after it place the one half of apple in one of the water, and the other place it on the other water.<br>3. Soak it 2-3 hours.<br>4. After a few hours check the apple and observe what happen.<br> <br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 11:43:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268591326</guid>
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      <item>
         <title></title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268591382</link>
         <description><![CDATA[<div>Grade 8&nbsp;<br>You can try other root crops like sayote or fruits like unripe mango.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 11:44:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268591382</guid>
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      <item>
         <title></title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268592260</link>
         <description><![CDATA[<div>Miase, Bea Clarisse<br>Evangelista, Christian Louie<br>Tariao, Darryl<br><br><br><strong>Materials:</strong><br>Basin<br>Water<br>Oil<br><br><strong>Procedures</strong>:<br><br>1. Prepare a 1 basin of half water inside.<br>2. Put a little of oil on the water<br>3. then put a drop of water on the oil.<br>4. Observe if what is happening in the after what you did.<br>5. Gather data<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-26 11:55:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268592260</guid>
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      <item>
         <title>Osmosis Experement </title>
         <author>mejaresw54</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268592822</link>
         <description><![CDATA[<div>Members:<br>Mejares, Wendell Angelo <br>Pette Bernard Coscos<br>Howard Godwin Delos Reyes<br>Materials:<br>•A potato<br>•salt<br>•water (if you have distilled water, that kind is best)<br>•couple of drinking glasses<br>Procedure:<br>Fill two glasses with water.<br>In one of the glasses add 2-3 tablespoons of salt, and stir it in.<br>Slice up a potato into French fry-like pieces.<br>Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.<br>Take a guess about how you think these slices might change by putting them into the different types of water.<br>Dunk the pieces in the water, and then let them sit overnight in it.<br>Remove the  pieces onto a plate and make your final observations.</div>]]></description>
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         <pubDate>2018-06-26 12:02:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268592822</guid>
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      <item>
         <title>OSMOSIS experiment activity</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268595809</link>
         <description><![CDATA[<div>sangatanan, shanlee<br>lorania, chorena grace<br><br>THE  WALKING WATER EXPERIMENT USING DIFFERENT TYPES OF PAPERS<br><br>I.<strong>objectives:</strong><br> To identify which type of paper is useful/effective on the walking water experiment.<br>II.<strong>materials</strong>:<br>*paper towel<br>*wet wipes<br>*manila paper<br>*3 empty clear glasses<br>*clean water<br>*3 sets of 2 colors of food coloring<br>   -red &amp; yellow<br>   -blue &amp; yellow<br>   -red &amp; blue<br>III. <strong>procedure<br>* </strong>add food coloring to a glass filled with water; and add another colr into the other.<strong><br></strong>* cut a paper towel into half and then fold it into quarters lengthwise.<br>* stick one end of the paper towel into the colored water and one end into the empty glass.<br>*repeat the procedures but using wet wipes instead of paper towels and then using manila paper.<br>*observe what action will happen.<br>note: this experiment is not yet conducted due to the addition of a new procedure, thus the observation and conclusion is not yet present.</div>]]></description>
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         <pubDate>2018-06-26 12:31:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268595809</guid>
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         <title>Osmosis Experiment</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268600910</link>
         <description><![CDATA[<div>Members:<br>Malunes, Jaesar Justin P.&nbsp;<br>Sangatanan, Shandee S.&nbsp;<br>Seromines, Zofia Amina S.&nbsp;<br><br><br>MATERIALS:<br>*water<br>*1 spoon of salt<br>*2 glasses<br>*3 gummy bears<br><br>PROCEDURE:<br>1. Pour half of water to the 2 glasses.&nbsp;<br>2. Put 1 spoon of salt into one of the glasses and mix it until the salt dissolves.&nbsp;<br>3. Put the 2 gummy bears in each of the glasses and leave the other 1 for measuring impact.&nbsp;<br>4. Leave the glasses for 2 hours and observe the glasses every 30 minutes.<br>5. Gather/collect data and make a conclusion.&nbsp;<br><br></div>]]></description>
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         <pubDate>2018-06-26 13:08:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268600910</guid>
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         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268684505</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-06-27 04:09:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268684505</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268684530</link>
         <description><![CDATA[<div><strong>Members:</strong><br>Bantes, Jannah H.<br>Hamajo, Mika C. (8-Aristotle)<br><br><strong>I. Title<br></strong>SCIENCE EXPERIMENT IN THE OSMOSIS OF AN ONION<br><br><strong>II. Objectives</strong><br>To know how much the salt water is absorbed by the onion<br><br><strong>III. Materials</strong><br>*onion<br>*water<br>*salt<br><br><strong>IV. Observations</strong><br>-As the onion is soaked in the water, there is something surrounding its body.<br>-The color of the onion is slowly being pale<br><br><strong>V.&nbsp; Conclusion</strong><br>-The weight of the onion will increase if it is soaked in the salt water.</div>]]></description>
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         <pubDate>2018-06-27 04:10:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268684530</guid>
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         <title>Osmosis Experiment</title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268717848</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-06-27 10:48:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268717848</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268717856</link>
         <description><![CDATA[<div>Members: (8- Mendel)<br>Palmaera, Kristine Joy B.<br>Agorilla, Julianne B.<br>Omawing, Merryl C.<br><br><strong>I. Title<br></strong>EXPERIMENT IN THE OSMOSIS OF A POTATO<br><br><strong>II. Objectives<br></strong>- to know if the potato will be fresher if soak in regular water or salt water.<br><br><strong>III. Materials<br></strong>- potato<br>- salt<br>- distilled water<br>- couple of drinking glass<br><br><strong>IV. Procedure<br></strong>1. Prepare all&nbsp; the materials needed.<br>2. Fill two glasses with water<br>3. In one of the glasses, add 2-3 tablespoons of salt and stir it in.<br>4. Slice up a potato into french fry like pieces.<br>5. Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.<br>6. Take a guess about how you think these slices might change by putting them into the different types of water.<br>7. Dunk the pieces in the water, and then let them sit overnight in it.<br>8. Remove the pieces onto a plate and make your final observation.</div>]]></description>
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         <pubDate>2018-06-27 10:48:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268717856</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268721485</link>
         <description><![CDATA[<div>Baluso, James Lian David V.&nbsp;<br>8-Mendel<br><br>I. Title<br>Experiment in The Osmosis of an Apple.<br><br>II. Objective<br>To know how much water or salt water can be absorbed by an apple in 2 hours.<br><br>III. Materials<br>-two small bowls<br>-apple&nbsp;<br>-tablespoon<br>-knife<br>-salt<br>-water<br><br>IV. Procedures<br>1.&nbsp; Fill the two bowls with same amount of water. And put one tablespoon of salt in one of the water.<br>2. Slice the apple into two, and after it place the one half of apple in one of the water, and the other place it on the other water.<br>3. Soak it 2-3 hours.<br>4. After a few hours check the apple and observe what happen.<br>&nbsp;<br>V. Observations<br>-As time passes the color of the apple's flesh became brown.<br>-The apple that soaked in a water with a salt became flexible than the apple that soaked in a normal water.<br><br>VI. Conclusion<br>-There will be change on form of an apple if it soak into a water with salt.</div>]]></description>
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         <pubDate>2018-06-27 11:29:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268721485</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268790057</link>
         <description><![CDATA[<div><strong>Members</strong>:<br>Berame,Bernadette Sue B.<br>Alberto, Achilles Alexis R.<br><strong><br>I.Title<br></strong>Permeability Of The Sponge<br><br><strong>II</strong>.<strong>Objective:<br></strong>1.)To know how the sponge responses to water and the salt.<br>2.)To know how the cell membrane function, represented by the sponge.<br><br><strong>lll.Materials:<br></strong>Sponge <br>Basin<br>1/2 cup of water<br>1/2 cup of salt<br><br><strong>lV.Observation:<br></strong>- The sponge can absorb water and water can pass through the sponge, but the salt can't.<br>-the sponge is heavier when poured with water, but when the salt is added the sponge became heavier but not too much unlike the water.<br><br><strong>V.Conclusion:<br></strong>The water can pass through the sponge that's why sponge is permeable to water, but the sponge is not permeable to salt. Just like the cell membrane, it only letscertain molecules to enter and exit.</div>]]></description>
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         <pubDate>2018-06-27 23:17:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268790057</guid>
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         <title>Osmosis </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268809490</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-28 02:45:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268809490</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268809493</link>
         <description><![CDATA[<div>Members:<br>Lata, Ronan Raphael C.<br>Lapating, Angelie Mae M.<br>Dejillo, Lanz S.<br><br>I. MATERIALS:<br>Cucumber<br>Peeler<br>Cutter<br>Spoon<br>Salt<br>Cups<br>Tap Water<br>Distilled Water<br>Digital Weighing scale<br><br>II. PROCEDURES:<br>1. Prepare all the materials.<br>2. Peel and slice the cucumber.<br>3. Fill an ordinary tap water on the 1st cup and a distilled water on the 2nd cup.<br>4. Add 2 spoons of salt on the 1st cup with a tap water.<br>5. Drop and leave the cucumbers on the cups with water for one hour.<br>6. Take out the slices and tap it gently on the paper.<br>7. Weigh the cucumber slices.<br>8. Drop and leave the cucumbers on the cups with water for one hour again.&nbsp;<br>9. Weigh the cucumber slices.<br><br></div>]]></description>
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         <pubDate>2018-06-28 02:45:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268809493</guid>
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      <item>
         <title>Al</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268815757</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-28 03:58:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268815757</guid>
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      <item>
         <title>Suarez, Alyanna Shin.                                              Sarmiento, Nadine </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268815758</link>
         <description><![CDATA[<div>I. Title: Dissolving Egg Shells With Vinegar<br><br>II. Objectives:<br>1.) See which liquid will dissolve the eggs.<br>2.) Understand how and why the egg shells dissolve or not dissolve, in a way the we can relate to cells.<br><br>III. Materials:<br>3 eggs&nbsp;<br>3 containers<br>Measuring cups and spoons<br>Butter knives<br>White vinegar<br>Cane vinegar<br>Distilled water<br>Corn syrup<br>Tongs<br>Masking Tape&nbsp;<br>Writing Utensil<br><br>IV. Procedure:<br>1.) Place one egg in each glass. Pour in enough liquid to cover each egg. Bubbles will start to form around the egg, an it'll float up. To keep it submerged, put a butter knife in the glass to hold it down.<br>2.) Put the three containers in the refrigerator for 24 hours.<br>3.) Gently holding the egg in the glass, pou out the old vinegar and replace with the fresh vinegars and water. Put them in the refrigrator for 24 hours. Repeat this process until the shells are fully dissolved and only the membrane remains. This should take 2-3 days.<br>4.) Remove the eggs using the tongs and rinse with tap water in the sink.<br>5.) Put the shell-less eggs aside for a moment on a plate.<br>6.) Prepare 3 diffeeent sugar water solutions. Weigh each of them first. Then, put each shell-less eggs on each solutions. Put them in the refrigerator for 24 hours.<br>7.) Remove the glasses from the refrigerator and gently put the eggs on a plate. If you weighed the eggs before putting them in each solution, weigh them again.<br><br>V. Observation:<br>The shells of each egg in each vinegar solutions dissolved slowly. When weighed, the eggs got slightly lighter. The shell-less eggs got shriveled up when it was out of the sugar solutions.<br><br>VI. Conclusion:<br>The Cane vinegar and the white vinegar acts as the nutrients, while the eggs shells act as the cell membrane. The cell membrane allows the some nutrients in, the vinegar. While it also lets some nutrients out, like the water. Thats why the the egg shells dissolved.</div>]]></description>
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         <pubDate>2018-06-28 03:58:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268815758</guid>
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      <item>
         <title>Alyanna</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268815759</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-06-28 03:58:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268815759</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268848191</link>
         <description><![CDATA[<div>Members:<br>Belicena, Michael Francis P.<br>Cagungao, Angela Lester<br><br></div><div>I.<strong>Title<br></strong>Define osmosis using Potato<br><br><strong>II.Objectives<br></strong>To know what will change in the potatoes' characteristic when left in the water.<br><br><strong>III. Materials</strong></div><ul><li>6 slices of potato(French Fries Like)</li><li>water</li><li>2 drinking glasses</li><li>salt</li></ul><div><strong>IV. Observations<br></strong>Before-<br>The potato slices are color yellow. It is fresh and not flexible.<br>After-<br>Regular water-<br>It look like fresh as if it is newly cut potato. It is firm and crisp.<br>Salted water-<br>The potato slices feels smooth but the color of it change and turn into brown. It is now flexible.<br><br><strong>V. Conclusion<br></strong>Water will move from an area with less salt than more salt,just like the regular water and the potato. The potato stay at its normal characteristics.While in the case of the salted water and the potato the potato get flimsy and not crisp because the water that is inside on it move out by osmosis</div>]]></description>
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         <pubDate>2018-06-28 10:32:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268848191</guid>
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      <item>
         <title>Kano, Safwan A.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268849377</link>
         <description><![CDATA[<div><strong>Osmosis Experiment</strong><br>I. Title<br> Dissolving Egg Shells With Vinegar<br>&nbsp;<br>II. Objectives:<br>1. To dissolve the egg shell.<br>2. To know which solution will dissolve the egg shell.<br>3. To know how and by why the egg dissolve by the vinegar. How can you relate this to the cell ?<br><br>III. Materials:<br>&nbsp;</div><ul><li>3 eggs</li><li>3 glasses (large enough to fit the egg plus liquid)</li><li>3 butter knives</li><li>White vinegar (about 3 cups)</li><li>Distilled water (about 2 cups)</li><li>Light corn syrup (about 1 ¼ cups)</li><li>Slotted spoon</li><li>Measuring cup (1 cup)</li><li>Measuring spoons (1 tablespoon and ½ tablespoon)</li><li>Sticky notes and marker</li><li>Scale (optional)&nbsp;</li></ul><div><br>IV. Procedure/s:<br>&nbsp;<em>Note</em>: It’s okay to touch the eggs, but remember to wash your hands afterwards to avoid any nasty surprises!&nbsp;<br><br></div><ol><li>Place one egg in each glass. Pour in enough vinegar to cover each egg. Bubbles will start to form around the egg, and it’ll float up. To keep it submerged, put a butter knife in the glass to hold it down.&nbsp;</li></ol><div><br>&nbsp; &nbsp; 2. Put the three glasses in the refrigerator and allow to sit for 24 hours. <br><br>&nbsp; &nbsp; &nbsp;3. <em>Gently</em> holding the egg in the glass, pour out the old vinegar. Replace with fresh vinegar, and let&nbsp; sit in the refrigerator for another 24 hours. Repeat this process until the shells are fully dissolved and only the membrane remains. This should take about 2-3 days. <br><br>&nbsp; &nbsp; &nbsp; 4. <em>Gently</em> remove the eggs using the slotted spoon and rinse with tap water in the sink. Rinse out the empty glasses as well. <br><br>&nbsp; &nbsp; &nbsp; &nbsp;5. <em>Gently</em> put the shell-less eggs aside for a moment on a plate. <br><br>&nbsp; &nbsp; &nbsp; &nbsp; 6. Prepare three different sugar-water solutions as follows, labeling with sticky notes: <br><br>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;7. Remove the glasses from the refrigerator, and <em>gently</em> put the eggs on a plate. If you weighed the eggs before putting them in each solution, weigh them again. What happened to each of the eggs?&nbsp;<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-06-28 10:48:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268849377</guid>
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         <title>Osmosis Experiment </title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268853454</link>
         <description><![CDATA[<div><strong>Members: <br></strong>Tan, JB Earl <br>Figuracion, Reanne Kathleen <br>Emata, Simone Anne <br><br><strong>I. Title<br></strong>Porosity of Floral Foam<br><br><strong>II. Objective<br></strong>1.) To know what will happen to the foam after poured with water. <br>2.) To see if the weight of the foam increased. <br><br><strong>III. Materials</strong><br>- floral foam <br>- water <br>- basin (optional) <br><br><strong>IV. Observation/s<br></strong>Before the experiment started the weight of the foam was lighter than the weight of it after the experiment since it became heavier . <br><br><strong>V. Conclusion<br></strong>The floral foam will be heavier when poured with water&nbsp;</div>]]></description>
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         <pubDate>2018-06-28 11:40:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268853454</guid>
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         <title>Omosis Experiment</title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268862500</link>
         <description><![CDATA[<div><strong>Miase</strong>, Bea Clarisse<br><strong>Evangelista</strong>, Christian Louie<br><strong>Tariao</strong>, Darryl<br><br><strong>I. Title: Who has greater density? Water or oil?<br><br>II. Objective</strong>:<br>1. To understand if which has more density: the water or the oil.<br>2. To relate the experiment in the function if a cell membrane.<br><br><strong>III. Materials:<br>Water<br>Oil<br>Basin<br></strong><br><strong>IV. Observation</strong>:<br>&nbsp; When we poured a few of oil in the water in the basin the oil spread itself with surprising swiftness upon the surface.<br>Then after we pour a few of water in the oil on the surface of water the water goes directly under the oil and it became a part of the water in the basin.<br>It looks like the function of cell membrane.<br><br><strong>V. Conclusion</strong>:<br>&nbsp; The function of the cell membrane is the same with the oil on the surface of the water, when we pour a few of water on the oil the oil just allow the water to go under it because of its less density, while in cell membrane only allows the ones which are needed by the cell and also separates the inner and outer environment if a cell</div>]]></description>
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         <pubDate>2018-06-28 13:04:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268862500</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268884032</link>
         <description><![CDATA[<div>Silva,Jasmin Imelda P.<br>Sulilap,Kenny Majewr T.<br>Calonia,Francis Albert L.<br><strong>(8-ARISTOTLE)<br><br>I.Materials:</strong><br>2 pieces of cloth <br>3 cups of water(full,partially full and 40 mL)<br>2 food coloring<br>3 empty paper cups<br><br><strong>II.Procedures:</strong><br>1. Prepare the materials.<br>2. Put a pinch of food coloring in one of each paper cups filled with designated amount of water.<br>3. Fold the tissue one half lengthwise and put it on the cups.<br>4.Observe.<br>5.Make a conclusion.</div>]]></description>
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         <pubDate>2018-06-28 16:27:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268884032</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268935044</link>
         <description><![CDATA[<div>Cabiles, Zyrreg Zhyller A.<br>Cutamora, Mizpah Gabrielle S.<br>Urdaneta, Caryl Jean P.<br><br><strong>I. Title<br>The Permeability of Cloth<br><br>II. Objectives<br></strong>•To know the permeability of cloth.<br>•To know how cell membrane works.<br><strong><br>III. Materials</strong><br>•Basin<br>•Cloth<br>•Water<br>•Coin<br><br><strong>IV. Observation</strong><br>When the coin was dropped, we observed that it was not passing through the cloth, but when we poured the water, we noticed that the water passed through the cloth and went to the basin.<br><br><strong>V. Conclusion</strong><br>The function of cell membrane is the same to the cloth because of it controls the movements of substances and lets specific nutrients in and out of the cell which makes it semipermeable like the cloth.</div>]]></description>
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         <pubDate>2018-06-29 03:53:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268935044</guid>
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      <item>
         <title>Osmosis Experiment</title>
         <author>christiansalas68</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268959398</link>
         <description><![CDATA[<div>Christian Ian Charles R. Salas<br>Alvin Jay A. Deleverio<br>Chrys Jan S. Benjamin<br><strong>I. Title<br>Osmosis In Potatoes<br>II. Objectives <br></strong>- To apply osmosis using potatoes.<br>- To know if water can get inside of raw and boiled potatoes.<strong><br>III. Materials<br></strong>-Cutter or Knife<br>-Potatoes ( 2 raw &amp; 1 boiled )<br>-Water<br>-Tray<br><strong>IV. Procedures<br></strong>- Prepare all the materials needed.<br>- Peel the boiled and raw potatoes.<br>- Make a cavity on the middle of the potatoes.<br>- Put each potatoes inside the tray.<br>- Add salt on boiled potato and on the raw potato ( only one ) with the same amount.<br>- Pour the tray with water.<br>- Observe what will happen<br>- Make conlusions.<strong><br>V. Observations<br></strong>We observe that the salt on the raw potato slowly melted but the salt on the boiled potato did not melt.<strong><br>VI. Conclusion<br></strong>Osmosis is a process in which water passes across the cell membrane. The salt in raw potato melted because the salt in raw potato drains out water from the cells lining the cavity due to osmosis. The salt in boiled potato did not melt because there is no collection of water which is because of the death of the cells due to boiling.<br><br>" So, cells need to be alive for osmosis take place."<br><strong><br></strong><br><br></div>]]></description>
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         <pubDate>2018-06-29 10:17:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268959398</guid>
      </item>
      <item>
         <title>Kano, Safwan A.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268966267</link>
         <description><![CDATA[<div><strong>Osmosis Experiments Continuation :<br></strong><br><strong>V. Observation</strong><br>As the day passes you can see that the drowned shell-less egg to the vinegar slowly get dissolve while the drowned shell-less egg drowned to the sugar solution get wrinkled. And their weight changes.<br><br><strong>VI. Conlusion<br></strong>The vinegar serve as the nutrients to the cell membrane (egg shell), By the function of the cell membrane that why the egg shell dissolve.<strong><br></strong><br></div>]]></description>
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         <pubDate>2018-06-29 12:19:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268966267</guid>
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      <item>
         <title></title>
         <author>mejaresw54</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268970233</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295999563/a9db7838c32174a5043b8391ab8ba89d/B_tech_c.docx" />
         <pubDate>2018-06-29 13:05:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268970233</guid>
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         <title>Osmosis Experiment</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/268971634</link>
         <description><![CDATA[<div>Juanico, DJ Christian<br>Natividad, Deniel Dave&nbsp;<br>Enojales, Desire Faith<br>(8- Aristotle)<br><br>I.TItle<br>&nbsp;- Walking Water ( Osmosis Experiment)<br><br>II. Objectives<br>&nbsp;- To know the permeability of the tissue on two liquids<br>&nbsp;- To know how cell membrane work.<br><br>III. Materials<br>&nbsp;- Ketchup<br>&nbsp;- Water<br>&nbsp;- Food Coloring<br>&nbsp;- Paper Towel<br>&nbsp;- Plastic Cups<br><br>IV. Observations<br>&nbsp;- After we left the two sets overnight we observed that the Set A (water) transferred half of its water to the other cup while the Set B (ketchup) have not transferred its ketchup in the other cup at all.<br><br>V. Conclusion&nbsp;<br>&nbsp;The function of cell membrane is the same as the paper towel it controls all the movement of substances going in and out of the cell like when the paper towel allows the water to pass through the cell (the other cup) because it have a specific function for the cell while the ketchup is not allowed to pass through because it can be considered as a foreign body to the cell.&nbsp;<br><br><br><br></div>]]></description>
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         <pubDate>2018-06-29 13:18:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/268971634</guid>
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         <title>Osmosis Experiment  </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269012386</link>
         <description><![CDATA[<div>Lata, Ronan Raphael C.<br>Lapating, Angelie Mae<br>Dejillo, Lanz S.<br>Gr. 8- Aristotle<br><br><strong>I. Title</strong><br>Osmosis Of a Cucumber<br><br><strong>II. Objectives</strong><br>To know how much water is absorbed by the cucumber.<br><br><strong>III. Materials<br></strong>• Cucumber<br>• Peeler<br>• Cutter/Knife<br>• Spoon<br>• 2 Cups<br>• Salt<br>• Tap Water<br>• Distilled Water<br>• Digital Weighing Scale<br><br><strong>IV. Procedures</strong><br>1. Prepare all the materials.<br>2. Peel and slice the cucumber.<br>3. Fill an ordinary tap water on the first cup &amp; fill a distilled water on the second cup.<br>4. Add two spoons of salt on the 1st cup with tap water.<br>5. Drop &amp; leave the cucumbers on the cups of water for one hour.<br>6. Take out the slices and tap it gently on the paper.<br>7. Weigh the cucumber slices.<br>8. Drop and leave the cucumbers for one hour again.<br>9. Weigh the cucumber slices. <br><br><strong>V. Observation</strong><br>• The absorbency of the cucumber slice of the fist cup with tap water &amp; two spoons of salt decrease. It loose three grams in two hours.<br>• The absorbency of the cucumber slice on the second cup increase. It gained two grams in two hours.<br><br><strong>VI. Conclusion</strong><br>The function iof cell membrane is to allow specific materials to move in and out like the cucumber increasing and decreasing its weight.</div>]]></description>
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         <pubDate>2018-06-30 03:53:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269012386</guid>
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         <title>Osmosis Experiment ( 8 - Mendel )</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269014401</link>
         <description><![CDATA[<div><strong>Members:<br>&nbsp;</strong>Valdez, Fajardo, Sabelita<br><strong>Title:</strong> <br>Osmosis using apple.<br><strong>Objective</strong>:<br>To identify the change and characteristic of the apple when left in the water and salt water.&nbsp;</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298129699/f1c82d38fb9e6845b85c649ad96ac76d/osmosis_experiment123.docx" />
         <pubDate>2018-06-30 05:08:01 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269014401</guid>
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         <title>osmosis experiment (8-aristotle)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269017829</link>
         <description><![CDATA[<div>Sangatanan,shanlee<br>Lorania,Chorena Grace<br><br>I.<strong>Title<br></strong>-The walking water experiment<br><br>II.<strong>Objective/s<br></strong>-to identify which type of paper will be effective in the transfer of fluids.<br>-to identify which type of paper is "healthy enough" to serve as a component of the cell membrane.<br>-to identify the relationship of the materials to the components of the cell membrane.<br>-to identify which type of paper could be identified as a component of the cell membrane.<br><br><strong>III. Materials<br></strong>-three types of papers:<br>~two strips of manila paper<br>~two strips of paper towel<br>~two wet wipes.<br>-three glasses of water<br>-two colors of any coloring (we used red and blue)<br><br><strong>IV.Procedures<br></strong>-prepare the materials.<br>-fill two glasses with water.<br>-add one color of coloring to each glass, mix well.<br>*as you drop/ add the coloring,you could see the diffusion of the colors to the water, this is the action when nutrients are being diffused to the liquid.<br>*the glasses with water and coloring will now serve as the "outside" of the animal cell,<br>-place the empty glass between the two glasses with water and coloring.<br>*the empty glass now serves as the animal cell.<br>*the glass itself now serves as the cell membrane.<br>-place one strip paper towel with one end to the glass with red coloring and the other to the empty glass; and the other strip of paper towel with one end to the glass with blue coloring and the other to the empty glass.<br>* the paper towel is now the proteins of the cell membrane that selectively transport channels into the cytoplasm or the inside of the animal cell.<br>-leave it like that until the level of the liquids are now all equal. then you're finish.<br>-observe what happens as the "channels" were transported inside the cell.<br>-repeat all procedures but using strips ofmanila paper instead of paper towels; wet wipes instead of paper towels.<br>*the paper towels are slightly dry; the wet wipes are wet (of course); and the manila paper is completely dry. we're going to identify which one is a "healthy" protein.<br><br><strong>V. Observations<br><br></strong>GENERAL OBSERVATION<br>*the colors of two glasses (red and blue) were mixed.<strong><br><br></strong>PAPER TOWEL:<br>*the transport was very slow that it took 28 hours before the level of the liquids became equal. however, the experiment became a success because the "blue water" was mixed with the "red water" and it turned into a "violet water".<br><br>MANILA PAPER:<br>*the transport only took 17 hours but it wasn't successful because only the water was able to be transported to the empty glass.<br><br>WET TOWEL<br>*the transport took 1.5 hours and its a success.<br><br><strong>VI. Conclusion</strong><br>* the wet wipes is highly effective for the transfer because it only took a short time for the activity to be finished.<br>*its also a near replica to the proteins of the cell membrane. we believed that the transport of the channels only need a short period of time so the wet wipes are really a near replica to the protein.<br><strong>Thus, we finally conclude that wet wipes are near replicas of protein because it transported the liquids successfully just as the protein transport the channels to the cytoplasm.</strong><br><br><br><br><br></div>]]></description>
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         <pubDate>2018-06-30 07:44:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269017829</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269019493</link>
         <description><![CDATA[GENERAL OBSERVATION
*the ]]></description>
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         <pubDate>2018-06-30 08:46:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269019493</guid>
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         <title></title>
         <author>kentfagutao</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269046080</link>
         <description><![CDATA[<div><strong>Osmosis experiment </strong><br><strong>Fagutao, Ramer Kent<br>Facurib, Jhonbert <br>Fajartin, Lrome<br>I. Title: </strong><br>The osmotic action of turnip in distilled water and distilled water with salt. <br><strong>II. Objective</strong>: <br>-To determine if the distilled water with salt can get inside of raw turnip. <br>-To determine how osmotic action affects the turnip. <br><strong>III. Materials: </strong><br>-2 slices of fresh turnip (French fries-like)<br>-Salt <br>-Distilled water <br>-Drinking glasses <br><strong>IV. Procedure:</strong><br>1. Fill two glasses with distilled water. <br>2. In one of the glasses add 2-3 tablespoons of salt, and stir it in. <br>3. Slice up a turnip into French fry-like pieces. <br>4. Make your observations on these pieces: pay attention to color, how flexible it is, smell, etc.<br>5. Take a guess about how you think these slices might change by putting them into the different types of water. <br>6.Dunk the pieces in the water, and then let them sit overnight in it. <br>7. Remove the pieces onto a plate and make your final observations&nbsp; <br><strong>V. Observation</strong>: <br><strong>Before</strong> <br>-The turnip slices are color white. It is fresh and cannot bend easily. <br><strong>After</strong> <br><strong>Distilled water </strong><br>-The slices turnip looks like fresh as if it is newly cut turnip. It is also firm, crisp and if you will break it, it snaps. <br><strong>Distilled water with Salt </strong><br>-The sliced turnip feels smooth and can be easily bend but some of its parts turn into brown. And it is now flexible. <br><strong>V. Conclusion</strong>&nbsp;<br>-In this case the sliced turnip in salt became more flexible and some of its parts turn into brown due to osmosis because of osmosis is a process in which after passes across the cell membrane. While in the sliced turnip with distilled water the turnip remains the same as if is newly cut turnip because there is no osmotic action happened to the experiment.&nbsp;<br><br></div>]]></description>
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         <pubDate>2018-06-30 21:47:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269046080</guid>
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         <title>Ç</title>
         <author>jhonbertfacurib27</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269109173</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-07-02 02:09:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269109173</guid>
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         <title>Osmosis experime</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269122128</link>
         <description><![CDATA[<div>Pertimos and fernandez<br>General observations&nbsp;<br>After 2 hours the measurement of the sugar solution increased and the potato didnt change its color. </div>]]></description>
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         <pubDate>2018-07-02 05:41:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269122128</guid>
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         <title>The </title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269154468</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-07-02 12:16:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269154468</guid>
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         <title>The Osmosis Experiment</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269154473</link>
         <description><![CDATA[<div>Members:<br>Malunes, Jaesar Justin P.&nbsp;<br>Seromines, Zofia Amina S.&nbsp;<br>Sangatanan, Shandee S.&nbsp;<br>8-Aristotle&nbsp;<br><br><strong>Title:&nbsp;</strong>Gummy Bear Osmosis Experiment&nbsp;<br><br><strong>Objectives:&nbsp;<br></strong>- To identify the effects of plain/salt water to the gummy bears. <br>-To know what is the effect of plain/salt water to the size of the gummy bears. <br><br><strong>Material</strong>: Water &nbsp; 1 table spoon of salt <br>2 Glasses &nbsp; 3 gummy bears<br><br><strong>Procedure:&nbsp;<br>1.&nbsp;</strong>Pour half water to the 2 glasses and label it, to determine which glass of water has salt.&nbsp;<br><strong>2.&nbsp;</strong>Put a 1 spoon of salt into one of the glasses and mix it until the salt dissolves.<br><strong>3.&nbsp;</strong>Put the 1 gummy bear in each of the glasses and leave the other 1 for measuring impact.&nbsp;<br><strong>4.&nbsp;</strong>Leave the glasses for 2 hours and observe the glasses every 30 minutes.&nbsp;<br><strong>5.&nbsp;</strong>Gather/Collect data and make conclusion.<br><br><strong>Observations:</strong><br>- The gummy bear absorbed the water in the plain water, while in the salt water the gummy bear released water.&nbsp;<br>- After the experiment the gummy bear inside the plain water gets bigger, while in the gummy bear inside the salt water shrieked.&nbsp;<br><br><strong>Conclusion:<br>&nbsp;</strong>Osmosis is a kind of diffusion. When diffusion occurs, molecules move from a higher concentration of water towards a lower concentration of water.&nbsp;<br><br>- The water outside the cell has less water than inside of the cell to the outside. That is what happened to the gummy bear in the salt. The water had to move out of the gummy bear to "even out" the concentration of water. So the gummy bear became smaller with less water to fill it up. Since there was a salt in the water and the gummy bear, the water didn't have to move as much to "even out" the concentration.&nbsp;<br><br>- The opposite happened to the gummy bear in the plain water. Water moved from the outside of the gummy bear to the inside to "even cut " the concentration of water. So the gummy bear became larger as move water filled it up. </div>]]></description>
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         <pubDate>2018-07-02 12:17:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269154473</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269228721</link>
         <description><![CDATA[<div>H</div>]]></description>
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         <pubDate>2018-07-03 05:20:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269228721</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269228722</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-07-03 05:20:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269228722</guid>
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         <title>Kano, Safwan. Baluso, James Lian David. Mato, Lacman</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269228724</link>
         <description><![CDATA[<div>PLASMOLYSIS:<br>Shrinking of the cytoplasm away from the wall of living cell due to outward osmotic flow of water.<br><br>HYPERTONIC:<br>Is a solution with a higher concentration of solute than some other specified solution.The solution with the lower concentration is then termed "hypertonic"<br><br>HYPOTONIC:<br>is a solution that has a lower solute concentration compared to anothet solution. Water molecules cluster around solutes, move them away from th , highest concentration s of solutes, and allow more water molecules to move in.<br><br>ISOTONIC:<br>it refers to two solutions having the same osmotic pressure accross a semipermeable membrane. This state allows for the free movement of water across the membrane without changing the concentration of solutes on other side.<br><br>Examples:<br>Examples of Plasmolysis<br>Although plasmolysis more commonly happens in a laboratory setting, it can happen in real-life settings as well. For example, during periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants on the affected land, killing them. Chemical weedicides are also used to kill unwanted plants through plasmolysis. This same process is also used when a lot of salt and/or sugar is added to preserve food and make jams, jellies, and pickles. The cells lose water and become less conducive to the growth of microorganisms such as bacteria, allowing these food items to be preserved.<br><br>Hypertonic<br>Glucose is the sugar present in an individual's blood. An example of a hypertonic solution that has glucose dissolved into it is any plain glucose solution with a concentration higher than 5%. One such example is a solution of 10% dextrose in water, also known by its name D10W. This is often used as an IV fluid.<br><br>Hypotonic<br>The type of solution provides free water, sodium and chloride but does not provide calories or other electrolytes. An example of a hypotonic solution is 0.45% sodium chloride (0.45% NS), commonly called half normal saline.<br><br>Isotonic: 0.9% NaCl (Normal Saline) Lactated Ringers. D5W (In the bag)</div>]]></description>
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         <pubDate>2018-07-03 05:20:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269228724</guid>
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         <title>Jefferson Llamera, Ej Carl Banlasan,  Patrick Lagang</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229028</link>
         <description><![CDATA[<div><strong>Plasmolysis<br></strong> is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than the cell does. This is known as a hypertonic solution.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:26:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229028</guid>
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         <title>Fagutao, Ramer Kent G. Fajartin, Lrme S. Facurib, Jhonbert U. </title>
         <author>kentfagutao</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229178</link>
         <description><![CDATA[<div><strong>Plasmolysis</strong> <br>- is a contraction of the protoplast of a plant cell as a result of loss of water from the cell. <br><strong>Plasmolysis example</strong><br>- Shrinkage of vegetables in hypertonic conditions.<br><strong>Hypertonic</strong><br>- A hypertonic solution is basically the opposite of a hypotonic solution. <br><strong>Hypertonic example</strong><br>- 5% Dextrose in Lactated Ringer’s<br><strong>Hypotonic</strong><br>- A hypotonic solution is a water that is less-concentrated than the cell the water surrounds.<br><strong>Hypotonic example</strong><br>- An example of a hypotonic solution is 0.45% sodium chloride (0.45% NS), commonly called half normal saline.<br><strong>Isotonic</strong> <br>- An isotonic solution, in a nutshell, is a balanced water-solute concentration. When a solution is isotonic, it is at equilibrium.<br><strong>Isotonic example</strong><br>- 5% dextrose in water (D5W)**also used as a hypotonic solution after it is administered because the body absorbs the dextrose BUT it is considered isotonic)</div>]]></description>
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         <pubDate>2018-07-03 05:26:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229178</guid>
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         <title>GALO, FERNANDEZ, PERTIMOS (8-Mendel)</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229433</link>
         <description><![CDATA[<div>PLASMOLYSIS <br>-is the process in which cells lose water in a hypertonic solution, the reverse process, cytolysis, can occur if the cell is in a hypotonic solution resulting in a lower external osmotic pressure and a net flow of water into the air.&nbsp;<br>Example:<br>Chemical weedicides are also used to kill unwanted plants on the affected land, killing them<br><br>HYPERTONIC<br>-A solution is one where the concentration of solutes is greater outside the cell than inside it&nbsp;<br>Example:<br>Glocuse is the sugar present in an individual's blood<br>Hypertonic solution that has glocuse dissolve into it is any plain glocuse solution with a concentration higher than 5<br>&nbsp;<br>HYPOTONIC<br>-A hypotonic solution is one which the concentration of the solute is greater inside the cell that outside of it.&nbsp;<br>Example:<br>Plants and fungi<br><br>ISOTONIC<br>-Denoting or relating to a solution having the same osmotic pressure as some other solution especially one in a cell or a body fuel<br>Example:<br>Blood cells-when plasma surrounding a blood cell is an isotonic solution.&nbsp;<br><br></div>]]></description>
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         <pubDate>2018-07-03 05:31:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229433</guid>
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         <title></title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229640</link>
         <description><![CDATA[<div>Members <br>-Patrick James Lagang<br>-Jefferson Llamera<br>-EJ Carl Banlasan<br><br>Plasmolysis<br>-is the process in which cells lose water in a hypertonic solution. The reverse process, cytolysis, can occur if the cell is in a hypotonic solution resulting in a lower external osmotic pressure and a net flow of water into the cell. Through observation of plasmolysis and deplasmolysis, it is possible to determine the tonicity of the cell's environment as well as the rate solute molecules cross the cellular membrane.<br><br><strong><em>Hypertonic</em></strong><br>-refers to a solution with higher osmotic pressure than another solution. In other words, a hypertonic solution is one in which there is a greater concentration or number of solute particles outside a membrane than there are inside it.<br>Example:<br>-Red blood cells are the classic example used to explain tonicity. When the concentration of salts (ions) is the same inside the blood cell as outside of it, the solution is isotonic with respect to the cells and they assume their normal shape and size.<br><br><strong><em>Isotonic<br></em></strong><br>When two solutions have the same concentration, and exchange water and solutes at the same rate.<br><br><strong><em>Hypotonic<br><br></em></strong>A hypotonic solution is a solution that has a lower solute concentration compared to another solution. <br><br></div>]]></description>
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         <pubDate>2018-07-03 05:36:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229640</guid>
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         <title></title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229778</link>
         <description><![CDATA[<div>8- Mendel<br>Members:<br>Fajardo, Nikko<br>Sabelita, Francine<br>Valdez, Leana<br><br>Plasmolysis - when plant cells lose water after being placed in a solution that has a higher concentration of solutes than the cell does.<br>Examples:</div><ul><li>During the periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants on the affected land, killing them</li><li>When a lot of salt and/or sugar is added to preserve food and make jams, jellies, and pickles.&nbsp;</li></ul><div><br>Hypertonic - A solution where the concentration of solutes is greater outside the cell than inside it.<br>Examples:</div><ul><li>Plants and Fungi - large plants and fungi control the environment around their cells, helping ensure the environment.</li><li>Animal Cells - animal cells do not have a cell wall. Typically, animals rely on their skin to separate the outside environment from their inside organs. The fluid inside of their body cavity can then be regulated by a series of membranes and proteins.</li></ul><div><br>Hypotonic -&nbsp; Any solution that has a lower osmotic pressure than another solution.&nbsp;</div><div>Examples:</div><ul><li>A lower concentration of salt than in normal cells of the body and blood. This solution usually contains 0.25% or 0.45% of saline and can also have dextrose.</li><li>The increase inthe extracellular fluid volume due to blood loss, surgery, dehydration, fluid loss that has been loss extracellularly..</li></ul><div><br></div><div>Isotonic - A solution that has the same osmolarity, or solute concentration, as another solution.&nbsp;</div><div>Examples:</div><ul><li>Blood Cells - when the plasma surrounding blood cells is an isotonic solution, compared to the solution inside the blood cells, the cells function normally.</li><li>The intravenously infused fluids in hospitalized patients. It is a muscular contraction in which the muscle remains to be a relatively constant tension while its length changes.</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:40:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229778</guid>
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         <title>Members:Hannah Arcilla,Tyra Arduo,Krizza Olar (Grade 8 Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269229793</link>
         <description><![CDATA[<div>&gt;Plasmolysis is the process in which cells lose water in a hypertonic solution. It is the reverse process,cytolysis can occur if a cell is in hypotonic solution resulting in a lower external osmotic pressure and a net flow of water into the cell.<br>example:<br>*Red blood cells shrink when placed in a salt solution.<br>&gt;Hypertonic is one where the concentration of solutes is greater outside the cell than inside it.<br>example:<br>*A solution of 10% dextrose in water or known as D10W<br>&gt;Hypotonic is one in which the concentration of solutes is greater inside the cell than outside of it.<br>example:<br>*0.45% sodium chloride (0.45% NS) commonly called half normal saline.<br>&gt;Isotonic is denoting or relating to a solution having the same osmotic pressure as some other solution,especially one in a cell or body fluid.<br>example:<br>*Saline solution consists of water mixed with sodium chloride (NACI) <br><br><br></div>]]></description>
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         <pubDate>2018-07-03 05:41:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269229793</guid>
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         <title>Plasmolysis and Tonicity  By: Jefferson Llamera, Ej Banlasan, Patrick Lagang               </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230042</link>
         <description><![CDATA[<div><strong>Plasmolysis</strong><br>-Plasmolysis is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than the cell does. This is known as a hypertonic solution.<br><br>For example, during periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants on the affected land, killing them. Chemical weedicides are also used to kill unwanted plants through plasmolysis. This same process is also used when a lot of salt and/or sugar is added to preserve food and make jams, jellies, and pickles. The cells lose water and become less conducive to the growth of microorganisms such as bacteria, allowing these food items to be preserved.<br><br><br><strong>Hypertonic</strong><br><br>A hypertonic solution contains a higher concentration of solutes compared to another solution.<br><br>Ex:<br><strong>Human Kidney<br></strong><br></div><div>To regulate the amount of water in the body, the human brain has special proteins called <em>osmoreceptors</em>, which can measure the osmolarity of the environment surrounding the cell.<br><strong>Plants in Hypertonic Solution<br></strong><br></div><div>Generally, plants prefer to live in hypotonic environments. In a hypotonic environment, water easily floods plant cells and they can remain <em>turgid</em>, or rigid, due to pressures exerted on their cell walls by the influx of water.<br><br><strong>Hypotonic</strong><br>A hypotonic solution is a solution that has a lower solute concentration compared to another solution.<br><br>Ex:<br><strong>Plants and Fungi<br></strong><br></div><div>Large plants and fungi control the environment around their cells, helping ensure the environment is always a hypotonic solution, compared to the cells. This creates cells that are <em>turgid</em>.<br><strong>Animal Cells<br></strong><br></div><div>Animal cells do not have a cell wall. Typically, animals rely on their skin to separate the outside environment from their inside organs.<br><br><strong>Isotonic</strong><br>An isotonic solution is one that has the same <em>osmolarity</em>, or solute concentration, as another solution. <br><br>Ex:<br><strong>Blood Cells<br></strong><br></div><div>When the plasma surrounding blood cells is an isotonic solution, compared to the solution inside the blood cells, the cells function normally.<br><strong>Osmoconformers and Osmoregulators<br></strong><br></div><div>In nature, there are two types of organisms: those who conform to the osmolarity of the environment, and those that regulate the osmolarity of their body to be different from the environment.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:49:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230042</guid>
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      <item>
         <title>Paguray </title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230244</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:55:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230244</guid>
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      <item>
         <title></title>
         <author>kentfagutao</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230257</link>
         <description><![CDATA[<div>Membes<br>Buladaco<br>Cerado<br>Fl</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:55:43 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230257</guid>
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      <item>
         <title>Searv</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230271</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:56:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230271</guid>
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      <item>
         <title>Biotech</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230273</link>
         <description><![CDATA[<div>Members:<br>Julianne Agorilla<br>Merryl Omawing<br>Kristine Joy Palmaera<br><br><strong>Plasmolysis</strong><br>- contraction of the protoplast of a plant cell as a result of water loss of a cell. <br>2 types:<br>Concave and Convex Plasmolysis<br>Examples:<br>Chemical weedicides- used to kill unwanted plants through plasmolysis.<br><br><strong>Hypertonic</strong><br>- having a high osmostic pressure than a particular fluid.<br>Example:<br>D10W- dextrose<br>* glucose dissolved into it is any plain glucose solution with a concentration higher than 5%<br><br><strong>Hypotonic<br></strong>- having a lower osmotic pressure than a particular fluid.<br>Example:<br>Sodium Chloride<br><br><strong>Isotonic</strong><br>- having same osmotic pressure as other solution.<br>Example:<br>Intravenous Solutions<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:56:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230273</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230284</link>
         <description><![CDATA[<div>Paguray,Krystle Ann Z. <br>Endaya, Princess Kyla<br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><br><strong>Plasmolysis</strong><br>- is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than the cell does.<br><strong>Ex.</strong><br>During periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants on the affected land, killing them.<br><br><strong>Hypertonic</strong><br>- having a higher osmotic pressure than a surrounding medium or a fluid under comparison.<br><strong>Ex.</strong><br>Hypertonic solution that has glucose dissolved into it is any plain glucose solution with a concentration higher than 5%.<br><br><strong>Hypotonic</strong><br>- having a lower osmotic pressure than a surrounding medium or a fluid under comparison.<br><strong>Ex.</strong><br>Large plants and fungi control the environment around their cells, helping ensure the environment is always a hypotonic solution, compared to the cells.<br><br><strong>Isotonic</strong><br>-refers to two solutions having the same osmotic pressure across a semipermeable membrane.<br><strong>Ex.</strong><br>A low-alcohol wine has the same proof as beer, it is isotonic to, or having the same ethanol concentration as ,beer.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:56:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230284</guid>
      </item>
      <item>
         <title>CUBETA</title>
         <author>gclikerz2134</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230310</link>
         <description><![CDATA[<div>MAGALLANO<br>MAHINAY<br><br>Plasmolysis<br>-Is the contraction of the protoplast of a plant cell as a result of loss of water from the cell<br><br>Example:<br>During periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants of the affected land , killing them<br><br>Hypertonic&nbsp;<br>-Is one where the concentration of solutes is greater outside the cell than inside it<br><br>Example:<br>Glucose<br><br>Hypotonic<br>-Is one which the concentration of solutes is greater inside the cell than outside it<br><br>Example:<br>-Large plants and fungi control the environment around their cells, helping sure the environment is always a hypotonic solution, compared to the cells. This create cells that are turgid. The turgid cells push outward on their cell walls, which push against each other creating a rigid structure&nbsp;<br><br>Isotonic<br>-Denoting or relating to a solution having the same osmotic press as some other solution especially one in a cell or a body fluid&nbsp;<br><br>Example:&nbsp;<br>When plasma surrounding blood cells is an isotonic solution, compared to the solution inside the blood cells, the cells function normally. The isotonic solution allow the cell to move water&nbsp;<br><br>Reason why late: slow i</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:57:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230310</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230341</link>
         <description><![CDATA[<div><strong>Members</strong><br>Buladaco<br>Cerado<br>Flores<br><br><strong>Turgidity</strong><br>-swollen ,distended,tumid<br><br><strong>Plasmolisis</strong> <br>- is the process in which cells lose water in hypotonic solution resulting in a lower external osmotic pressure and a net flow if water into the cell<br><br><strong>Hypotonic</strong><br>-is one in which the concentration of solutes is greater inside the cell than the outside of it <br><br><strong>Hypertonic</strong> <br>-is on where the concentration of sloutes is greater outside the cells than inside it.<br><br><strong>Isotonic</strong> <br>-refers to two solution have same osmotic pressure across a <em>semipermidable</em> membrane.<br><br><strong>Examples</strong><br><strong>Hypotonic</strong><br>- sodium chloride (<em>half normal saline</em>)<br><br><strong>Hypertonic</strong><br>-Glucose<br><br><strong>Isotonic</strong> <br>-saline solution</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 05:57:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230341</guid>
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      <item>
         <title>Activity</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230713</link>
         <description><![CDATA[<div>Members</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:04:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230713</guid>
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         <title>Activity</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230714</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:04:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230714</guid>
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         <title>Ac</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230716</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:04:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230716</guid>
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      <item>
         <title>Activity</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269230717</link>
         <description><![CDATA[<div>Mem</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:04:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269230717</guid>
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         <title>Mercado, Vergara, Cadiz </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231115</link>
         <description><![CDATA[<div><strong><br>Search for the Ff.<br>PLASMOLYSIS-Is the process in which cells lose in a hypertonic solution<br></strong>Ex. When a lot of salt or sugar is added to preserve food and make jams, jelly, and pickles.<strong><br>HYPERTONIC- a solution that contains a higher concentration of solutes compared to another solution <br></strong>Ex. Glocuse is the sugar present in an individuals blood hypertonic solution that has glocuse desolves into it is any plain Glocuse solution with a concentration higher than 5<strong><br><br>HYPNOTIC - A solution in which the concentration of solutes is greater inside the cell than outside of it.<br>Biology *Having a lower osmotic pressure than a particular fluid, tipically a body fluid or intracellular fluid.<br>Physiology *of or in a state of abnormaly low muscle tone. <br></strong>Ex.large Plants and fungi control the environment around there cells helping ensure the environment is always a hypotonic solution, compared to the cells.these creates cells that are turgid. <strong><br><br>ISOTONIC-Denoting or relating to a solution <br></strong>Ex.push ups are isotonic because they involved raising the body and lowering it in a plank position. They produce isotonic masles because they work both sides of the body equally when performed&nbsp;correctly <br><strong><br><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:11:47 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231115</guid>
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         <title>(howard gogodwin </title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231243</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:14:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231243</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231274</link>
         <description><![CDATA[<div>Paguray, </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:14:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231274</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231275</link>
         <description><![CDATA[<div>Paguray, Krystle</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:14:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231275</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231276</link>
         <description><![CDATA[<div>Paguray, keys</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:14:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231276</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231283</link>
         <description><![CDATA[<div>Paguray, Krystle Ann.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:14:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231283</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231399</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231399</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231400</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231400</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231401</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- <br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231401</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231402</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than. The&nbsp;cell<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231402</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231403</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a&nbsp;higher<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231403</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231404</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a higher concentration of&nbsp;solutes <strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231404</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231405</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is&nbsp;when <strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231405</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231406</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than.The&nbsp;cel<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231406</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231407</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla&nbsp;<br>Brocoy, Angelle Kate Y.<br>8-Mendel<br>Plasmolysis</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231407</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231408</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a higher concentration of&nbsp;solute<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231408</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231409</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a&nbsp;solution<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231409</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231410</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after&nbsp;being<strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231410</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231411</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>-</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231411</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231412</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>-i<br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231412</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231414</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>-is when <br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231414</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231416</link>
         <description><![CDATA[<div>Paguray, Krystle Ann Z.<br>Endaya, Princess Kyla <br>Brocoy, Angelle Kate Y.<br>8-Mendel<br><strong>Plasmolysis<br>- </strong>is when plant cells lose water after being placed in a solution that has a higher concentration of solutes than the cell does.<br>Ex.<br>During periods of extreme coastal flooding, ocean water deposits salt onto land. Too much salt causes the water to flow out of any plants on the affected land, killing them.<br><br><strong>Hypertonic<br>- </strong>having a higher osmotic pressure than a surrounding medium or a fluid under comparison.<br>Ex.<br>A solution of 10% dextrose in water, also known by it's name D10W.<br><br><strong>Hypotonic</strong><br>- a solution that has a lower solute concentration compared to another solution.<br>Ex.<br>Large plants and fungi control the environment around their cells, helping ensure the environment is always a hypotonic solution, compared to the cells.<br><br>Isotonic<br>-refers to two solutions having the same osmotic&nbsp;pressure <br><strong><br></strong><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:16:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231416</guid>
      </item>
      <item>
         <title>Howard godwin, wendell angelo, </title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231454</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:17:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231454</guid>
      </item>
      <item>
         <title>Howard godwin, wendell angelo, pette bernard</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231457</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:17:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231457</guid>
      </item>
      <item>
         <title>Howard godwin, wendell angelo, pette bernard</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231461</link>
         <description><![CDATA[<div>Isotonic<br><br></div><ol><li>1.</li><li>(of muscle action) taking place with normal contraction.</li><li>2.</li><li>denoting or relating to a solution having the same osmotic pressure as some other solution, especially one in a cell or a body fluid.</li></ol><div>Hypertonic<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:17:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231461</guid>
      </item>
      <item>
         <title>Howard godwin delosreyes,wendell angelo, pette bernard(hypotonic)</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231639</link>
         <description><![CDATA[<div>having reduced pressure or tone, in particular.</div><ul><li>BIOLOGY</li><li>having a lower osmotic pressure than a particular fluid, typically a body fluid or intracellular fluid.<strong>hypotonic</strong></li><li>PHYSIOLOGY</li><li>of or in a state of abnormally low muscle tone.</li></ul>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:20:50 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231639</guid>
      </item>
      <item>
         <title>Howard godwin, wendell angelo , pette berlat</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269231792</link>
         <description><![CDATA[<div>(plasmolysis)Contraction of the protoplast of a plant cell as a result of loss of water from the cell.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:23:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269231792</guid>
      </item>
      <item>
         <title>Pagut</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232493</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:31:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232493</guid>
      </item>
      <item>
         <title>Paguray</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232495</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:31:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232495</guid>
      </item>
      <item>
         <title>Paguray, Endaya</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232510</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:31:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232510</guid>
      </item>
      <item>
         <title>Paguray, </title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232511</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:31:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232511</guid>
      </item>
      <item>
         <title>Paguray, Endaya, </title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232514</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:32:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232514</guid>
      </item>
      <item>
         <title>Paguray, Endaya, Brocoy</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232520</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:32:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232520</guid>
      </item>
      <item>
         <title>Paguray, Endaya, Brocoy</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232559</link>
         <description><![CDATA[<div><strong>PlasmolysusPlasbdhhsjsdhdhgz</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:32:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232559</guid>
      </item>
      <item>
         <title>Paguray, Endaya, Brocoy</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/269232626</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-03 06:33:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/269232626</guid>
      </item>
      <item>
         <title>Berame, Bernadette Sue B.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270482998</link>
         <description><![CDATA[<div><strong>SLIDE 1</strong><br>1. The shape of the plant cell is circle.<br>2.The plant cells vacuole is larger than the animal cells.<br>3.Nucleus<br>4.Cytoplasm<br>5.Cell membrane<br>6.It is rectangular in shape.<br>7. Its vacuole is smaller than the plant cell.<br><strong>SLIDE 2</strong><br>1.Plantcell<br>2.Animal cell<br>3.Nucleus<br><strong>SLIDE 3<br></strong>1.Plant cell<br><strong>SLIDE 4<br></strong>1.Nucleus<br>2.Cell wall<br>3.Vacuole<br>4.Mitchodrion<br><strong>SLIDE 5<br></strong>1.Mitochondrion<br>2.Ribosomes<br>3.Cell membrane<br>4.Nucleus<strong><br>SLIDE 6<br></strong>1.Inside to outside<br><strong>SLIDE 7</strong><br>1.Osmosis<br>2.Hypertonic Solution<br>3.Hypotonic Solution</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:06:48 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270482998</guid>
      </item>
      <item>
         <title>Cutamora, Mizpah Gabrielle S. 8—Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483136</link>
         <description><![CDATA[<div>I.<br>1. Rectangular<br>2. Small and not bigger than the nucleus<br>3. Nucleus<br>4. Cell Membrane<br>5. Mitochondria<br>6. Circular<br>7. Large and bigger than the nucleus<br>II.<br>1. Plant Cell<br>2. Animal Cell<br>3. Cell Membrane<br>III.<br>1. Plant Cell<br>IV.<br>1. Nucleus<br>2. Cell Membrane<br>3. Vacuole<br>4. Mitochondria<br>V.&nbsp;<br>1. Mitochondria<br>2. Rough Endoplasmic Reticulum<br>3. Cell membrane<br>4. Nucleolus<br>VI.&nbsp;<br>1. Inside of the cell to outside of the cell<br>2. Plasmolysis<br>VII.<br>Direction: in to out of the cell<br>Process: plasmolysis<br>Solution:<br>Left — Hypotonic<br>Right — Hypertonic</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483136</guid>
      </item>
      <item>
         <title>Reanne Kathleen Figuracion</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483137</link>
         <description><![CDATA[<div>S1<br>1. Square-like<br>2.The vacuole of the plant cells are used as storage<br>3. Chlorophlast and cell wall<br>4.nucleus,lysosomes, vacuole and cell membrane<br>5. Mitochondria<br>6. Round -like<br>7. The vacuole of the animal cells are also used as storage<br>S2<br>1. Plant cell<br>2 . Animal cell<br>3.<br>S3<br>1. Plant cell<br>S4<br>1.nucleus<br>2. Cell membrane<br>3. golgi bodies<br>4. Mitochondria<br>S5<br>1.mitochondria<br>2. Endoplasmic reticulum<br>3. Cell membrane<br>4. Nucleulus <br>S6<br>•the direction is from low concentration of solute to high water<br>• hypotonic<br>S7<br>Process- Cytolisis<br>Direction- From high concentration of water to low concentration<br>Hypotonic<br>hyperton<br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483137</guid>
      </item>
      <item>
         <title>Angela L</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483156</link>
         <description><![CDATA[<div><br>1.<br>2. The vacp</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483156</guid>
      </item>
      <item>
         <title>Achilles Alexis R. Alberto</title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483168</link>
         <description><![CDATA[<div><strong>S-1</strong><br>1. It has own shape <br>2. The Vacuole of the Plant Cell is larger than Animal Cell. <br>3. Nucleus <br>4. Cytoplasm <br>5. Cell Membrane <br>6. Circular Shape<br>7. The Vacuole in Animal Cell is smaller than Plant Cell. <br><br><strong>S-2<br></strong>1. Plant Cell<br>2. Animal Cell<br>3. Nucleus<br><br><strong>S-3</strong><br>1. Plant Cell<br><br><strong>S-4</strong><br>1. Nucleus<br>2.Cell Membrane <br>3. Golgi Bodies<br>4. Mitochondria <br><br><strong>S-5</strong><br>1. Mitochondria<br>2. Endoplasmic Reticulum <br>3.Cell membrane<br>4. Nucleus<br><br><strong>S-6<br></strong>1. In and out <br>2. Hypertonic Solution <br><br><strong>S-7<br></strong>1. In and Out<br>2. Cytolisis <br>3. Hypotonic Solution <br>4. Hypertonic Solution<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483168</guid>
      </item>
      <item>
         <title></title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483201</link>
         <description><![CDATA[<div>1</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483201</guid>
      </item>
      <item>
         <title>Christian Louie N. Evangelista    8 Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483202</link>
         <description><![CDATA[<div>slide 1<br>1. rectangular shape<br>2. large vacoule<br>3. nucleus<br>4. cytoplasm<br>5. cell mebrane<br>6. round shape<br>7. small vacoule<br>slide 2<br>1. plant cell <br>2. animal cell<br>3. nucleus<br>slide 3<br>1. animal cell<br>slife 4<br>1. nucleus<br>2. cell membrane<br>3. vacuole<br>4. mitochondria<br>slide 5<br>1. mitochondria<br>2. Endoplasmic Reticulum<br>3. cell mebrane <br>4. nucleus<br>slide 6<br>1. outside the cell &gt; hypotonic solution &gt; passive transportation<br>slide 7<br>2. hypotonic solution &gt; Active transportation</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483202</guid>
      </item>
      <item>
         <title>Sarmiento, Nadine A.   8-Aristotle</title>
         <author>nadinesarmiento2004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483207</link>
         <description><![CDATA[<div><strong>Slide 1.</strong><br>1.) Rectangular<br>2.) The vacuole of the plant cell is just as big if not much bigger than the nucleus. It is the empty space where proteins and others are stored.<br>3.) Nucleus<br>4.) Vacuole<br>5.) Golgi Bodies<br>6.) Circular or round<br>7.) The vacuole in the animal cell is smaller than the nucleus. It is an empty space where some things are stored.<br><strong>Slide 2.</strong><br>1.) Plant Cell<br>2.) Animal Cell<br>3.) Nucleus<br><strong>Slide 3.<br></strong>1.) Plant Cell<br><strong>Slide 4.<br></strong>1.) Nucleus<br>2.) Cell membrane <br>3.) Vacuole<br>4.) Chloroplast<br><strong>Slide 5.</strong><br>1.) Mitochondria<br>2.) Smooth Endoplasmic Reticulum<br>3.) Rough Endoplasmic Reticulum <br>4.) Nucleus<br><strong>Slide 6.<br></strong>1.) Plasmolysis<br>2.) Cytolisis <br><strong>Slide 7.<br></strong>1.) Hypertonic- in to out<br>2.) Hypotonic- out to in<br>&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483207</guid>
      </item>
      <item>
         <title>Emata, Simone Anne C.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483211</link>
         <description><![CDATA[<div>Slide 1<br>1. The shape of the plant cell is 'rectangular' like&nbsp;<br><br>2. The vacuole of a plant cell is <br><br>3. Cell membrane<br>4.&nbsp;Nucleus<br>5. Vacuole <br><br>6. The shape of the animal cell is somewhat circle in shape.<br><br>7.&nbsp;The vacuole of <br><br><br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483211</guid>
      </item>
      <item>
         <title>Urdaneta Caryl Jean</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483212</link>
         <description><![CDATA[<div>I.<br>1. Square<br>2. Are sacs which serve as the storage unit.<br>3. Cell mebrane<br>4. Cytoplasm<br>5. Mitochondria<br>6. Round<br>7. Storage unit<br>II.<br>1. Plant cell<br>2. Animal cell<br>3. Nucleous<br>III.<br>1.Plant cell<br>IV.<br>1.Nucleous<br>2.cell membrane<br>3. Vacoule<br>4. Cytoplasm<br>V.<br>1. Mitochondria<br>2. Endoplasmic Reticulum<br>3. Cell membrane<br>4. Nucleous<br>VI.<br>1.&nbsp; out of the cell<br>2. Hypertonic solution<br>VII.<br>1. In to out of the cell<br>2. Hypotonic solution</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:08:54 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483212</guid>
      </item>
      <item>
         <title></title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483228</link>
         <description><![CDATA[<div>1</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:09:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483228</guid>
      </item>
      <item>
         <title>Alvin Jay</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483229</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:09:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483229</guid>
      </item>
      <item>
         <title>Bea Clatisse A. Miase </title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483231</link>
         <description><![CDATA[<div>1.&nbsp;<br>2. The one that contains the </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:09:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483231</guid>
      </item>
      <item>
         <title>en</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483288</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:09:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483288</guid>
      </item>
      <item>
         <title>Angelie Mae M. Lapating</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483289</link>
         <description><![CDATA[<div>1. Square like&nbsp;<br>2. The Vacuole of the Plant Cell is larger than the Vacuole of the Animal Cell. It store water and waste.<br>3.Cell Membrane<br>4. Nucleus<br>5. Cytoplasm<br>6.Circle<br>7.Tshe vacuole of the Animal cell is called vesicle, it is smaller than the vacuole of the plant cell. It store water and waste.<br>S2.<br>1. Plant Cell<br>2. Animal cell<br>3. Nucleus<br>S3.<br>1. Plant cell<br>S4.<br>1. Nucleus<br>2. Cell Membrane<br>3. Golgi Bodies<br>4. Mitochondria<br>S5.<br>1. Mitochondria<br>2. Endoplasmic Reticulum<br>3. Cell Membrane<br>4. Nucleus<br>S6.<br>1. In to out<br>Hypertonic&nbsp;<br>2. In to out<br>Hypertonic<br>S7.<br>1. In to out<br>2. Cytolisis<br>3. Hypotonic Solution<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:09:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483289</guid>
      </item>
      <item>
         <title>Lorania, Chorena Grace G.  8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483324</link>
         <description><![CDATA[<div>S-1</div><ol><li>Round rectangular shape</li><li>More/big vacuole</li><li>Nucleus</li><li>Endoplasmic reticulum</li><li>Cytoplasm</li><li>Round</li><li>Less/smal vacuole</li></ol><div>S-2</div><ol><li>Plant cell</li><li>Animal cell</li><li>Nucleus</li></ol><div>S-3</div><ol><li>Animal cell</li></ol><div>S-4</div><ol><li>Nucleus</li><li>Cell wall</li><li>Vacuole</li><li>Mitochondria</li></ol><div>S-5</div><ol><li>mitochondria</li><li>Ribosome</li><li>Cell membrane</li><li>Nucleus</li></ol><div>S-6</div><ol><li>Outward</li><li>Cytolysis</li></ol><div>S-7</div><ol><li>Plasmolysis</li><li>Hypertonic solution</li><li>Hypotonic solution</li></ol><div><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:10:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483324</guid>
      </item>
      <item>
         <title>Mika C. Hamajo 8-Aristotle</title>
         <author>cimagalamary</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483682</link>
         <description><![CDATA[<div>Slide #1<br>1. Circle-like<br>2. Vacuole of the plant cell is bigger than the animal cell and it stores water<br>3. Vacuole<br>4. Nucleus<br>5. Mitochondria<br>6. Square/ Box-like<br>7. Vacuole of the animal cell is smaller than the plant cell and it also stores water<br>Slide #2<br>1. Animal Cell<br>2.Plant Cell<br>3. Nucleus<br>Slide #3<br>1. Plant Cell<br>Slide #4<br>1. Mitochondria<br>2. Cell Membrane<br>3.&nbsp;<br>4.<br>Slide #5<br>1. Ribosomes<br>2. Endoplasmic Reticulum<br>3. Cell Wall<br>4. Vacuole<br>Slide #6<br>1. Inside to Outside; Hypotonic<br>Slide #7<br>1. From higher water concentration to lower concentration<br>2. Hypertonic<br>3. Hypoyonic</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:13:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483682</guid>
      </item>
      <item>
         <title>Suarez,Alyanna Shin D.     8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483695</link>
         <description><![CDATA[<div>Slide no.1<br>1.rectangular shape<br>2.The vacoule in the plant cell is big.<br>3.Cell Membrane<br>4.Cytoplasm<br>5.Nucleus<br>6.round shape<br>7.The vacoules are small.<br>Slide no.2<br>1.Plant Cell<br>2.Animal cell<br>3.Cytoplasm<br>Slide no.3<br>1.Plant Cell<br>Slide no.4<br>1.Nucleus<br>2.Cell Wall<br>3.vacuole<br>4.mitochondria<br>Slide no.5<br>1.Mitochondria<br>2.Endoplasmic Reticulum<br>3.Cell membrane<br>4.Nucleus<br>Slide no.6<br>1.Out to in<br>2.Plasmolysis<br>Slide no.7<br>1.Osmosis<br>2.Hypertonic solution- the movement is from in to out<br>3.Hypotonic solution-the movement is from out to in<br><br><br><br><br><br><br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:13:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483695</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483770</link>
         <description><![CDATA[<div>R</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:14:01 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483770</guid>
      </item>
      <item>
         <title>Desire Faith C. Enojales</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483786</link>
         <description><![CDATA[<div>Rectangular shape<br>Vacuole-<br>Cell membrane<br>Cytoplasm<br>Nucleus<br>Circle<br>Vacuole-<br><br>Slide 2<br>Vacuole<br>Cell membrane<br>Slide 3<br>Plant cell<br>Slide 4<br>Mitochondria<br>Endoplasmic Reticulum<br><br><br>Slide 5<br>Mitochondria<br>Endoplasmic Reticulum<br>Cell membrane<br>Vacuole<br>Slide 6<br>In and out<br>Hypertonic<br>Slide 7<br>In and out<br>Osmosis<br>Hypotonic<br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:14:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483786</guid>
      </item>
      <item>
         <title>Malunes, Jaesar Justin</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270483908</link>
         <description><![CDATA[<div>1. Square<br>2. Big<br>3.Cell membrane&nbsp;<br>4. Cytoplasm&nbsp;<br>5.Nuclear membrane&nbsp;<br>6. Circle<br>7. Small<br>8.Onion Cell<br>9. Cheeck cell<br><br>10. Cell membrane&nbsp;<br>11. Plants cell<br>12.<br>S-4<br>A. Nucleus&nbsp;<br>B. Cell membrane&nbsp;<br>C. Golgi bodies<br>D.&nbsp;<br>1. Rough ER<br>2.Smooth ER<br>3. Cell membrane&nbsp;<br>4. Nucleus&nbsp;<br>S-6<br>14. out of the cell&nbsp;<br>15.cytolysis<br>S-7<br>16. Plasmolysis&nbsp;<br>17. Hypertonic solution&nbsp;<br>18. Hypotonic solution&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:15:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270483908</guid>
      </item>
      <item>
         <title>Emata, Simone Anne C.</title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270486056</link>
         <description><![CDATA[<div>Slide 1  <br>1. The shape of the plant cell is 'rectangular' like <br>2. The vacuole of a plant cell is big.    <br>3. Cell membrane<br>4. Nucleus<br>5. Vacuole <br>6. The shape of the animal cell is somewhat circle in shape.   <br>7. The vacuole of the animal cell is small.   <br><br><br>Slide 2 <br><br>1. Plant Cell<br>2. Animal Cell<br><br>Slide 3<br>1. Plant Cell<br><br><br><br>Slide 4<br>1. Nucleus <br>2. Cell Wall<br>3. Cell Membrane <br>4.  Vacuole    <br><br>Slide 5    <br>1. Vacuole <br>2. Cell Membrane <br>3. Vacuole <br>4. Nucleus     <br><br>Slide 6<br>1. The movement is going in of a cell.   <br>2. The direction of the water is going in and out of the cell.<br><br>Slide 7<br>1. Plasmolysis <br>2. Hypertonic Solution <br>3. Hypotonic Solution</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 04:34:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270486056</guid>
      </item>
      <item>
         <title>Fernand</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489697</link>
         <description><![CDATA[<div>S-1. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:14:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489697</guid>
      </item>
      <item>
         <title>Baluso, James Lian David V. 8-mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489701</link>
         <description><![CDATA[<div>S1<br>1. Rectangular Shape<br>2. Large Vacuole<br>3. Cell membrane<br>4. Nucleus<br>5. Cytoplasm<br>6. Irregular shape<br>7. Small vacuole<br>S2<br>1. Plant cell<br>2. Animal cell<br>3. Nucleus<br>S3<br>1.plant cell<br>S4<br>1. Nucleus<br>2. cell wall<br>3. Golgi body<br>4. Chloroplasts<br>S5<br>1. Lysosomes<br>2. Mitochondria<br>3. Cell membrane<br>4. Nucleus<br>S6<br>1. INSIDE-OUTSIDE<br>2. Plasmolysis<br>S7<br>1. Left - right<br>2. Osmosis<br>3.hypertonic<br>4. Hypotonic<br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:14:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489701</guid>
      </item>
      <item>
         <title>Name: Ashley Marie Pertimos</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489728</link>
         <description><![CDATA[<div>S-1<br>1. The shape of a plant cell is rectangular.<br>2. The vacuole of a plant cell is large.<br>3. Mitochondria<br>4. Endoplasmic Reticulum<br>5. Nucleus<br>6. The shape of an animal cell is round<br>7.&nbsp; The vacuole of an animal cell is small<br><br>S-2<br>1. plant cell<br>2. animal cell&nbsp;<br>3. nucleus<br><br>S-3<br>1. animal cell<br><br>S-4<br>1. nucleus<br>2. cell wall<br>3.&nbsp;<br>4. ribosomes<br><br>S-5&nbsp;<br>1. mitochondria<br>2. endoplasmic reticulum<br>3. cell wall<br>4. nucleus<br><br>S-6<br>1. outside-inside<br>2. osmosis<br><br>S-7<br>1. diffusion<br>2. inside-outside<br>3. hypertonic<br>4. hypotonic<br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:14:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489728</guid>
      </item>
      <item>
         <title>Francine </title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489729</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:14:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489729</guid>
      </item>
      <item>
         <title>Aira Shyn Leigh Vergara </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489741</link>
         <description><![CDATA[<div>Slide 1<br>&nbsp;1. Rectangular shape<br>2.large vacoule<br>3.nucleus<br>4.cytoplasm<br>5. Cell membrane&nbsp;<br>6.round shape<br>7. Small vacoule<br>Slide 2<br>1.Plant Cell<br>2.Nucleus&nbsp;<br>3.Animal Cell<br>Slide 3<br>1. Animal Cell<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:14:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489741</guid>
      </item>
      <item>
         <title>Jhonbert Facurib</title>
         <author>jhonbertfacurib27</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489749</link>
         <description><![CDATA[<div>S-1<br>1.Rectangular<br>2.large<br>3.mitochondria<br>4.nucleus<br>5.golgi apparatus<br>6.Round<br>7.small<br>S-2<br>1.plant cell<br>2.animal cell<br>3. Nucleus<br>S-3<br>1.plant cell<br>S-4<br>1.nucleus<br>2.cell membrane<br>3.chloroplast<br>4.vacuole<br>S-5<br>1.mitochondrion<br>2.Endoplasmic reticulum<br>3.cell membrane<br>4.nucleus<br>S-6<br>1.from inside to outside<br>2.passive transport<br>S-7<br>1.from left to right<br>2.passive transport<br>3.Hypertonic solution<br>4.Hypotonic solution<br>S-8<br>1.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:15:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489749</guid>
      </item>
      <item>
         <title>Princess Kyla M.Endaya</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489756</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:15:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489756</guid>
      </item>
      <item>
         <title>Agorilla, Julianne B.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489779</link>
         <description><![CDATA[<div><strong>SLIDE 1</strong><br>1. Rectangular shape<br>2. The vacuole of the plant cell is bigger than the animal cell<br>3. Nucleus<br>4. Cell Membrane<br>5. Cytoplasm<br>6. Angular/Rigid shape or circular shape<br>7. The vacuole ofanimal cell is small compared to the plant cell<br><strong>SLIDE 2<br></strong>8. plant cell<br>9. Animal cell<br>10.<br>Slide 3<br>1.plant cell<br>Slide 4<br>1. Chloroplast<br>2 plasma membrane<br>3.&nbsp;<br>4. Mitochondria<br>Slide 5<br>1. Mitochondria<br>2. Ribosomes<br>3. Plasma membrane<br>4. Nucleus<br>Slide 6<br>1.from outside to inside the cell<br>2 osmosis<br>Slide 7<br>1. Water moves out of the cell<br>2. Plasmolysis<br>3.hypertonic solution</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:15:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489779</guid>
      </item>
      <item>
         <title>Fernandez 8-mendel</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489804</link>
         <description><![CDATA[<div>Slide 1<br>1. Ractangular shape<br>2. The vacuole is large<br>3.cell wall<br>4. Mitochondria&nbsp;<br>5. Cell membrane<br>6. Round shaped<br>7. The vacuole is small<br>Slide 2<br>1. The cells appearance is rectangular and it has a lot of cells present&nbsp;<br>2. The cells are round shaoed<br>3.nucleus<br>Slide 3<br>1.plant cell<br>Slide 4<br>1.nucleus<br>2.cell wall<br>3. Endoplasmic reticulum<br>4.golgi bodies<br>Slide 5<br>1. Endoplasmic reticulum<br>2.goldi bodies<br>3.cell wall<br>4. Nucleus<br>Slide 6<br>1.From outside to inside&nbsp;<br>2.osmosis<br>Slide 7<br>1.from left to right<br>2.osmosis<br>3.hypertonic solution<br>4. Hypotonic solution<br>Slide 8<br><br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:16:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489804</guid>
      </item>
      <item>
         <title>Safwan A. Kano</title>
         <author>Bloopbleeep426</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489826</link>
         <description><![CDATA[<div>SLIDE 1<br>1. Square<br>2. Big<br>3. Nucleus<br>4. Cytoplasm<br>5. Cell Membrane<br>6. Circle<br>7. Small<br><br>SLIDE 2</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:16:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489826</guid>
      </item>
      <item>
         <title>Princess Kyla M. Endaya 8-Mendel</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489837</link>
         <description><![CDATA[<div>S-1:<br>1: The shape is a square. But it is not a perfect square.<br>2: It is a part of a cell that stores water.<br>3: Cell Membrane<br>4: Nucleus<br>5: Golgi Bodies<br>6: The shape is a circle. But it is not a perfect circle.<br>7: Like the vacuole of the plant cell, the vacuole of the animal cell also stores water.<br><br>S-2: Nucleus<br><br>S-3: Plant cell<br><br>S-4:&nbsp;<br>1. Nucleus<br>2. Cell membrane<br>3. Vacuole<br>4. Ribosomes<br><br>S-5:<br>1. Golgi bodies<br>2.Ribosomes<br>3. Cell wal<br>4. Nucleus<br><br>S-6:<br>1. From inside to outside<br>2. Hypotonic<br><br>S-7:<br>1. From outside to inside&nbsp;<br>2. Osmosis<br>3. The solution to the left is hypertonic because its water is decreasing.<br>4. The solution to the right is hypotonic because its water is increasing.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:16:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489837</guid>
      </item>
      <item>
         <title>Jefferson Llamera 8-Mendel</title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489841</link>
         <description><![CDATA[<div>Compare and Contrast<br>S-1<br>1. Rectangular in shape<br>&nbsp;2. Large Vacoule<br>&nbsp;3. Nucleus<br>&nbsp;4. Cytoplasm<br>5. Cell membrane<br>6. Circular in shape<br>7. Small in size<br>S-2<br>1. Plant cell<br>2. Animal cell<br>3. Nucleus<br>S-3<br>1. Plant cell<br>S-4<br>1. Nucleus<br>2. Cell membrane<br>3. Vacoule<br>4. Chloroplast<br>S-5<br>1. Mitochondria<br>2. Endoplasmic Reticulum<br>3. Cell Membrane<br>4. Nucleus<br>S-6<br>1. Inside to outside<br>2. Plasmolysis<br>S-7<br>1. Outside to inside<br>2. Osmosis<br>3. Hypotonic<br>4. Hypertonic<br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:16:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489841</guid>
      </item>
      <item>
         <title>Wendell mejares</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489849</link>
         <description><![CDATA[<div>1.plants have a box shape. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:16:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489849</guid>
      </item>
      <item>
         <title>Name: Krystle Ann Z. Paguray</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489874</link>
         <description><![CDATA[<div><strong>8-MENDEL</strong><br><strong>S-1</strong><br>1. The shape of the plant cell is rectangular.<br>2. Large vacoules<br>3. nucleus <br>4. mitochondria<br>5. Vacoules<br>6. The shape of the animal cell is circle.<br>7. Small vacoules<br><strong>S-2</strong><br>1. plant cell<br>2. animal cell<br>3. Cell membrane<br><strong>S-3</strong><br>1. Plant cell<br><strong>S-4</strong><br>1. Nucleus<br>2. Cell Membrane<br>3. Vacoules<br>4. Centrioles<br><strong>S-5</strong><br>1. Vacoules <br>2. Endoplasmic Reticulum <br>3. Cell Wall <br>4. Nucleus<br><strong>S-6</strong> <br>1. The water will flow from higher concentration to lower concentration of water<br>2.&nbsp; Osmosis<br><strong>S-7</strong><br>1. From higher concentration to lower concentration of water.<br>2. Osmosis<br>3. Hypertonic Solution<br>4. Hypotonic Solution<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:17:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489874</guid>
      </item>
      <item>
         <title>Name:Mercado,Grejie Lou G</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489887</link>
         <description><![CDATA[<div>S-1<br>1. Kind of oval,and rectangular shape.<br>2.big<br>3.cell mebrane<br>4.cytoplasm<br>5.nucleus<br>6.not the same with the others,circular<br>7.small vacuole<br>Slide 2<br>1.plant cell<br>2.animal cell<br>3.nucleus<br>Slide #<br>1.animal cell<br>Slide 4<br>1.nucleus<br>2.cell membrane<br>3.vacuole<br>4.mitochondria<br>S-5<br>1.vacuoles<br>2.endoplasmic reticulum<br>3.cell membrane<br>4.nucleous<br>S-6<br>1.inside to outside<br>Hypotonic<br>S-7<br>1.outside to inside<br>2.osmosis<br>3.hypotonic<br>4.hypertonic</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:17:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489887</guid>
      </item>
      <item>
         <title>La</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489909</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:17:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489909</guid>
      </item>
      <item>
         <title>Slixe</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489924</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:17:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489924</guid>
      </item>
      <item>
         <title>Patrick james Lagang</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489938</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:18:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489938</guid>
      </item>
      <item>
         <title>Lrome S fajartin</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489982</link>
         <description><![CDATA[<div>S1<br>1.rectangular shape<br>2.large vacoule<br>3.nUcleus<br>4.cytoplasm<br>5.cell membrane<br>6.round shape<br>7.small vacoule<br>S2<br>1.plant cell<br>2.animal cell<br>3.nucleus<br>S3<br>1.animal cell<br>S4.<br>1.nucleus<br>2.cell Membrane<br>3.mitochondria<br>4.Cytoplast<br>S5.<br>1.ribosomes<br>2.Golgi body<br>3.cell wall<br>4.NucleuS<br>S6<br>1.active transport<br>2.inside to outside<br>S7<br>1.Osmosis<br>2.Outside to inside<br>3.HypertonIc<br>4.hypotonic<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:19:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489982</guid>
      </item>
      <item>
         <title>Fagutao, Ramer Kent G. 8-mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489989</link>
         <description><![CDATA[<div>S-1<br>1.the plant cell is in a rectangular shape<br>2.the vacoule in a plant cell is bigger than animal cell<br>3.mitochondria<br>4.Nucleus<br>5.endoplasmic reticulum<br>6.in circular shape<br>7.smaller than plant cell<br>S-2<br>1.plant cell<br>2.animal cell<br>3.nucleus <br>S-3<br>1.plant cell<br>S-4<br>1.nucleus<br>2.cell membrane<br>3.vacoule<br>4.chloroplast<br>S-5<br>1.mitochondrion<br>2.endoplasmic reticulum<br>3.cell membrane<br>4.nucleus<br>S-6<br>1. Passive transport<br>2.plasmolysis<br>S-7<br>1.active transport<br>2. Osmosis<br>3.hypotonic solution<br>4.hypertonic solution<br>S-8<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:19:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489989</guid>
      </item>
      <item>
         <title>Nikkko fajardo</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489990</link>
         <description><![CDATA[<div>S-1<br>1. it shape is oval/rectangular<br>2. Large<br>3.chloroplast<br>4. Centrioles<br>5.cell wall<br>6.it shape is circular<br>7. Small<br>S-2<br>8. Plant cell<br>9. Animal cell<br>10. Vacoule<br>S-3<br>11. Vacoule<br>S-4<br>12.cytoplasm<br>13. Centrioles<br>14. Ribosomes<br>S-5<br>15. Chloroplast<br>16. Endoplasmic reticulum<br>17. Cytoplasm<br>S-6<br>18.outside<br>19.difussion&nbsp;<br>S-7<br>20.the direction of the water is from high to&nbsp;<br>Low<br>21. Diffusion<br>22. Hypotonic<br>23. The water is going inside.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:19:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489990</guid>
      </item>
      <item>
         <title>Ej Carl Banlasan 8 Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270489991</link>
         <description><![CDATA[<div>Slide 1<br>1. ) rectangular<br>2. ) vacoule<br>3. )&nbsp; nucleus<br>4. )&nbsp; cytoplasm </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:19:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270489991</guid>
      </item>
      <item>
         <title>Ej</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270490446</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:28:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270490446</guid>
      </item>
      <item>
         <title>Ej Carl Banlasan 8 Menddl</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270490447</link>
         <description><![CDATA[<div>S-1<br>1.rectangular shape <br>2. Big Vacoule<br>3. Nucleus<br>4. Endoplasmic Reticulum<br>5. Cytoplasm <br>6. Round<br>7. Small Vacoule<br>S-2<br>1. Plant cell<br>2. Animal Cell<br>3. Nucleus<br>S-3<br>1. Animal cell<br>S-4<br>1. Nucleus<br>2. Cell Membrane<br>3. Vacoule<br>4. Mitochondria<br>S-5<br>1.mitochondrion<br>2. Endoplasmic reticulum<br>3. Cell Membrane<br>4. Nucleus<br><br>S-6<br>1. It will go to the right<br>2. Hyootonic<br><br>S-7<br>1. Plasmol<br>2. Hypertonic<br>3. Hypotonic</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:28:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270490447</guid>
      </item>
      <item>
         <title>Mato, Lacman L. red</title>
         <author>luckygrande05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270490823</link>
         <description><![CDATA[<div>S-1<br>1.Rectangular shape<br>2.Large <br>3.Nucleus<br>4.Cytoplasm <br>5.Cell Membrane<br>6.Round shape<br>7.Small<br>S-2<br>8.Plant cell<br>9.Animal Cell<br>10.Nucleus<br>S-3<br>11.Animal Cell<br>S-4<br>12.Nucleus<br>13.Cell Membrane<br>14.Vacuole<br>15.Mitochondria<br>S-5<br>16.Mitochondria<br>17.Endoplasmic Reticulum<br>18.Cell Membrane<br>19.Plasma Membrane<br>S-6<br>20.Outside of the cell<br>21.Hypotonic Solution<br>22.Passive Transport<br>S-7<br>23.Osmosis<br>24.Hypertonic<br>25.Hypotonic<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:35:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270490823</guid>
      </item>
      <item>
         <title>Jhonbert Facurib</title>
         <author>jhonbertfacurib27</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270491216</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:44:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270491216</guid>
      </item>
      <item>
         <title>Aira </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270491226</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:44:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270491226</guid>
      </item>
      <item>
         <title>Aira Shyn Leigh Vergara </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270491227</link>
         <description><![CDATA[<div>S1<br>1.Recatangular Shape<br>2. Large Vacoule<br>3.Nucleus<br>4.Cytoplasm<br>5.Cell Membrane&nbsp;<br>6.Round Shape<br>7.Small vacuole&nbsp;<br>S2<br>1.Plant Cell<br>2. Nucleus<br>3.Animal Cell<br>S3<br>1.Animal cell<br>S4<br>1.Nucleus<br>2.Cell Membrane&nbsp;<br>3.Vacoule<br>4.Metochondria<br>S5<br>1.Mitochondria<br>2.Endoplasmic<br>3.Cell Membrane&nbsp;<br>4.Nucleus<br>S6<br>1.Inside to Outside&nbsp;<br>Hypotonic<br>S7<br>Outside to Inside<br>Oismosis<br>Hypotonic<br>Hypertonic</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:44:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270491227</guid>
      </item>
      <item>
         <title>M</title>
         <author>luckygrande05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/270491311</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-18 05:47:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/270491311</guid>
      </item>
      <item>
         <title>Kenny Mcjewr Sulilap &amp; Angela Lester Cagungao</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271057663</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:08:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271057663</guid>
      </item>
      <item>
         <title>Darryl Tariao and Chorena Grace Lorania 8-Aristotle July 25, 2018</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060349</link>
         <description><![CDATA[<div>S1.</div><ol><li>  Hydrophilic phospate head</li><li>  Transport protein</li></ol><div>II.</div><ol><li>Low to high concentration</li><li>The movement is from low to high concentration thus it an Active transport</li></ol><div>S2</div><ol><li>Exocytosis</li><li>Endocytosis</li><li>It breaks down the wastes from the golgi apparatus/body</li></ol><div>S3</div><ol><li>Diffusion</li><li>Facilitated diffusion</li><li>Active transport</li><li>Passive transport</li></ol><div>S4<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296646070/62954284b0be1e8b169ad5edd3d09449/15324958516561972283092.jpg" />
         <pubDate>2018-07-25 04:34:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060349</guid>
      </item>
      <item>
         <title></title>
         <author>cimagalamary</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060411</link>
         <description><![CDATA[<div>Hamajo, Mika C.<br>Bantes, Jannah H.<br><br>07-25-18<br><br>Cellular Transport<br><br>S1<br>No.2&nbsp;<br>1. Phospate Head &amp; Transport Protein<br>2. Facilitated Diffusion<br>3. It is active because it is from lower to higher concentration<br><br>S2<br>1. Exocytosis<br>2. Endocytosis<br>3. Breaks down the molecules into sub-units<br><br>S3<br>1. Diffusion<br>2. Facilitated Diffusion<br>3. Osmosis<br>4. Passive Transport<br><br>S4</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/256728337/be625d0593ad4a3289c2617e29252ffd/1532494480151_216564311.jpg" />
         <pubDate>2018-07-25 04:35:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060411</guid>
      </item>
      <item>
         <title>Calonia,Francis Albert   Suarez,Alyanna Shin</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060414</link>
         <description><![CDATA[<div>Suarez,Alyanna S.&nbsp; 8- Aristotle&nbsp;<br>07-25-18<br>S-1<br>1.<br>A.Cell membrane<br>B.Molecules transported<br>C.Transport Protein<br>2.Endocytosis<br>3.Passive because it does need energy and the transport proteins helps the molecules to get in.<br>S-2<br>1.Exocytosis<br>2.Endocytosis<br>3.Breaks down micromolecules into submits.<br>S-3<br>1.Pinocytosis<br>2.Endocytosis<br>3.Exocytosis<br>4. Active Transport<br>S-4<br>&nbsp; Cellular Transport<br>Active Transport<br>*Exocytosis-excrete waste<br>*Endocytosis-let the nutrients to enter the cell<br>*Pinocytosis-drinking of cells in the form of liquid<br><br>Passive Transport<br>*Diffusion-passive transport of substances across the plasma membrane<br>*Osmosis-diffusion of water across the cell membrane<br>*Facilitated Diffusion-transport substances with the help of transport proteins<br>&nbsp; &nbsp;&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:35:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060414</guid>
      </item>
      <item>
         <title>CELLULAR TRANSPORT (8-Aristotle)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060428</link>
         <description><![CDATA[<div><strong>Names:<br></strong>Dejillo, Lanz S.<br>Berame, Bernadette Sue B.<br><br><strong>Date:</strong><br>July 25,2018<br><br><strong>S-1:<br></strong>1<strong>. </strong>Lipid Bilayer &amp; Channel Protein<br>2.Facilitated Diffusion<br>3.Passive transport because particles move from areas with higher concentration to areas with lower concentration.<br><br><strong>S-2:</strong><br>1.Exocytosis<br>2.Endocytosis<br>3.The lysosome breaks down the macromolecules<br>into subunits.<br><br><strong>S-3:<br></strong>1.Diffusion<br>2.Facilitated Diffusion<br>3.Osmosis<br>4.Passive Transport<br><br><strong>S-4:<br></strong><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298455354/bab0364a4841bd3725ef8b5ce704c6f3/1532496299114_1210082898.jpg" />
         <pubDate>2018-07-25 04:35:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060428</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060436</link>
         <description><![CDATA[<div>S-1<br>2. Endocytosis<br>3. Active transport,because</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:35:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060436</guid>
      </item>
      <item>
         <title>Zofia Amina Seromines and Jasmin Imelda Silva</title>
         <author>zofiaseromines123</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060452</link>
         <description><![CDATA[<div>Of</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:35:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060452</guid>
      </item>
      <item>
         <title>Salas, Christian</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060458</link>
         <description><![CDATA[<div>Belicena, Michael<br>072518<br>Cellular Transport<br>1transport protein<br>2.facilitated diffusion<br>3.passive transport<br>S2<br>1exocytosis<br>2Endocytosis<br>3breakdown macromolecules into submits<br>S3&nbsp;<br>1.Pinocytos<br>2endocytosis<br>3exocytosis<br>4active transport<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:35:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060458</guid>
      </item>
      <item>
         <title>Malunes  &amp;  Benjamin </title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060502</link>
         <description><![CDATA[<div><strong>S-1</strong><br>1. Phosphate head &amp; Fatty acid tails<br>2. Facilitated Diffusion&nbsp;<br>3. Passive transport,because it needs transport protein.<br><strong>S-2<br>1. </strong>Exocytosis<br>2. Endocytosis<br>3. It is the membrane-bound organelles that is used to store the molecules that is taken by cell<br><strong>S-3<br></strong>1.Diffusion<br>2. Facilitated Diffusion<br>3. Osmosis<br>4. Passive Transport&nbsp;<br><strong>S-4<br></strong><br>CONCEPT MAP OF CELLULAR TRANSPORT <br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:36:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060502</guid>
      </item>
      <item>
         <title>R</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060503</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:36:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060503</guid>
      </item>
      <item>
         <title>Reanne Kathleen B. Figuracion</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060504</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:36:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060504</guid>
      </item>
      <item>
         <title>Cutamora Mizpah, Miase Bea</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060513</link>
         <description><![CDATA[<div>I.<br>1. Phosphate head, Transport Protein<br>2. Exocytosis<br>3. It is active because the molecules move from lower to higher concentration<br>II.<br>1. Exocytosis<br>2. Endocytosis<br>3. Break down macromolecules into subunits and contain drolytic enzymes. It is also where wastes go.<br>III.<br>1. Osmosis<br>2. Facilitated Diffusion<br>3. Diffusion<br>4. Passive Transport<br>IV.</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295865171/d128ba5076627fc25d686cae9cf96cf7/15324987767741049884975.jpg" />
         <pubDate>2018-07-25 04:36:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060513</guid>
      </item>
      <item>
         <title></title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060542</link>
         <description><![CDATA[<div><strong>Members:<br><br></strong><br>Alberto, Achilles Alexis R.<br>Tan, Jb Earl G.&nbsp;<br>July 25, 2018&nbsp;</div><pre>          <strong>Cellular Transport </strong></pre><div><br><strong>S-1</strong><br>1. Hydrophilic and Transport Protein <br>2. From low to high concentration <br>3. Active Transport, because it moves from low concentration to high concentration. <br><br><strong>S-2</strong><br>1. Exocytosis <br>2. Endocytosis<br>3. It break downs macromolecules into o submits. <br><br><strong>S-3</strong><br>1. Diffusion <br>2. Facilitated Diffusion <br>3. Osmosis <br>4. Passive Transport <br><br><strong>S-4<br></strong><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296009012/97a2c49019f6e25a71dc206a9e5b1130/1532494365419276982374.jpg" />
         <pubDate>2018-07-25 04:36:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060542</guid>
      </item>
      <item>
         <title>Cabiles Zyrreg Zhyller , Urdaneta Caryl Jean</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060562</link>
         <description><![CDATA[<div>U</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:36:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060562</guid>
      </item>
      <item>
         <title>R</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060612</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:37:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060612</guid>
      </item>
      <item>
         <title>Lata, Ronan Raphael C.                           Lapating, Angelie Mae M.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060621</link>
         <description><![CDATA[<div>July 25, 2018<br><strong>Cellular Transport<br><br></strong>S-1<br>2.) Phospate Head<br>Carrier Protein<br>Facilitated Diffusion<br>3.) It is a passive transport because the molecules move from higher to lower concentration and facilitated diffusion is a type of passive transport.<br><br>S-2<br>1.) Exocytosis<br>2.) Endocytosis<br>3.) The lysosome helps in the digestion with its powerful enzymes.<br><br>S-3&nbsp;<br>1.) Osmosis&nbsp;<br>2.) Facilitated diffusion<br>3.) Diffusion<br>4.) Passive transport<br><br>S-4&nbsp;<br>Make a concept map of cellular transport base on the ppt.<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298457666/b49af7fa38a87c254ca9a9a8d1b9d7f8/1532494612240_14787442.jpg" />
         <pubDate>2018-07-25 04:37:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060621</guid>
      </item>
      <item>
         <title>lll</title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060739</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:39:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060739</guid>
      </item>
      <item>
         <title>Christian Louie N. Evangelista     Desire Faith C. Enojales</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060806</link>
         <description><![CDATA[<div>July 25, 2018<br>CELLULAR TRANSPORT<br>S1<br>1.cell membrane<br>2.facilitated diff<br>3.active transport, because it uses energy for the molecule to get in<br><br>S2&nbsp;<br>1-2. Endocytosis and Exocytosis<br>3. Lysosome become the storage center. This is the place where the molecules are stored.<br><br>S3<br>1-3. osmosis, diffusion ,and facilitated diffusion<br>4. Passive Transport<br>S4<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:40:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060806</guid>
      </item>
      <item>
         <title>Alvin Jay Deleverio, Nadine Sarmiento  8-Aristotle   7-25-18      Topic: Cellular Transport</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060812</link>
         <description><![CDATA[<div>S-1<br>1. Cell membrane, carrier protein<br>2.Facilitated Diffusion<br>3. Passive Transport, because facilitated diffusion doesn't need energy and it works naturally or on impulse.<br><br>S-2</div><ol><li>Exocytosis, Endocytosis</li><li>The lysosome is eating the molecules near it.</li></ol><div>S-3</div><ol><li>Diffusion</li><li>Channel Protein (Facilitated diffusion)</li><li>Carrier Protein (Facilitated diffusion)</li><li>Passive transport</li></ol><div><br>S-4<br>Passi</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302054721/5ae1104f7c3eec28de0b4169a98aa948/IMG20180725140130.jpg" />
         <pubDate>2018-07-25 04:40:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060812</guid>
      </item>
      <item>
         <title>Deniel Daved Natividad</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060900</link>
         <description><![CDATA[<div>dj christian juanico<br>8-aristotle<br>Date: July 25,2018<br>Title: Cellular Transport<br>S-1<br>1. Cell membrane and channel protein<br>2. Exocytosis<br>3.Active transport because it needed energy to transport.<br>S-2<br>1. Exocytosis<br>2. Endocytosis<br>3. Lysosome eats the foreign bodies that is not needed and harmful to the cells.<br>S-3<br>1.diffusion<br>2.facilitated diffusion<br>3.active transport<br>4.passive transport<br>s-4<br><br></div><blockquote><br></blockquote><div><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/301416325/c11c7ffbc1d77ddf0a53ff751a9bb54b/1532497080629_575389380.jpg" />
         <pubDate>2018-07-25 04:41:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060900</guid>
      </item>
      <item>
         <title>ZOFIA amina Seromines and Jasmin Imelda Silva</title>
         <author>zofiaseromines123</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271060979</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:41:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271060979</guid>
      </item>
      <item>
         <title>Cabiles Zyrreg Zhyller , Urdaneta Caryl Je</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061063</link>
         <description><![CDATA[<div>July 25, 2018<br>TOPIC: Cellular Transport<br>S1.<br>1. Channel Protein<br>2. Exocytosis<br>3.Active Transport because it moves from low to h</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:42:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061063</guid>
      </item>
      <item>
         <title>CELLULAR TRAN</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061238</link>
         <description><![CDATA[<div>July 24, 2018<br>Chorena Grace Lorania<br>Darryl Tariao<br><br><br>S1<br>1.Hydrophillic membrane<br>&nbsp;&nbsp; <br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:44:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061238</guid>
      </item>
      <item>
         <title>Seromines, Sofia Amina. S.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061468</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:47:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061468</guid>
      </item>
      <item>
         <title>Kenny Mcjewr Sulilap&amp;Angela Lester Cagungao</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061625</link>
         <description><![CDATA[<div>Date: July 25 2018Title: <br><br></div><div><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:49:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061625</guid>
      </item>
      <item>
         <title>Kenny Mxj</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061636</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:49:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061636</guid>
      </item>
      <item>
         <title>Zofia Amina Seromines and Jasmin Imelda Silva</title>
         <author>zofiaseromines123</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271061760</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:50:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271061760</guid>
      </item>
      <item>
         <title>Angela Lester V. Cagungao, Kenny Macjewr T. Sulilap</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062114</link>
         <description><![CDATA[<div>July 25, 2018<br>CELLULAR TRANSPORT<br>S1<br>1. Hydrophilic head<br> Vesicle<br>2. Endocytosis<br>3 Active transport because it is from low concentration to high concentration.<br>S2<br>1. Endocytosis<br>2. Exocytosis<br>3. It separates wastes from nutrients.<br>S3<br>1. Facilitated diffusion<br>2. Diffusion<br>3. Osmosis <br>4. Passive Transport</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 04:54:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062114</guid>
      </item>
      <item>
         <title>Shandee s.Sangatanan &amp; Shanlee S. Sangatanan</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062160</link>
         <description><![CDATA[<div>July 25, 2018<br>CELLULAR TRANSPORT<br><br>Slide 1:<br>1.- Phosphate head<br>- Carrier protein<br>2. Endocytosis<br>3. Active transport because it is from low concentration to high concentration<br><br>Slide 2:<br>1. Exocytosis<br>2. Endocytosis<br>3. The role of lysosome are membrane-bound organelle and break down macromolecules into subunits<br><br>Slide 3:<br>1. Diffusion<br>2. Facilitated diffusion<br>3. Osmosis<br>4. Passive transport<br><br>Slide 4:<br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/299451781/c1417a28c2ba87218dd1e8b69aed817f/15324960828321648814537.jpg" />
         <pubDate>2018-07-25 04:55:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062160</guid>
      </item>
      <item>
         <title>Christian Ian Charles Salas and Michael Belicena 8- Aristotle (real)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062355</link>
         <description><![CDATA[<div>S1<br>1.Facilitated diffusion<br>2.Carrier protein<br>3Passive transport because the process is facilitated diffusion and facilitated diffusion is a passive transport.<br>S2<br>1.Exocytosis<br>2.Endocytosis<br>3.Breakdowns the macromolecules into submits.<br>S3.<br>1.Diffusion<br>2.Facilitated diffusion<br>3.Osmosis<br>S4<br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302057253/254ded4c1cef2dbff3db17023632f2f9/1532498562025_136894550.jpg" />
         <pubDate>2018-07-25 04:57:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062355</guid>
      </item>
      <item>
         <title></title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062366</link>
         <description><![CDATA[<div><strong>Figuracion, Reanne Kathleen B. <br>Emata, Simone Anne C. <br></strong><br>S - 1:<br>1. Molecule and Cell Membrane <br>2. Endocytosis<br>3. Active Transport because energy is required to move materials through a cell membrane. It is against concentration gradient. <br><br>S - 2 <br>1.  Exocytosis <br>2. Endocytosis <br>3. Breaks down macromolecules into subunits. <br><br>S - 3 <br>1. Endocytosis <br>2. Endocytosis <br>3. Endocytosis<br>4. Active Transport <br><br>S - 4 <br>(Concept Map) <br> </div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298124724/a1ce76f42ba3f259be1c4112bc0899d6/image_22754d89_01ed_409c_8747_fadebb91d1e0.jpg" />
         <pubDate>2018-07-25 04:58:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062366</guid>
      </item>
      <item>
         <title>Malunes &amp; Benjamin</title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062528</link>
         <description><![CDATA[<div><strong>S-1</strong><br>1. Phosphate head &amp; Fatty acid tails<br>2. Facilitated Diffusion <br>3. Passive transport,because it needs transport protein.<br><strong>S-2<br>1. </strong>Exocytosis<br>2. Endocytosis<br>3. It is the membrane-bound organelles that is used to store the molecules that is taken by cell<br><strong>S-3<br></strong>1.Diffusion<br>2. Facilitated Diffusion<br>3. Osmosis<br>4. Passive Transport <br><strong>S-4<br>CONCEPT MAP <br></strong><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296539826/0f543c88feab7e21f3ad7fa11d54d3a6/1532498736731913578542.jpg" />
         <pubDate>2018-07-25 05:00:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062528</guid>
      </item>
      <item>
         <title>Pertimos, Ashley &amp;  Buladaco, Lei</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271062977</link>
         <description><![CDATA[<div>July 25, 2018<br>Cellular Transport<br><br>S-1<br>1. Lipids, Carrier Protein<br>2.&nbsp; Facilitated Diffusion<br>3. Passive, because it doesn't use energy<br><br>S-2<br>1. Endocytosis<br>2. Exocytosis<br>3. The lysosome breaks them into units.&nbsp;<br><br>S-3<br>1. Diffusion<br>2. Facilitated Diffusion<br>3. Active Transport<br>4. Passive transport<br><br>S-5<br>A particle is trying to enter the cell membrane➡The carrier protein identyfies it so it could enter➡It is identified, so the carrier proteins let them in ➡now the particle is in.&nbsp;<br>S-4<br>(Concept Map)</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296103241/1ab8ea043a61a37bbef5c411985f6677/15324987321971525860512.jpg" />
         <pubDate>2018-07-25 05:06:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271062977</guid>
      </item>
      <item>
         <title>Howard Godwin Delosreyes and Wendell Mejares </title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271063001</link>
         <description><![CDATA[<div>07/25/18<br>8-Mendel.&nbsp;<br>S-1<br>1)cell membrane ,<br>Carrier protein<br>2)facilitated diffusion and diffusiom<br>3)passive, because it does not require energy and it&nbsp;<br>also have carrier protein whinch can be seen in passive transport only<br>S-2<br>1-2)exoxytosis -<br>Endocytosis<br>3)Breaking the nutrieNts,that enters the cell<br>S-3)<br>1-3)diffusion<br>Facilitate d diffusion<br>Endocytosis<br>4)Passive transport and active transpoRt<br>S4)<br>Concept map<br>S-5)<br>Picture 1-the solute Plans tO enter the cell .<br>Picture 2-the solute was carried by the carrier protein on the process.<br>Picture3- the solute hace now entered the cell<br>&nbsp;Picture 4-others will do it&nbsp;Too<br>or do the process tOo(solutes)</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:06:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271063001</guid>
      </item>
      <item>
         <title>Jam</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271063026</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:07:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271063026</guid>
      </item>
      <item>
         <title>Ej </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271063137</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:09:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271063137</guid>
      </item>
      <item>
         <title>Ej Carl Banlasan and Pette Coscos 8 Mendel july 25 2O18</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271063142</link>
         <description><![CDATA[<div>S1<br>1. Phosphate Head, Carrier Protein<br>2. Endocytosis<br>3. Active Transport because molecules move from lower to higher concentration.&nbsp;<br><br>S2<br>1. Exocytosis<br>2. Endocytosis <br>3. It breaksdown the micromolecules into submit. It is also&nbsp;<br>Where waste goes<br>S3<br>1. Osmosis<br>2. Facilitated Diffusion<br>3. Diffusion<br>4. Passive Transport<br><br>S4</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302059184/6d35ad495a54643f5d666db153390a8f/1532498133536361546073.jpg" />
         <pubDate>2018-07-25 05:09:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271063142</guid>
      </item>
      <item>
         <title>Facurib, Jhonbert Fagutao, Ramer Kent</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271063669</link>
         <description><![CDATA[<div>July 25 2018<br>S-1<br>1.cell membrane,, particle<br>2.endocytosis<br>3.passive, because it does not require energy<br>S-2<br>1.Exocytosis<br>2.Endocytosis<br>3.it breaks down the macro molecules into sub units<br>S-3<br>1.diffusion<br>2.facilitated diffusion<br>3.osmosis<br>4. Passive transport<br>S-4<br>S-5<br>Figure1<br>-the solute is outside the cell<br>Figure2<br>-the solute is now halfway through entering the cell<br>Figure3<br>-the solute reaches the end of the path to entering the cell<br>Figure4<br>-the solute is now inside the cell</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/301017709/6f24356130c81a65ea5fd0d223619fd6/1532498469436442090071.jpg" />
         <pubDate>2018-07-25 05:18:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271063669</guid>
      </item>
      <item>
         <title></title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064706</link>
         <description><![CDATA[<div>Fernandez cadiz<br>July 25,2018<br>CELLULAR TRANSPORT <br>S-1<br>1. CELLULAR MEMBRANE AND LIPID<br>2. ACTIVE TRANSPORT <br>3.ACTIVE BECAUSE IT DOESN'T USE ANY KIND OF ENERGY OR ATP TO PASS THROUGH. <br>S-2<br>1. EXOCYTOSIS<br>2. ENDOCYTOSIS<br>3.. MEMBRANE-BOUND ORGANELLE. <br>S-3<br>1. DIFFUSION<br>2. FACILITATED DIFFUSION <br>3. PROTEIN PUMP<br>4 PASSIVE TRANSPORT <br>SS-5<br>THE SOLUTE ARE OUTSIDE ABOUT TO ENTER THE CELL--&gt;THE SOLUTE IS PASSING THE CARRIER PROTEIN --&gt; THE SOLUTE HAS ENTERED THE CELL - - &gt; ANOTHER SOLUTE IS ABOUT TO ENTER THE CELL. <br>S-4</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298129698/ecd6c2d080e6371c74aac34d64940782/1532498087766403799297328677386.jpg" />
         <pubDate>2018-07-25 05:36:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064706</guid>
      </item>
      <item>
         <title>Nikko Fajardo and Princess Kyla M. Endaya 8-Mendel</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064709</link>
         <description><![CDATA[<div>S-1:<br>1. Cell membrane and phosphate head.<br>2. Facilitated diffusion<br>3. Passive transport<br>S-2:<br>1-2. Exocytosis and endocytosis<br>3. The suicide center of the cell.<br>S-3:<br>1. Diffusion<br>2. Osmosis<br>3. Facilitated diffusion<br>4. Passive transport<br><br>S-5<br>1. The molecules go to cell membrane.<br>2. Extracellular potassium binds to exposed sites.<br>3. Binding of potassium causes dephosphorylation of protein.<br>4. Dephosphorylation of protein triggers change back to original conformation, potassium moves into cell, and the cycle repeats.<br><br>S-4:</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295896009/317764ba6d26d42e93e70c9391bb3d3f/1532498773066_1670978800.jpg" />
         <pubDate>2018-07-25 05:36:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064709</guid>
      </item>
      <item>
         <title>Cell Transport (July 25, 2018)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064728</link>
         <description><![CDATA[<div>Agorilla, Julianne<br>Palmaera, Kristine<br>Fajartin, Lrome<br>8- Mendel<br><br>S-1 <br>1. Plasma Membrane<br>2. Active Transport<br>3. Active Transport because it requires energy to move.<br>S-2<br>1. Exocytosis<br>2. Endocytosis<br>3. Encharge of breaking down macromolecules into sub- units<br>S-3<br>1. Simple Diffusion<br>2. Facilitated Diffusion<br>3. Osmosis<br>4. Passive transport<br>S-4<br>Concept Map<br><br>S-5<br>The solute from outside of the cell will enter the to carrier protein -----&gt; the solute from outside of the cell enters the carrier protein -----&gt; the shape of the solute is compatible in the carrier protein-----&gt; the solute  </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:37:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064728</guid>
      </item>
      <item>
         <title>Allyza Joy Cerqdo and Ma. Krizza Olwr</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064744</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:37:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064744</guid>
      </item>
      <item>
         <title>Allyza Joy Cerqdo and Ma. Krizza Olwr 8 Mendel.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064749</link>
         <description><![CDATA[<div>S1<br>1. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:37:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064749</guid>
      </item>
      <item>
         <title>Flores, Julija</title>
         <author>Bloopbleeep426</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064757</link>
         <description><![CDATA[<div>Kano, Safwan A.&nbsp;<br>07-25-2018<br><br>CELLULAR TRANSPORT<br>S1<br>1. Protein Carrier<br>2. Facilitated Diffusion<br>3. Passive because the transportation doesn't require energy for the molecules to pass through<br>S2<br>1-2.endocytosis and exocytosis<br>3. The lysosmes breaks the big particles<br>S3<br>1-3. Diffusion, Facilitated Diffusion, Active transport<br>4. Passive Transport<br>S5<br>1. A particle going to enter the cell ---&gt; A particle pass through the carrier protein to get inside the cell---&gt; A particle entered the cell successfully---&gt; Another particle is going to enter the cell again.<br>S4</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296203130/c94b02b6ab99000ee10a10819ec47a68/15324984744571789796929.jpg" />
         <pubDate>2018-07-25 05:38:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064757</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064808</link>
         <description><![CDATA[<div>S1<br>1.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:39:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064808</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064825</link>
         <description><![CDATA[<div>S1<br>1. Cell embrane, transport pro</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064825</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064826</link>
         <description><![CDATA[<div>S1<br>1. Cell embrane, transport protein<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064826</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064827</link>
         <description><![CDATA[<div>S1<br>1. Cell embrane, transport protein<br>2. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064827</guid>
      </item>
      <item>
         <title>James Baluso, Lacman Mato 8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064832</link>
         <description><![CDATA[<div>S1<br>1. Cell membrane, transport protein<br>2. Endocytosis<br>3. Active transport because it uses energy<br>S2<br>1. Exocytosis<br>2. Endocytosis<br>3.<br>s3<br>1.simple diffusion<br>2. Facilitated diffusion<br>3. Carrier protein<br>4. passive transport<br>S4 <br>S5<br>1.solute passes through carrier protien.<br>2. Carrier protein recognizes its shape<br>3. Carrier protein let solute pass.<br>4. Solute goes inside the cell.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064832</guid>
      </item>
      <item>
         <title>Bit</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064837</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064837</guid>
      </item>
      <item>
         <title>July 25,2018.                                                     Neil Mahinay and Rynch Magallano cellular transport</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064839</link>
         <description><![CDATA[<div>S1 cell membrane<br>: endocytosis<br>&nbsp;active transport because its from lower to higher concentration<br>S2:<br>Endocytosis<br>Exocytosis<br>S3:<br>Diffusion<br>Facilitated diffusion<br>Osmosis<br>Active transport<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302061545/c03fc673d3b29f31bd0d5b5696cad22b/IMG_20180725_140544.jpg" />
         <pubDate>2018-07-25 05:40:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064839</guid>
      </item>
      <item>
         <title>Cabiles Zyrreg Zhyller, Urdaneta Caryl Jean</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064841</link>
         <description><![CDATA[<div>July 25,2018<br>Topic: Cellular transport<br>S-1<br>1. Channel Protein<br>2. Exocytosis<br>3. Active transport because the molecules moves from low concentration to high concentration and it moves against the concentration gradient. <br>S-2<br>1Endocytosis<br>2.Exocytosis<br>3.break down large molecules / remove waste.<br>S-3 <br>1. osmosis <br>2. Facilitated diffusion <br>3. Diffusion <br>4. Passive Transport <br>S-4</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:40:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064841</guid>
      </item>
      <item>
         <title>Mercado,Brocoy</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064860</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br>1.hydrophilic head<br>Carrier protein<br>2.Osmosis<br>3.passive transport,because osmosis is a type of passive transport and it doesn't uses energy.<br>S-</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:41:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064860</guid>
      </item>
      <item>
         <title>Cerado Allyza Joy and Ma. </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064898</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:43:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064898</guid>
      </item>
      <item>
         <title>Cerado Allyza Joy and Ma. Krizza Mae Olar </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064900</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:43:12 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064900</guid>
      </item>
      <item>
         <title>Cerado, Allyza Joy and Olar, Ma. Krizza Mae 8- Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064902</link>
         <description><![CDATA[<div>S-1<br>1. Hydropholic Head<br>2. Vesicle<br>3. Endocytosis<br>4. It is an Active Transport because the partcles are entering the cell membrane using an energy.<br><br>S-2<br>1. Passive Transport<br>2.Diffusion<br><br>S-3<br>1. Endocytosis<br>2. Diffusion<br>3. Protein pumps<br>4. Active Transport<br><br>S-5<br>Facilitated Diffusion<br>It is the transport of substances across the plasma membrane. It aslo involves molecule diffusion across the cell membrane.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:43:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064902</guid>
      </item>
      <item>
         <title>Francine Sabelita and Gabrielle Galo</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064913</link>
         <description><![CDATA[<div>Cellular Transport<br>S1<br>1.Hydrophilic Head Vesicle<br>2.Endocytis<br>3.Active Transport&nbsp;Because it uses energy for the molecule to get </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:43:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064913</guid>
      </item>
      <item>
         <title>Cellular Transport</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271064919</link>
         <description><![CDATA[<div><strong>Krystle Ann Z. Paguray<br>Kelly Ashton A. Cubeta<br>8-Mendel<br>S-1<br></strong>1A. Lipids<br>1B. Carrier Protein<br>2. Cellular Transport&nbsp;<br>3. Passive Transport because it doesn't need energy and it passed through the cell by the carrier potein.<br><strong>S-2<br></strong>1. Exocytosis<br>2. Endocytosis<br>1-2.<br>3. It breaks the molecules into sub units.<br><strong>S-3<br></strong>1. Simple diffusion<br>2. Facilitated Diffusion<br>3. Active Transport<br>4. Passive Transport<br><strong>S-5<br></strong>1. The solute gets inside the cell passing through the carrier protein.<br>2. The solute is inside the carrier protein passing.<br>3. The solute just got out of the carrier protein so it is now inside the cell.<br>4. The solute is already inside the cell.<br><strong>S-4<br></strong>Concept Map</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/299548702/e38fcc0af1fb0e948da4cc6280072a78/15324995803441463852448.jpg" />
         <pubDate>2018-07-25 05:43:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271064919</guid>
      </item>
      <item>
         <title>MERRY </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065000</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:45:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065000</guid>
      </item>
      <item>
         <title>MERRY C. Oma</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065020</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:45:50 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065020</guid>
      </item>
      <item>
         <title>MERRY C. Omawi</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065026</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:46:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065026</guid>
      </item>
      <item>
         <title>MERRY C. Omawing</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065027</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:46:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065027</guid>
      </item>
      <item>
         <title>ME</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065084</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:47:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065084</guid>
      </item>
      <item>
         <title>ME</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065094</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:47:48 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065094</guid>
      </item>
      <item>
         <title>OMawing   and </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065200</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065200</guid>
      </item>
      <item>
         <title>OMawing   </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065201</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065201</guid>
      </item>
      <item>
         <title>OM</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065202</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065202</guid>
      </item>
      <item>
         <title>OMawing   and arcilla</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065203</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065203</guid>
      </item>
      <item>
         <title>OMawing   and arcilla</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065204</link>
         <description><![CDATA[<div>JULY 25, </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065204</guid>
      </item>
      <item>
         <title>OMawing</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065205</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065205</guid>
      </item>
      <item>
         <title>OMawing   and arcilla</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065206</link>
         <description><![CDATA[<div>JU</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065206</guid>
      </item>
      <item>
         <title>O</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065207</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065207</guid>
      </item>
      <item>
         <title>Merryl C. Omawing and Hannah M. Arcilla 8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065208</link>
         <description><![CDATA[<div>JULY 25, 2018 <br>cellular transport<br>S-1<br>1. hydrophilic head vesicle<br>2.endocytosis<br>3. active transport because the concentration is from low to high.<br>S-2<br>1.endocytosis<br>2.Exocytosis<br>3.it separates waste  from nutrients<br>S-3<br>1.Facilitated diffusion<br>2.DIffusion<br>3.osmosis<br>4.passive transport<br>S-4<br>Refer to the picture at the end. (concept map)<br>S-5<br>1.The molecules want to pass by the cell membrane<br>2.The molecules pass through the transport protein.<br>3.The molecules is transferring now to another side.<br>4.The molecules has already pass through the transport protein.<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298647553/22a18b41548cdb0a8f82c9625eb30f95/1532501974082_466134584.jpg" />
         <pubDate>2018-07-25 05:50:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065208</guid>
      </item>
      <item>
         <title>Aira Vergara and Leana Valdez</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065294</link>
         <description><![CDATA[<div>S-1<br>1.Plasma Membrane&nbsp;<br>2.Endocytosis<br>3.Active Transport Because it uses energy for the molecules to enter the cell<br>S-2<br>1.Exocytosis<br>2.Endocytosis<br>3. Become the storage center<br>S-3<br>1.Facilitated diffusion&nbsp;<br>2.Diffusion<br>3.Oismosis<br>4.Passive transport<br>S-5<br>1.The solute is in the outside and planning to go inside the carrier protein.<br>2.The solute is now in the carrier protein and the shape of it adjust so that the solute will fit.<br>3.The solute is going inside of the cell and the carrier protein was adjusting to go back in his normal shape.<br>4.The solute is now inside of the cell and the carrier protein is now back in the original shape<br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296137965/3e18d218efafbcf88c31b025e742c598/1532499325458_157680707.jpg" />
         <pubDate>2018-07-25 05:51:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065294</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065366</link>
         <description><![CDATA[<div>Cellular Transport<br>S1<br>1.Hydrophilic Head Vesicle<br>2.Endocytis<br>3.Active Transport Because it uses energy for the molecule to get in</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:53:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065366</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065371</link>
         <description><![CDATA[<div>Cellular Transport<br>S1<br>1.Hydrophilic Head Vesicle<br>2.Endocytis<br>3.Active Transport Because it uses energy for the molecule to get in</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:53:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065371</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065507</link>
         <description><![CDATA[<div>Cellular Transport<br>S1<br>1.Hydrophilic Head Vesicle<br>2.Endocytis<br>3.Active Transport Because it uses energy for the molecule to get in</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:55:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065507</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065510</link>
         <description><![CDATA[<div>Cellular Transport<br>S1<br>1.Hydrophilic Head Vesicle<br>2.Endocytis<br>3.Active Transport Because it uses energy for the molecule to get in</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:55:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065510</guid>
      </item>
      <item>
         <title>Gabrielle Galo and Francince </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065519</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:56:07 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065519</guid>
      </item>
      <item>
         <title>Howard Godwin Delosreyes</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065529</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:56:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065529</guid>
      </item>
      <item>
         <title>Howard Godwin Delosreyes and wendell Mejares</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065535</link>
         <description><![CDATA[<div>07/25/18<br>8-Mendel.&nbsp;<br>S-1<br>1)cell membrane ,<br>Carrier protein<br>2)facilitated diffusion and diffusiom<br>3)passive, because it does not require energy and it&nbsp;<br>also have carrier protein whinch can be seen in passive transport only<br>S-2<br>1-2)exoxytosis -<br>Endocytosis<br>3)Breaking the nutrieNts,that enters the cell<br>S-3)<br>1-3)diffusion<br>Facilitate d diffusion<br>Endocytosis<br>4)Passive transport and active transpoRt<br>S4)<br>Concept map<br>S-5)<br>Picture 1-the solute Plans tO enter the cell .<br>Picture 2-the solute was carried by the carrier protein on the process.<br>Picture3- the solute hace now entered the cell<br>&nbsp;Picture 4-others will do it Too<br>or do the process tOo(solutes)<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:56:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065535</guid>
      </item>
      <item>
         <title>Gabrielle Galo,Francine Sabelita                Cellular TransportS11.Hydrophilic Head Vesicle2.Endocytis3.It is Active Transport because it uses energy for the molecule to get in</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065623</link>
         <description><![CDATA[<div>S2<br>1-2 Endocytosis and Exocytosis<br>3.It seperates wastes from Nutrients<br>S3<br>1.Facilitated Diffusion<br>2.Diffusion<br>3.Osmosis&nbsp;<br>4.Passive Transport<br>S4<br><br><br><br>S5<br>1.Solute passes through carrier proteins<br>2.Carrier proteins recognize it shape<br>3.Carrier proteins let solute pass<br>4.Solute goes inside the cell<br><br><br><br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:57:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065623</guid>
      </item>
      <item>
         <title>M</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065639</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065639</guid>
      </item>
      <item>
         <title>Mercado,</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065643</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065643</guid>
      </item>
      <item>
         <title>Mercado,Bro</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065648</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065648</guid>
      </item>
      <item>
         <title>Mercado,Brocoy</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065653</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065653</guid>
      </item>
      <item>
         <title>Mercado,Brocoy </title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065656</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065656</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065657</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065657</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065659</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065659</guid>
      </item>
      <item>
         <title>Cellular Transport</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065661</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065661</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065664</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065664</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065668</link>
         <description><![CDATA[<div>Jul</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065668</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065677</link>
         <description><![CDATA[<div>July 25,2018</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:47 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065677</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065681</link>
         <description><![CDATA[<div>July 25,2018<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:48 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065681</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065685</link>
         <description><![CDATA[<div>July 25,2018<br>1</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065685</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065689</link>
         <description><![CDATA[<div>July 25,2018<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065689</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065692</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065692</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065693</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br>1.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:58:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065693</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065729</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br>1.L</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:59:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065729</guid>
      </item>
      <item>
         <title>Mercado,Brocoy 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065732</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br>1.Lipid's hydrophilic head<br>-transport protein<br>2.Endocytosis<br>3.Transport ,because endocytosis is a type of active transport and it use energy.<br>S2<br>1.Exocytosis<br>2.Endocytosis<br>3.break down macromolecules into subunits.<br>S3<br>1.diffusion<br>2.façilitated diffusion<br>3.passive transport<br>S5<br>The solute will go inside the cell through carrier protein<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 05:59:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065732</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065833</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295898994/d25c79b05ea30d9c07a5fb4bc55def97/1532498426072_2113136260.jpg" />
         <pubDate>2018-07-25 06:01:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065833</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271065998</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295865171/6845e6aabd3ced53353de3b9284948aa/1532498630056443140872.jpg" />
         <pubDate>2018-07-25 06:04:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271065998</guid>
      </item>
      <item>
         <title>7/25/18</title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271066124</link>
         <description><![CDATA[<blockquote>Jefferson Llamera and Patrick James Lagang</blockquote><div><br>S1<br>1. Cell membrane, transport protein<br>2. Osmosis<br>3. Passive transport, because it doesnt require energy<br>S2<br>1. Exocytosis<br>2. Endocytosis<br>3. To break down macromolecules into subunits<br>S3<br>1. Diffusion<br>2. Facilitated diffusion<br>3. Osmosis<br>4. Passive transport<br>S4<br>(Concept map at the very end) <br>S5<br>1. Solutes will enter inside the cell by passing the carrier protein<br>2. It is inside the carrier protein<br>3. Solute is inside the cell<br>4. The carrier protein will close <br>S4<br><br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295892210/2188c7f38c3016efe8e1fad555056133/1532499648584.jpg" />
         <pubDate>2018-07-25 06:07:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271066124</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271066130</link>
         <description><![CDATA[<div> <br>Zofia Amina Seromines and Jasmin Imelda Silva<br><br>Slide 1<br>1. Phosphate head <br>2. Pinocytosis<br>3. Active Transport<br>Slide 2<br>1.Exocytosis<br>2. Endocytosis<br>3. Break downs the molecules<br>Slide 3<br>1. Osmosis<br>2. Passive<br>3. Ac tive</div>]]></description>
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         <pubDate>2018-07-25 06:07:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271066130</guid>
      </item>
      <item>
         <title>Cagungao Angela, Sulilap Kenny</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271066804</link>
         <description><![CDATA[<div>Slide 4</div>]]></description>
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         <pubDate>2018-07-25 06:17:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271066804</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271066892</link>
         <description><![CDATA[<div><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 06:18:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271066892</guid>
      </item>
      <item>
         <title>Cerdo</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271067680</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 06:33:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271067680</guid>
      </item>
      <item>
         <title>Cerado, Allyza Joy B. and Ma. Krizza Mae B.  8 - Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271067686</link>
         <description><![CDATA[<div>S-1<br>1. Hydrophilic Head<br>2. Vesicle<br>3. Endocytosis<br>4. It is an Active Transport because in enters the cell membrane using energy to enter.<br><br>S-2<br>1.Endocytosis<br>2.Exocytosis<br>3. Lysosomes serves as the storage center of thr cell.<br><br>S-3<br>1. Endocytosis<br>2. Diffusion<br>3. Protein pumps<br>4. Active Transport<br><br>S-5<br>Facilitated Diffusion<br>Is the transport of particles across the cell membrane.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 06:33:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271067686</guid>
      </item>
      <item>
         <title>Brocoy, Angelle Kate y    </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271069100</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 06:53:19 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271069100</guid>
      </item>
      <item>
         <title>Mercado, Bro</title>
         <author>rejiefernandez88</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271069311</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 06:57:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271069311</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271069429</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/301410471/af2a2a03dfe83e3485312fda01c1346b/1532501869721_1022361680.jpg" />
         <pubDate>2018-07-25 06:59:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271069429</guid>
      </item>
      <item>
         <title>Credo a</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271069471</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/301410471/af2a2a03dfe83e3485312fda01c1346b/1532501869721_1022361680.jpg" />
         <pubDate>2018-07-25 07:00:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271069471</guid>
      </item>
      <item>
         <title>Cerado and Olar 8- Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271069479</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/301410471/af2a2a03dfe83e3485312fda01c1346b/1532501869721_1022361680.jpg" />
         <pubDate>2018-07-25 07:00:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271069479</guid>
      </item>
      <item>
         <title>Mercado,Brocoy</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271070570</link>
         <description><![CDATA[<div>July 25,2018<br>S1<br>1.Lipid's hydrophilic head<br>-transport protein<br>2.Endocytosis<br>3.Transport ,because endocytosis is a type of active transport and it use energy.<br>S2<br>1.Exocytosis<br>2.Endocytosis<br>3.break down macromolecules into subunits.<br>S3<br>1.diffusion<br>2.facilitated diffusion<br>3.passive transport<br>&nbsp;S5<br>-The solute will go inside the cell through carrier protein<br>- The carrier protein checks if the solute is allowed to go inside the cell,if it is not allowed,the solute will be rejected.<br>-The solute is accepted,so the solute enters the cell.<br>-The solute already enters the cell,and the process repeats.<br>S4<br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-25 07:19:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271070570</guid>
      </item>
      <item>
         <title>E</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271422342</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-30 05:46:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271422342</guid>
      </item>
      <item>
         <title>Ej Carl Banlasan</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271422344</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-07-30 05:46:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271422344</guid>
      </item>
      <item>
         <title>Howard GodwIn Delos Reyes</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271437646</link>
         <description><![CDATA[<div>What is Mitosis?<br>A type of cell division that results in two daughter cells each having the same number and kind of chromosomes as the parent nucleus, typical of ordinary tissue growth<br>There are 4 stages in Mitosis<br>1)Prophase-<br>Chromosomes become visible, crossing-over occurs, the nucleolus disappears, the meiotic spindle forms, and the nuclear envelope disappears.<br>2)Interphase<br>-During this time, a high amount of protein synthesis occurs and the cell grows (to about double its original size) – more organelles are produced and the volume of the cytoplasm increases.<br>3)TeLophase<br>-<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-30 10:57:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271437646</guid>
      </item>
      <item>
         <title>Howard Godwin</title>
         <author>howardfodwindelosreyes</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271438951</link>
         <description><![CDATA[<div>Telophase&nbsp;<br>-the final phase of cell division, between anaphase and interphase, in which the chromatids or chromosomes move to opposite ends of the cell and two nuclei are formed<br>Anaphase<br>-the final phase of cell division, between anaphase and interphase, in which the chromatids or chromosomes move to opposite ends of the cell and two nuclei are formed<br>What is the purpose of Metosis?<br>It is a gteat purpose itself on thth body of the organism for it is a way of repairing,reproducting and developiNg of the cell of the organism .It is making our body cells more Defending of itself ang it also Help reproducts for it Is a very fast and great way of reproducting </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-30 11:29:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271438951</guid>
      </item>
      <item>
         <title></title>
         <author>leibuladaco12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271440830</link>
         <description><![CDATA[<div>lei buladaco 8-mendel<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/299442175/9f50f80f52fd1b75a969dcb5a69a3163/Light_and_dark_photosynthesis.pptx" />
         <pubDate>2018-07-30 12:03:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271440830</guid>
      </item>
      <item>
         <title>Patrick Lagang 8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271441078</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302577712/a6232b925368a79fa46bb6025eb147c7/MEIOSIS.pptx" />
         <pubDate>2018-07-30 12:07:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271441078</guid>
      </item>
      <item>
         <title>Microbes Used In Biotech PPT</title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271444088</link>
         <description><![CDATA[<div>Kristine Joy B. Palmaera 8- Mendel<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298160052/4db67a56b99529e735dd831c611186c3/Microbes_Used_In_Biotech.pptx" />
         <pubDate>2018-07-30 12:57:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271444088</guid>
      </item>
      <item>
         <title>Metaphase </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271445891</link>
         <description><![CDATA[<div>Francine Nicole P. Sabelita 8-Mendel </div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302585217/23a7c56d64b63e3519c9c8ab2437c655/Metaphase_2.pptx" />
         <pubDate>2018-07-30 13:21:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271445891</guid>
      </item>
      <item>
         <title>Techniques and Practices in traditional Biotchnology</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271454748</link>
         <description><![CDATA[<div>Julianne B. Agorilla 8-Mendel</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295898994/29216f7e077bfe10a6c7123100a1d9c4/Techniques_And_Practices_In_Traditional_Biotechnology.pptx" />
         <pubDate>2018-07-30 14:52:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271454748</guid>
      </item>
      <item>
         <title>For mendel and aris</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271521821</link>
         <description><![CDATA[<div>Go to library and make the activity i gave yesterday. The individual activity in table form. Instead of doing that during report you will be the one to to fill up that. Make about mitosis first.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-07-31 04:39:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271521821</guid>
      </item>
      <item>
         <title>The Proper Use of Pipette </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271659492</link>
         <description><![CDATA[<div>Name: Bernadette Sue B. Berame<br>8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302844344/35ff159b073fe778eb8118f71753242a/The_Proper_Use_Of_Pipette.pptx" />
         <pubDate>2018-08-01 12:03:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271659492</guid>
      </item>
      <item>
         <title>INTRODUCTION TO CELL DIVISION</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271659967</link>
         <description><![CDATA[<div>Jannah H. Bantes<br>8- Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296034928/d7b60cb95473098f364e22dc8e1fa828/INTRODUCTION_TO_CELL_DIVISION.pptx" />
         <pubDate>2018-08-01 12:09:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271659967</guid>
      </item>
      <item>
         <title>DESCRIBE OR DIFFERENTIATE RNA FROM DNA</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271660001</link>
         <description><![CDATA[<div>Reanne Kathleen B. Figuracion<br>Grade 8- Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302843477/ddb622bb00dc00cd37d25a813f32d07b/BIOTECH_REPORT.pptx" />
         <pubDate>2018-08-01 12:09:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271660001</guid>
      </item>
      <item>
         <title>MITOSIS STAGE I: PROPHASE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271661988</link>
         <description><![CDATA[<div>Mika C.Hamajo<br>Grade 8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302847775/7724226456d4e85b393fb2d609f2f245/MITOSIS_STAGE_1.pptm" />
         <pubDate>2018-08-01 12:36:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271661988</guid>
      </item>
      <item>
         <title>Sterilization and Disinfection</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271668486</link>
         <description><![CDATA[<div>Nadine A. Sarmiento<br>Grade 8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302856016/8d36800515607aa878ed68491cb62884/BIOTECHNOLOGY_POWERPOINT_yeahyah.pptx" />
         <pubDate>2018-08-01 13:40:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271668486</guid>
      </item>
      <item>
         <title>ASEPTIC TECHNIQUE</title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271668623</link>
         <description><![CDATA[<div>Simone Anne C. Emata <br>8 - Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298124724/2b2ad6de38316e277b0150a4ab4a8e36/Aseptic_technique.pptm" />
         <pubDate>2018-08-01 13:41:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271668623</guid>
      </item>
      <item>
         <title>Preparation Of Biological Specimen</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271674978</link>
         <description><![CDATA[<div>Lanz Dejillo<br>Grade 8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302863774/dc4b3cf7b3de7bc2901af125592d822b/Verges___SlidesCarnival__1_.pptx" />
         <pubDate>2018-08-01 14:40:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271674978</guid>
      </item>
      <item>
         <title>MEIOSIS ANAPHASE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271850183</link>
         <description><![CDATA[<div>DJ CHRISTIAN A. JUANICO<br>GRADE 8- ARISTOTLE</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296100250/e219917cb9f70e992c1e1acc4eca43bb/Meiosis_Anaphase_1.pptx" />
         <pubDate>2018-08-03 02:49:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271850183</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271867853</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296100250/cc9789a1dcde26faa28f2ca6496b96c1/Prophase_1.pptx" />
         <pubDate>2018-08-03 06:55:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271867853</guid>
      </item>
      <item>
         <title>INOCULATION &amp; INCUBATION</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271925950</link>
         <description><![CDATA[<div>Angelie Mae M. Lapating<br>Grade 8-Aristotle<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296007188/46ffdbf78d76a556a6169b22e248e249/INOCULATION___INCUBATION.pptx" />
         <pubDate>2018-08-04 00:16:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271925950</guid>
      </item>
      <item>
         <title>techniques used in traditional biotechnology</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271926244</link>
         <description><![CDATA[<div>Chorena Grace G. Lorania&nbsp;<br>8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/303163316/f83c281dfa330903b86879300acc384d/Techniques_used_in_traditional_biotechnology.pptx" />
         <pubDate>2018-08-04 00:30:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271926244</guid>
      </item>
      <item>
         <title>prophase 1</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271932612</link>
         <description><![CDATA[<div>Francis Albert L. Calonia 8-aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/303172310/6e696de90eb4f489b1daf1206b0d215f/Prophase_1.pptx" />
         <pubDate>2018-08-04 03:23:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271932612</guid>
      </item>
      <item>
         <title>ATP PRODUCTION AND RESPIRATION</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271938046</link>
         <description><![CDATA[<div>Alyanna Shin D. Suarez 8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298341848/cc6589329b3a6a3ecf5d5d4960f0543c/BIOTECHNOLOGY_1.pptm" />
         <pubDate>2018-08-04 06:46:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271938046</guid>
      </item>
      <item>
         <title>Preparation Of Microbial Media</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/271944353</link>
         <description><![CDATA[<div>Lata, Ronan Raphael C.<br>Gr. 8-Aristotle<br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298457666/9deb6f6ef84d808d307a35665eb68636/Solanio___SlidesCarnival.pptx" />
         <pubDate>2018-08-04 11:32:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/271944353</guid>
      </item>
      <item>
         <title>Mitosis Telophase </title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/272069385</link>
         <description><![CDATA[<div>Achilles Alexis R. Alberto<br>8-Aristotle <br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296009012/fbe894a769f49495354b87482b318d64/Mitosis_Telophase.pptx" />
         <pubDate>2018-08-06 12:21:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/272069385</guid>
      </item>
      <item>
         <title>Comparison of Meiosis and Mitosis</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/272078652</link>
         <description><![CDATA[<div>Miase, Bea Clarisse A.<br>8-Aristotle</div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/303385497/a5af9b15d06a6adf5c3132f737a40934/PPTBIOT.ppt" />
         <pubDate>2018-08-06 13:48:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/272078652</guid>
      </item>
      <item>
         <title>USAGE OF  BURETTE </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/272217966</link>
         <description><![CDATA[<div>Jasmin Imelda  P. Silva <br>8-Aristotle </div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298291541/bbba9920b53c108f5862e8d0fa80f20f/USAGE_OF_BURETTE.pptx" />
         <pubDate>2018-08-07 15:36:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/272217966</guid>
      </item>
      <item>
         <title>SAMPLE 1 by  Sheena 🤪</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197724</link>
         <description><![CDATA[<div>Attainable objectives within a given amount of time but not congruent with the activities ,evaluation,and it is not appropriate in 5E's<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:45:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197724</guid>
      </item>
      <item>
         <title>Sample</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197734</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:45:19 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197734</guid>
      </item>
      <item>
         <title>Sample 1b</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197741</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:45:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197741</guid>
      </item>
      <item>
         <title>Its</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197811</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:46:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197811</guid>
      </item>
      <item>
         <title>Its </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197815</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:46:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197815</guid>
      </item>
      <item>
         <title> The objective of the activity is not congruent to the objective</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273197947</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:47:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273197947</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/273199611</link>
         <description><![CDATA[<div><br>Objectives are not attainable for that day it should be<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-15 06:59:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/273199611</guid>
      </item>
      <item>
         <title>Group 4 - 8 -Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274515197</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 08:58:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274515197</guid>
      </item>
      <item>
         <title>Group 4 VIII Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274515902</link>
         <description><![CDATA[<div>How long does it take to compleTe a cell cycle? </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:03:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274515902</guid>
      </item>
      <item>
         <title>Group 4 (Grade 8-Aristotle)</title>
         <author>sueberame890</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274519886</link>
         <description><![CDATA[<div><strong>Members</strong>:<br>Berame, Bernadette Sue B.<br>Cutamora, Mizpah Gabrielle S.<br>Urdaneta, Caryl Jean P.<br>Alberto, Achilles Alexis R.<br>Natividad, Deniel Dave C.<br>Cabiles, Zyrreg Zhyller A.<br><br><strong>How long does it take to complete a cell cycle?<br></strong>-It takes 24 hours to complete the cell cycle.<br><strong>INTERPHASE</strong>:<br><strong>G1</strong>- 10 hours<br><strong>S Phase- </strong>5-6 hours<br><strong>G2</strong>- 3-4 hours<br><strong>M Phase- </strong>2 hours</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:41:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274519886</guid>
      </item>
      <item>
         <title>How many hours will it takes to finish interphase ?</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274520420</link>
         <description><![CDATA[<div><strong>GROUP 6(GRADE 8 ARISTOTLE)<br></strong>MEMBERS:<br>Calonia,Francis Albert<br>Suarez, Alyanna Shin D.<br>Evangelista, Christian Louie N.<br>Enojales, Desire Faith C.<br>Tariao, Darryl B.<br><br>Representative:<br>Suarez, Alyanna Shin D.<br><br>Answer:<br> </div><div>G1 is typically the longest phase of the cell cycle. This can be explained by the fact that G1 follows cell division in mitosis; G1 represents the first chance for new cells have to grow. Cells usually remain in G1 for about 10 hours of the 24 total hours of the cell cycle. The length of S phase varies according to the total DNA that the particular cell contains; the rate of synthesis of DNA is fairly constant between cells and species. Usually, cells will take between 5 and 6 hours to complete S phase. G2 is shorter, lasting only 3 to 4 hours in most cells. In sum, then, interphase generally takes between 18 and 20 hours. <br><br><br>*G1=10 hours<br>*G2=3 to 4 hours<br>*Synthesis=5 to 6 hours<br><br>total interphase= 20 to 24 hours<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:46:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274520420</guid>
      </item>
      <item>
         <title>Group 1 (Grade 8-Aristotle) Lata,  Ronan Raphael C.Tan, JB Earl Dejillo,  Lanz S. Emata, Simone Anne C. Lapating, Angelie MaeFiguracion,  Reanne Kathleen How long does it take to complete cell cycle? </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274520496</link>
         <description><![CDATA[<div><br><br>Gap 1= 10 hours<br>Synthesis= 5 to 6 hours<br>Gap 2= 3 to 4 hours<br>Mitosis= 2 hours<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:47:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274520496</guid>
      </item>
      <item>
         <title>Group 3 aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274520670</link>
         <description><![CDATA[<div>Interpha</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:48:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274520670</guid>
      </item>
      <item>
         <title>GROUP 3 (8-Aristotle)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274521596</link>
         <description><![CDATA[<div>Members:<br>Chorena Grace Lorania<br>Shandee Sangatanan<br>Shanlee Sangatanan<br>Jannah Bantes<br>Nadine Sarmiento<br>Dj Christian Juanico<br><br><strong>How many hours will it takes to complete interphase?<br></strong>Gap 1=10 hours<br>Synthesis=between 5 and 6 hours<br>Gap 2=3 to 4 hours<br><strong>-So interphase generally takes between 18 and 20 hours.</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 09:59:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274521596</guid>
      </item>
      <item>
         <title>Group 3 (8-Aristotle)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274522349</link>
         <description><![CDATA[<div>MEMBERS:<br>Chorena Grace Lorania<br>Jannah Bantes<br>Shandee Sangatanan<br>Shanlee Sangatanan<br>Nadine Sarmiento<br>DJ Christian Juanico<br><br><strong>How long does it take to finish a cell cycle?<br></strong> G1 is typically the longest phase of the cell cycle. Cells usually remain in G1 for about 10 hours of the 24 total hours of the cell cycle. The length of S phase varies according to the total DNA that the particular cell contains. Usually, cells will take between 5 and 6 hours to complete S phase. G2 is shorter, lasting only 3 to 4 hours in most cells. In sum, then, interphase generally takes between 18 and 20 hours. Mitosis, during which the cell makes preparations for and completes cell division only takes about 2 hours. <br><br>Overall, it takes 24 hours to complete the cell cycle.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 10:07:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274522349</guid>
      </item>
      <item>
         <title>Group 5 (Aristotle)</title>
         <author>pmmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274522396</link>
         <description><![CDATA[<div><strong>How long does it take to complete the cell cycle?<br></strong><br><strong><em>Over all, it takes 22-24 hours to complete<br>G1- 10 hrs<br>S Phase- 5-6 hrs<br>G2- 3-4 hrs<br>Mitosis- 2 hrs<br><br>Members:</em></strong><br><strong>Miase</strong>, Bea Clarisse<br><strong>Silva</strong>, Jasmin<br><strong>Sulilap</strong>, Kenny<br><strong>Cagungao</strong>, Angela<br><strong>Belicena</strong>, Francis<br><strong>Salas</strong>, Christian</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 10:07:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274522396</guid>
      </item>
      <item>
         <title>Group 1 (Grade 8-Aristotle) Lata,  Ronan Raphael C.Tan, JB Earl Dejillo,  Lanz S. Emata, Simone Anne C. Lapating, Angelie MaeFiguracion,  Reanne Kathleen                                                                             How long does it take to complete cell cycle? </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274523872</link>
         <description><![CDATA[<div><br><br>Gap 1= 10 hours<br>Synthesis= 5 to 6 hours<br>Gap 2= 3 to 4 hours<br>Mitosis= 2 hours<br><br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 10:19:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274523872</guid>
      </item>
      <item>
         <title>Group 2 (Grade 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/274544049</link>
         <description><![CDATA[<div>How long does it take to complete the cell cycle?<br><br>*Usually, cells will take between 5 and <strong>6 hours</strong> to complete S phase. G2 is shorter, lasting only <strong>3 to 4 hours</strong> in most cells. In sum, then, interphase generally takes between 18 and <strong>20 hours</strong>. Mitosis, during which the cell makes preparations for and completes cell division only takes <strong>about 2 hours<br><br>Members:<br>Hamajo, Mika C.<br>Seromies, Zofia Amina<br>Deleverio, Alvin Jay<br>Benjamin, Chrys Jan<br>Malunes, Jaesar Justin</strong></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-22 12:36:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/274544049</guid>
      </item>
      <item>
         <title>Scores of a</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/275065306</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-08-24 04:37:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/275065306</guid>
      </item>
      <item>
         <title>Scores of aristole.</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/275065312</link>
         <description><![CDATA[<div>Group 1<br>Mastery- 10. <br> Delivery-10<br>Content-10<br>Group 6- +5  asking questions<br>Group 2<br>M- 9<br>D-8<br>C - 10<br>Group - 4<br>M- 10 <br>D- 10<br>C-10<br>Group 3 ask questions +5<br>Group 3 <br>M-10<br>D-8<br>C- 10<br>Group 5<br>M- 10<br>D-8 not collsborative<br>C- 10<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-24 04:37:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/275065312</guid>
      </item>
      <item>
         <title>Scores of aristole.</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/275065316</link>
         <description><![CDATA[<div>G</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-08-24 04:37:41 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/275065316</guid>
      </item>
      <item>
         <title></title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277420859</link>
         <description><![CDATA[<div>A</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 04:49:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277420859</guid>
      </item>
      <item>
         <title>Michael Francis P. Belicena Grade 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277472787</link>
         <description><![CDATA[<div>Questions:<br>1.What is Mitochondrial Disease?<br>Answer:<strong>Mitochondrial diseases</strong> are a group of disorders caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<strong>Mitochondria</strong> are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions<br>2.What are the symptoms of mitochondrial disease?<br>Answer:Depending on which cells of the body are affected, symptoms might include:<br><br></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Mental retardation</li><li>Heart, liver, or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Respiratory disorders</li><li>Diabetes</li><li>Increased risk of infection</li><li>Neurological problems, seizures</li><li>Thyroid dysfunction</li><li>Dementia (mental disorder characterized by confusion, disorientation, and memory loss)</li></ul><div>3.Who discovered the mitochondrial disease?<br>Answer: Much of what we know about these <strong>diseases</strong> has been<strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers<strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4.What happen when mitochondria is not working?<br>Answer:For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a<strong>problem</strong> and they may become weaker and get tired faster.<br>5.What are some examples of mitochondrial disease and can be cure?<br>Answer:A number of specific <strong>mitochondrial disorders</strong> have been associated with Complex I deficiency including: Leber's hereditary optic neuropathy (LHON), MELAS, MERRF, <strong>and</strong> Leigh Syndrome (LS). ... Myopathy (muscle <strong>disease</strong>) – starting in childhood or adulthood,<strong>and</strong> characterized by weakness or exercise intolerance.<br><strong>CASE STUDY ABOUT MITOCHONDRIAL DISEASE LOKE LEIGH SYNDROME</strong></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/308910661/1d8bc4798d0b92835400c27e34ca49e5/ijss_may_18.pdf" />
         <pubDate>2018-09-04 09:41:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277472787</guid>
      </item>
      <item>
         <title>Francis Albert L. Calonia</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277482931</link>
         <description><![CDATA[<div>GRADE 8-ARISTOTLE</div><div>Questions:<br>1. What is Mitochondrial Disease?<br><strong>Mitochondrial diseases</strong> are a group of <strong>disorders </strong>caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell. In some patients, only one organ is affected, while in other patients all the organs are involved. Depending on how severe the <strong>mitochondrial</strong> disorder is, the illness can range in severity from mild to <strong>fatal</strong>.<br>2. What are the symptoms of mitochondrial disease?Mitochondrial Disease should be suspected when three or more organ systems are involved.<br>BRAIN</div><div>Developmental delays, Migraines, Seizure, and Dementia<br>NERVES</div><div>Weakness (may be intermittent), Fainting, Absent reflexes and<br>Neuropathic pain<br>MUSCLES</div><div>Weakness, Irritable bowel syndrome, Gastroesophogeal reflux and Cramping and more.</div><div>3.Who discovered the mitochondrial disease?<br>In 1962, the first patient was diagnosed with a <strong>mitochondrial </strong>disorder. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria </strong>have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) <strong>found</strong> in the cells' nucleus.<br>4.What happen when mitochondria is not working?<br>For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a <strong>problem</strong> and they may become weaker and get tired faster.<br>5.What are some examples of mitochondrial disease and can it be cure?<br>Examples of mitochondrial diseases include chronic progressive external ophthalmoplegia, Kearns–Sayre syndrome, Leigh’s disease, Leber’s hereditary optic neuropathy (LHON), mitochondrial myopathy, and myoclonic epilepsy with lactic acidosis and stroke-like episodes (MELAS syndrome). Treatment options are limited, and primarily supportive. Carnitine may lessen muscle weakness and fatigue in some children, and does not interact with anesthetic agents.<br><br>C<strong>ASE STUDY: </strong></div><h1><strong> Mitochondrial myopathy</strong></h1><div>PATIENT<br><strong>An 11-month-old female is scheduled for a diagnostic muscle biopsy and open gastrostomy tube insertion. She has a history of hypotonia, developmental delay, and failure to thrive secondary to poor feeding effort. Chromosomal analysis is normal, and her neurologist suspects she may have a mitochondrial myopathy.</strong><br>MITOCHONDRIAL MYOPATHY:<br>A mitochondrial myopathy is a type of genetic disease that is encompassed within a broad category of entities whose origin is a defect in mitochondrial function, and thus interfere with normal adenosine triphosphate (ATP) production.  ATP depletion results in accumulation of lactate, a byproduct of anaerobic metabolism. Clinical manifestations include abnormalities of the heart, skeletal muscle, and central nervous system, among many others.<br>TREATMENT:<br>Preanesthetic assessment of a child with a suspected or confirmed mitochondrial disease includes evaluation of comorbidities – in particular, cardiac, respiratory, hepatic, and renal function. Premedication should be tailored to the individual patient; respiratory depressants should be avoided in children with weak ventilatory drive. The overall goal of anesthetic management is avoidance of metabolic stressors, such as hypoxemia and hypoglycemia, which may potentially exacerbate lactic acidosis. <br>S</div><div><br><br><br></div><div><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 10:37:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277482931</guid>
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      <item>
         <title>Alyanna Shin D. Suarez          8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277486589</link>
         <description><![CDATA[<div>Questions:<br>1. What is Mitochondrial Disease?<br>- Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.<br>2.What are the symptoms of mitochondrial disease?<br>- Symptoms can range from fatigue and exercise intolerance to hearing loss, seizures, strokes, heart failure, diabetes and kidney failure. Depending on the severity of the disorder, symptoms can appear at birth or they may not appear until adulthood &nbsp;<br>&nbsp;</div><div><strong>BRAIN</strong></div><div>Developmental delays, Migraines, Seizure, and Dementia&nbsp;<br>&nbsp;</div><div><strong>NERVES</strong></div><div>Weakness and Fainting <br><br>&nbsp;<strong>KIDNEYS</strong></div><div>Renal tubular acidosis or wasting</div><div><br>3.Who discovered the mitochondrial disease?<br>-In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mt DNA), which is different than the nuclear DNA (n DNA) found in the cells' nucleus.<br>4.Whathappens if mitochondria is not working?<br>- Mitochondria are called the powerhouse of cells, as they converts nutrients into energy using biochemical processes. These processes are also known as cellular respiration, where carbon dioxide and water are released. Therefore, if Mitochondria stop working, that means there is no respiration and the cells would die. <br>5.What are some examples of mitochondrial disease?Can this be cure?<br>- <strong>Alpers Disease -</strong> a progressive, neuro developmental, mitochondrial DNA depletion syndrome that begins in early childhood and is characterized by three co-occurring clinical symptoms: psychomotor regression (dementia); seizures; and liver disease. <br>-&nbsp; <strong>Autosomal Dominant Optic Atrophy (ADOA)&nbsp; </strong>is a neuro-ophthalmic condition which tends to begin in the first ten years of life and is characterized by degeneration of the optic nerves, causing&nbsp; visual loss.&nbsp;<br><br>Case Study:&nbsp;</div><h1>&nbsp;Sporadic Myopathy&nbsp;</h1><div><br>&nbsp;A 38-year-old man complained of lifelong intolerance to exercise. He described premature <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/fatigue">fatigue</a> and <a href="https://www.sciencedirect.com/topics/neuroscience/myalgia">myalgia</a> even after moderate exercise, such as walking on level ground for 20 min. Walking <a href="https://www.sciencedirect.com/topics/agricultural-and-biological-sciences/uphill">uphill</a> or upstairs, he could only tolerate for a few minutes. If he forced himself to exercise, he felt exhausted on the following day, and the muscles he used felt sore. He never noted pigmenturia. He believed his legs were more affected than his arms. A <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/cardiovascular-system">cardiovascular</a> consultation revealed no cardiac dysfunction. The neurological examination was normal, including muscle bulk and strength. Resting serum CK was normal. <a href="https://www.sciencedirect.com/topics/neuroscience/lactic-acid">Lactic acid</a> was 5.2 mEq/L (normal values, 0.5–2.2 mEq/L) and rose excessively after a standardized <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/aerobic-exercise">aerobic exercise</a> (9.2 mEq/L after 1 min). <a href="https://www.sciencedirect.com/topics/neuroscience/electromyography">Electromyography</a> findings were normal. His family history was negative for <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/neuromuscular-disease">neuromuscular disorders</a>; specifically, his mother and two sisters did not complain of <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/exercise-intolerance">exercise intolerance</a>. Biochemical analysis showed a distinct <a href="https://www.sciencedirect.com/topics/neuroscience/nadh-dehydrogenase-ubiquinone">complex I</a> deficiency in his frozen <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/muscle-biopsy">muscle biopsy</a>, and <a href="https://www.sciencedirect.com/topics/medicine-and-dentistry/histochemistry">histochemistry</a> revealed scattered hyper-SDH stained and COX-positive (ragged blue) fibers; Sanger <a href="https://www.sciencedirect.com/topics/biochemistry-genetics-and-molecular-biology/sequencing">sequencing</a> identified a pathogenic mutation (m.11832G&gt;A) in <em>MTND4</em> encoding the ND4 subunit of complex I.&nbsp;<br><br><br><br></div><div><br><br></div>]]></description>
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         <pubDate>2018-09-04 10:55:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277486589</guid>
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         <title>Alvin Jay A. Deleverio Grade 8-Aristotle</title>
         <author>audreydejillo</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277486623</link>
         <description><![CDATA[<div>Questions:<br>1. What is Mitochondrial Disease?<br>Answer:&nbsp; <strong>Mitochondrial diseases</strong> are a group of disorders caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<br>2. What are the symptoms?<br>Answer: The symptoms can be progressive neurological disease with motor and intellectual development delay and raised lactate levels in blood and cerebrospinal fluid.<br>3. Who discovered the mitochondrial disease?<br>Answer: In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.&nbsp;<br>4. What happens when mitochondria is not working?<br>Answer: when mitochondria is not working,&nbsp; less and less energy is generated within the cell.&nbsp;<br>5. What are some examples of mitochondrial disease? Can this be cured?<br>Answer: Some examples that cannot be cured are Leigh Syndrome, Pearson Syndrome, and MERRF Syndrome. Some examples that can be cured</div>]]></description>
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         <pubDate>2018-09-04 10:55:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277486623</guid>
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      <item>
         <title>Angela Lester V. Cagungao</title>
         <author>kimangela1595</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277487884</link>
         <description><![CDATA[<div>Grade 8- Aristotle<br>Questions:<br>1. What is Mitochondrial Disease?<br>Answer: &nbsp;</div><div>Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells).<br><br></div><div>Mitochondria are responsible for creating more than 90% of the energy needed by the body to sustain life and support organ function. When they fail, less and less energy is generated within the cell. Cell injury and even cell death follow. If this process is repeated throughout the body, whole organ systems begin to fail.&nbsp;<br><br>2. What are the symptoms of mitochondrial disease?<br><br>Answer: The symptoms of Mitochondrial disease(Mitochondrial Myopathy ) are&nbsp; shortness of breath, low blood pressure, tachycardia (heart rhythm disorder), constipation, exercise intolerance, muscle weakness, headaches, balance problems, anemia, and anxiety/depression.&nbsp;<br><br>3. Who discovered Mitochondrial diseases or disorders?<br>Answer:&nbsp; Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.&nbsp;<br><br>4. What happens when mitochondria are not working?<br>Answer:&nbsp; If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br><br>5. What are some examples of mitochondrial disease and can it be cured?<br>Answer:<br>Alpers Disease-&nbsp; This disease is not curable.<br>Autosomal Dominant Optic Atrophy (ADOA) - This disease is not curable.<br>Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy)- &nbsp; There is no specific treatment for Barth syndrome.<br>Mitochondrial myopathy-&nbsp; This disease is not yet curable.<br>Diabetes mellitus and deafness (DAD) - This disease is Curable.<br>Leigh syndrome -&nbsp; There is no cure for Leigh's Disease.&nbsp;<br><br></div><div>CASE STUDY:<br><br>&nbsp;</div><h1><strong>Women with Mitochondrial Disease More Prone to Pregnancy Complications, Study Shows</strong></h1><div><br>&nbsp;</div><div>Women with <a href="https://mitochondrialdiseasenews.com/">mitochondrial disease</a> or mitochondrial dysfunction are more susceptible to complications during pregnancy, and their newborns are more likely to have congenital defects, a retrospective study shows.<br><br></div><div>The study, “<a href="https://www.sciencedirect.com/science/article/pii/S1567724918301089?via%3Dihub">Effects of mitochondrial disease/dysfunction on pregnancy: A retrospective study</a>,” was published in <a href="https://www.sciencedirect.com/journal/mitochondrion"><em>Mitochondrion</em></a>.<br><br></div><div>Primary mitochondrial disease (MD) comprises several rare disorders caused by genetic mutations in nuclear and mitochondrial DNA, whereas <a href="https://my.clevelandclinic.org/health/diseases/15612-mitochondrial-diseases">mitochondrial dysfunction</a> (Md) may develop through environmental exposure, medications, infection or other non-genetic factors.<br><br></div><div>Both MD and Md lead to mitochondria malfunction, in particular decreased production of adenosine triphosphate (ATP) — the energy molecule used by all cells in the body — by the mitochondrial respiratory chain (MRC), which has a direct impact on cell function.&nbsp;<br><br></div><div>During pregnancy, metabolic demands increase to allow fetal development and growth, which requires optimal and efficient energy production. But there is little information regarding the impact of pregnancy on women with MD/Md who might not be able to sustain normal energy production and have, in theory, a higher chance of pregnancy complications.<br><br></div><div>In this study, authors analyzed obstetric effects and potential complications in women with MD/Md and their children to gather information that could help improve maternal and fetal outcomes in MD/Md in the future.<br><br></div><div>The study analyzed 103 women diagnosed with MD/Md between 16 and 75 years old who had been pregnant at least once. Information about their demographic backgrounds, gynecological history, fertility, pregnancy history, postnatal issues, MD/Md symptoms and use of dietary supplements was gathered in online surveys.<br><br></div><div>Most were Caucasian (94.2%) and were diagnosed with <a href="https://mitochondrialdiseasenews.com/category/mitochondrial-myopathy-news/">mitochondrial myopathy</a>(34%). A total of 370 pregnancies (average 3.6 pregnancies/woman), with 248 leading to live deliveries, were reported by the women.<br><br></div><div>In 68.1% of all pregnancies, newborns were delivered vaginally at an average gestational age of 38.2 weeks, within the normal range of 38 to 42 weeks. But 10.9% of these newborns had breathing difficulties, and 8.4 percent were diagnosed with congenital defects, mostly associated with heart problems.&nbsp;<br><br></div><div>On average, the women had their first period at age 13, nearly identical to the general U.S. population (12.5 years old), first got pregnant at 24.4 years old, and 46.6% said they had at least one miscarriage during the first or second trimester (average loss was 2.4 pregnancies/woman).<br><br></div><div>Most (78%) experienced pregnancy complications with varying degrees of severity, including vaginal bleeding (37.9%), abnormally high levels of proteins in the urine (26.2%), high blood pressure (24.3%), gestational diabetes (23.3%), fetuses who were small for gestational age (21.4%), and pre-eclampsia (18.4%).<br><br></div><div>In general, pregnancy seemed to worsen chronic MD/Md symptoms, including shortness of breath, low blood pressure, tachycardia (heart rhythm disorder), constipation, exercise intolerance, muscle weakness, headaches, balance problems, anemia, and anxiety/depression.<br><br></div><div>The findings indicate that MD/Md tends to exacerbate women’s disease symptoms during pregnancy and raise the likelihood of common gestation complications, but all these effects do not appear to lead to drastically worse pregnancy outcomes.<br><br></div><div>“[These findings] highlight the need for future studies to better assess complications and health concerns of pregnant women affected with primary mitochondrial disease and how they differ from mitochondrial dysfunction in a larger population of patients. In the future, a multi-center prospective analysis of pregnant women with MD may aid in providing more concrete information about the specific risks of complications during pregnancy when compared to a healthy cohort of females,” the researchers wrote. <br><br><strong>Insights</strong><br>The according to the article, the Mitochondrial disease affects pregnancy.  Pregnant women must take care of their health to avoid Mitochondrial diseases or disorders.</div><div><br>&nbsp;</div><div><br><br><br></div>]]></description>
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         <pubDate>2018-09-04 11:01:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277487884</guid>
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      <item>
         <title>Christian Ian Charles R. Salas 8- Aristotle        </title>
         <author>christiansalas68</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277488993</link>
         <description><![CDATA[<div>Questions:<br><strong>1. What is Mitochondrial Disease?</strong><br>Answer:<strong>Mitochondrial diseases</strong> are a group of disorders caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>Answer: &nbsp;</div><div><strong>BRAIN</strong></div><div>Developmental delays, migraines, seizures, dementia, strokes and more</div><div><strong>NERVES</strong></div><div>Weakness, fainting, absent reflexes,dysautonomia and more.<br><strong>Muscles<br></strong>&nbsp;Weakness, irritable bowel syndrome, Gastroesophogeal reflux,&nbsp; cramping and more.&nbsp;</div><div><strong>3. Who discovered Mitochondrial disease?</strong><br>Answer:<br>In 1962, the first patient was diagnosed with a <strong>mitochondrial </strong>disorder. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria </strong>have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) <strong>found</strong> in the cells' nucleus.<br><strong>4. What happens when mitochondria is not working? Can it be cured?</strong><br>Answer:<br>-For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are not <strong>working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br>-Mitochondrai cannot be cured but there are treatments on this disease.<br><strong>5. What are examples of mitochondrial disease?</strong><br>Answer:<br> Examples of mitochondrial diseases include chronic progressive external ophthalmoplegia, Kearns–Sayre syndrome, Leigh’s disease, Leber’s hereditary optic neuropathy (LHON), mitochondrial myopathy, and myoclonic epilepsy with lactic acidosis and stroke-like episodes (MELAS syndrome). Treatment options are limited, and primarily supportive. Carnitine may lessen muscle weakness and fatigue in some children, and does not interact with anesthetic agents. <br><br><strong>Case Study:<br></strong>&nbsp;</div><div>The study reports the case of a 45-year-old man who presented to <a href="http://www.chop.edu/centers-programs/mitochondrial-genetic-disease-clinic">The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic</a> for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a <strong>mitochondrial myopathy</strong>, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br></div><div>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br></div><div>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.<br><br></div><div>The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.&nbsp;<br><br></div><div><br><br><br><br><br><br></div><div><br></div>]]></description>
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         <pubDate>2018-09-04 11:07:12 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277488993</guid>
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         <title>Christi</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491338</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 11:18:35 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491338</guid>
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         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491586</link>
         <description><![CDATA[<div>s i1. W</div>]]></description>
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         <pubDate>2018-09-04 11:19:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491586</guid>
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      <item>
         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491637</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 11:19:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491637</guid>
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      <item>
         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491646</link>
         <description><![CDATA[<div>w</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 11:19:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491646</guid>
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      <item>
         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491650</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 11:20:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491650</guid>
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      <item>
         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author>christianlouie9244</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277491676</link>
         <description><![CDATA[<div>1. What is Mitochondrial Disease?<br>Answer:<br>Mitochondrial disease is an inherited chronic illness that can be present at birth or develop later: in life. It causes debilitating physical, developmental, and cognitive disabilities .<br>2. What are the symptoms of mitochondrial disease/disorder?<br>Answer:<br>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br><br></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss.&nbsp;</li><li>3.Who discovered mitochondrial disease?</li><li>Answer:</li><li><strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers<strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.</li><li>4. What happen when mitochondrial are not working?</li><li>Answer</li><li>mitochondria fail to produce enough energy for the body to function properly.</li><li>5. What are some examples of mitochondrial disease? Can these be cure?</li><li><strong>Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy), Autosomal Dominant Optic Atrophy (ADOA), Fatty Acid Oxidation Disorders / Beta-oxidation Defects / Fatty Acid Transport and Mitochondrial Oxidation Disorders and Alpers Disease.</strong></li><li>Cas</li></ul>]]></description>
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         <pubDate>2018-09-04 11:20:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277491676</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277494964</link>
         <description><![CDATA[Autosomal Dominant Optic Atrophy (ADOA)  is a neuro-ophthalmic condition which tends to begin in the first ten years of life and is characterized by degeneration of the optic nerves, causing  visual loss.]]></description>
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         <pubDate>2018-09-04 11:35:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277494964</guid>
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      <item>
         <title>Shandee S. Sangatanan Gr. 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277495251</link>
         <description><![CDATA[<div><strong>QUESTION and ANSWERS:<br>1. WHAT IS MITOCHONDRIAL DISEASE?</strong><br>-Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br><br><strong>2. WHAT ARE THE SYMPTOMS OF MITOCHONDRIAL DISEASE?</strong></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory</li></ul><div><br><strong>3. WHO DISCOVERED MITOCHONDRIAL DISEASE OR DISORDER?</strong><br>-&nbsp; Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. WHAT HAPPENS WHEN MITOCHONDRIA ARE NOT WORKING?</strong><br>-If mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and then may become weaker and get tired faster.<br><br><strong>5. WHAT ARE SOME EXAMPLES OF MITOCHONDRIAL DISEASE? CAN THESE BE CURED?</strong><br>* Mitochondrial myopathy<br>* Diabetes mellitus and deafness (DAD)<br>* Lebee's hereditary optic neuropathy (LHON)<br>* Leigh syndrome<br>* Neuropathy,ataxia,retinitis pigmentosa, and ptosis (NARP)<br>* Myoneurogenic Epilepsy withRagged Red Fibers (MERRF)<br>* Mitochondrial myopathy, encephalomyopathy, lactic acidosis, stroke-like sympyoms ((MELAS)<br>* Mitochondrial neurogastrointestinal encephalomyopathy (MNGIE)<br><br>- Treatments that can ease some of the symptoms or slow the progression of mitochondrial disease do exist, but as yet, none can cure them.<br><br><strong>A CASE STUDY OF MITOCHONDRIAL DISEASE: Mit</strong><strong><em>ochondrial Neurogastrointestinal Encephalomyopathy: Diagnose and response to peritoreal dialysis</em></strong><strong><br></strong>A 26-year-old man born to nonconsanguineous parents was admitted with complaints of muscle cramps, fatigue, recurrent abdominal pain, and vomiting since 10 years of age. Since 16 years of age, he noted progressive weakness of the proximal and distal muscles of all 4 limbs with intermittent worsening precipitated by febrile illnesses. He reported dysesthesias of the extremities and gait unsteadiness, which worsened in the dark. There was deterioration of symptoms with progressive dyspnea, orthopnea, dysphagia, early satiety, abdominal pain, and postprandial vomiting associated with a weight loss of 13 kg in the 6 months prior to presentation. Previous treatment received elsewhere included intermittent oral corticosteroids and azathioprine with suboptimal benefit. There was history of proximal weakness, recurrent abdominal pain, and respiratory involvement in his younger sibling (age at onset 20 years), who had died of the illness within 4 years of disease onset.<br><br></div><div>On examination, the patient was grossly emaciated. Body weight was 42 kg, height 174 cm with body mass index (BMI) of 13.9 kg/m<sup>2</sup> (normal population BMI 18.5–22.9 kg/m<sup>2</sup>). He was tachypneic with chest expansion of 1.5 cm and paradoxical breathing. There was bilateral ptosis, horizontal gaze restriction, bifacial weakness, reduced palatal movements, and an impaired gag reflex. Motor examination revealed moderate neck weakness and symmetric weakness in upper and lower limbs (proximal Medical Research Council [MRC] grade 4/5 and distal MRC grade 2/5). There was reduction in pain and temperature perception in a glove-stocking pattern. Joint, position sense, and vibration were impaired in all 4 limbs. All deep tendon reflexes were absent. Romberg sign was positive and there was sensory ataxia.<br><br></div><div>Routine investigations including blood counts and liver and renal function were normal. Autonomic function testing revealed resting tachycardia and orthostatic hypotension within 3 minutes necessitating termination of tilt table test (<a href="http://n.neurology.org/content/86/14/e147#F1">figure, A</a>). Other investigations included creatine kinase 349 IU/mL (normal 45–195 IU/mL), venous blood lactate 3.2 mmol/L (normal 0.3–1.3 mmol/L), and arterial blood gas analysis showing hypercapneic respiratory failure needing intermittent noninvasive ventilation (NIV). Pulmonary function testing suggested severe restrictive lung disease. Nerve conduction studies (NCS) showed a mixed axonal-demyelinating polyneuropathy with phrenic nerve involvement. Sympathetic skin response was absent. On needle EMG, there was spontaneous activity in the form of fibrillations and positive sharp waves from the tibialis anterior and vastus lateralis. The motor unit potentials were small amplitude and short duration with increased interference. CSF analysis showed 2 cells with elevated protein (148 gm/dL) and normal sugar (73 mg/dL). MRI of the brain showed patchy leukoencephalopathy. Muscle biopsy revealed myopathic changes without ragged-red fibers. There was evidence of cytochrome <em>c</em> oxidase (COX) activity with COX and COX–succinate dehydrogenase stains. Nerve biopsy showed extensive loss of myelin sheath and axons with myelin ovoid formation without inflammation.<br><br></div><div><strong><br><br></strong><br></div>]]></description>
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         <pubDate>2018-09-04 11:36:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277495251</guid>
      </item>
      <item>
         <title>Shanlee . Sangatanan Grade 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277495413</link>
         <description><![CDATA[<div><strong>Questions:</strong><br><strong>1. What is Mitochondrial Disease?</strong><br><strong>Answer:</strong> Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells).</div><div>Mitochondria are responsible for creating more than 90% of the energy needed by the body to sustain life and support organ function. When they fail, less and less energy is generated within the cell. Cell injury and even cell death follow. If this process is repeated throughout the body, whole organ systems begin to fail.<br><strong>2. What are the symtoms of Mitochondrial Diseases?<br>Answer:</strong> </div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss</li></ul><div><strong>3. Who discovered the Mitochondrial Disease or Disorder?<br>Answer:</strong> In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><strong>4. What happens when mitochondria are not working?<br>Aswer:</strong> For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem<strong> </strong>and they may become weaker and get tired faster.<br><strong>5. What are the examples of Mitochondrial Diseases? can these be cured?<br>Answer:</strong> </div><ul><li>Mitochondrial myopathy</li><li>Diabetes mellitus and deafness (DAD)</li><li>Leber's hereditary optic neuropathy (LHON)</li><li>Leigh syndrome, subacute sclerosing encephalopathy</li><li>Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)</li><li>Myoneurogenic gastrointestinal encephalopathy (MNGIE)</li><li>Myoclonic Epilepsy with Ragged Red Fibers(MERRF)</li><li>Mitochondrial myopathy, encephalomyopathy, lactic acidosis, stroke-like symptoms (MELAS)</li><li>mitochondrial neurogastrointestinal encephalomyopathy (MNGIE)</li></ul><div>-The Mitochondrial Diseases cannot be cured but it can ne prevented.<br><br></div><div><strong>Case Study:</strong></div><h1><br></h1><h1><strong>Mitochondrial myopathy presenting as fibromyalgia: a case report</strong></h1><ul><li>Mishal Abdullah,</li><li>Sahana Vishwanath,</li><li>Amro Elbalkhi and</li><li>Julian L AmbrusJr<a href="mailto:jambrus@buffalo.edu">Email author</a></li></ul><h1><em>Journal of Medical Case Reports</em>2012<strong>6</strong>:55<br><a href="https://doi.org/10.1186/1752-1947-6-55">https://doi.org/10.1186/1752-1947-6-55</a>©  Abdullah et al; licensee BioMed Central Ltd. 2012</h1><div><br><a href="https://jmedicalcasereports.biomedcentral.com/articles/10.1186/1752-1947-6-55#Casepresentation"><strong>Case presentation</strong></a><br>Our patient was a 41-year-old Caucasian woman who presented to our Rheumatology clinic for evaluation of progressive exercise intolerance, fatigue, diffuse myalgias, arthralgias and difficulty sleeping. The pain primarily involved her entire back and arms, and she reported multiple tender points all over her body. On the basis of her symptoms, our patient had also been diagnosed with fibromyalgia, and was treated with multiple different medications without any relief.<br><br>Her other medical history included hypothyroidism, cervical disc disease, hypertension and Raynaud's disease. Notable family history included breast carcinoma and hypertension in her mother, and lymphoma in her father. Her medications at presentation included lisinopril 5 mg daily and levothyroxine 25 μg daily. Other pertinent medications she had previously used included pregabalin, amitriptyline and gabapentin. Her physical examination results were normal except for mild tenderness to palpation along her upper and lower back, and shoulders.<br><br>Significant laboratory results included creatine kinase (CK) = 325 U/L (normal range 0 to 150 U/L), normal comprehensive metabolic panel (CMP) and complete blood count (CBC), liver function tests and thyroid functions tests. A six-minute walk test revealed a normal resting lactic acid = 1.6 mmol/L, and an elevated post-six-minute-walk lactic acid = 5 mmol/L (normal result &lt; 2 mmol/L). Her ammonia levels were normal at rest and after six-minute walk (14 μmol/L and 38 μmol/L respectively; normal 0 to 40 μmol/L). A muscle biopsy was performed for biochemical evaluation, which revealed several abnormalities including decreased levels of citric acid synthase (49% of normal), cytochrome <em>c</em> oxidase (53% of normal), succinate dehydrogenase (72% of normal), and nicotinamide adenine dinucleotide (NADH) dehydrogenase (73% of normal), thereby demonstrating a defect in the mitochondrial respiratory chain. Genome sequencing was performed, which revealed multiple POLG1 polymorphisms (C-T polymorphism at 2254, and G-T polymorphism at 3708) and several mitochondrial genome polymorphisms (1438 A-G, 3992 C-T, 14365 C-T, 14582 A-G, and 4042 A-G).<br><br>Our patient was started on a compound of Co-Q10 200 mg, creatine 1000 mg, carnitine 200 mg and folic acid 1 mg to be taken four times a day. She gradually showed significant improvement in her symptoms over a course of several months.<br><br><a href="https://jmedicalcasereports.biomedcentral.com/articles/10.1186/1752-1947-6-55#Conclusions"><strong>Conclusions</strong></a><br>This case postulates the possible role of mitochondrial disease in the pathogenesis of the symptom complex known as fibromyalgia, whereby not only is the underlying defect identified at the molecular and genomic level, but with appropriate therapy, significant symptomatic improvement is also noted.<br><br>Underlying mitochondrial disease may not be the only explanation for such a symptom complex, but the exact role of mitochondrial myopathy in the development of fibromyalgia needs to be studied further for a better understanding of the disease, and to ensure adequate and effective patient care. All patients with fibromyalgia should be evaluated for sleep disorders, endocrine disorders such as hypothyroidism and metabolic disorders before a diagnosis of primary fibromyalgia is given. The relative frequency of these medical problems in patients currently diagnosed with fibromyalgia is unclear, but would be worthy of future study.</div>]]></description>
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         <pubDate>2018-09-04 11:37:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277495413</guid>
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         <title></title>
         <author>zofiaseromines123</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277500806</link>
         <description><![CDATA[<div>Zofi</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 12:00:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277500806</guid>
      </item>
      <item>
         <title></title>
         <author>zofiaseromines123</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277500840</link>
         <description><![CDATA[<div><strong>Zofia Amina S.&nbsp; Seromines <br>Aristotle <br>1.What is mitochondrial disease? </strong><br>-Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells). In the case study i chose, Leigh syndrome is a rare progressive neurodegenerative, mitochondrial disorder of childhood with only a few<br>cases documented from India. The clinical presentation of Leigh syndrome is highly variable. However, in most<br>cases it presents as a progressive neurological disease with motor and intellectual developmental delay and signs<br>and symptoms of brain stem and/or basal ganglia involvement. Raised lactate levels in blood and/or cerebrospinal<br>fluid is noted. It is the neuroimaging, mainly the Magnetic Resonance Imaging showing characteristic symmetrical<br>necrotic lesions in the basal ganglia and/or brain stem that leads to the diagnosis. Here, we report a case of<br>7 months old female child presenting to us with status epilepticus, delayed developmental milestones and<br>regression of the achieved milestones suspected to be a case of neurodegenerative disorder, which on MRI was<br>diagnosed as Leigh syndrome. Leigh syndrome is a rare genetic neurometabolic disorder.In most cases, Leigh syndrome is inherited as an autosomal recessive trait. However, X-linked recessive and maternal inheritance, due to a mitochondrial DNA mutation, are additional modes of transmission.<br><br><strong>2. What are the symptoms of mitochondrial disease? </strong><br>-Symptoms of Leigh's disease usually progress rapidly. The earliest signs may be poor sucking ability,and the loss of head control and motor skills. These symptoms may be accompanied by loss of appetite, vomiting, irritability, continuous crying, and seizures. As the disorder progresses, symptoms may also include generalized weakness, lack of muscle tone, and episodes of lactic acidosis, which can lead to impairment of respiratory and kidney function.<br><br><strong>3. Who discovered mitochondrial diseases/disorders?</strong><br>-Researchers have discovered a genetic mutation underlying late-onset Leigh syndrome, a rare inherited metabolic disorder characterized by the degeneration of the central nervous system. The study provides vital insights into the cell biology of this neurological disorder and will lead to the development of diagnostic and predictive tests allowing for family and genetic counseling. Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria is not working?</strong><br>-For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster. When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.<br><br><strong>5. What are some examples of mitochondrial diseases?&nbsp; Can these be cured? </strong><br>-Examples of mitochondrial diseases include:<br>1. Mitochondrial myopathy.<br>Diabetes mellitus and deafness (DAD)<br>2. Leber's hereditary optic neuropathy (LHON)<br>3.Leigh syndrome, subacute sclerosing encephalopathy<br>4. Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)<br>5. Myoneurogenic gastrointestinal encephalopathy (MNGIE)<br>-The diseases can be present at birth or develop later in life. Mitochondria are tiny enclosures inside cells that produce energy from food. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br><strong><em>Conclusion</em></strong><br>The diagnosis of Leigh’s disease should be considered in<br>appropriate clinical and laboratory settings whenever<br>symmetrical hypodensities are encountered in the puta-<br>mina and midbrain on CT and further investigated with<br>MRI. Our experience suggested that bilateral symmetric<br>T2 prolongation involving multiple brainstem nuclei/<br>structures associated with basal ganglia abnormalities in<br>a child with neurological problems should prompt the<br>clinician to consider Leigh syndrome and conduct<br>further investigations such as measurement of blood<br>and/or CSF lactate, and respiratory chain enzymes activ-<br>ities. Neuro-sradiological discriminative observation is<br>very useful in guiding the clinicians for the most appro-<br>priate enzymatic and genetic study in their patients.<br>Mitochondrial disease cannot be cured completely.<br>Efforts for prevention and prenatal diagnosis are still in<br>the nascent stage. With appropriate investigations, accu-<br>rate diagnosis and prompt institution of adequate sup-<br>portive therapy, symptomatic amelioration can be<br>achieved, thereby adding life to the limited years of sur-<br>vival of these children. Further research aimed at prena-<br>tal identification of the responsible mutations and<br>prevention of the disease is warranted.<br><br>*Insights:<br>•Treatment: According to the web,&nbsp; The most common treatment for Leigh's disease is thiamine or Vitamin B1. Oral sodium bicarbonate or sodium citrate may also be prescribed to manage lactic acidosis. Researchers are currently testing dichloroacetate to establish its effectiveness in treating lactic acidosis.<br><br>Source:<br>Shrikhande et al.: A rare mitochondrial disorder:<br>Leigh sydrome - a case report. Italian Journal of Pediatrics 2010 36:62. What is mitochondrial disease?&nbsp;<br>-Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells). In the case study i chose, Leigh syndrome is a rare progressive neurodegenerative, mitochondrial disorder of childhood with only a few<br>cases documented from India. The clinical presentation of Leigh syndrome is highly variable. However, in most<br>cases it presents as a progressive neurological disease with motor and intellectual developmental delay and signs<br>and symptoms of brain stem and/or basal ganglia involvement. Raised lactate levels in blood and/or cerebrospinal<br>fluid is noted. It is the neuroimaging, mainly the Magnetic Resonance Imaging showing characteristic symmetrical<br>necrotic lesions in the basal ganglia and/or brain stem that leads to the diagnosis. Here, we report a case of<br>7 months old female child presenting to us with status epilepticus, delayed developmental milestones and<br>regression of the achieved milestones suspected to be a case of neurodegenerative disorder, which on MRI was<br>diagnosed as Leigh syndrome. Leigh syndrome is a rare genetic neurometabolic disorder.In most cases, Leigh syndrome is inherited as an autosomal recessive trait. However, X-linked recessive and maternal inheritance, due to a mitochondrial DNA mutation, are additional modes of transmission.<br><br>2. What are the symptoms of mitochondrial disease?&nbsp;<br>-Symptoms of Leigh's disease usually progress rapidly. The earliest signs may be poor sucking ability,and the loss of head control and motor skills. These symptoms may be accompanied by loss of appetite, vomiting, irritability, continuous crying, and seizures. As the disorder progresses, symptoms may also include generalized weakness, lack of muscle tone, and episodes of lactic acidosis, which can lead to impairment of respiratory and kidney function.<br><br>3. Who discovered mitochondrial diseases/disorders?<br>-Researchers have discovered a genetic mutation underlying late-onset Leigh syndrome, a rare inherited metabolic disorder characterized by the degeneration of the central nervous system. The study provides vital insights into the cell biology of this neurological disorder and will lead to the development of diagnostic and predictive tests allowing for family and genetic counseling. Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4. What happens when mitochondria is not working?<br>-For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster. When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.<br><br>5. What are some examples of mitochondrial diseases?&nbsp; Can these be cured?&nbsp;<br>-Examples of mitochondrial diseases include:<br>1. Mitochondrial myopathy.<br>Diabetes mellitus and deafness (DAD)<br>2. Leber's hereditary optic neuropathy (LHON)<br>3.Leigh syndrome, subacute sclerosing encephalopathy<br>4. Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)<br>5. Myoneurogenic gastrointestinal encephalopathy (MNGIE)<br>-The diseases can be present at birth or develop later in life. Mitochondria are tiny enclosures inside cells that produce energy from food. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Conclusion<br>The diagnosis of Leigh’s disease should be considered in<br>appropriate clinical and laboratory settings whenever<br>symmetrical hypodensities are encountered in the puta-<br>mina and midbrain on CT and further investigated with<br>MRI. Our experience suggested that bilateral symmetric<br>T2 prolongation involving multiple brainstem nuclei/<br>structures associated with basal ganglia abnormalities in<br>a child with neurological problems should prompt the<br>clinician to consider Leigh syndrome and conduct<br>further investigations such as measurement of blood<br>and/or CSF lactate, and respiratory chain enzymes activ-<br>ities. Neuro-sradiological discriminative observation is<br>very useful in guiding the clinicians for the most appro-<br>priate enzymatic and genetic study in their patients.<br>Mitochondrial disease cannot be cured completely.<br>Efforts for prevention and prenatal diagnosis are still in<br>the nascent stage. With appropriate investigations, accu-<br>rate diagnosis and prompt institution of adequate sup-<br>portive therapy, symptomatic amelioration can be<br>achieved, thereby adding life to the limited years of sur-<br>vival of these children. Further research aimed at prena-<br>tal identification of the responsible mutations and<br>prevention of the disease is warranted.<br><br>*Insights:<br><br>•Treatment: According to the web,&nbsp; The most common treatment for Leigh's disease is thiamine or Vitamin B1. Oral sodium bicarbonate or sodium citrate may also be prescribed to manage lactic acidosis. Researchers are currently testing dichloroacetate to establish its effectiveness in treating lactic acidosis.<br><br>Source:<br>Shrikhande et al.: A rare mitochondrial disorder:<br>Leigh sydrome - a case report. Italian Journal of Pediatrics 2010 36:62.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 12:00:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277500840</guid>
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      <item>
         <title>Name: Cerado, Allyza Joy B. and Valdez, Leana Jan Riz           8- Mendel (9/4/18) (8:26 PM)</title>
         <author>allyzajoycerado</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277503681</link>
         <description><![CDATA[<div>&nbsp;</div><div><strong>What is Mitochondrial Disease? </strong>Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function. Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells). When a person has Mitochondrial Disease, the mitochondria in the cells are not producing enough energy. Sometimes they are not very efficient or they do not work at all.&nbsp;</div><div>There is huge variety in the symptoms and severity of Mitochondrial Disease. It depends on how many cells are affected and where they are in the body.&nbsp;<br>&nbsp;</div><div><strong>What are the symptoms of mitochondria disease?&nbsp;</strong></div><div>&nbsp;</div><div>-Poor growth&nbsp;</div><div>-Loss of muscle coordination, muscle weakness&nbsp;</div><div>-Neurological problems, seizures&nbsp;</div><div>Autism, autistic spectrum, autistic-like features&nbsp;</div><div>-Visual and/or hearing problems&nbsp;</div><div>-Developmental delays, learning disabilities&nbsp;</div><div>-Heart, liver or kidney disease&nbsp;</div><div>-Gastrointestinal disorders, severe constipation&nbsp;</div><div>-Diabetes&nbsp;</div><div>-Increased risk of infection&nbsp;</div><div>-Thyroid and/or adrenal dysfunction&nbsp;</div><div>-Autonomic dysfunction&nbsp;</div><div>-Neuropsychological changes characterized by confusion, disorientation, and memory loss.&nbsp;<br>&nbsp;</div><div><strong>Who discovered mitochondrial disease?</strong></div><div>&nbsp;</div><div>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.&nbsp;<br><br>&nbsp;</div><div><strong>What happens when mitochondria is not working? Can it be cured?</strong>&nbsp;</div><div>&nbsp;</div><div>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;</div><div>Unfortunately, there is no cure for Mitochondrial Disease at present.</div><div>Treatment is usually supportive, relieving the symptoms that can develop, for example, treating seizures with medication.</div><div>Doctors can also try to make energy production more efficient, using co-factors and vitamins. Examples of these are Ubiquinone (Coenzyme Q10), Thiamine and Riboflavin.<br><br></div><div><strong>What are some examples of mitochondrial diseases?&nbsp; Can these be cured?&nbsp;<br></strong><br></div><ul><li><a href="https://en.wikipedia.org/wiki/Mitochondrial_myopathy">Mitochondrial myopathy</a></li><li><a href="https://en.wikipedia.org/wiki/Diabetes_mellitus_and_deafness">Diabetes mellitus and deafness</a> (DAD)</li><li><a href="https://en.wikipedia.org/wiki/Leber%27s_hereditary_optic_neuropathy">Leber's hereditary optic neuropathy</a> (LHON)</li><li><a href="https://en.wikipedia.org/wiki/Leigh_syndrome">Leigh syndrome</a></li><li><a href="https://en.wikipedia.org/wiki/Neuropathy,_ataxia,_retinitis_pigmentosa,_and_ptosis">Neuropathy, ataxia, retinitis pigmentosa, and ptosis</a> (<a href="https://en.wikipedia.org/wiki/Neuropathy,_ataxia,_and_retinitis_pigmentosa">NARP</a>)</li><li><a href="https://en.wikipedia.org/wiki/Myoneurogenic_gastrointestinal_encephalopathy">Myoneurogenic gastrointestinal encephalopathy</a> (MNGIE)</li><li><a href="https://en.wikipedia.org/wiki/Myoclonic_Epilepsy_with_Ragged_Red_Fibers">Myoclonic Epilepsy with Ragged Red Fibers</a> (MERRF)</li></ul><div>&nbsp;</div><ul><li><a href="https://en.wikipedia.org/wiki/MELAS">Mitochondrial myopathy, encephalomyopathy, lactic acidosis, stroke-like symptoms</a> (MELAS)</li><li><a href="https://en.wikipedia.org/w/index.php?title=MtDNA_depletion&amp;action=edit&amp;redlink=1">mtDNA depletion</a></li><li><a href="https://en.wikipedia.org/wiki/Mitochondrial_neurogastrointestinal_encephalomyopathy">mitochondrial neurogastrointestinal encephalomyopathy</a> (<a href="https://en.wikipedia.org/wiki/MNGIE">MNGIE</a>)</li></ul><div>Unfortunately, there is no cure for Mitochondrial Disease at present.&nbsp;</div><div><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 12:10:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277503681</guid>
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         <title>Jannah H. Bantes VIII- Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277505005</link>
         <description><![CDATA[<div><strong>Questions and answers:<br>1. What is Mitochondrial Disease?<br>-</strong> Mitochondrial diseases are chronic (long-term), genetic, often inherited disorders that occur when mitochondria fail to produce enough energy for the body to function properly. (Inherited means the disorder was passed on from parents to children.) Mitochondrial diseases can be present at <br>birth, but can also occur at any age.<br>Mitochondrial diseases can affect almost any part of the body, including the cells of the brain, nerves, muscles, kidneys, heart, liver, eyes, ears or pancreas. <br><strong>2. What are the symptoms of Mitochondrial Diseases?<br>-</strong>&nbsp; Symptoms of mitochondrial diseases can include:<br>&nbsp; &nbsp; &nbsp;-Poor growth<br>&nbsp; &nbsp; &nbsp;-Muscle weakness, <a href="https://my.clevelandclinic.org/health/symptoms/17669-muscle-pain">muscle pain</a>, low muscle tone, exercise intolerance<br>&nbsp; &nbsp; &nbsp;-Vision and/or hearing problems<br>&nbsp; &nbsp; &nbsp;-Learning disabilities, delays in development, mental retardation<br>&nbsp; &nbsp; &nbsp;-Heart, liver or <a href="https://my.clevelandclinic.org/health/diseases/15096-kidney-disease-chronic-kidney-disease">kidney</a> diseases<br>&nbsp; &nbsp; &nbsp;-Diabetes<br><strong>3. Who discovered Mitochondrial Diseases or disorders?<br>-</strong> Mitochondrial medicine is a new and rapidly developing medical subspecialty. Many specialists are involved in researching mitochondrial diseases. Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus. <br><strong>4. What happens when Mitochondria is not working?<br>-</strong> For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster. <br><strong>5. What are some examples of Mitochondrial Diseases? Can these be cured?<br></strong>&nbsp;<strong>Examples of mitochondrial diseases include:</strong></div><ul><li>Mitochondrial myopathy</li><li>Diabetes mellitus and deafness (DAD)</li><li>Leber's hereditary optic neuropathy (LHON)</li><li>Leigh syndrome, subacute sclerosing encephalopathy</li><li>Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)</li><li>Myoneurogenic gastrointestinal encephalopathy (MNGIE)<br>- The <strong>diseases</strong> can be present at birth or develop later in life. Treatments that can ease some of the symptoms or slow the progression of <strong>mitochondrial diseases</strong> do exist, but as yet, none can <strong>cure</strong> them.<br><br><strong>CASE STUDY<br>Finding A Cure for Mitochondrial Diseases</strong><br>Selected Case Studies of those with Mitochondrial Diseases from<strong> </strong><a href="http://www.umdf.org/pdf/MITOCYTO.PDF"><strong>a Paper by Dr. Cohen</strong></a><strong><br></strong>&nbsp;1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.</li></ul>]]></description>
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         <pubDate>2018-09-04 12:14:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277505005</guid>
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      <item>
         <title>Caryl Jean Urdaneta - 8 Arist</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277509416</link>
         <description><![CDATA[<div>Questions;Answers<br>1. What is mitochondrial disease?<br>- </div>]]></description>
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         <pubDate>2018-09-04 12:26:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277509416</guid>
      </item>
      <item>
         <title>Taria</title>
         <author>keane56k</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277511988</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 12:33:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277511988</guid>
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      <item>
         <title>Christian Louie N. Evangelista    8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277514435</link>
         <description><![CDATA[<div>1. What is Mitochondrial Disease?<br>Answer:<br>Mitochondrial disease is an inherited chronic illness that can be present at birth or develop later: in life. It causes debilitating physical, developmental, and cognitive disabilities .<br>2. What are the symptoms of mitochondrial disease/disorder?<br>Answer:<br>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br><br>Poor growth<br>Loss of muscle coordination, muscle weakness<br>Neurological problems, seizures<br>Autism, autistic spectrum, autistic-like features<br>Visual and/or hearing problems<br>Developmental delays, learning disabilities<br>Heart, liver or kidney disease<br>Gastrointestinal disorders, severe constipation<br>Diabetes<br>Increased risk of infection<br>Thyroid and/or adrenal dysfunction<br>Autonomic dysfunction<br>Neuropsychological changes characterized by confusion, disorientation, and memory loss. <br>3.Who discovered mitochondrial disease?<br>Answer:<br>discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchersdiscovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4. What happen when mitochondrial are not working?<br>Answer<br>mitochondria fail to produce enough energy for the body to function properly.<br>5. What are some examples of mitochondrial disease? Can these be cure?<br>Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy), Autosomal Dominant Optic Atrophy(ADOA), Fatty Acid Oxidation Disorders/Betaoxidation Defects/Fatty Acid Transport and Mitochondrial Oxidation Disorders, Leigh Disease, Lactic Acidosis, Kearns-Sayre syndrome(KSS), Chronic Progressive External Opthalmoplegia(CPEO), Co-Enzymes Q10 Deficiency, Creatine Deficiency Syndrome, Carnitine Deficiency and Alpers Disease.<br><strong>Case study of Mitochondrial Disease<br></strong> 1982 DF was born at term and had no problems until she was 4 months old when it was found she was anemic during an evaluation for fever. She had multiple courses of antibiotics for UTIs and otitis media. She developed a picture of overwhelming sepsis and her lactic acid was elevated at 12 mM. She subsequently went on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome.<br> </div>]]></description>
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         <pubDate>2018-09-04 12:40:01 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277514435</guid>
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      <item>
         <title></title>
         <author>bcmiase</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277519626</link>
         <description><![CDATA[<div><strong>1. What is Mitochondrial disease</strong><br>-Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.<br><strong>2. What are the symptoms of mitochondrial diseases</strong></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss.</li></ul><div>3. Who discivered Mitochondrial disease?<br>It has been discivered since 1962, the furst patient was diagnosed witha mitochondrial disored.</div><div><br></div>]]></description>
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         <pubDate>2018-09-04 12:52:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277519626</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277522069</link>
         <description><![CDATA[The diseases can be present at birth or develop later in life. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.]]></description>
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         <pubDate>2018-09-04 12:57:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277522069</guid>
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      <item>
         <title>Mendel (9/4/18) </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277524566</link>
         <description><![CDATA[<div><strong>Krystle Ann Z. Paguray<br>Angelle Kate Y. Brocoy<br><br>I. Questions:<br></strong>1.)&nbsp;<strong>What is a mitochondrial disease?<br></strong>Mitochondrial Diseases are disorders caused by the failures of mitochondrial functions. <br>2.) <strong>What are the symptoms of mitochondrial disease?<br></strong>&nbsp;• Poor Growth<strong><br> </strong>• Loss of muscle coordination<br>&nbsp;• Muscle Weakness<br>&nbsp;• Visual/Hearing Problems<br>&nbsp;• Develomental Delay<br>&nbsp;• Mental Retardation<br>&nbsp;• Heart, Liver, or Kidney Disease<br>&nbsp;• Gastro Intestinal Disorders<br>&nbsp;• Severe Constipation<br>&nbsp;• Respiratory Disorders<br>&nbsp;• Diabetes<br>&nbsp;• Increase risk of infection<br>&nbsp;• Neuroogical Problems<br>&nbsp;• Seizures<br>&nbsp;• Thyroid Dysfunction<br>&nbsp;• Dementia<br>3.) <strong>Who discovered the Mitochondrial Diseases or Disorders?<br></strong>4.) <strong>What happens when Mitochondria are not working?<br>" </strong>If your Mitochondria are not working properly then you are less able to convert your food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may became weaker and get tired faster." <strong>(General Information About Mitochondrial Disease)<br></strong>5.) <strong>What are some examples of Mitochondrial Diseases?<br>•MITOCHONDRIAL DNA DEPLETION SYNDROME<br></strong>(Currently no Curative Treatment)<br>•<strong>MITOCHONDRIAL MYOPATHY<br></strong>(Not Curable)<br>• <strong>MELAS SYNDROME<br></strong>( No cure; medical care is largely supportive )<br>• <strong>MERRF SYNDROME<br>(</strong>No cure; Various Medications and Therapies are helpful in managing symptoms)<strong><br></strong>&nbsp;<br>II. Case Study<strong><br>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.<br><br>III. Insight<br>JAUNDICE IS A DISEASE BY WHICH A PERSON BECOMES YELLOWISH. THE SYMPTOMS ARE DIARRHEA AND THE YELLOWISH COLOR OF RHE SKIN THE ACTIONS TO CURE IT IS BY TAKING UP GLUCOSE AND PROPHYLACTIC ANTIBIOTICS THE RESULT IS THAT THE JAUNDICE DISEASE WILL NOT BE OCCURING ANYMORE IN YOUR BODY.<br><br>(9:54 PM)<br><br></strong><br></div>]]></description>
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         <pubDate>2018-09-04 13:02:19 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277524566</guid>
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         <title>Kenny Macjewr T. Sulilap 8-Aristotle </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277527779</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:09:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277527779</guid>
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         <title>Miase, Bea Clarisse A.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277528027</link>
         <description><![CDATA[<div><strong>8-Aristotle<br>1. what is mitochondrial disease?<br></strong><br>Mitochondrial disease are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy from the cell.<br><br><strong>2. What are the symptoms in mitochondrial diseases?</strong><br>&nbsp;</div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss.&nbsp;</li></ul><div><strong>3. Who discovered mitochondrial disease/disorder?<br></strong><br>&nbsp;Much of what we know about these <strong>diseases</strong> has been <strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus. <br><br><strong>4. What happens when mitochondria is not working?<br></strong><br>&nbsp;For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a <strong>problem</strong> and they may become weaker and get tired faster, you cant enjoy life to its fullest because of your disease.<br><br><strong>5.What are some examples of mitochondrial disease? Can this be cured?<br></strong><br><strong>&nbsp;-Alpers' disease</strong>- no cure;&nbsp; no way to slow its progression <br>- <strong>Autosomal Dominant Optic Atrophy (ADOA)</strong> <br>- <strong>Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy)</strong> - Treatment of Barth syndrome is generally symptomatic, requiring the coordinated efforts of a team of medical professionals which includes a pediatrician, pediatric cardiologist,&nbsp; hematologist, specialist in the treatment of bacterial infections, physical therapist, occupational therapist, and/or other healthcare professionals. <br>- <strong>Systemic Primary Carnitine Deficiency</strong> - treatment includes carnitine supplementation. <br>- <strong>Long Chain Fatty Acid Transport Deficiency</strong> -&nbsp;</div><ul><li>Treatment includes liver transplantation.</li></ul><div><strong>&nbsp;-</strong> <strong>Carnitine Palmitoyl Transferase I (CPT-I) Deficiency</strong> - Treatment includes avoidance of fasting, frequent feeding, high-dose glucose during acute episodes and replacement of long-chain dietary fat with medium-chain fats. <br><strong>-other more<br><br>Case Study<br></strong>&nbsp;1982 DF was born at term and had no problems until she was 4 months old when it was found she was anemic during an evaluation for fever. She had multiple courses of antibiotics for UTIs and otitis media. She developed a picture of overwhelming sepsis and her lactic acid was elevated at 12 mM. She subsequently went on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome.<br><br><strong>Another case study:</strong><br>&nbsp; 1984 CP was born at term weighing 1900 gm after an uncomplicated pregnancy, labor and delivery. He had mild dysmorphic features, along with the fact he was SGA, with rocker bottom feet, small upturned nose and epicanthal folds. He was noted to be hypotonic at birth and at 19 hours of life had his first seizure. His initial evaluation showed a metabolic acidosis with a lactic acid of 6 mM, with a lactate to pyruvate ratio of 10:1. Seizures continued and he was transferred to our hospital. Over the next several days his acidosis worsened, and fluids of D10+ 1/2 Normal bicarbonate were used to attempt to correct his acidosis. A urine organic acid showed enormous lactate excretion. A tentative diagnosis of pyruvate dehydrogenase deficiency was made, and the fluids were changed to a D2.5 solution with amino acids and intralipids. Within hours, the acidosis reversed and the child was extubated 2 days later. He was treated with high dose B1 and lipoic acid (stimulators of PDH), as well as with polycitra. He was given a regular infant formula with MCT oil and the lactic acid remained in the 4-6 mM range. He died in his sleep at 8 months of life. The E1-alpha subunit of PDH was absent. This is an X-linked disease.<br> <br><strong>Insights:</strong><br><em>Mitochondrial disease/disorder is a really harmful and dangerous disease. Although some can be cured, there's also high risks that will be taken in which it talks about the patient's life on the line.</em></div>]]></description>
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         <pubDate>2018-09-04 13:09:48 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277528027</guid>
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         <title>Deniel Dave C. Natividad              8- Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277529273</link>
         <description><![CDATA[<div>Questions<br>&nbsp;<strong>1. What is Mitochondrial Disease?<br></strong>- Mitochondria are responsible for creating more than 90% of the energy needed by the body to sustain life and support organ function. When they fail, less and less energy is generated within the cell. Cell injury and even cell death follow. If this process is repeated throughout the body, whole organ systems begin to fail.<br>2. <strong>What are the symptoms of mitochondrial disease?<br></strong>&nbsp;- Symptoms might include:</div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Mental retardation</li><li>Heart, liver, or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Respiratory disorders</li><li>Diabetes</li><li>Increased risk of infection</li><li>Neurological problems, seizures</li><li>Thyroid dysfunction</li><li>Dementia (mental disorder characterized by confusion, disorientation, and memory loss)&nbsp;</li></ul><div><strong>3. Who discovered mitochondrial disease or disorders?<br>- </strong>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria are not working?<br>- </strong>If the mitochondria is removed from the cell, then the cell will lose its capacity to further perform any activities. Since energy leads to work done, if there is no energy, there will be no work done. The cell will die.<br><br><strong>5. What are some examples of mitochondrial diseases? Can these be cured?<br>- </strong>MELAS syndrome- No specific treatment is available for MELAS syndrome. Anti-convulsant drugs are used to help prevent and control seizures associated with MELAS syndrome. Valproic acid should not be used as an anticonvulsant. Cochlear implants have been used to treat sensorineural deafness.<br>- MERRF syndrome- Anti-convulsant drugs are used to help prevent and control seizures associated with MERRF syndrome. Levetiracetam has been effective in controlling myoclonus in a small number of patients. Therapies are sometimes used to increase energy production by the mitochondria and slow the effects of the condition.<br>- Leigh syndrome- The most common treatment for Leigh's disease is thiamine or Vitamin B1. Oral sodium bicarbonate or sodium citrate may also be prescribed to manage lactic acidosis. Researchers are currently testing dichloroacetate to establish its effectiveness in treating lactic acidosis.<br>- Leber's hereditary optic neuropathy- Leber hereditary optic neuropathy is an inherited mitochondrial disease with devastating visual consequences for those affected. Current treatment options are limited to supportive measures and therapies with questionable benefits. Idebenone can be considered for the patient early in the course of the disease.<br>- &nbsp;Kearns–Sayre syndrome- No disease-modifying therapy is available for Kearns-Sayre syndrome (KSS). Management is supportive vigilance for detection of associated problems. In the future, potential treatment in patients with Kearns-Sayre syndrome may attempt to inhibit mutant mtDNA replication or encourage replication of wild-type mtDNA<br><br><strong>Case Study (MERRF Syndrome):<br><br></strong>A 25-year-old male presented with paroxysmal left upper limb tics and weakness that had been ongoing for two years. The involuntary limb tics exhibited a sudden onset and lasted for seconds, but were not accompanied by consciousness disturbance. The patient had approximately 10 attacks per day, which were accompanied by limb weakness. A magnetic resonance imaging (MRI) scan was performed initially and was found to be normal. The patient had received irregular diazepam administration from the onset of the disease; however, the symptoms became increasingly more serious. The patient was prescribed 600 mg per day valproate sodium on admission to hospital to control the seizures, but experienced one or two attacks per month subsequent to the administration of valproate sodium. The past medical history of the patient was unremarkable. On examination, the patient was alert and his pupils adjusted to light. Neurological examination revealed intact cranial nerves, but decreased deep tendon reflexes and a decreased sensation of touch, pain and vibration. The gait of the patient was broad and he was unable to walk in a straight line. Full strength was observed in all the muscle groups. The results of the Romberg, heel-knee-shin and finger-to-nose tests were normal. An electroencephalogram (EEG) revealed diffuse spikes and slow waves, predominantly in the frontal and temporal lobes (Fig. 1). A further MRI scan was performed and revealed increased signal density on T2-weighted imaging and decreased signal density on T1-weighted imaging in the right temporal occipital cortical lesions. Local cortical atrophy was also observed in the left temporal-occipital cortex. In addition, the lactic acid concentration (5.2 mmol/l) had markedly increased. The results of the carotid ultrasound and electromyography were normal. A biopsy of the biceps muscle demonstrated a variation in fiber size and the presence of ragged-red fibers (Fig. 2). In addition to the prescribed 600 mg per day valproate sodium, the patient was administered 10 mg per day coenzyme Q10 for approximately 2 years. Two years later his symptoms relieved and an EEG showed less spikes and slow waves than it had previously shown.<br><br></div>]]></description>
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         <pubDate>2018-09-04 13:12:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277529273</guid>
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      <item>
         <title>Sarmiento, Nadine A.              8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277529416</link>
         <description><![CDATA[<div><strong>Questions:</strong><br>1.) What is Mitochondrial Disease?<br>2.) What are its symptoms?<br>3.) Who discovered mitochondrial diseases or disorders?<br>4.) What happens when mitochondria is not working?<br>5.)What are some examples of mitochondrial diseases? Can these be cured?<br><br>Answers:<br>1.) Mitochondrial diseases are caused by the dysfunction of the mitochondria.<br>2.) Mitochondrial diseases affect the whole body, causing diseases everywhere in the body. This causes poor growth, muscle weaknesses, visual and hearing problems, experiencing developmental delays, learning disabilities, and lots of other disabilities and diseases.<br>3.) This disease was discovered since 1940, but in 1963, researchers discovered that mitochondria have their own DNA or "blueprint".<br>4.) As we all have learned, the food we eat is turned into ATP by the mitochondria, so therefore, if the mitochondria is not functioning well, there will be less ATP or energy produced, making it easier for you to get tired and weak.<br>5.) * Mitochondrial Myopathy<br>&nbsp; &nbsp; &nbsp;-causes prominent muscle problems<br>&nbsp; &nbsp; &nbsp; * Leigh syndrome<br>&nbsp; &nbsp; &nbsp;-a&nbsp; severe neurological disorder that usually becomes apparent in the first year of life<br>&nbsp; &nbsp; &nbsp; &nbsp;-characterized by progressive loss of mental and movement abilities<br>&nbsp; &nbsp; &nbsp;-can result to death within 2 to 3 years, usuallydue to respiratory failure<br>&nbsp; &nbsp; &nbsp; &nbsp;* Mitochondrial neurogastrointestinal-encephalopath disease<br>&nbsp; &nbsp; &nbsp;- affects several parts of the body, like the digestive and nervous system, symptoms often begin by age 20<br>* All of these diseases can be cured depending on the severeness of the diseases.<br><strong>Case Study:</strong><br><br> Of 71 index cases with histologically defined mitochondrial myopathy, 13 (18%) had relatives who were definitely affected with a similar disorder. Eight familial cases from four families were confined to a single generation. In five families maternal transmission to offspring occurred. There were no instances of paternal transmission, but one patient had an affected cousin in the paternal line. No consistent clinical syndrome or pattern of inheritance emerged for any identified defect of the mitochondrial respiratory chain, localised biochemically in 41 cases. Overall, the recurrence rate was 3% for sibs and 5.5% for offspring of index cases. Review of published reports of familial cases of mitochondrial myopathy suggests that the ratio of maternal to paternal transmission is about 9:1. We conclude that these disorders may be caused by mutations of either nuclear or mitochondrial genes.&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:12:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277529416</guid>
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      <item>
         <title>Bernadette Sue B. Berame     8- Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277535090</link>
         <description><![CDATA[<div><strong>Questions:<br>1.) What is mitochondrial disease?<br></strong>- Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.  Mitochondrial disease, or 'mito', is the term given to a group of medical disorders caused by mutations in mitochondria, the tiny organelles that are present in every cell in our bodies and which generate about 90% of the energy we need to live. Cells cannot function properly without healthy mitochondria, so when they fail the consequences can be serious and wide-ranging. <br><br><strong>2.) What are the symptoms of mitochondrial disease?<br></strong>- The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br><br></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss. </li></ul><div><br><strong>3.)  Who discovered mitochondrial disease or disorder?<br></strong>- In 1962, the first patient was diagnosedwith a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus. <br><br><strong>4.) What happens when mitochondria is not working?<br></strong>- For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster. <br><br><strong>5.) What are some examples of mitochondrial disease. Can this be cured?<br></strong>-  <strong> Leber’s Hereditary Optic Neuropathy</strong>- In this mitochondrial disease, a person can lose vision in both eyes. <br> </div><div> </div><div><strong>Neurogenic weakness, Ataxia and Retinitis Pigmentosa (NARP)-</strong>NARP stands for neurogenic weakness, ataxia and retinitis pigmentosa. Neurogenic weakness means the muscles are weak because of abnormalities in the nerves that send signals to the muscles. <br><br> <strong>Myoclonic Epilepsy with Ragged Red Fibers (MERRF)-</strong> people with this mitochondrial disorder may have seizures, ataxia and dementia. They may also have abnormalities in their muscles because the mitochondria are accumulating within them. <br><br> <strong>Mitochondrial Encephalomyopathy, lactic acidosis and stroke (MELAS)-</strong> People with this disorder experience several episodes that resemble strokes because of problems primarily located in the back of their brain. Other symptoms may include seizures, reccurring headaches, nausea and vomiting. <br><br> </div><div><strong>Kearns-Sayre Syndrome-</strong> In this mitochondrial disorder, people usually have paralysis in some of the muscles in their eyes and problems in their retinas before they are 20 years old. Some may also develop heart problems, ataxia, diabetes mellitus and hearing loss. <br><br><br><strong> CASE STUDY:<br></strong> </div><h1><strong>Case Report of a New Mitochondrial Disease Linked to Desmin Gene </strong></h1><div><br> </div><div>A study recently published in the journal Frontiers in Genetics reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA” and was conducted by researchers at The Children’s Hospital of Philadelphiaand the Thomas Jefferson University Hospital Philadelphia.<br><br>The study reports the case of a 45-year-old man who presented to The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.<br><br>The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria. <br><br></div><div><strong><br></strong><br></div><div><br><br></div><div><br></div>]]></description>
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         <pubDate>2018-09-04 13:22:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277535090</guid>
      </item>
      <item>
         <title>Alberto, </title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277535222</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:23:01 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277535222</guid>
      </item>
      <item>
         <title>Lata,   Ronan Raphael  C.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277536988</link>
         <description><![CDATA[<div>Gr</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:26:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277536988</guid>
      </item>
      <item>
         <title>Kenny Macjewr T. Sulilap 8-Aristotle </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277537496</link>
         <description><![CDATA[<div><br>1. What is mitochondrial disease?&nbsp;<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br>2.What are the symptoms of mitochondrial disease?&nbsp;<br>Poor growth<br>Loss of muscle coordination, muscle weakness<br>Visual and/or hearing problems<br>Developmental delays, learning disabilities<br>Mental retardation<br>Heart, liver, or kidney disease<br>Gastrointestinal disorders, severe constipation<br>Respiratory disorders<br>Diabetes<br>Increased risk of infection<br>Neurological problems, seizures<br>Thyroid dysfunction<br>Dementia (mental disorder characterized by confusion, disorientation, and memory loss)<br>3.Who discovered mitochondrial disease?&nbsp;<br>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4.What happens when mitochondria is not working?&nbsp;<br>If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br>5. What are some examples of mitochondrial disease? Can this be cured?<br>A number of specific mitochondrial disorders have been associated with Complex I deficiency including: Leber's hereditary optic neuropathy (LHON), MELAS, MERRF, and Leigh Syndrome (LS). ... Myopathy (muscle disease) – starting in childhood or adulthood, and characterized by weakness or exercise intolerance.<br>Mitochondrial diseases can't be cured but can be treated to help reduce symptoms or slow the decline in health.<br>6. Case study about mitochondrial disease.<br>4. S.K. 57 year old man came from India for evaluation of his cardiac conduction defect. His family history is significant for his mother dying at a young age of a cardiomyopathy, and that his brothers have similar problems to his. He was the president of a successful business in India. As a young man he was diagnosed with calcifying pancreatitis, and had been on replacement digestive enzymes for years. He has had numerous admissions for non-surgical bowel obstruction and has had several exploratory laporatomies, the etiology was never determined. Over the last 15 years he lost 75 pounds, and developed aching in his limbs. In January 1996 he was admitted for a stroke, and a cardiac evaluation determined he was in A-fib. He was placed on Coumadin. He was readmitted for a stroke several months later, and was again found to be in A-fib. His neurologist and cardiologist referred him to CCF Cardiology for placement of a pacemaker. In October 1996, after a 24 hour journey, he visited our cardiology department, and was told that a pacemaker was not indicated. While resting in the waiting room, trying to gather the energy to walk back to the hotel, he collapsed. He spent the next two weeks in a stupor in the NICU, and again, another stroke was detected, along with his A-fib. The patient was found to have an elevated lactate, ammonia and CK (MM). Polarography revealed decreased oxidation of substrates that donate reducing equivulants to complex I and beta oxidation. An evaluation determined that he had a deficiency in carnitine palmitoyl transferase II activity. Treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates, along with levo-carnitine, CoQ10 and other vitamins. He has had no further events since his hospitalization in October 1996. Comments: In order for the mitochondria to burn fats, the free fatty acid must first enter the mitochondrial inner membrane. CPT I catalyzes the conversion of the activated free FA (acyl CoA) + carnitine to the acyl-carnitine. A carnitine translocase exchanges the acylcarnitine across the inner membrane for a free carnitine molecule. CPT II catalyzes the conversion of the acyl-carnitine to the acyl-CoA and free carnitine. The acyl-CoA can then enter the beta-oxidation spiral. CPT II deficiency usually results in exercise intolerance, muscle cramping and fatigue in young adults, but is also known to cause an early cardiomyopathy.<br>http://www.umdf.org/pdf/MITOCYTO.PDF</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:26:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277537496</guid>
      </item>
      <item>
         <title>Lata,  Ronan Raphael  C.   8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277539159</link>
         <description><![CDATA[<div>Questions:<br><br>1.) What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br><br>2.) What are the symptoms of mitochondrial diseases? <br>*Poor growth<br>*Muscle weakness, muscle pain, low muscle tone, exercise intolerance<br>*Vision and/or hearing problems<br>*Learning disabilities, delays in development, *mental retardation<br>*Autism, autism-like features<br>*Heart, liver or kidney diseases<br>*Gastrointestinal disorders, swallowing difficulties, diarrhea or constipation, unexplained vomiting, cramping, reflux<br>*Diabetes<br>*Increased risk of infection<br>*Neurological problems, seizures, migraines, strokes<br>*Movement disorders<br>*Thyroid problems<br>*Respiratory (breathing) problems<br>*Lactic acidosis (a buildup of lactate)<br>*Dementia<br><br>3.) Who discovered mitochondrial diseases?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4.) What happens when mitochondria is bot working? <br>You are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br>5.) What are examples of mitochondrial diseases?  Can this be cured?<br>*Neuropathy, ataxia, and retinitis pigmentosa<br>*MERRF syndrome<br>*Mitochondrial myopathy<br>*Leigh syndrome<br>The diseases can be present at birth or develop later in life. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Insights: From the article that I read,I could say that mitochondria is a very important part not just for the cell but also for our body. It may be small,but it has a large impact to our body. Overall,  mitochondria is important especially in energy production. <br><br>Passed in 9:35 P. <br><br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/308980394/9ec40c595d1cb7d0fc028d091cc73c66/91045_Mito_Case_Study.pdf" />
         <pubDate>2018-09-04 13:29:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277539159</guid>
      </item>
      <item>
         <title>Mitochondrial myopathy.Diabetes mellitus and deafness (DAD) ...Leber&#39;s hereditary optic neuropathy (LHON) ...Leigh syndrome, subacute sclerosing encephalopathy. ...Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP) ...Myoneurogenic gastrointestinal encephalopathy (MNGIE)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277540180</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:31:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277540180</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277542754</link>
         <description><![CDATA[1982 DF was born at term and had no problems until she was 4 months old when it was found she was anemic during an evaluation for fever. She had multiple courses of antibiotics for UTIs and otitis media. She developed a picture of overwhelming sepsis and her lactic acid was elevated at 12 mM. She subsequently went on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome.
]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:36:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277542754</guid>
      </item>
      <item>
         <title>Keny M</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277545494</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:41:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277545494</guid>
      </item>
      <item>
         <title>Kenny Macjewr T. Sulilap 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277546228</link>
         <description><![CDATA[<div><br>1. What is mitochondrial disease?&nbsp;<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br>2.What are the symptoms of mitochondrial disease?&nbsp;<br>Poor growth<br>Loss of muscle coordination, muscle weakness<br>Visual and/or hearing problems<br>Developmental delays, learning disabilities<br>Mental retardation<br>Heart, liver, or kidney disease<br>Gastrointestinal disorders, severe constipation<br>Respiratory disorders<br>Diabetes<br>Increased risk of infection<br>Neurological problems, seizures<br>Thyroid dysfunction<br>Dementia (mental disorder characterized by confusion, disorientation, and memory loss)<br>3.Who discovered mitochondrial disease?&nbsp;<br>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4.What happens when mitochondria is not working?&nbsp;<br>If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br>5. What are some examples of mitochondrial disease? Can this be cured?<br>A number of specific mitochondrial disorders have been associated with Complex I deficiency including: Leber's hereditary optic neuropathy (LHON), MELAS, MERRF, and Leigh Syndrome (LS). ... Myopathy (muscle disease) – starting in childhood or adulthood, and characterized by weakness or exercise intolerance.<br>Mitochondrial diseases can't be cured but can be treated to help reduce symptoms or slow the decline in health.<br>6. Case study about mitochondrial disease.<br>4. S.K. 57 year old man came from India for evaluation of his cardiac conduction defect. His family history is significant for his mother dying at a young age of a cardiomyopathy, and that his brothers have similar problems to his. He was the president of a successful business in India. As a young man he was diagnosed with calcifying pancreatitis, and had been on replacement digestive enzymes for years. He has had numerous admissions for non-surgical bowel obstruction and has had several exploratory laporatomies, the etiology was never determined. Over the last 15 years he lost 75 pounds, and developed aching in his limbs. In January 1996 he was admitted for a stroke, and a cardiac evaluation determined he was in A-fib. He was placed on Coumadin. He was readmitted for a stroke several months later, and was again found to be in A-fib. His neurologist and cardiologist referred him to CCF Cardiology for placement of a pacemaker. In October 1996, after a 24 hour journey, he visited our cardiology department, and was told that a pacemaker was not indicated. While resting in the waiting room, trying to gather the energy to walk back to the hotel, he collapsed. He spent the next two weeks in a stupor in the NICU, and again, another stroke was detected, along with his A-fib. The patient was found to have an elevated lactate, ammonia and CK (MM). Polarography revealed decreased oxidation of substrates that donate reducing equivulants to complex I and beta oxidation. An evaluation determined that he had a deficiency in carnitine palmitoyl transferase II activity. Treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates, along with levo-carnitine, CoQ10 and other vitamins. He has had no further events since his hospitalization in October 1996. Comments: In order for the mitochondria to burn fats, the free fatty acid must first enter the mitochondrial inner membrane. CPT I catalyzes the conversion of the activated free FA (acyl CoA) + carnitine to the acyl-carnitine. A carnitine translocase exchanges the acylcarnitine across the inner membrane for a free carnitine molecule. CPT II catalyzes the conversion of the acyl-carnitine to the acyl-CoA and free carnitine. The acyl-CoA can then enter the beta-oxidation spiral. CPT II deficiency usually results in exercise intolerance, muscle cramping and fatigue in young adults, but is also known to cause an early cardiomyopathy.<br>http://www.umdf.org/pdf/MITOCYTO.PDF</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:43:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277546228</guid>
      </item>
      <item>
         <title>  Lata,  Ronan Raphael C.     8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277546369</link>
         <description><![CDATA[<div>Questions:<br><br>1.) What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br><br>2.) What are the symptoms of mitochondrial diseases? <br>*Poor growth<br>*Muscle weakness, muscle pain, low muscle tone, exercise intolerance<br>*Vision and/or hearing problems<br>*Learning disabilities, delays in development, *mental retardation<br>*Autism, autism-like features<br>*Heart, liver or kidney diseases<br>*Gastrointestinal disorders, swallowing difficulties, diarrhea or constipation, unexplained vomiting, cramping, reflux<br>*Diabetes<br>*Increased risk of infection<br>*Neurological problems, seizures, migraines, strokes<br>*Movement disorders<br>*Thyroid problems<br>*Respiratory (breathing) problems<br>*Lactic acidosis (a buildup of lactate)<br>*Dementia<br><br>3.) Who discovered mitochondrial diseases?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4.) What happens when mitochondria is bot working? <br>You are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br>5.) What are examples of mitochondrial diseases?  Can this be cured?<br>*Neuropathy, ataxia, and retinitis pigmentosa<br>*MERRF syndrome<br>*Mitochondrial myopathy<br>*Leigh syndrome<br>The diseases can be present at birth or develop later in life. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Insights: From the article that I read,I could say that mitochondria is a very important part not just for the cell but also for our body. It may be small,but it has a large impact to our body. Overall,  mitochondria is important especially in energy production. <br><br>Passed 9:35 P. M. <br><br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/308988218/5b9a4b5bc32cd913674540603d9435e0/91045_Mito_Case_Study.pdf" />
         <pubDate>2018-09-04 13:43:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277546369</guid>
      </item>
      <item>
         <title>Zyrreg Zhyller A. Cabiles 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277547248</link>
         <description><![CDATA[<div>1. What is a mitochondrial disease?<br><br>• A mitochondrial disease is a group of disorders caused by dysfunctional mitochondria caused by mutations in the mitochondrial DNA (mtDNA) or in the nuclear DNA (nDNA) whose gene products are imported into the mitochondria (mitochondrial proteins) as well as acquired mitochondrial conditions.&nbsp;<br><br>2. What are the symptoms of mitochondrial diseases?<br><br>• Some symptoms of mitochondrial diseases are poor growth, loss of muscle coordination, muscle weakness, visual problems, hearing problems, learning disabilities, heart disease, liver disease, kidney disease, gastrointestinal disorders, respiratory disorders, neurological problems, autonomic dysfunction and dementia.<br><br>3. Who discovered mitochondrial diseases?&nbsp;<br><br>• Knowledge about these diseases are not sufficient and we only know that it has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. Later on,&nbsp; they discovered that mitochindria has DNA on itself.&nbsp;<br><br>4. What happens when mitochondria is not working?<br><br>• If mitochondria are not working properly then the body is less able to convert food into ATP. The person may feel from strong, weak to weaker and weaker. The person may die depending on the disease.&nbsp;<br><br>5. What are the examples of mitochondrial diseases?&nbsp;<br><br>•&nbsp; Currently, there is mitochondrial myopathy&nbsp;<br>diabetes mellitus and deafness (DAD), leber'shereditary optic neuropathy (LHON), leigh syndrome, subacute sclerosing encephalopathy, neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP) , and myoneurogenic gastrointestinal encephalopathy (MNGIE). But there is no cure in all of these diseases but only treatments like vitamins.<br><br>Case Study<br><br>MITOCHONDRIAL DYSFUNCTION AND APOPTOSIS IN MYOPARHIC MICE WITH COLLAGEN VI DEFICIENCY&nbsp;<br><br>•William A Irwin1,2,6, Natascha Bergamin1,6, Patrizia Sabatelli3, Carlo Reggiani4, Aram Megighian4, Luciano Merlini5,<br>Paola Braghetta1, Marta Columbaro5, Dino Volpin1, Giorgio M Bressan1, Paolo Bernardi2 &amp; Paolo Bonaldo1<br><br>Collagen VI is an extracellular matrix protein that forms amicrofilamentous network in skeletal muscles and otherorgans1–3. Inherited mutations in genes encoding collagen VI in<br>humans cause two muscle diseases, Bethlem myopathy andUllrich congenital muscular dystrophy4,5. We previouslygenerated collagen VI–deficient (Col6a1–/–) mice and showedthat they have a muscle phenotype that strongly resemblesBethlem myopathy6. The pathophysiological defects andmechanisms leading to the myopathic disorder were not known.Here we show that Col6a1–/– muscles have a loss of contractilestrength associated with ultrastructural alterations ofsarcoplasmic reticulum (SR) and mitochondria and spontaneousapoptosis. We found a latent mitochondrial dysfunction inmyofibers of Col6a1–/– mice on incubation with the selectiveF1FO-ATPase inhibitor oligomycin, which caused mitochondrialdepolarization, Ca2+ deregulation and increased apoptosis. Thesedefects were reversible, as they could be normalized by platingCol6a1–/– myofibers on collagen VI or by addition of cyclosporinA (CsA), the inhibitor of mitochondrial permeability transitionpore (PTP). Treatment of Col6a1–/– mice with CsA rescued themuscle ultrastructural defects and markedly decreased thenumber of apoptotic nuclei in vivo. These findings indicate thatcollagen VI myopathies have an unexpected mitochondrialpathogenesis that could be exploited for therapeutic intervention.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:45:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277547248</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277547257</link>
         <description><![CDATA[ Mitochondrial myopathy]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:45:21 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277547257</guid>
      </item>
      <item>
         <title>Tariao, Darryl</title>
         <author>keane56k</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277548255</link>
         <description><![CDATA[<div>8 aristorle<br><br>1. What is Mitochondrial disease?<br>Mitochondrial disease&nbsp;is a chronic, genetic&nbsp;disorder&nbsp;that occurs when the&nbsp;mitochondria&nbsp;of the cell fail to produce enough energy for cell or organ function.&nbsp;<br><br>2. What are the symptoms of mitochondrial diseases?<br>-Poor growth<br>-Loss of muscle coordination, mneurological problems, seizures<br>-Autism, autistic spectrum, autistics like features&nbsp;<br>-visual and/or hearing problem&nbsp;<br>-developmental delays, learning disabilities&nbsp;<br>-Heart, liver or kidney disease<br>-Gastrointestinal disorders, severe constipation<br>-Diabetes<br>-Increased risk of infection<br>-Thyroid and/or adrenal dysfunction<br>-Autonomic dysfunction<br>-Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br>3. Who discovered mitochondrial diseases?<br>The first disorder of mitochondrial function was described by Professor Rolf Luft in 1959.<br><br>4. What will happen if the mitochondria is not working?<br>Can it be cured?<br>When the mitochondria are defective, the cells do not have enough energy. The unused oxygen and fuel molecules build up in the cells and cause damage.<br>No<br>There are no cures for these diseases, but treatments may help with symptoms and slow down the disease. They may include physical therapy, vitamins and supplements, special diets, and medicines<br><br>5. What are examples of mitochondrial diseases?<br><br>Diabetes mellitus and deafness<br><br>Friedreich's ataxia<br><br>GFER Syndrome<br><br>HUPRA syndrome<br><br>Kearns–Sayre syndrome<br><br>Leber's hereditary optic neuropathy<br><br>Leigh syndrome<br><br>May–White syndrome<br><br>MELAS syndrome<br><br>MERRFsyndrome<br><br>Mitochondrial DNA depletion syndrome<br><br>CASE STUDY:<br><br>Mattie Stepanek, a poet, peace advocate, and motivational speaker who suffered from dysautonomic mitochondrial myopathy, and who died at age 13<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:47:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277548255</guid>
      </item>
      <item>
         <title>Lata,  Ronan Raphael C.     8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277550370</link>
         <description><![CDATA[<div>Questions:<br><br>1.) What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br><br>2.) What are the symptoms of mitochondrial diseases?&nbsp;<br>*Poor growth<br>*Muscle weakness, muscle pain, low muscle tone, exercise intolerance<br>*Vision and/or hearing problems<br>*Learning disabilities, delays in development, *mental retardation<br>*Autism, autism-like features<br>*Heart, liver or kidney diseases<br>*Gastrointestinal disorders, swallowing difficulties, diarrhea or constipation, unexplained vomiting, cramping, reflux<br>*Diabetes<br>*Increased risk of infection<br>*Neurological problems, seizures, migraines, strokes<br>*Movement disorders<br>*Thyroid problems<br>*Respiratory (breathing) problems<br>*Lactic acidosis (a buildup of lactate)<br>*Dementia<br><br>3.) Who discovered mitochondrial diseases?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4.) What happens when mitochondria is bot working?&nbsp;<br>You are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br>5.) What are examples of mitochondrial diseases?&nbsp; Can this be cured?<br>*Neuropathy, ataxia, and retinitis pigmentosa<br>*MERRF syndrome<br>*Mitochondrial myopathy<br>*Leigh syndrome<br>The diseases can be present at birth or develop later in life. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Insights: From the article that I read,I could say that mitochondria is a very important part not just for the cell but also for our body. It may be small,but it has a large impact to our body. Overall,&nbsp; mitochondria is important especially in energy production.&nbsp;<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:51:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277550370</guid>
      </item>
      <item>
         <title>Caryl Jean P. Urdaneta 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277552436</link>
         <description><![CDATA[<div>Questions &amp; Answers<br>1. What is mitochondrial disease?<br>&nbsp;- Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br>&nbsp;- In the case report, It talks about the new mitochondrial disease linked to Demin gene.<br>&nbsp;<br>&nbsp;2. What are the symptoms of the mitochondrial diseases?<br>&nbsp;- The symptoms of the mitochondrial disease as experienced by a 45-year-old man are; cardiac gastrointestinal, neuromuscular and mood disorders.<br>&nbsp;<br>&nbsp;3. Who discovered the mitochondrial disease?<br>&nbsp;- The researchers discovered the disease at the Children's Hospital of Philadelphia and the Thomas Jeferson University Hospital Philadelphia.<br>&nbsp;<br>&nbsp;4. What happens when mitochondria are not working?<br>&nbsp;- For our bodies the conversion from food energy to ATP happens inmitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br>&nbsp;<br>&nbsp;5. What are some examples of mitochondrial diseases? Can this be cured?<br>&nbsp;•LHON - it can be cured, because there is one approved treatment in Europe.<br>&nbsp;•Leigh's disease- There is no cure for leigh's disease. Treatments generally involve variations of vitamin and supplement therapies, often in a “cocktail” combination, and are only partially effective.&nbsp;<br>&nbsp;•Pyruvate dehydrogenase complex deficiency - It can be cured. The goal of the treatment for pyruvate dehydrogenase complex (PDC) deficiency is to stimulate the pyruvate dehydrogenase complex to produce as much energy as possible. This can prevent immediate worsening of the disease.Treatment options typically include supplementing cofactors including carnitine, thiamine, and lipoic acid. These are substances in the body that help the chemical reactions in the cells occur.<br>&nbsp;•Automosal Dominant Optic Atrophy - There currently is no treatment or cure ADOA.&nbsp; The supplement, Idebenone, according to one study, showed some improvement in visual acuity in patients with ADOA.<br>&nbsp;•Mitochondrial myophathy - There is currently no available disease-modifying therapy for MM. Several agents (mostly nutritional supplements) have been investigated with double-blind, placebo-controlled studies.<br>&nbsp;<br>&nbsp;"THE CASE STUDY" Case Report of a New Mitochondrial Disease Linked to Desmin Gene<br>&nbsp;By: Patricia Silva PhD<br>&nbsp;<br>&nbsp;A study recently published in the journal Frontiers in Genetics reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA” and was conducted by researchers at The Children’s Hospital of Philadelphia and the Thomas Jefferson University Hospital Philadelphia.<br>&nbsp;<br>&nbsp;The study reports the case of a 45-year-old man who presented to The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br>&nbsp;<br>&nbsp;The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br>&nbsp;<br>&nbsp;The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.<br>&nbsp;<br>&nbsp;The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.<br>&nbsp;<br>&nbsp;•My insights about this case is that there is a cure for the disease that is new and about demin gene. There are many symptomps or diseases that directly interact into the mitochondria that causes dysfuctional of it.<br>&nbsp;<br>&nbsp;<br>&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:54:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277552436</guid>
      </item>
      <item>
         <title>Lata,  Ronan Raphael  C.    8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277554821</link>
         <description><![CDATA[<div>Questions:<br><br>1.) What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br><br>2.) What are the symptoms of mitochondrial diseases?&nbsp;<br>*Poor growth<br>*Muscle weakness, muscle pain, low muscle tone, exercise intolerance<br>*Vision and/or hearing problems<br>*Learning disabilities, delays in development, *mental retardation<br>*Autism, autism-like features<br>*Heart, liver or kidney diseases<br>*Gastrointestinal disorders, swallowing difficulties, diarrhea or constipation, unexplained vomiting, cramping, reflux<br>*Diabetes<br>*Increased risk of infection<br>*Neurological problems, seizures, migraines, strokes<br>*Movement disorders<br>*Thyroid problems<br>*Respiratory (breathing) problems<br>*Lactic acidosis (a buildup of lactate)<br>*Dementia<br><br>3.) Who discovered mitochondrial diseases?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4.) What happens when mitochondria is bot working?&nbsp;<br>You are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br>5.) What are examples of mitochondrial diseases?&nbsp; Can this be cured?<br>*Neuropathy, ataxia, and retinitis pigmentosa<br>*MERRF syndrome<br>*Mitochondrial myopathy<br>*Leigh syndrome<br>The diseases can be present at birth or develop later in life. ... Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Insights: From the article that I read,I could say that mitochondria is a very important part not just for the cell but also for our body. It may be small,but it has a large impact to our body. Overall,&nbsp; mitochondria is important especially in energy production.&nbsp;<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 13:59:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277554821</guid>
      </item>
      <item>
         <title>MENDEL (9/4/2018)</title>
         <author>mtskmisa</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277562577</link>
         <description><![CDATA[<div>PAGURAY, Krystle Ann Z.<br>BROCOY, Angelle Kate Y.<br><br>Questions:<br>1. What is a Mitochondrial Disease?<br>Mitochondrial Disease is a Disorder that is caused by the failure of mitochondria functions.<br><br>2. What are the symptoms of Mitochondrial Disease?<br>Symptoms:<br>-Poor Growth<br>-Loss of muscle coordination , muscle weakness<br>-Visual /Hearing Problems<br>-Developmental Delays<br>-Mental Retardation<br>-Heart, Liver, Kidney Disease<br>-Gastro Intestinal Disorders<br>-Respiratory Disorders<br>-Diabetes<br>-Increased Risk of Infection<br>-Nuerological problems<br>-Thyroid Dysfunction<br>-Dementia<br><br>3. Who discovered the Mitochondrial Disease?<br>In 1940 Mitochondrial Disease is discovered. The first patient to be diagnosed with Mitochondrial Disease is in 1962.<br><br>4. What happens when mitochondria are not working?<br>According to Newscastle,"If your mitochondria are not working properly then you are less able to convert food into ATP".<br><br>5. What are some examples of Mitochondrial Diseases? Can they be cured?<br><br>&nbsp;MITOCHONDRIAL DNA DEPLETION SYNDROME<br>(Currently no curative treatment)<br>MITOCHONDRIAL MYOPATHY<br>(not curable)<br>MELAS SYNDROME<br>(No cure; medical care is largely supportive)<br><br>Case Study:&nbsp;<br>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.<br><br>Insight:<br>JAUNDICE IS A DISEASE BY WHICH A PERSON BECOMES YELLOWISH. THE SYMPTOMS ARE DIARRHEA AND THE YELLOWISH COLOR OF RHE SKIN THE ACTIONS TO CURE IT IS BY TAKING UP GLUCOSE AND PROPHYLACTIC ANTIBIOTICS THE RESULT IS THAT THE JAUNDICE DISEASE WILL NOT BE OCCURING ANYMORE IN YOUR BODY.<br><br>(10:49 PM)</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:13:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277562577</guid>
      </item>
      <item>
         <title>Emata, Simone Anne C.  (8 - Aristotle)</title>
         <author>annieace</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277564479</link>
         <description><![CDATA[<div>&nbsp; &nbsp; &nbsp; 1.) What is Mitochondrial Disease?<br><strong>A:</strong> Mitochondrial diseases<strong> </strong>are a group of disorders caused by dysfunction in the mitochondria, the organelles that generate energy for the cell. <br><br>2.) What is/are the symptom/s of Mitochondrial Disease?<br><strong>A: <br></strong>- fatigue <br>- exercise intolerance <br>- headache <br>- multiple trigger points <br>-&nbsp; Neuropathic Pain <br>- weakness <br>- developmental delays <br>- Diarrhea <br>-&nbsp; cramping <br><br>3.)&nbsp; Who discover mitochondrial disease/s?<br><strong>A: </strong>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint", which is different than the nuclear DNA found in the cells' nucleus.<br><br>4.) What happens when mitochondria is not working?<br><strong>A: </strong>&nbsp;Everything that the body does, from staying warm, to moving and thinking needs energy. The energy that we use comes from the proteins, carbohydrates and fats in our food. We are unable to use these directly, but instead need to convert them in to a form of energy that cells can use called ATP. You can think of this like a battery. This converts chemical energy into a different form (in this case electrical) that is easier to use. For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br><br>5.) What are some examples of mitochondrial disease? Can this be cured?&nbsp;<br><strong>A:&nbsp;</strong>&nbsp;1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.&nbsp;</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:16:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277564479</guid>
      </item>
      <item>
         <title>Jasmin Imelda P. Silva *-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277581857</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:47:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277581857</guid>
      </item>
      <item>
         <title>La</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277582141</link>
         <description><![CDATA[<div><strong>Aristotle<br></strong> <br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:48:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277582141</guid>
      </item>
      <item>
         <title>MENDEL (9/4/2018)</title>
         <author>mtskmisa</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277588150</link>
         <description><![CDATA[<div>PAGURAY, Krystle Ann Z.<br>BROCOY, Angelle Kate Y.<br><br>Questions:<br>1. What is a Mitochondrial Disease?<br>Mitochondrial Disease is a Disorder that is caused by the failure of mitochondria functions.<br><br>2. What are the symptoms of Mitochondrial Disease?<br>Symptoms:<br>-Poor Growth<br>-Loss of muscle coordination , muscle weakness<br>-Visual /Hearing Problems<br>-Developmental Delays<br>-Mental Retardation<br>-Heart, Liver, Kidney Disease<br>-Gastro Intestinal Disorders<br>-Respiratory Disorders<br>-Diabetes<br>-Increased Risk of Infection<br>-Nuerological problems<br>-Thyroid Dysfunction<br>-Dementia<br><br>3. Who discovered the Mitochondrial Disease?<br>In 1940 Mitochondrial Disease was discovered.<br><br>4. What happens when mitochondria are not working?<br>According to Newscastle,"If your mitochondria are not working properly then you are less able to convert food into ATP".<br><br>5. What are some examples of Mitochondrial Diseases? Can they be cured?<br><br>&nbsp;MITOCHONDRIAL DNA DEPLETION SYNDROME<br>(Currently no curative treatment)<br>MITOCHONDRIAL MYOPATHY<br>(not curable)<br>MELAS SYNDROME<br>(No cure; medical care is largely supportive)<br><br>Case Study:&nbsp;<br>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.<br><br>Insight:<br>JAUNDICE IS A DISEASE BY WHICH A PERSON BECOMES YELLOWISH. THE SYMPTOMS ARE DIARRHEA AND THE YELLOWISH COLOR OF RHE SKIN THE ACTIONS TO CURE IT IS BY TAKING UP GLUCOSE AND PROPHYLACTIC ANTIBIOTICS THE RESULT IS THAT THE JAUNDICE DISEASE WILL NOT BE OCCURING ANYMORE IN YOUR BODY.<br><br>(10:58 PM)</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:55:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277588150</guid>
      </item>
      <item>
         <title>MENDEL (9/4/18)</title>
         <author>mtskmisa</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277590224</link>
         <description><![CDATA[<div>PAGURAY, Krystle Ann Z.<br>BROCOY, Angelle Kate Y.<br><br>Questions:<br>1. What is a Mitochondrial Disease?<br>Mitochondrial Disease is a Disorder that is caused by the failure of mitochondria functions.<br><br>2. What are the symptoms of Mitochondrial Disease?<br>Symptoms:<br>-Poor Growth<br>-Loss of muscle coordination , muscle weakness<br>-Visual /Hearing Problems<br>-Developmental Delays<br>-Mental Retardation<br>-Heart, Liver, Kidney Disease<br>-Gastro Intestinal Disorders<br>-Respiratory Disorders<br>-Diabetes<br>-Increased Risk of Infection<br>-Nuerological problems<br>-Thyroid Dysfunction<br>-Dementia<br><br>3. Who discovered the Mitochondrial Disease?<br>Unknown.<br><br>4. What happens when mitochondria are not working?<br>According to Newscastle,"If your mitochondria are not working properly then you are less able to convert food into ATP".<br><br>5. What are some examples of Mitochondrial Diseases? Can they be cured?<br><br>&nbsp;MITOCHONDRIAL DNA DEPLETION SYNDROME<br>(Currently no curative treatment)<br>MITOCHONDRIAL MYOPATHY<br>(not curable)<br>MELAS SYNDROME<br>(No cure; medical care is largely supportive)<br><br>Case Study:&nbsp;<br>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.<br><br>Insight:<br>JAUNDICE IS A DISEASE BY WHICH A PERSON BECOMES YELLOWISH. THE SYMPTOMS ARE DIARRHEA AND THE YELLOWISH COLOR OF RHE SKIN THE ACTIONS TO CURE IT IS BY TAKING UP GLUCOSE AND PROPHYLACTIC ANTIBIOTICS THE RESULT IS THAT THE JAUNDICE DISEASE WILL NOT BE OCCURING ANYMORE IN YOUR BODY.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 14:59:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277590224</guid>
      </item>
      <item>
         <title>Jasmin Imelda P. Silva 8-AristotleAnswers to QUESTIONS :1.what is mitochondrial disease?-Mitochondrial diseases are long-term, genetic, often inherited disorders that occur when mitochondria fail to produce enough energy for the body to function properly.2.What are the symptoms?muscle weakness/exercise intoleranceheart failuredementiamovement disordersstroke-like episodesdeafnessblindnessvomitingDiabetesIncreased risk of infectionNeurological problems, seizures</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277591624</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 15:02:14 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277591624</guid>
      </item>
      <item>
         <title>Lapating, Angelie Mae M. Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277593366</link>
         <description><![CDATA[<div>1.	What is mitochondrial disease?<br>Mitochondrial disease are a group of clinically heterogeneous inherited metabolic disorders caused by defects in mitochondrial ATP production<br>2.	What is the symptoms of mitochondrial disease? <br>•	Nervous system tissues are ataxia <br>•	Myoclonia<br>•	P sychomotor retardation<br>•	Psychomotor regression <br>•	Dystonia <br>•	Muscle weakness<br>•	Intolerance to physical exercise<br>•	Sensorineural hearing loss <br>•	Movement coordination issues<br>•	Seizures <br>•	Learning disabilities <br>•	Optic atrophy<br>•	 Pigmentary retinopathy <br>•	Ophthalmoplegia <br>•	Cardiomyopathy<br>•	Diabetes, autism <br>•	Growth atrophy<br>•	Peripheral neuropathy<br>•	Dementia<br>•	Multiple lipomas <br>•	Respiratory issues<br>3.	Who discover mitochondrial disease?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4.	What happens when mitochondria is not working? <br>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br>5.	What are some examples of mitochondrial disease? Can this be cured?<br>•	Kearns-Sayre Syndrome<br>•	MELAS<br>•	MERRF<br>•	PEO<br>•	Pearson Syndrome<br>•	Leigh encephalopathy<br>•	NARP<br>The Mitochondrial Diseases cannot be cured but it can be prevented.<br>INSIGHTS: Mitochondria is a very important part for the cell especially in energy production. It is so useful to our body. If It will be damaged It may cause a lot of different Mitochondrial Diseases that cannot be cured but it can be prevented.<br>Case Study:<br>The patient is an 11-year-old boy diagnosed with mitochondrial disease who has been under the care of a specialized genetics clinic since he was one year old, and has since then been taking medications to replace the amino acids his body cannot produce: l-carnitine, thiamine, coenzyme, vitamins C and E, riboflavin and folic acid. He undergoes periodic hearing and vision evaluations, with results within normal standards. He goes to a regular private school and during the study was attending the second and third years of elementary school. The school’s main complaint was about his behavior, especially in regard to his interaction with other students and his aggressiveness, both of which were associated with the patient’s communication difficulties. His communication with his family was based mostly on oral language, with the support of some non-symbolic gestures like pointing12, always associated with vocalizations. His relatives reported having trouble understanding him. The speech-language evaluation found: presence of rare vocalizations or monosyllabic words (as the patient’s phoneme articulation is limited, it is often difficult to understand the word) and use of gestures and body language, with low functionality due to his limited discursive autonomy and communicative intentions, associated with repetitive speech, i.e., he repeats his own speech. All this led to irritability – he would slap his interlocutor or his own head – whenever he was not understood.<br>Source: http://www.scielo.br/pdf/rcefac/v18n4/en_1982-0216-rcefac-18-04-01001.pdf</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 15:05:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277593366</guid>
      </item>
      <item>
         <title>Jasmin Imelda P. Silva 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277593753</link>
         <description><![CDATA[<div><br>ANSWERS TO QUESTIONS:<br><strong>1. what is mitochondrial disease?</strong></div><div>-Mitochondrial diseases are long-term, genetic, often inherited disorders that occur when mitochondria fail to produce enough energy for the body to function properly.</div><div> </div><div><strong>2.What are the symptoms?</strong></div><div>muscle weakness/exercise intolerance</div><div>heart failure</div><div>dementia</div><div>movement disorders</div><div>stroke-like episodes</div><div>deafness</div><div>blindness</div><div>vomiting</div><div>Diabetes</div><div>Increased risk of infection</div><div>Neurological problems, seizures<br><br><strong>3.Who discovered mitochondrial disease?</strong></div><div><strong> </strong></div><div>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondriahave their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.</div><div> </div><div><strong>4. What happens when mitochondria is not working?</strong></div><div> </div><div>-If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is aproblem and they may become weaker and get tired faster.</div><div> </div><div><strong>5.What are some examples of mitochondrial disease?Can this be cured?</strong></div><div><br>-Mitochondrial diseases can affect almost any part of the body, including the cells of the brain, nerves, muscles, kidneys, heart, liver, eyes, ears or pancreas.<br><br></div><div><br>-Mitochondrial dysfunction occurs when the mitochondria do not work as well as they should due to another disease or condition. Many conditions can lead to secondary mitochondrial dysfunction and affect other diseases, including <a href="https://my.clevelandclinic.org/health/diseases/9164-alzheimers-disease-an-overview">Alzheimer’s disease</a>, <a href="https://my.clevelandclinic.org/health/diseases/14128-muscular-dystrophy">muscular dystrophy</a>, <a href="https://my.clevelandclinic.org/health/diseases/16729-amyotrophic-lateral-sclerosis-als">Lou Gehrig’s disease</a>, <a href="https://my.clevelandclinic.org/health/diseases/7104-diabetes-mellitus-an-overview">diabetes</a> and <a href="https://my.clevelandclinic.org/health/diseases/12194-cancer-overview">cancer</a>. Individuals with secondary mitochondrial dysfunction do not have primary genetic mitochondrial disease and do not need to be concerned about the ongoing development or worsening of symptoms.It is curable but it needs a lot of years to await for the tests results.<br><strong><br>CASE STUDY<br></strong><br></div><div><br> <br><br></div><h1>Mitochondrial Disorder Medical Information</h1><div><em>Visit the BCMJ May 2011 issue </em><a href="http://www.bcmj.org/node/3783"><em>here</em></a><em> to read a compilation of medical articles about mitochondrial disease.<br></em><br></div><div><strong>What are mitochondria?<br></strong>A mitochondrion (singular of mitochondria) is part of every cell in the body that contains genetic material. Mitochondria are responsible for processing oxygen and converting substances from the foods we eat into energy for essential cell functions. Mitochondria produce energy in the form of adenosine triphosphate (ATP), which is then transported to the cytoplasm of a cell for use in numerous cell functions.<br><br></div><div><strong>What are mitochondrial and metabolic diseases?<br></strong>Mitochondrial medicine is a new and rapidly developing medical subspecialty. Many specialists are involved in researching mitochondrial diseases, including doctors specializing in metabolic diseases, cell biologists, molecular geneticists, neurologists, biochemists, pathologists, immunologists, and embryologists. Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br></div><div>Mitochondrial and metabolic medical conditions are now referred to as <em>mitochondrial cytopathies. </em>Mitochondrial cytopathies actually include more than 40 different identified diseases that have different genetic features. The common factor among these diseases is that the mitochondria are unable to completely burn food and oxygen in order to generate energy.<br><br></div><div>The process of converting food and oxygen (fuel) into energy requires hundreds of chemical reactions, and each chemical reaction must run almost perfectly in order to have a continuous supply of energy. When one or more components of these chemical reactions does not run perfectly, there is an energy crisis, and the cells cannot function normally. As a result, the incompletely burned food might accumulate as poison inside the body.<strong> <br></strong><br></div><div>This poison can stop other chemical reactions that are important for the cells to survive, making the energy crisis even worse. In addition, these poisons can act as free radicals (reactive substances that readily form harmful compounds with other molecules) that can damage the mitochondria over time, causing damage that cannot be reversed. Unlike nuclear DNA, mitochondrial DNA has very limited repair abilities and almost no protective capacity to shield the mitochondria from free radical damage.<br><br></div><div><strong>What are the symptoms of mitochondrial diseases?<br></strong>The types of mitochondrial diseases are categorized according to the organ systems affected and symptoms present. Mitochondrial diseases might affect the cells of the brain, nerves (including the nerves to the stomach and intestines), muscles, kidneys, heart, liver, eyes, ears, or pancreas. In some patients, only one organ is affected, while in other patients all the organs are involved. Depending on how severe the mitochondrial disorder is, the illness can range in severity from mild to fatal.<br><br></div><div>Depending on which cells of the body are affected, symptoms might include:<br><br></div><div>· Poor growth</div><div>· Loss of muscle coordination, muscle weakness</div><div>· Visual and/or hearing problems</div><div>· Developmental delays, learning disabilities</div><div>· Mental retardation</div><div><strong> </strong></div><div><strong> </strong></div><div><strong> </strong></div><div><strong> </strong></div><div><strong> </strong></div><div><strong>What causes mitochondrial disease?<br></strong>For many patients, mitochondrial disease is an inherited condition that runs in families (genetic). An uncertain percentage of patients acquire symptoms due to other factors, including mitochondrial toxins.</div><div>It is important to determine which type of mitochondrial disease inheritance is present, in order to predict the risk of recurrence for future children.<br><br></div><div>The types of mitochondrial disease inheritance include:<br><br></div><div>· nDNA (DNA contained in the nucleus of the cell) inheritance. Also called autosomal inheritance.<br>-- If this gene trait is recessive (one gene from each parent), often no other family members appear to be affected. There is a 25 percent chance of the trait occurring in other siblings.<br>-- If this gene trait is dominant (a gene from either parent), the disease often occurs in other family members. There is a 50 percent chance of the trait occurring in other siblings.</div><div>· mtDNA (DNA contained in the mitochondria) inheritance.<br>-- There is a 100 percent chance of the trait occurring in other siblings, since all mitochondria are inherited from the mother, although symptoms might be either more or less severe.</div><div>· Combination of mtDNA and nDNA defects:<br>-- Relationship between nDNA and mtDNA and their correlation in mitochondrial formation is unknown</div><div>· Random occurrences<br>Diseases specifically from deletions of large parts of the mitochondrial DNA molecule are usually sporadic without affecting other family member<br>-- Medicines or other toxic substances can trigger mitochondrial disease</div><div> <br><br></div><div><strong>How are mitochondrial diseases diagnosed?<br></strong>Diagnosis of mitochondrial disease can be invasive, expensive, time-consuming, and labor-intensive. Therefore, evaluation is not taken lightly. Doctors experienced in diagnosing and treating these diseases will take either a step-wise approach to diagnosis or, in some centers, the evaluation takes place over a few days. The evaluation includes a combination of clinical observations and laboratory tests.<br><br></div><div>Under ideal circumstances, the evaluation will produce an answer. However, even after a complete evaluation, the doctor might not be able to confirm a specific diagnosis or put a name to the disorder. In many cases, however, the physician will be able to identify which patients do and don't have metabolic diseases.<br><br></div><div>Mitochondrial disease is diagnosed by:<br><br></div><div>· Evaluating the patient's family history</div><div>· Performing a complete physical examination</div><div>· Performing a neurological examination</div><div>· Performing a metabolic examination that includes blood, urine, and optional cerebral spinal fluid tests</div><div>· Performing other tests, depending on the patient's specific condition and needs. These tests might include:<br>-- Magnetic resonance imaging (MRI) or scan (MRS) if neurological symptoms are present<br>-- Retinal exam or electroretinogram if vision symptoms are present<br>-- Electrocardiogram (EKG) or echocardiogram if heart disease symptoms are present<br>-- Audiogram or BAEP if hearing symptoms are present<br>-- Blood test to detect thyroid dysfunction if thyroid problems are present<br>-- Blood test to perform genetic DNA testing</div><div>More invasive tests, such as a skin or muscle biopsy, might be performed as needed and recommended by your doctor.<br><br></div><div> <br><br></div><div><strong>How are mitochondrial diseases treated?<br></strong>There are no cures for mitochondrial diseases, but treatment can help reduce symptoms, or delay or prevent the progression of the disease.<br><br></div><div>Treatment is individualized for each patient, as doctors specializing in metabolic diseases have found that every child and adult is "biochemically different." That means that no two people will respond to a particular treatment in a specific way, even if they have the same disease.<br><br></div><div><strong>· Certain vitamin and enzyme therapies,</strong> along with occupational and physical therapy, might be helpful for some patients.</div><div>Vitamins and supplements prescribed might include:<br><br></div><div>- Coenzyme Q10<br>- B complex vitamins: thiamine (B1), riboflavin (B2), niacin (B3), B6, folate, B12, biotin, pantothenic acid<br>- Vitamin E, lipoic acid, selinium, and other antioxidants<br>- L-carnitine (Carnitor®)<br>- Intercurrent illness supplement: vitamin C, biotin<br><br></div><div><strong>· Diet therapy</strong>, as prescribed by your doctor along with a registered dietitian, might be recommended.</div><div><strong>· Antioxidant treatments</strong> as protective substances are currently being investigated as another potential treatment method.</div><div><strong>Important:</strong> <strong>Specific treatments should always be guided by a metabolic specialist. </strong>Patients should not take any of these supplements or try any of the treatments unless prescribed by a doctor. Taking inappropriate supplements or treatments might lead to delays or failure in establishing an accurate diagnosis.<br><br></div><div> <br><br></div><div><strong>What is the prognosis or outlook?<br></strong>Once a patient is diagnosed with a specific mitochondrial disease, the patient's medical problems have already been identified or can be identified with proper testing so treatment can be initiated to relieve symptoms and delay the progression of the disease.<br><br></div><div>There is no way to predict the course of mitochondrial diseases. They might progress quickly or slowly, even over decades. The disease might also appear stable for years.<br><br></div><div>For parents considering having other children, genetic counseling is available. Although complex, prenatal testing is only available for a few types of mitochondrial disorders. Please discuss your concerns with your doctor.<br><br></div><div><br> <br><br></div>]]></description>
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         <pubDate>2018-09-04 15:06:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277593753</guid>
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         <title>Malunes,  Jaesar Justin P.  8-Aristotle </title>
         <author>janicepalacios_bj</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277594858</link>
         <description><![CDATA[<div><strong>1.What is mitochondrial disease?&nbsp;</strong></div><div>Answer:<br>&nbsp;Mitochondrial disease includes a group of neuromuscular diseases caused by damage to intracellular structures that produce energy, the mitochondria<br>&nbsp;<br>&nbsp;2.<strong>What is the symptom of mitochondrial disease?&nbsp;</strong></div><div>Answer:<br>&nbsp;The symptoms of mitochondrial myopathies include muscle weakness or exercise intolerance, heart failure or rhythm disturbances, dementia, movement disorders, stroke-like episodes, deafness, blindness, droopy eyelids, limited mobility of the eyes, vomiting, and seizures. <br>&nbsp;<br>&nbsp;3.<strong>Who discover mitochondrial disease?&nbsp;</strong></div><div>Answer:<br>We know about these disease that it has been disscovered since 1940.<br>&nbsp;<br>&nbsp;4.<strong>What happens when mitochondria is not working?</strong>&nbsp;</div><div>Answer:<br>&nbsp;For our bodies the conversion from food energy to ATP happens in mitochondria.<br>&nbsp;If our mitochondria are not working properly, it means that we are less able to convert food into ATP. For cells that requires a lot of ATP.<br>&nbsp;<br>&nbsp;5. <strong>What are some examples of mitochondrial disease?can this be cured?&nbsp;</strong></div><div>Answer:<br>&nbsp;A number of specific mitochondrial<strong> </strong>disorders have been associated with Complex I deficiency including: Leber's hereditary optic neuropathy (LHON), MELAS, MERRF, and Leigh Syndrome (LS), Myopathy (muscle disease) – starting in childhood or adulthood, and characterized by weakness or exercise intolerance.&nbsp;<br><br>-There is currently no cure for mitochondrial disease. However, important steps have been made to make diagnosis faster, easier and less invasive to the patient, and some promising research into effective treatments is underway<br><br></div><div><strong>Case Study</strong><br> -1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.&nbsp;</div><div><strong>Source</strong>:&nbsp;<br> http://www.circuitblue.com/mito/caseStudies.shtml&nbsp;<br><br><strong>Insights:&nbsp;</strong><br>The mitochondria is very important to us because it is where the conversion of food into energy happens. So, if the mitochondria will be damaged and we will have a mitochondrial disease it means we are less able to convert food into ATP and we will be weak. </div>]]></description>
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         <pubDate>2018-09-04 15:08:05 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277594858</guid>
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         <title>Figuracion, Reanne Kathleen B.     Grade 8- Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277613880</link>
         <description><![CDATA[<div>Questions:<br><strong>1. What is mitochondrial disease?</strong><br>Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells).<br><br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>Poor growth<br>Muscle weakness, muscle pain, low muscle tone, exercise intolerance<br>Vision and/or hearing problems<br>Learning disabilities, delays in development, mental retardation<br>Autism, autism-like featuresHeart, liver or kidney diseases<br>Gastro intestinal disorders, swallowing difficulties, diarrhea or constipation, unexplained vomiting, cramping, reflux<br>Diabetes <br>Increased risk of infection<br>Neurological problems, seizures, migraines, strokes<br>Movement disorders<br>Thyroid problems<br>Respiratory (breathing) problems<br>Lactic acidosis (a buildup of lactate)<br>Dementia<br><br><strong>3. Who discovered mitochondrial diseases or disorders?</strong><br>These diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria are not working?</strong><br>If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br><strong>5. What are some examples of mitochondrial diseases?</strong><br>Leigh syndrome<br>Leber's hereditary optic neuropathy<br>Kearns–Sayre syndrome<br>MELAS syndrome<br>MERRF syndrome<br>Chronic progressive external ophthalmoplegia<br>Neuropathy, ataxia, and retinitis pigmentosa<br>Pearson syndrome<br>Diabetes mellitus and deafness<br><br><strong>Can this be cured?</strong><br>The diseases can be present at birth or develop later in life. ... Treatments thatcan ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br><strong>Case study:</strong><br><br>1982 DF was born at term and had no problems until she was 4 months old when it was found she was anemic during an evaluation for fever. She had multiple courses of antibiotics for UTIs and otitis media. She developed a picture of overwhelming sepsis and her lactic acid was elevated at 12 mM. She subsequently went on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome.</div>]]></description>
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         <pubDate>2018-09-04 15:39:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277613880</guid>
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         <title></title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277716454</link>
         <description><![CDATA[<div>1. What is a mitochondrial disease?<br>A: Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br><br>2. What are the symptoms of mitochondrial disease?<br>A: The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br><br>•Poor growth<br><br>Loss of muscle coordination, muscle weakness<br><br>Neurological problems, seizures<br><br>Autism, autistic spectrum, autistic-like features<br><br>Visual and/or hearing problems<br><br>Developmental delays, learning disabilities<br><br>Heart, liver or kidney disease<br><br>Gastrointestinal disorders, severe constipation<br><br>Diabetes<br><br>Increased risk of infection<br><br>Thyroid and/or adrenal dysfunction<br><br>Autonomic dysfunction<br><br>Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br><br></div>]]></description>
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         <pubDate>2018-09-04 18:52:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277716454</guid>
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         <title>H</title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277733122</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-04 19:33:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277733122</guid>
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         <title>JB Earl Tan Grade 8- Aristotle</title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277733807</link>
         <description><![CDATA[<div><strong>1. What is a mitochondrial disease?</strong><br>A: Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br><br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>A:<br>•Poor growth<br>•Loss of muscle coordination, muscle weakness<br>•Neurological problems, seizures<br>•Autism, autistic spectrum, autistic-like features<br>•Visual and/or hearing problems<br>•Developmental delays, learning disabilities<br>•Heart, liver or kidney disease<br>•Gastrointestinal disorders, severe constipation<br>•Diabetes<br>•Increased risk of infection<br>•Thyroid and/or adrenal dysfunction<br>•Autonomic dysfunction<br>•Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br><strong>3. Who discovered the mitochondrial disease?</strong><br>A: In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria is not working?</strong><br>A: If mitochondria is not working, you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br><strong>5. What are the examples of mitochondrial disease?&nbsp; Can this be cured?</strong><br>A: <br>•LHON<br>•LHON Plus<br>•Leigh's Disease<br>•Pyruvate Dehydrogenase Complex Deficiency (PDCD/PDH)<br>•Automosal Dominant Optic Atrophy (ADOA)<br><br>There are treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br><strong>Case Study:</strong><br><br><em>"Mitochondrial Disease as HCM"<br><br>A 26 year-</em>old male presents with hypertrophic cardiomyopathy (left ventricular wall thickness 1.8 cm) on echocardiogram. Full panel testing was <br>ordered for hypertrophic cardiomyopathy (HCM). A homoplasmic mitochondrial DNA mutation (A4300G) was found in the MT-TI gene, which is <br>known to cause maternally inherited HCM. Unlike other mitochondrial mutations which are known to have phenotypic variability, this mutation is <br>currently believed to cause isolated HCM.<br><br><strong>Insights:<br></strong>If mitochondria is not working properly, it may affect the parts of the body (requiring the greatest amount of energy such as heart, brain, muscles, and lungs)</div>]]></description>
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         <pubDate>2018-09-04 19:35:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277733807</guid>
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         <title>JB Earl Tan Grade 8- Aristotle</title>
         <author>jeybbb113004</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277735477</link>
         <description><![CDATA[<div><strong>1. What is a mitochondrial disease?</strong><br>A: Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br><br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>A:<br>•Poor growth<br>•Loss of muscle coordination, muscle weakness<br>•Neurological problems, seizures<br>•Autism, autistic spectrum, autistic-like features<br>•Visual and/or hearing problems<br>•Developmental delays, learning disabilities<br>•Heart, liver or kidney disease<br>•Gastrointestinal disorders, severe constipation<br>•Diabetes<br>•Increased risk of infection<br>•Thyroid and/or adrenal dysfunction<br>•Autonomic dysfunction<br>•Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br><strong>3. Who discovered the mitochondrial disease?</strong><br>A: In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria is not working?</strong><br>A: If mitochondria is not working, you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br><strong>5. What are the examples of mitochondrial disease?&nbsp; Can this be cured?</strong><br>A: <br>•LHON<br>•LHON Plus<br>•Leigh's Disease<br>•Pyruvate Dehydrogenase Complex Deficiency (PDCD/PDH)<br>•Automosal Dominant Optic Atrophy (ADOA)<br><br>There are treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br><strong>Case Study:<br></strong><br><em>"Mitochondrial Disease as HCM"<br></em><br>A 26 year-old male presents with hypertrophic cardiomyopathy (left ventricular wall thickness 1.8 cm) on echocardiogram. Full panel testing was <br>ordered for hypertrophic cardiomyopathy (HCM). A homoplasmic mitochondrial DNA mutation (A4300G) was found in the MT-TI gene, which is <br>known to cause maternally inherited HCM. Unlike other mitochondrial mutations which are known to have phenotypic variability, this mutation is <br>currently believed to cause isolated HCM.<br><br><strong>Insights:</strong> If mitochondria is not working properly, it may affect the parts of the body (requiring the greatest amount of energy such as heart, brain, muscles, and lungs)</div>]]></description>
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         <pubDate>2018-09-04 19:41:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277735477</guid>
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         <title>Alberto, Achilles Alexis R.   8-Aristotle </title>
         <author>achillesalberto05</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277773196</link>
         <description><![CDATA[<div><strong>Questions:<br></strong>1. What is Mitochondrial Disease? </div><pre>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions. </pre><div><br>2. What are the symptoms in mitochondrial disease?<br> - Poor growth</div><div> - Loss of muscle coordination, muscle weakness</div><div> - Neurological problems, seizures</div><div> - Autism, autistic spectrum, autistic-like features</div><div> - Visual and/or hearing problems</div><div> - Developmental delays, learning disabilities</div><div> - Heart, liver or kidney disease</div><div> - Gastrointestinal disorders, severe constipation</div><div> - Diabetes</div><div> - Increased risk of infection</div><div> - Thyroid and/or adrenal dysfunction</div><div> - Autonomic dysfunction</div><div> - Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br>3. Who discovered Mitochondrial Disease? </div><pre>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus. </pre><div><br>4. What happens when mitochondria are not working? </div><pre>When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.</pre><div><br>5. What are some examples of Mitochondrial Diseases? Can this be cured? <br> - LHON</div><div> - LHON Plus</div><div> - LEIGH’S DISEASE</div><div> - PYRUVATE DEHYDROGENASE COMPLEX DDEFICIENCY (PDCD/PDH)</div><div>AUTOSOMAL DOMINANT OPTIC ATROPHY (ADOA)<br><br>Case Study</div>]]></description>
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         <pubDate>2018-09-04 22:13:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277773196</guid>
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         <title>Tan, JB Earl Grade 8- Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277774016</link>
         <description><![CDATA[<div><strong> 1. What is a mitochondrial disease?</strong><br>Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br><br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>•Poor growth<br>•Loss of muscle coordination, muscle weakness<br>•Neurological problems, seizures<br>•Autism, autistic spectrum, autistic-like features<br>•Visual and/or hearing problems<br>•Developmental delays, learning disabilities<br>•Heart, liver or kidney disease<br>•Gastrointestinal disorders, severe constipation<br>•Diabetes<br>•Increased risk of infection<br>•Thyroid and/or adrenal dysfunction<br>•Autonomic dysfunction<br>•Neuropsychological changes characterized by confusion, disorientation, and memory loss.<br><br><strong>3. Who discovered the mitochondrial disease?</strong><br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br><strong>4. What happens when mitochondria is not working?<br></strong>If mitochondria is not working, you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br><strong>5. What are the examples of mitochondrial disease?  Can this be cured?</strong><br>•LHON<br>•LHON Plus<br>•Leigh's Disease<br>•Pyruvate Dehydrogenase Complex Deficiency (PDCD/PDH)<br>•Automosal Dominant Optic Atrophy (ADOA)<br><br>There are treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br><strong>Case Study:</strong><br><br><em>"Mitochondrial Disease as HCM"<br></em><br>A 26 year-old male presents with hypertrophic cardiomyopathy (left ventricular wall thickness 1.8 cm) on echocardiogram. Full panel testing was <br>ordered for hypertrophic cardiomyopathy (HCM). A homoplasmic mitochondrial DNA mutation (A4300G) was found in the MT-TI gene, which is <br>known to cause maternally inherited HCM. Unlike other mitochondrial mutations which are known to have phenotypic variability, this mutation is <br>currently believed to cause isolated HCM.<br><br><strong>Insights:</strong> <br>If mitochondria is not working properly, it may affect the parts of the body (requiring the greatest amount of energy such as heart, brain, muscles, and lungs)<br><br></div><div><em>Source: </em><a href="https://www.google.com.ph/url?sa=t&amp;source=web&amp;rct=j&amp;url=https%3A%2F%2Fmitochondrialdiseasenews.com%2F2015%2F06%2F26%2Fcase-report-new-mitochondrial-disease-linked-desmin-gene%2F%3Famp&amp;ved=2ahUKEwjC8LiHiaLdAhUEv7wKHXRNBlQQFjACegQICRAB&amp;usg=AOvVaw2bj0Xq4_tg4bR81mFtYcmI&amp;ampcf=1&amp;cshid=1536089037242"><em>https://www.google.com.ph/url?sa=t&amp;source=web&amp;rct=j&amp;url=https://mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/%3Famp&amp;ved=2ahUKEwjC8LiHiaLdAhUEv7wKHXRNBlQQFjACegQICRAB&amp;usg=AOvVaw2bj0Xq4_tg4bR81mFtYcmI&amp;ampcf=1&amp;cshid=1536089037242</em></a><em> </em></div>]]></description>
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         <pubDate>2018-09-04 22:18:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277774016</guid>
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      <item>
         <title>Ro</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277776055</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 22:32:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277776055</guid>
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      <item>
         <title>Rod</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277776056</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 22:32:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277776056</guid>
      </item>
      <item>
         <title>Rodu</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277776057</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 22:32:42 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277776057</guid>
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      <item>
         <title>Roduard Rynch </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277776079</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 22:32:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277776079</guid>
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      <item>
         <title>Rodua</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277776679</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-04 22:37:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277776679</guid>
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      <item>
         <title>Roduard Rynch Magallano and Romer Fagutao(8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277777038</link>
         <description><![CDATA[<div><strong>What is mitochondrial diseases?</strong><br>It is the disfunctional of mitochondria which caused by several disorders.<br><strong>What are the symptoms of mitochondrial diseases? </strong><br>There are several symptoms of mitochondrial diseases which is cornemeri and these are poor growth,loss of muscle coordination neurological problems, seizures and Development delays. <br><strong>Who discoverd mitochondrial diseases or disorder? <br></strong>In 1963,researcher discovered that mitochondria have their own or (m+DNA) which is different than the nuclear DNA (nDNA) found in the cell's nucleus. <br><strong>What happens when Mitochondria are not working ?</strong><br>If mitochindria is not working then eventually the cell will lose its capacity to further perform any activities such as ATP sunthesis. <br><strong>What are some examples of Mitochondrial diseases</strong>? <strong><br></strong>Some examples of them are the Mitochondrial DNA Depletion Sundrome, Mitochondrial Myopathy, Melas sundrome and Merrs syndrome and all of them are not curable but some of them has medications and the rapies which can be helpful in managing symptoms. <br><br><strong>Case study on Mitochondrial</strong> <br>A fundamental new understanding of programs that control <br><br>Mitochondria are microscopic parts of human cells that produce the energy required for life. Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria and while symptoms vary, they can include poor growth, loss of muscle coordination, muscle weakness, visual problems, hearing problems, learning disabilities, heart disease, liver disease, kidney disease, gastrointestinal disorders, respiratory disorders, neurological problems, autonomic dysfunction, and dementia.<br><br>“We have found how mitochondrial genes regulate the energy requirements in the human body’s tissues,” one of the study authors said. For example, tissues that need more energy such as the heart or brain have more active mitochondrial genes than tissues with lower energy demands such as the skin.”</div>]]></description>
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         <pubDate>2018-09-04 22:40:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277777038</guid>
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      <item>
         <title>Caryl Jean P. Urdaneta 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277790778</link>
         <description><![CDATA[<div>Questions &amp; Answers<br>1. What is mitochondrial disease?<br>&nbsp;- Mitochondrial diseases are a group of disorders caused by dysfunctionalmitochondria, the organelles that generate energy for the cell.Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br>&nbsp;- In the case report, It talks about the new mitochondrial disease linked to Demin gene.<br>&nbsp;<br>&nbsp;2. What are the symptoms of the mitochondrial diseases?<br>&nbsp;- The symptoms of the mitochondrial disease as experienced by a 45-year-old man are; cardiac gastrointestinal, neuromuscular and mood disorders.<br>&nbsp;<br>&nbsp;3. Who discovered the mitochondrial disease?<br>&nbsp;- The researchers dicovered the disease at the Children's Hospital of Philadelphia and the Thomas Jeferson Universith Hospital Philadelphia.<br>&nbsp;<br>&nbsp;4. What happens when mitochondria are not working?<br>&nbsp;- For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br>&nbsp;<br>&nbsp;5. What are some examples of mitochondrial diseases? Can this be cured?<br>&nbsp;•LHON - it can be cured, because there is one approved treatment in Europe<br>&nbsp;•Leigh's disease- There is no cure for leigh's disease. Treatments generally involve variations of vitamin and supplement therapies, often in a “cocktail” combination, and are only partially effective.&nbsp;<br>&nbsp;•Pyruvate dehydrogenase complex deficiency - It can be cured. The goal of the treatment for pyruvate dehydrogenase complex (PDC) deficiency is to stimulate the pyruvate dehydrogenase complex to produce as much energy as possible. This can prevent immediate worsening of the disease.Treatment options typically include supplementing cofactors including carnitine, thiamine, and lipoic acid. These are substances in the body that help the chemical reactions in the cells occur.<br>&nbsp;•Automosal Dominant Optic Atrophy - There currently is no treatment or cure ADOA.&nbsp; The supplement, Idebenone, according to one study, showed some improvement in visual acuity in patients with ADOA.<br>&nbsp;•Mitochondrial myophathy - There is currently no available disease-modifying therapy for MM. Several agents (mostly nutritional supplements) have been investigated with double-blind, placebo-controlled studies.<br>&nbsp;<br>&nbsp;"THE CASE STUDY" Case Report of a New Mitochondrial Disease Linked to Desmin Gene<br>&nbsp;By: Patricia Silva PhD<br>&nbsp;<br>&nbsp;A study recently published in the journal Frontiers in Genetics reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA” and was conducted by researchers at The Children’s Hospital of Philadelphia and the Thomas Jefferson University Hospital Philadelphia.<br>&nbsp;<br>&nbsp;The study reports the case of a 45-year-old man who presented to The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br>&nbsp;<br>&nbsp;The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br>&nbsp;<br>&nbsp;The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.<br>&nbsp;<br>&nbsp;The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.<br>&nbsp;<br>&nbsp;•My insights about this case is that there is a cure for the disease that is new and about demin gene. There are many symptomps or diseases that directly interact into the mitochondria that causes dysfuctional of it.<br>&nbsp;<br>&nbsp;<br>&nbsp;</div>]]></description>
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         <pubDate>2018-09-05 00:07:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277790778</guid>
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      <item>
         <title>Aira Shyn Leigh Vergara and JUlijah Flores 8-Mende</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277792198</link>
         <description><![CDATA[<div>Question &amp; Answer<br><strong>1.What is Mitochondrial Disease<br>-Mitochonrial Disease </strong>is a chronic, genetic disorder thst occurd when the mitochindria of the cwll fail to produce enough energy for the cell or organ function.<br><strong>2.What are the symptoms of mitochondrial Disease<br></strong>&nbsp;The most common symptoms are:</div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorientation, and memory loss.</li></ul><div><strong>3.Who discover the motochondrial DIsease?<br></strong>In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria</strong>have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><strong>4.What happened when&nbsp; mitochondria is not working?<br></strong>For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. <br><strong>5.What are some examples of mitochondrial disease and can be cure?<br></strong>&nbsp;The example of mitochondrial disease are:<br>&nbsp; •LHON - it can be cured, because there is one approved treatment in Europe.<br>&nbsp;•Leigh's disease- There is no cure for leigh's disease. Treatments generally involve variations of vitamin and supplement therapies, often in a “cocktail” combination, and are only partially effective. <br>&nbsp;•Pyruvate dehydrogenase complex deficiency - It can be cured. The goal of the treatment for pyruvate dehydrogenase complex (PDC) deficiency is to stimulate the pyruvate dehydrogenase complex to produce as much energy as possible. This can prevent immediate worsening of the disease.Treatment options typically include supplementing cofactors including carnitine, thiamine, and lipoic acid. These are substances in the body that help the chemical reactions in the cells occur.<br>&nbsp;•Automosal Dominant Optic Atrophy - There currently is no treatment or cure ADOA.&nbsp; The supplement, Idebenone, according to one study, showed some improvement in visual acuity in patients with ADOA.<br>&nbsp;•Mitochondrial myophathy - There is currently no available disease-modifying therapy for MM. Several agents (mostly nutritional supplements) have been investigated with double-blind, placebo-controlled studies. <br><br><strong>CASE STUDY<br></strong><strong><em>"</em></strong><em>Pediatrics"<br></em>&nbsp;1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet. <br><strong><br>Insigh:</strong> Juadice is a disease that the person infected by it becomes YELLOWISH. THe symptoms are the yellowish color of the skin and DIARREHA. It can be cured by taking the glucose and prophylatic antibiotics the result id JUANDICE DISEASE will&nbsp; not occure anymore to the infected person body.<br><br>Sources:<br>*<a href="http://mitoaction.org/mito-faq">http://mitoaction.org/mito-faq</a><br>* www.mitoaction.org/medical information<br>*http://en.wikipedia.org/wiki/Mitochondrial disease <br>*<a href="http://www.circuitblue.com/mito/caseStudies.shtml">http://www.circuitblue.com/mito/caseStudies.shtml</a><br>* <a href="http://www.umdf.org/pdf/MITOCYTO.PDF">http://www.umdf.org/pdf/MITOCYTO.PDF</a>&nbsp;</div>]]></description>
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         <pubDate>2018-09-05 00:15:59 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277792198</guid>
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      <item>
         <title>Enojales, Desire Faith C.    8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277797100</link>
         <description><![CDATA[<div><br>1. What is mitochondrial disease?<br>&nbsp;</div><div>Mitochondrial disease is an inherited chronic illness that can be present at birth or develop later in life. Mitochondrial disease is inherited in a number of ways.&nbsp; <br>2. What are the symptoms of mitochondrial diseases?<br><br>-Diabetes<br>-Heart, liver and kidney disease <br>-Autonomic dysfunction<br>-Increase risk of infection<br>-Muscle weakness<br>-Seizures<br>3. Who discovered mitochondrial disease/disorder?<br><br>&nbsp;Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus. <br>4. What happens when mitochondria is not working ?<br><br>When mitochondria malfunction or not working, organs start to fail - people get sick, and even die.<br>5. What are some examples of mitochondrial diseases?&nbsp; Can these be cured? <br><br>-Alpers Disease <br>Autosomal Dominant Optic Atrophy (ADOA)<br>-Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy)<br>-Fatty Acid Oxidation Disorders / Beta-oxidation Defects / Fatty Acid Transport and Mitochondrial ---- &nbsp; Oxidation Disorders<br>-Carnitine Deficiency<br>-Creatine Deficiency Syndromes <br>-Co-Enzyme Q10 Deficiency <br>-Chronic Progressive External Ophthalmoplegia (CPEO) <br>- Lactic Acidosis <br>- Leighs Disease <br>Some of these are curable. There are treatments that can be used to treat some of the mitochondrial diseases. But not all of these are curable only few. <br><br>Case Study: <br><br>The study, “<a href="http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4782535/#!po=4.16667">Adult-onset Leigh’s disease: A rare entity</a>,” was published in the <a href="http://dx.doi.org/10.4103/0972-2327.175437"><em>Annals of Indian Academy of Neurology</em></a><em>. </em>It describes the case of a 37-year-old woman who had long-term abdominal pain and vomiting as well as brain stem syndrome characterized by dizziness, headache, and double vision.<br><br></div><div>Following neurological examination, brain MRI (magnetic resonance imaging) and blood and urine tests, she was provisionally diagnosed with adult-onset Leigh’s disease. When she was treated with a mitochondrial cocktail, she showed dramatic improvement and continued to be free of symptoms after one year.<br><br></div><div>“Mitochondrial cocktail” is a mixture of several vitamins and supplements commonly used by people with mitochondrial disease to decrease symptoms of the condition. The mitochondrial cocktail used in the recent study contained vitamin B1, vitamin B2, coenzyme-Q, L-carnitine and L-arginine.<br><br></div><div>Genetic analyses showed no mutations in the patient’s mitochondrial DNA (mtDNA), which can sometimes cause Leigh’s disease. But the study authors noted: “Recognized mtDNA mutations only account for a small proportion of cases of mitochondrial disease;” some people never get a definitive genetic diagnosis for their condition.<br><br></div><div>Leigh’s disease is a rare but severe neurodegenerative condition that usually affects babies and is diagnosed by the first birthday. The condition is characterized by progressive loss of the ability to move and results in death within two years, usually caused by respiratory failure. In some rare cases, Leigh’s disease appears in adulthood and progresses more slowly.<br><br></div><div>The condition can be caused by a mutation in one of more than 30 different genes. Some of the genes are found in the mitochondria and are involved in energy production. The condition can be inherited or result from a new mutation.<br><br></div><div>Although no proven therapies for Leigh’s disease exist, some case studies and personal observations show improvements following certain therapies.<br><br></div><div><br><br><br><strong><br></strong><br><br></div><div><br></div>]]></description>
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         <pubDate>2018-09-05 00:42:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277797100</guid>
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      <item>
         <title>Jefferson</title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277798052</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 00:48:12 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277798052</guid>
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      <item>
         <title>Jefferson Llamera and Patrick James Lagang</title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277798145</link>
         <description><![CDATA[<div>Grade 8-Mendel<br> </div><div>1.)What is mitochondrial disease?<br>-Mitochondrial diseases are a group of disorders caused by 8dysfunctional mitochondria, the organelles that generate energy for the cell. <br><br>2.)what are the symptoms of mitochondrial disease?<br>-Symptoms include: poor growth, loss of muscle coordination, muscle weakness, visual problems, hearing problems, learning disabilities, heart disease, liver disease, kidney disease, gastrointestinal disorders, respiratory disorders, neurological problems, autonomic dysfunction and dementia.<br><br>3.)who discovered the mitochondrial disease /disoreder?<br>-In 1962, the first patient was diagnosed with a mitochondrial disorder. The researcher (1963), discovered that mitochondria have their own DNA or "blueprint" <br><br>4.)what happens when mitochondria are not working?<br>-If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.<br><br>5.) Give examples of mitochondrial disease?<br>Can it be cured?<br>·Mitochondrial myopathy.<br>·Diabetes mellitus and deafness (DAD) <br>·Leber's hereditary optic neuropathy (LHON)<br>·Leigh syndrome, subacute sclerosing encephalopathy. <br>·Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP) <br>·Myoneurogenic gastrointestinal encephalopathy (MNGIE)<br>-Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them</div><div><br></div><div><strong>Case Study:</strong></div><div>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria and while symptoms vary, they can include poor growth, loss of muscle coordination, muscle weakness, visual problems, hearing problems, learning disabilities, heart disease, liver disease, kidney disease, gastrointestinal disorders, respiratory disorders, neurological problems, autonomic dysfunction, and dementia.<br> ·In 1982 DF was born at term and had no problems until she was 4 months old when it was found she was anemic during an evaluation for fever. She had multiple courses of antibiotics for UTIs and otitis media. She developed a picture of overwhelming sepsis and her lactic acid was elevated at 12 mM. She subsequently went on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome. </div><div><br></div><div>Insights:<br>Mitochondrial disease/disorder can range from mild to fatal. If your mitochondrion is not working properly, it affects your body's ability to convert the food you eat into energy. Thus, tissues that needs more energy such as heart, brain and lungs may not work properly because of mitochondrial disease. It also means that tissues that needs more energy have more active mitochondrial genes, and can only be affected if a person has mitochondrial disorder.</div><div><br></div><div>Source:</div><div><a href="https://theconversation.com/insight-into-mitochondrial-diseases-3073">https://theconversation.com/insight-into-mitochondrial-diseases-3073</a> <br><a href="http://www.circuitblue.com/mito/caseStudies.shtml">http://www.circuitblue.com/mito/caseStudies.shtml</a> (par. 1) </div>]]></description>
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         <pubDate>2018-09-05 00:48:54 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277798145</guid>
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      <item>
         <title>Hamajo, Mika C. 8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277803302</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?<br>* <strong>Mitochondrial diseases</strong> are a group of <strong>disorders </strong>caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<strong>Mitochondria</strong> are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions. <br><br>2.  What are the symptoms of mitochondrial disease?<br>* BRAIN</div><div>Developmental delays | Migraines | Seizures<br>Dementia | Autistic Features | Atypical cerebral palsy<br>Neuro-psychiatric disturbances | Intellectual Disabilities | Strokes</div><div>NERVES</div><div>Weakness (may be intermittent) | Fainting | Absent reflexes<br>Neuropathic pain | Dysautonomia | Temperature Instability</div><div>MUSCLES</div><div>Weakness | Irritable bowel syndrome | Gastroesophogeal reflux<br>Cramping | Diarrhea or constipation | Hypotonia<br>Gastrointestinal problems | Pseudo-obstruction | Dysmotility</div><div>KIDNEYS</div><div>Renal tubular acidosis or wasting</div><div>HEART</div><div>Cardiac conduction defects (heart blocks) | Cardiomyopathy | </div><div>LIVER</div><div>Hypoglycemia (low blood sugar) | Liver failure | </div><div>EARS &amp; EYES</div><div>Visual loss and blindness | Optic atrophy | Acquired strabismus<br>Ptosis | Ophthalmoplegia | Retinitis pigmentosa<br>Hearing loss | Deafness | </div><div>PANCREAS &amp; OTHER GLANDS</div><div>Diabetes | Exocrine Pancreatic Failure | Parathyroid failure</div><div>SYSTEMIC</div><div>Failure to gain weight | Unexplained vomiting | Respiratory problems<br>Fatigue | Short stature | <br><br>3. Who discovered the mitochondrial disease?</div><div>*Much of what we know about these <strong>diseases</strong> has been <strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers <strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4. What happens when mitochondria are not working?<br>*For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria are not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a<strong>problem</strong> and they may become weaker and get tired faster.</div><div><br>5. Give examples of mitochondrial disease. Can it be cured?</div><ul><li><strong>Mitochondrial myopathy</strong>.</li><li><strong>Diabetes mellitus and deafness</strong> (DAD) ...</li><li>Leber's <strong>hereditary optic neuropathy</strong> (<strong>LHON</strong>) ...</li><li><strong>Leigh syndrome</strong>, <strong>subacute sclerosing encephalopathy</strong></li><li><strong>Neuropathy</strong>, ataxia, <strong>retinitis pigmentosa</strong>, and ptosis (<strong>NARP</strong>) </li><li><strong>Myoneurogenic gastrointestinal encephalopathy</strong> (<strong>MNGIE</strong>)</li></ul><div>*Mutations in <strong>mitochondrial</strong> DNA leave cells without enough energy to develop <strong>and </strong>function properly. <strong>Treatments</strong> that <strong>can</strong> ease some of the symptoms or slow the progression of <strong>mitochondrial diseases</strong> do exist, but as yet, none <strong>can cure </strong>them.<br><br>INSIGHTS:<br>From the article I have read, Mitochondrial Disease are diverse and influenced by genetic, environmental, and social-economic factors. Also, the article I have read from the book   help students, physicians, scientists, health care students, and families recognize and accurately diagnose mitochondrial disease and learn about potential treatments. <br><br><br><a href="https://www.sciencedirect.com/science/article/pii/B9780128008775000012">https://www.sciencedirect.com/science/article/pii/B9780128008775000012</a></div>]]></description>
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         <pubDate>2018-09-05 01:20:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277803302</guid>
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      <item>
         <title>Baluso, James &amp; Cadiz, Elias   8-Mend</title>
         <author>zingycactus4808</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277808372</link>
         <description><![CDATA[<div>Question:<br>What is mitochondrial disease</div>]]></description>
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         <pubDate>2018-09-05 01:48:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277808372</guid>
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         <title>Sabelita, Francine Nicole P. &amp; Fajardo, Nikko G.</title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277809707</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 01:56:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277809707</guid>
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         <title>Lorania, Chorena Grace G.  8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277810055</link>
         <description><![CDATA[<ol><li>What is MI</li></ol>]]></description>
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         <pubDate>2018-09-05 01:58:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277810055</guid>
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      <item>
         <title>Sabelita, Francine Nicole P. &amp; Fajardo Nikko </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277812477</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?<br>Mitochondrial disease is the failure to produce an adequate amount of ATP or energy which results in multisystemic disorders.<br><br>2. What are the symptoms of mitochondrial disease?<br><br></div>]]></description>
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         <pubDate>2018-09-05 02:13:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277812477</guid>
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         <title>Juanico, dj Christian A. 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277815226</link>
         <description><![CDATA[<div>1.) <strong>What is mitochondrial disease?</strong><br>Mitochondrial respiratory chain disorders, also known as mitochondrial cytopathies, mitochondrial diseases or oxidative phosphorylation disorders, are a group of clinically heterogeneous inherited metabolic disorders caused by defects in mitochondrial ATP production.<br><br>2.) <strong>What are the symptoms of mitochondrial disease?</strong><br>The main clinical presentations involving central nervous system tissues are ataxia, myoclonia, psychomotor retardation, psychomotor regression, dystonia, muscle weakness, intolerance to physical exercise, sensorineural hearing loss, movement coordination issues, seizures, learning disabilities, optic atrophy, pigmentary retinopathy, ophthalmoplegia, cardiomyopathy, diabetes, autism, growth atrophy, peripheral neuropathy, dementia, and multiple lipomas, in addition to frequent episodes of respiratory issues.<br><br>3.) <strong>Who discover mitochondrial disease?</strong><br>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>&nbsp;</div><div>4.) <strong>What happens when mitochondria are not working?</strong>&nbsp;<br>&nbsp;</div><div>The prognosis for MD is poor, with high mortality rates and premature death. Mortality ranges from 10% to 50%/year after diagnosis, among children, and from 5% to 20%/year after the beginning of clinical treatment among adults. Cases that show good clinical progress are rare. Its shows that if mitochondria will not work there’s a possibility that the prognosis for MD not be cured and all the patients that being diagnose will die.&nbsp;<br>&nbsp;</div><div>5.) <strong>Example of mitochondrial disease can be cure? </strong>&nbsp;</div><div>There’s no mitochondrial diseases that can be cured.&nbsp;<br><br></div><div>Mitochondrial diseases have many, many causes and therefore will have many, many cures. Many drug trials take years -- and treatment advances happen one small step at a time. Which means in the case of mitochondrial diseases that the one cure, one timeline, simply does not exist but with research we will move forward faster.<br>&nbsp;<br>&nbsp;Drug development for mitochondrial disease has only just begun. The road to successful new drugs and drug therapies is riddled with incredible successes and forgotten failures. Recent studies estimate an average timeline to bring a drug to market is 13 years and $1 billion; 75% of the costs of drug development are associated with compounds failing in early stage development. Additionally, 90% of new drugs fail in the second and third phases.<br>&nbsp;The Foundation for Mitochondrial Medicine identifies one barometer of success: supporting and accelerating the development of the most promising mitochondrial research and treatments of the many, many forms of mitochondrial disease.<br>&nbsp;At this point there are only a few drugs for mitochondrial disease that have reached early phases of the FDA drug development approval process. The more resources that are available, the more researchers can augment the path to the cure including an increase in clinical drug trials. The Foundation’s accelerator fuels connections, encourages scientific collaboration, and will bring forth people, ideas and partnerships to set the wheels in motion to develop treatments, sooner and quicker.<br>&nbsp;</div><div>CASE STUDY <br><strong>Mitochondrial disease and augmentative and alternative communication: a clinical case study Doença mitocondrial e comunicação suplementar e alternativa: estudo de caso clínico</strong>&nbsp;<br><br>This study has been approved by the home institution’s Research Ethics Committee as per report no. 1.227.183 CAAE: 48729315.5.0000.5482. The participant’s legal custodians have signed the Free and Informed Consent Term, thus agreeing with the conduction and disclosure of the research and its results.<br>The patient is an 11-year-old boy diagnosed with mitochondrial disease who has been under the care of a specialized genetics clinic since he was one year old, and has since then been taking medications to replace the amino acids his body cannot produce: l-carnitine, thiamine, coenzyme, vitamins C and E, riboflavin and folic acid. He undergoes periodic hearing and vision evaluations, with results within normal standards. He goes to a regular private school and during the study was attending the second and third years of elementary school. The school’s main complaint was about his behavior, especially in regard to his interaction with other students and his aggressiveness, both of which were associated with the patient’s communication difficulties. His communication with his family was based mostly on oral language, with the support of some non-symbolic gestures like pointing12, always associated with vocalizations. His relatives reported having trouble understanding him. The speech-language evaluation found: presence of rare vocalizations or monosyllabic words (as the patient’s phoneme articulation is limited, it is often difficult to understand the word) and use of gestures and body language, with low functionality due to his limited discursive autonomy and communicative intentions, associated with repetitive speech, i.e., he repeats his own speech. All this led to irritability – he would slap his interlocutor or his own head – whenever he was not understood.<br>RESULTS The material analyzed regards the speechlanguage therapy based on the AAC approach during the period between May 2014 and May 2015, in 45-minute sessions conducted once a week. The therapeutic procedures emphasized the social use of language through meaningful, contextualized activities, supported by photos and drawings of daily activities provided by the Picture Communication Symbols (PCS), the system employed by AAC. The activities were chosen according to the patient’s interests, and sometimes he picked the symbol himself. Games, history books and toys (ball, toy cars) were explored, while at the same time presenting the pictures so they might assist the communication. The child used the pictures spontaneously, usually by pointing, but any gesture, such as looking, grabbing, or even biting, was accepted as a response and interpreted orally by the therapist. During the therapeutic process a notebook-shaped communication board was created and used as a means of supporting oral language within different interactional contexts (speech therapy, school and home activities), represented by theme boards (pictures organized according to themes associated to the patient’s most significant experiences). The speech-language therapy included monthly guidelines given to the patient’s educators, with the aim of helping them make use of AAC strategies, thus maximizing the child’s interactions. These strategies basically refer to the presence of pictures in therapy, used the therapist along with speech, and at times spontaneously pointed at or picked up by the patient himself. These pictures were composed of drawings on paper representing activities usually associated with the context. The family approach consisted of individual interviews with the parents, as well as their participation in some sessions. The results of this intervention will be presented in accordance with the following parameters: communicative intention, discursive autonomy, language functionality and non-verbal resources. The use of AAC led to significant changes: after one year of therapy, the patient expanded his vocalizations with the support of AAC symbols, which proved to be effective resources to supplement speech. His communicative intention improved, leading to some discursive autonomy, and, consequently, an increase in the functionality of his spoken language.<br>DISCUSSION In this study, AAC proved effective to establish and sustain the dialogue. However, to make this possible it was necessary to constantly attribute meaning to the patient’s utterances14, which are still linguistically precarious. That is, so as to go beyond mere metalinguistic activities (naming, repeating) and towards a discursive structure aimed at expressing subjective content, even faced with the significant limitations imposed by the patient’s pathology15. Only then may the AAC symbols evolve from being mere signs to acquiring a polysemic, variable and flexible character within the intersubjective context between interlocutors14, stepping away from the conceptionthat overvalues the formal aspects of language to the detriment of its discursive function and effectiveness in interactions15. Thus the use of AAC in the present case became an alternative to low-functionality spoken language, allowing for the development of some discursive autonomy, albeit still precarious. On the other hand, the significant presence of unintelligible elements in the subject’s speech is compatible with the orofacial myofunctional disabilities inherent to the pathology11.&nbsp; However, such limitation did not prevent the dialogue, as the T sought to interpret these segments according to the interactional context, which proved effective, particularly when the AAC was employed16.<br>CONCLUSION The results of the present study indicate that AAC, associated with a non-formalistic conception of language (i.e., that which privileges the expression of subjectivity) has expanded the functionality of the subject’s language by benefitting his discursive practices, especially regarding the communicative intention in the interactional context. Therefore, the authors recommend that further research into ACC focus on and give emphasis to a conception of language that underpins its procedures, which is not typically the case in the existing literature on this issue.<br>&nbsp;</div><div>Authors&nbsp;<br><br>Luciana Maria Wolff-Barnabé(1) Bruna Diógenes(1) Maria Claudia Cunha(2) Regina Maria Ayres de Camargo Freire(2)<br><br><br></div><div><br><br>&nbsp;&nbsp;<br>&nbsp;<br>&nbsp;</div><div>Sources:&nbsp;<br><br></div><div>no. 3 source: http://www.mitoaction.org/medical-information&nbsp;</div><div>No. 5 source: https://www.sharecare.com/health/genetic-disorders/there-cure-for-mitochondrial-diseases&nbsp;<br>http://www.scielo.br/pdf/rcefac/v18n4/en_1982-0216-rcefac-18-04-01001.pdf</div>]]></description>
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         <pubDate>2018-09-05 02:28:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277815226</guid>
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         <title>LoRania, </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277815342</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 02:29:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277815342</guid>
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         <title>Lorania, Chorena Grace G.  8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277816774</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?<br>It is the malfunction of the mitochondria occuring in all cells in the body except red blood cells.<br>2. What are the symptoms of mitochondrial diseases?<br>-developmental delays</div><div>-migraines</div><div>-dementia</div><div>-weakness</div><div>-</div><div>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.ypoglycemia (low blood sugar)</div><div>-ptosis<br>3. Who discovered the mitochondrial disease?<br><br></div>]]></description>
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         <pubDate>2018-09-05 02:36:19 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277816774</guid>
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         <title>Princess Kyla M. Endaya and Ej Carl Banlasan</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277820204</link>
         <description><![CDATA[<div>Grade 8-Mendel<br><br>A.<br>1. Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.<br>2. Poor quality, loss of muscle coordination, seizures, visual and/or hearing problems, heart and liver or kidney disease.&nbsp;<br>3. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963 l, researcher discovered that mitochondria have their own DNA or blueprint (mtDNA).<br>4. If the mitochondria are not working properly then you are less able to convert food into ATP.<br>5. - Mitochuondrial myopathy- there in no cure<br>- Neuropathy ataxia, retinitis pigmentosa, and ptosis (NARP)- there is no cure &nbsp;<br>- Mitochondrial neurogastrointestinal encephalophathy (MNGIE)- there is a cure<br><br>B.<br>1. The case is about the 57 year old man who came from India for evaluation of his cardiac conduction defect. He is also diagnosed with calcifying pancreatitis. He has a elevated lactate, ammonia and CK(MM). An evaluation determined that he had a deficiency in carnitive palmitoyl transterase II activity.<br>2. Non-surgical bowel and the limbs are aching.<br>3. Treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates, along with levo-carnitine, CoQ10 and other vitamins.<br>4.He had no further events since his hospitalization in October 1996.</div>]]></description>
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         <pubDate>2018-09-05 02:54:39 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277820204</guid>
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      <item>
         <title>Lorania, Chorena Grace G.  8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277821189</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?</div><div>Mitochondrial diseases are the result of either inherited or spontaneous mutations in mtDNA or nDNA which lead to altered functions of the proteins or RNA molecules that normally reside in mitochondria. Problems with mitochondrial function, however, may only affect certain tissues as a result of factors occurring during development and growth that we do not yet understand. Even when tissue-specific isoforms of mitochondrial proteins are considered, it is difficult to explain the variable patterns of affected organ&nbsp;<br>systems in the mitochondrial disease syndromes seen clinically.<br><br></div><div>2. What are the symptoms of mitochondrial disease?</div><div>-weakness</div><div>-dementia</div><div>-developmental delays</div><div>-ptosis</div><div>-autism<br><br>3. Who discovered mitochondrial disease?<br>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitocdigestive problems, heart disease, seizures and many other symptoms.hondria have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4. What happens when the mitochondria stops working?</div><div>When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.<br><br>5. What are some examples of nitochondria disease? Can these be cured?</div><div>-Mitochondrial myopathy.</div><div>-Diabetes mellitus and deafness (DAD)&nbsp;</div><div>-Leber's hereditary optic neuropathy (LHON)&nbsp;</div><div>Sadly, none of which is curable.<br><br>INSIGHTS:<br>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.<br>Source: http://www.circuitblue.com/mito/caseStudies.shtml</div>]]></description>
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         <pubDate>2018-09-05 03:00:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277821189</guid>
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         <title>Baluso &amp; Cadiz  8-M</title>
         <author>zingycactus4808</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277821322</link>
         <description><![CDATA[<div>Questions:<br>1. What is mitochondrial disease?<br>-are a group of disorders cause by not functioning properly of mitochondria which generates energy for the cell.<br><br>2. What are the symptoms of mitochondrial disease?<br>- Symptoms can range from fatigue and exercise intolerance to hearing loss, seizures, strokes, heart failure, diabetes and kidney failure.<br><br>3. Who discovered the mitochondrial disease?<br>- No one mentioned the name of the person who discovered it but mitochondrial discovered since 1940 and in 1962, the first patient was diagnosed with a mitochondrial disease.<br><br>4. What happens when mitochondria are not working?<br>-Mitochondria has a big role to our cell, if it will not working properly then mitochondria can't produce energy and your muscles will become weaker and get tired easily. Then the result of this, you will get a mitochondrial disease.<br><br>5. What are some examples of mitochondrial disease? Can these be cured?<br>- Examples of it are alpers disease, autosomal dominant optic atrophy, barth syndrome, beta-oxidation defects, carnitine defiency, and many more. Only few types of mitochondrial disease can be cured such as beta-oxidation defects by dietery modification and carnitine deficiency by carnitine supplementation and other types cannot be cured.<br><br>Case study:<br>A study recently published in the journal Frontiers in Genetics reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA” and was conducted by researchers at The Children’s Hospital of Philadelphia and the Thomas Jefferson University Hospital Philadelphia.<br><br>The study reports the case of a 45-year-old man who presented to The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy. The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.<br><br>Insights:<br>If mitochondria is not functioning then your cell can't receive energy and you will get tired easily and   by this your one of those who has mitochondrial disease <br><br>Sources:<br>https://www.google.com.ph/amp/s/mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/%3famp<br>http://www.umdf.org/types/</div>]]></description>
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         <pubDate>2018-09-05 03:01:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277821322</guid>
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         <title>Lacman L. Mato.   </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277821383</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 03:01:49 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277821383</guid>
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         <title>Baluso and Cadiz   8-Mendel (11:09am)</title>
         <author>zingycactus4808</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277822345</link>
         <description><![CDATA[<div>Questions:<br>1. What is mitochondrial disease?<br>-are a group of disorders cause by not functioning properly of mitochondria which generates energy for the cell.<br><br>2. What are the symptoms of mitochondrial disease?<br>- Symptoms can range from fatigue and exercise intolerance to hearing loss, seizures, strokes, heart failure, diabetes and kidney failure.<br><br>3. Who discovered the mitochondrial disease?<br>- No one mentioned the name of the person who discovered it but mitochondrial discovered since 1940 and in 1962, the first patient was diagnosed with a mitochondrial disease.<br><br>4. What happens when mitochondria are not working?<br>-Mitochondria has a big role to our cell, if it will not working properly then mitochondria can't produce energy and your muscles will become weaker and get tired easily. Then the result of this, you will get a mitochondrial disease.<br><br>5. What are some examples of mitochondrial disease? Can these be cured?<br>- Examples of it are alpers disease, autosomal dominant optic atrophy, barth syndrome, beta-oxidation defects, carnitine defiency, and many more. Only few types of mitochondrial disease can be cured such as beta-oxidation defects by dietery modification and carnitine deficiency by carnitine supplementation and other types cannot be cured.<br><br>Case study:<br>A study recently published in the journal Frontiers in Genetics reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA” and was conducted by researchers at The Children’s Hospital of Philadelphia and the Thomas Jefferson University Hospital Philadelphia.<br><br>The study reports the case of a 45-year-old man who presented to The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy. The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.<br><br>Insights:<br>If mitochondria is not functioning then your cell can't receive energy and you will get tired easily and &nbsp; by this your one of those who has mitochondrial disease&nbsp;<br><br>Sources:<br>https://www.google.com.ph/amp/s/mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/%3famp<br>http://www.umdf.org/types/</div>]]></description>
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         <pubDate>2018-09-05 03:07:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277822345</guid>
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         <title>Mato, Lacman  L.     8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277822401</link>
         <description><![CDATA[<div>Answeres t</div>]]></description>
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         <pubDate>2018-09-05 03:08:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277822401</guid>
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      <item>
         <title>Pette Bernard B. Coscos &amp; Kelly Ashton Cubeta 8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277825157</link>
         <description><![CDATA[<div>08-5-2018,12:14 PM<br>1. What is mitochondrial diseaese?<br>&nbsp;<strong>Mitochondrial diseases</strong> are a group of disorders caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<strong>Mitochondria</strong> are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.&nbsp;<br>2.What are the symptoms of mitochondrial disease?<br>&nbsp;</div><div>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br><br></div><ul><li>Poor growth</li><li>Loss of muscle coordination, muscle weakness</li><li>Neurological problems, seizures</li><li>Autism, autistic spectrum, autistic-like features</li><li>Visual and/or hearing problems</li><li>Developmental delays, learning disabilities</li><li>Heart, liver or kidney disease</li><li>Gastrointestinal disorders, severe constipation</li><li>Diabetes</li><li>Increased risk of infection</li><li>Thyroid and/or adrenal dysfunction</li><li>Autonomic dysfunction</li><li>Neuropsychological changes characterized by confusion, disorien&nbsp;</li></ul><div>3. Who discovered the mitochondrial disease? <br><br>Much of what we know about these <strong>diseases</strong> has been <strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers<strong>discovered</strong> that <strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br><br>4.What happens when mitochondria are not working? <br><br> For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria are not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a <strong>problem</strong> and they may become weaker and get tired faster. <br>5.What are some examples of mitochondrial disease can these be cured?<br><br> Treatment: As with all mitochondrial diseases, there is no cure for Complex I deficiency. A variety of treatments, which may or may not be effective, can include such <strong>metabolic</strong> therapies as: riboflavin, thiamine, biotin, co-enzyme Q10, carnitine, and the ketogenic diet.<br><br><br><br><br><strong><mark>Case study about mitochondrial disease<br><br></mark></strong>&nbsp;<strong>INSIGHTS</strong></div><div>A study recently published in the journal <a href="http://journal.frontiersin.org/journal/genetics"><em>Frontiers in Genetics</em></a> reported on a case report of a man with a new mitochondrial disease associated with the desmin (DES) gene. The study is entitled “<a href="http://journal.frontiersin.org/article/10.3389/fgene.2015.00199/abstract">Desmin common mutation is associated with multi-systemic disease manifestations and depletion of mitochondria and mitochondrial DNA</a>” and was conducted by researchers at <a href="http://www.chop.edu/">The Children’s Hospital of Philadelphia</a>and the <a href="http://hospitals.jefferson.edu/">Thomas Jefferson University Hospital Philadelphia</a>.<br><br></div><div>The study reports the case of a 45-year-old man who presented to <a href="http://www.chop.edu/centers-programs/mitochondrial-genetic-disease-clinic">The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic</a> for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a <strong>mitochondrial myopathy</strong>, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br></div><div>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br></div><div>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.<br><br></div><div>The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.&nbsp;<br><br><br>&nbsp;</div><div>&nbsp;<strong>INSIGHTS</strong>&nbsp;</div><div>If the mitochondria is not working, then the ATP synthesis cannot function very well, so the process will gone into a diseases or body problems.<br><br>&nbsp;</div><div>1. Cardiac, gastrointestinal, neuromuscular and mood disorders - Usually on heart problems. Doctors put pacemaker on the patient to avoid some symptoms like this.<br>2. Distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves) - A <strong>mitochondrial myopathy disorders</strong></div><div>So the team revealed and conducted a research about the diseases involved on that patient and so the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.&nbsp;</div><div><br>Source: <a href="https://mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/">https://mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/</a><br><br><br><br></div>]]></description>
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         <pubDate>2018-09-05 03:29:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277825157</guid>
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         <title>H</title>
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         <link>https://padlet.com/lalaine65biboso/biotech/wish/277825227</link>
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         <pubDate>2018-09-05 03:29:43 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277825227</guid>
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         <title>J</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277825368</link>
         <description><![CDATA[<div>gjHco</div>]]></description>
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         <pubDate>2018-09-05 03:30:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277825368</guid>
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         <title>, lAmjfdbkufs kjrsbkursfsb</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277827539</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 03:46:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277827539</guid>
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         <title></title>
         <author>bchrysjan</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277828132</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/309248224/9e16ea4d2291a24a5f488da50fccf8d1/Benjamin.docx" />
         <pubDate>2018-09-05 03:50:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277828132</guid>
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         <title>Benjamin, Chrys Jan S. 8¬-Aristotle                                      1. What is mitochondrial disease?                  Mitochondrial disease is an inherited chronic illness that can be present at birth or develop later in life. It causes debilitating physical, developmental, and cognitive disabilities with symptoms including poor growth; loss of muscle coordination; muscle weakness and pain; seizures; vision and/or hearing loss; gastrointestinal issues; learning disabilities; and organ failure.  It is estimated that 1 in 4,000 people has Mito. It’s progressive and there is no cure.                                             2. What are the symptoms of mitochondrial diseases?  Developmental delays	Migraines	SeizuresDementia	Autistic Features	Atypical cerebral palsyNeuro-psychiatric disturbances	Intellectual Disabilities	Strokes                3. Who discovered mitochondrial disease/disorder?              Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or &quot;blueprint&quot; (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells&#39; nucleus.       4. What happens when mitochondria is not working ?We are unable to use these directly, but instead need to convert them in to a form of energy that cells can use called ATP. ... For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP.   5. What are some examples of mitochondrial diseases?  Can these be cured?•	LHON•	LHON Plus•	LEIGH’S DISEASE•	PYRUVATE DEHYDROGENASE COMPLEX DEFICIENCY (PDCD/PDH)•	AUTOSOMAL DOMINANT OPTIC ATROPHY (ADOA)   Case Study: Mitochondrial disease presenting as HCMClinical Overview A 26 year-old male presents with hypertrophic cardiomyopathy (left ventricular wall thickness 1.8 cm) on echocardiogram. Full panel testing was ordered for hypertrophic cardiomyopathy (HCM). A homoplasmic mitochondrial DNA mutation (A4300G) was found in the MT-TI gene, which is known to cause maternally inherited HCM. Unlike other mitochondrial mutations which are known to have phenotypic variability, this mutation is currently believed to cause isolated HCM.Diagnostic Summary: POSITIVE. Homoplasmic A4300G mutation in MT-TI. GeneDx tests not only for genes associated with isolated HCM, but also genes associated with multisystem disorders such as amyloidosis, mitochondrial disease, and Fabry disease. Distinguishing the different genetic causes of heart muscle thickening is extremely important, as the treatment for HCM can differ based upon the etiology. The GeneDx 18 gene HCM panel detects mutations in over 60% of patients with a clinical diagnosis of hypertrophic cardiomyopathy. Diagnostic Implications: Mitochondrial disorders are clinically and biochemically very diverse, but typically present in tissues with high energy requirements such as the brain, heart, and skeletal muscle first. In some cases, the patients can present with isolated cardiomyopathy. Genetic testing as part of a clinical evaluation for cardiomyopathy can define the diagnosis, prognosis, and assist in determining appropriate management. Individuals with mitochondrial mutations should be screened for other mitochondrial-related diseases such as myopathy, diabetes, hearing loss, visual impairment, strokes, and cognitive deficitsReferences: 1. Casali C et al. A novel mtDNA point mutation in maternally inherited cardiomyopathy.Biochem and Biophys Res Commun 213(2):588-593, 1995. 2. Taylor R et al. A homoplasmic mitochondrial transfer ribonucleic acid mutation as a cause of maternally inherited hypertrophic cardiomyopathy. J Am CollCardiol. 41(10):1786-1796, 2003.</title>
         <author>bchrysjan</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277828370</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/309248224/9e16ea4d2291a24a5f488da50fccf8d1/Benjamin.docx" />
         <pubDate>2018-09-05 03:51:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277828370</guid>
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         <title>Julianne B. </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277830036</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 04:04:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277830036</guid>
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      <item>
         <title>Pri</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277831803</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-05 04:17:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277831803</guid>
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         <title>Princess Kyla M. Endaya and Ej Banlasan</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277832194</link>
         <description><![CDATA[<div>Grade 8-Mendel<br>A.&nbsp;<br>1. Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.<br>2. Poor growth, loss of muscle coordination, seizures, visual and/or hearing problems, heart and liver or kidney disease.<br>3. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researcher discovered that mitochondria have their own DNA or blueprint (mtDNA).<br>4. If the mitochondria are not working properly then you are less able to convert food into ATP.&nbsp;<br>5. •Mitochondria myopathy- there is no cure<br>•Neuropathy ataxia, refinitis pigmentosa, and ptosis (NARP)- there is no cure<br>•Mitochondrial neurogastrointestinal encephalophathy (MNGIE)- there is a cure.<br><br>Case study:<br>The case is all about the 57 year old man who came from India for evaluation of his cardiac conduction defect. He is also diagnosed with calcifying pancreatitis. He has a elevated lactate , ammonia and CK(MM). An evaluation determined that he had a deficiency in carnitine palmitoyl transterase II activity. The symptoms are non-surgical bowel and the aching of limbs. The treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates along with levo-carnitine, CoQ10 and other vitamins. He had no further events since his hospitalization in October 1996.<br><br>Sources:<br>1.https://en.m.wikipedia.org/wiki/Mitochondrial_disease<br>2.http://www.mitoaction.org/mito-faq<br>3.http://www.mitoaction.org/medical-information<br>4.http://www.newcastle-mitochondria.com/patient-and-public-home-page/general-information-about-mitochondrial-disease/<br>5.https://en.m.wikipedia.org/wiki/Mitochondrial_disease<br><br>Case study source:<br>http://www.circuitblue.com/mito/caseStudies.shtml</div>]]></description>
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         <pubDate>2018-09-05 04:19:50 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277832194</guid>
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         <title>Kristine Joy B.Palmaera and Julianne B. Agorilla 8- Mendel (09/05/18 1:30 pm)</title>
         <author>kristhism</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277833015</link>
         <description><![CDATA[<div><br><strong>A. Questions:<br><br>1. What is mitochondrial disease?<br></strong>Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fails to produce enough energy for the cell or organ function.<br><br><strong>2. What are the symptoms of mitochondrial disease?<br></strong>Common symptoms includes poor growth, loss of muscle coordination, neurological problems/seizures, autism, visual/hearing problems, developmental delays, heart, liver or kidney disease, severe constipation, diabetes and memory loss.<br><br><strong>3. Who discovered the mitochondrial disease?</strong><br>Much of what they know, it has been discovered since 1940, the first patient was diagnosed in 1962. <br><br><strong>4. What happens when mitochondria is not working?<br></strong>As we all know, mitochondria is the powerhouse of the cell and the conversion from food energy to ATP happens here. If mitochondria are not working properly, then you are less able to convert food into ATP. Since muscle requires a lot of ATP, this is a problem and they may become weaker and get tired faster. <br><br><strong>5. What are some examples of mitochondrial disease? Can it be cured?<br></strong>Some examples are Alpers Disease, Autosomal Dominant Optic Atrophy (ADOA), Barth Syndrome/ Lethal Infantible Cardiomyopathy (LIC), Carnitine Deficiency, Friedreich's Ataxia, Kearns-Sayre Syndrome (KSS) and Leigh's Disease. The disease can be present at birth or develop later in life. Treatments can ease some of the symptoms or slow the progression of mitochondrial disease do exist, but as of now, none can cure them.<br><br><strong>Sources:<br></strong><a href="http://www.mitoaction.org/mito-faq">http://www.mitoaction.org/mito-faq</a>, <a href="http://www.umdf.org/types/">http://www.umdf.org/types/</a>, <a href="https://www.medicalnewstoday.com/articles/296836.php">https://www.medicalnewstoday.com/articles/296836.php</a>,&nbsp; <a href="http://www.mitoaction.org/medical-information">http://www.mitoaction.org/medical-information</a><br><br><strong>B. Article: Case Study on Mitochondrial Diseases<br><br>Article:<br></strong>A study recently in the journal <em>Frontier in Genetics </em>reported on a case report&nbsp; of a man with new mitochondrial disease associated with the desmin (DES) gene.&nbsp; The study is entitled <em>“Desmin common mutation is associated with multi-systemic disease manifestation and depletion of mitochondria and mitochondrial DNA"&nbsp;</em>and was conducted by researchers at the Children's Hospital of Philadelphia and the Thomas Jefferson University Hospital Philadelphia.&nbsp;<br><br>The study reports the case of a 45-year old man who presented to The Children's Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a mitochondrial myopathy, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and mitochondrial DNA (of 35%).<br><br>The patient’s family history revealed cardiac conditions and myopathy in several maternal relatives. Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).<br><br>The results are relevant because mutations in the DES gene have not been previously identified as a cause of multi-systemic mitochondrial disease, and symptoms like neuropathy and depletion of both mitochondria and mitochondrial DNA have not been previously associated with desmin-related myopathy.&nbsp;<br>&nbsp;<br>The team concluded that mitochondrial dysfunction occurs in desmin-related myopathy, making it a new mitochondrial disorder. In this way, the team suggests that therapies aimed at improving mitochondrial function could be effective therapeutic strategies to improve the effects associated with the often fatal desmin-related myopathy. Researchers also hypothesize that there might be other neuromuscular diseases caused by mutations in structural proteins that directly interact with the mitochondria.&nbsp;<br><br><strong>Insights:<br></strong>Mitochondria are tiny organelles found in almost every cell in the body. They are known as the "powerhouse of the cell."<br>They are responsible for creating more than 90 percent of cellular energy. They are necessary in the body to sustain life and support growth. They are composed of tiny packages of enzymes that turn nutrients into cellular energy.<br><br>Mitochondrial failure causes cell injury that leads to cell death. When multiple organ cells die there is organ failure. Desmin (DES) is a major muscle scaffolding protein that also functions to anchor mitochondria. Pathogenic DES mutations, however, have not previously been recognized as a cause of multi-systemic mitochondrial disease.<br><br>To cure a mitochondrial disease,<br>The goal is to improve symptoms and slow progression of the disease. Use vitamin therapy, conserve energy, pace activities, maintain an ambient environmental temperature, avoid exposure to illness, ensure adequate nutrition and hydration.<br><br><strong>Source:<br></strong><a href="https://mitochondrialdiseasenews-com.cdn.ampproject.org/v/s/mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/?amp=&amp;usqp=mq331AQCCAE%3D&amp;amp_js_v=0.1#referrer=https%3A%2F%2Fwww.google.com&amp;amp_tf=From%20%251%24s&amp;ampshare=https%3A%2F%2Fmitochondrialdiseasenews.com%2F2015%2F06%2F26%2Fcase-report-new-mitochondrial-disease-linked-desmin-gene%2F">https://mitochondrialdiseasenews-com.cdn.ampproject.org/v/s/mitochondrialdiseasenews.com/2015/06/26/case-report-new-mitochondrial-disease-linked-desmin-gene/?amp=&amp;usqp=mq331AQCCAE%3D&amp;amp_js_v=0.1#referrer=https%3A%2F%2Fwww.google.com&amp;amp_tf=From%20%251%24s&amp;ampshare=https%3A%2F%2Fmitochondrialdiseasenews.com%2F2015%2F06%2F26%2Fcase-report-new-mitochondrial-disease-linked-desmin-gene%2F</a></div>]]></description>
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         <pubDate>2018-09-05 04:25:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277833015</guid>
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      <item>
         <title>Princess Kyla M. Endaya &amp; Ej Banlasan</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277833098</link>
         <description><![CDATA[<div>Grade 8-Mendel<br>A.&nbsp;<br>1. Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell.<br>2. Poor growth, loss of muscle coordination, seizures, visual and/or hearing problems, heart and liver or kidney disease.<br>3. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researcher discovered that mitochondria have their own DNA or blueprint (mtDNA).<br>4. If the mitochondria are not working properly then you are less able to convert food into ATP.&nbsp;<br>5. •Mitochondria myopathy- there is no cure<br>•Neuropathy ataxia, refinitis pigmentosa, and ptosis (NARP)- there is no cure<br>•Mitochondrial neurogastrointestinal encephalophathy (MNGIE)- there is a cure.<br><br>Case study:<br>The case is all about the 57 year old man who came from India for evaluation of his cardiac conduction defect. He is also diagnosed with calcifying pancreatitis. He has a elevated lactate , ammonia and CK(MM). An evaluation determined that he had a deficiency in carnitine palmitoyl transterase II activity. The symptoms are non-surgical bowel and the aching of limbs. The treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates along with levo-carnitine, CoQ10 and other vitamins. He had no further events since his hospitalization in October 1996.<br><br>Sources:<br>1.https://en.m.wikipedia.org/wiki/Mitochondrial_disease<br>2.http://www.mitoaction.org/mito-faq<br>3.http://www.mitoaction.org/medical-information<br>4.http://www.newcastle-mitochondria.com/patient-and-public-home-page/general-information-about-mitochondrial-disease/<br>5.https://en.m.wikipedia.org/wiki/Mitochondrial_disease<br><br>Case study source:<br>http://www.circuitblue.com/mito/caseStudies.shtml</div>]]></description>
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         <pubDate>2018-09-05 04:26:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277833098</guid>
      </item>
      <item>
         <title>Mato, Lacman L.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277836593</link>
         <description><![CDATA[<div>Answers to Questions:<br>1.What is Mitochondrial disease?<br><strong>Mitochondrial diseases</strong> are a group of disorders caused by dysfunctional <strong>mitochondria</strong>, the organelles that generate energy for the cell.<strong>Mitochondria</strong> are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.<br>2.What are the symptoms of mitochondrial disease?<br><strong>The most common symptoms are:</strong></div><ul><li>Poor growth.</li><li>Loss of <strong>muscle</strong> coordination, <strong>muscle weakness</strong>.</li><li><strong>Neurological problems</strong>, seizures.</li><li>Autism, autistic spectrum, autistic-like features.</li><li>Visual and/or <strong>hearing problems</strong>.</li><li>Developmental delays, learning disabilities.</li><li>Heart, liver or kidney disease.</li></ul><div><br></div><div>3.Who discovered mitochondrial diseases or disorders?<br>Much of what we know about these <strong>diseases</strong> has been <strong>discovered</strong> since 1940. In 1962, the first patient was diagnosed with a <strong>mitochondrial disorder</strong>. In 1963, researchers <strong>discovered</strong> that<strong>mitochondria</strong> have their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4.What happens when mitochondria stops working?<br>For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. <strong>If</strong> your<strong>mitochondria</strong> are <strong>not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a <strong>problem</strong> and they may become weaker and get tired faster.<br>5.What are examples of mitochondrial disease? Can this be cured?<br><br></div><ul><li><a href="http://www.umdf.org/types/lhon/"><br>LHON<br></a><br></li><li><a href="http://www.umdf.org/types/lhon-plus/"><br>LHON Plus<br></a><br></li><li><a href="http://www.umdf.org/types/leighs-disease/"><br>LEIGH’S DISEASE<br></a><br></li><li><a href="http://www.umdf.org/types/pyruvate-dehydrogenase-complex-deficiency/"><br>PYRUVATE DEHYDROGENASE COMPLEX DEFICIENCY (PDCD/PDH)<br></a><br></li><li><a href="http://www.umdf.org/types/12332-2/"><br>AUTOSOMAL DOMINANT OPTIC ATROPHY (ADOA)</a></li></ul><div>The <strong>diseases can</strong> be present at birth or develop later in life. ... <strong>Treatments</strong> that <strong>can</strong> ease some of the symptoms or slow the progression of<strong>mitochondrial diseases do</strong> exist, but as yet, none<strong>can cure</strong> them.</div><div><br>ARTICLE AND INSIGHT:<br><br><a href="http://www.circuitblue.com/mito/caseStudies.shtml">http://www.circuitblue.com/mito/caseStudies.shtml</a><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 04:51:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277836593</guid>
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         <title>Howard Godwin Delos Reyes Wendell Angelo Mejares       8-Mendel</title>
         <author>mejaresw54</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277840258</link>
         <description><![CDATA[<div>&nbsp;</div><div><strong>What is mitochondrial disease?</strong>&nbsp;<br><br></div><div>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.&nbsp;<br><br></div><div>&nbsp;<br><br></div><div><strong>What are the Symptoms of Mitochondrial Disease? </strong><br><br></div><div>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are: Poor growth Loss of muscle coordination, muscle weakness Neurological problems, seizures Autism, autistic spectrum, autistic-like features Visual and/or hearing problems Developmental delays, learning disabilities Heart, liver or kidney disease Gastrointestinal disorders, severe constipation Diabetes Increased risk of infection Thyroid and/or adrenal dysfunction Autonomic dysfunction Neuropsychological changes characterized by confusion, disorientation, and memory loss.&nbsp;<br><br></div><div>&nbsp;<br><br></div><div><strong>Who discovered mitochondrial disease or disorders?</strong>&nbsp;<br><br></div><div>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br><br></div><div>(UNIDETIFIED RESEARCHERS DISCOVER MITOCHONDRIAL DISEASES)&nbsp;<br><br></div><div>&nbsp;<br><br></div><div><strong>What happens when Mitochondria disease or diseases?</strong>&nbsp;<br><br></div><div>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster.&nbsp;<br><br></div><div><strong>What are some examples of mitochondrial diseases that these can be cured?</strong>&nbsp;<br><br></div><div>Examples of mitochondrial diseases include: Mitochondrial myopathy. Diabetes mellitus and deafness (DAD) ... Leber's hereditary optic neuropathy (LHON) ... Leigh syndrome, subacute sclerosing encephalopathy. ... Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP) ... Myoneurogenic gastrointestinal encephalopathy (MNGIE)&nbsp;<br><br></div><div><br><br></div><div><strong>Case Study </strong><br><br></div><div>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.&nbsp;<br><br></div><div><br></div><div><em>What is the disorder all about?</em>&nbsp;<br><br></div><div>Its all about galactosemia. The disorder is all about the low production of the second sugar on the body called galactose. the disease disables the capability of body to breakdown the sugar called galactose into a serious threat.&nbsp;<br><br></div><div><em>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp;</em><br><br></div><div><em>What are the symptoms?</em>&nbsp;<br><br></div><div>Symptoms are listed. Depends on the phase of the disease. Common symptoms are&nbsp;<br><br></div><div>Feeding difficulties, lack of energy (lethargy), failure to gain weight.&nbsp;<br><br></div><div><em>Doctors actions.</em>&nbsp;<br><br></div><div>The child was immediately treated by putting glucose on his body. Also have prophylactic antibiotics. Treatments last until few days. Also have a soy-based formula until 3 weeks.&nbsp;<br><br></div><div><em>Doctors Actions results</em>.&nbsp;<br><br></div><div>The child was immediately recovered before a month. But he developed e. coli. Sepsis. A disease that is some quite of friendly but if enters urinary tract will soon developed UTI. Its beneficial sometimes to his health as a galactosemia patient. Despite of it, the child is aliv aside from learning problems and have a lactose free diet.&nbsp;<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 05:23:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277840258</guid>
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      <item>
         <title>Grade 8 Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277850349</link>
         <description><![CDATA[<div>Grejie Lou Mercado<br>Gabrielle Galo<br><br><br><br><br>The parasitic plant manages to go without a component of mitochondria found in all other multicellular life forms.<br>A transgenic approach allows researchers to collect the organelles from specific cells in nematodes with unprecedented efficiency.<br>Adding a fusion gene to certain mitochondria in C. elegans enables researchers to collect and analyze them.<br>Contrary to some hypotheses, the organelles did not descend from any known lineage of Alphaproteobacteria, researchers find.<br>Experiments in mice show that mitochondria, both within the tumor and beyond, can make the difference between promoting or inhibiting cancer spread.<br>Mitochondria may sustain temperatures more than 10 °C warmer than human cells, say researchers.&nbsp;<br><br>1.)What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria<br><br>2.)what are the symptoms of mitochondrial disease?The most common symptoms are:<br>Poor growth.<br>Loss of muscle coordination, muscle weakness.<br>Neurological problems, seizures.<br>Autism, autistic spectrum, autistic-like features.<br>Visual and/or hearing problems.<br><br>3.)who discovered the mitochondrial disease /disorder?<br>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder.&nbsp;<br><br>4.)what happens when mitochondria are not working?<br>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP.&nbsp;<br><br>5.) Give examples of mitochondrial disease?<br>Mitochondrial DNA depletion syndrome<br>Mitochondrial myopathy<br>MELAS syndrome<br>MERRF syndrome<br><br>Can it be cured?<br>The diseases can be present at birth or develop later in life Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Case Study:<br>Numerous cellular changes underlie the decline of muscle mass and strength in the elderly.<br>&nbsp;How Muscles Age, and How Exercise Can Slow It<br>How Muscles Age, and How Exercise&nbsp;<br>Researchers untangle the multifarious nature of muscle aging. So far, the only reliable treatment is exercise.<br>&nbsp;Mitochondrial Infusions Given to Babies with Heart Damage<br>Among 11 infants treated to date, most survived and their heart function improved.<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:30:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277850349</guid>
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         <title>Grade 8 MendeoGree Lou Me</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277852599</link>
         <description><![CDATA[<div>jirc</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:40:18 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277852599</guid>
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         <title>fernandez, nicole d.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277853058</link>
         <description><![CDATA[<div>arduo,tyra<br><br>1. What is mitochondrial disease?<br>*Mitochondrial disease is a chronic, genetic disorder that occurs when the mitochondria of the cell fail to produce enough energy for cell or organ function.<br>&nbsp;2. What are the symptoms of mitochondrial disease?<br>*Poor growth<br>*Loss of muscle coordination, muscle weakness<br>*Neurological problems, seizures<br>*Autism, autistic spectrum, autistic-like features<br>*Visual and/or hearing problems<br>*Developmental delays, learning disabilities<br>*Heart, liver or kidney disease<br>*Gastrointestinal disorders, severe constipation<br>*Diabetes<br>*Increased risk of infection<br>*Thyroid and/or adrenal dysfunction<br>*Autonomic dysfunction<br>*Neuropsychological changes characterized by confusion, disorientation, and memory loss <br>&nbsp;3. Who discovered mitochondrial disease or disorders?<br>* These diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" <br><br>&nbsp;4. What happens when mitochondria are not working?<br>*If your mitochondria are not working properly then you are less able to convert food into ATP. <br>&nbsp;5. What are some examples of mitochondrial disease?<br>*Mitochondrial myopathy<br>*Diabetes mellitus and deafness (DAD)<br>*Leber's hereditary optic neuropathy (LHON)<br>*Leigh syndrome, subacute sclerosing encephalopathy<br>*Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)<br>*Myoneurogenic gastrointestinal encephalopathy (MNGIE)<br>*Myoclonic Epilepsy with Ragged Red Fibers (MERRF)<br>*Mitochondrial myopathy, encephalomyopathy, lactic acidosis, stroke-like symptoms (MELAS) <br><br><br><br>case study:<br>&nbsp;The study reports the case of a 45-year-old man who presented to <a href="http://www.chop.edu/centers-programs/mitochondrial-genetic-disease-clinic">The Children’s Hospital of Philadelphia Mitochondrial-Genetics Diagnostic Clinic</a> for assessment of a suspected mitochondrial disease based on the patient’s symptoms — cardiac, gastrointestinal, neuromuscular and mood disorders. The patient had a pacemaker placed at 30 years of age due to cardiac problems, yet his cardiac function still continued to deteriorate. The individual experienced progressive leg weakness and was no longer able to run by the age of 45. The patient was diagnosed with distal myopathy (muscular disorder) and mild neuropathy (disorder in the peripheral nerves). Muscle biopsy results suggested a <strong>mitochondrial myopathy</strong>, with muscle cells having a pronounced decrease in mitochondrial content (16% compared to the control) and&nbsp; mitochondrial DNA (of 35%).&nbsp;<br>&nbsp; Sequencing studies were unrevealing regarding known deleterious mutations or deletions; however, a particular mutation (p.S13F) in the DES gene was identified by whole exome sequencing. DES is a key scaffolding protein in muscles that anchors mitochondria. The p.S13F mutation is known to be associated with desmin-related myopathy, a condition characterized by muscle weakness, arrhythmias and right ventricular hypertrophic cardiomyopathy (disorder in which the heart muscle function becomes compromised).&nbsp;<br>the disorder is about the heart muscle funcyion becomes compromised.</div><div><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:42:08 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277853058</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277853296</link>
         <description><![CDATA[<div>Grade 8 Mendel<br>Grejie Lou Mercado<br>Gabrielle Tyresse C. Galo<br><br>Questions<br>1.)What is mitochondrial disease?<br>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria<br><br>2.)what are the symptoms of mitochondrial disease?The most common symptoms are:<br>Poor growth.<br>Loss of muscle coordination, muscle weakness.<br>Neurological problems, seizures.<br>Autism, autistic spectrum, autistic-like features.<br>Visual and/or hearing problems.<br><br>3.)who discovered the mitochondrial disease /disorder?<br>Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder.&nbsp;<br><br>4.)what happens when mitochondria are not working?<br>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP.&nbsp;<br><br>5.) Give examples of mitochondrial disease?<br>Mitochondrial DNA depletion syndrome<br>Mitochondrial myopathy<br>MELAS syndrome<br>MERRF syndrome<br><br>Can it be cured?<br>The diseases can be present at birth or develop later in life Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.<br><br>Case Study:<br>Numerous cellular changes underlie the decline of muscle mass and strength in the elderly.<br>&nbsp;How Muscles Age, and How Exercise Can Slow It<br>How Muscles Age, and How Exercise&nbsp;<br>Researchers untangle the multifarious nature of muscle aging. So far, the only reliable treatment is exercise.<br>&nbsp;Mitochondrial Infusions Given to Babies with Heart Damage<br>Among 11 infants treated to date, most survived and their heart function improved.<br>The parasitic plant manages to go without a component of mitochondria found in all other multicellular life forms.<br>A transgenic approach allows researchers to collect the organelles from specific cells in nematodes with unprecedented efficiency.<br>Adding a fusion gene to certain mitochondria in C. elegans enables researchers to collect and analyze them.<br>Contrary to some hypotheses, the organelles did not descend from any known lineage of Alphaproteobacteria, researchers find.<br>Experiments in mice show that mitochondria, both within the tumor and beyond, can make the difference between promoting or inhibiting cancer spread.<br>Mitochondria may sustain temperatures more than 10 °C warmer than human cells, say researchers.&nbsp;<br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:43:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277853296</guid>
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         <title>Grade 8 MendelGrejie Lou MercadoGabrielle Tyresse C. GaloQuestions1.)What is mitochondrial disease?Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria2.)what are the symptoms of mitochondrial disease?The most common symptoms are:Poor growth.Loss of muscle coordination, muscle weakness.Neurological problems, seizures.Autism, autistic spectrum, autistic-like features.Visual and/or hearing problems.3.)who discovered the mitochondrial disease /disorder?Much of what we know about these diseases has been discovered since 1940. In 1962, the first patient was diagnosed with a mitochondrial disorder. 4.)what happens when mitochondria are not working?For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. 5.) Give examples of mitochondrial disease?Mitochondrial DNA depletion syndromeMitochondrial myopathyMELAS syndromeMERRF syndromeCan it be cured?The diseases can be present at birth or develop later in life Treatments that can ease some of the symptoms or slow the progression of mitochondrial diseases do exist, but as yet, none can cure them.Case Study:Numerous cellular changes underlie the decline of muscle mass and strength in the elderly. How Muscles Age, and How Exercise Can Slow ItHow Muscles Age, and How Exercise Researchers untangle the multifarious nature of muscle aging. So far, the only reliable treatment is exercise. Mitochondrial Infusions Given to Babies with Heart DamageAmong 11 infants treated to date, most survived and their heart function improved.The parasitic plant manages to go without a component of mitochondria found in all other multicellular life forms.A transgenic approach allows researchers to collect the organelles from specific cells in nematodes with unprecedented efficiency.Adding a fusion gene to certain mitochondria in C. elegans enables researchers to collect and analyze them.Contrary to some hypotheses, the organelles did not descend from any known lineage of Alphaproteobacteria, researchers find.Experiments in mice show that mitochondria, both within the tumor and beyond, can make the difference between promoting or inhibiting cancer spread.Mitochondria may sustain temperatures more than 10 °C warmer than human cells, say researchers. </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277853476</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:43:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277853476</guid>
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         <title>Safwan A. Kano.  8-Mendel</title>
         <author>Bloopbleeep426</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277856721</link>
         <description><![CDATA[<div>ANSWERS TO QUESTIONS:<br>1. <strong>What is mitochondrial disease?</strong><br>- Mitochondrial diseases are long-term, genetic, often inherited disorders that occur when mitochondria fail to produce enough energy for the body to function properly.<br>2.<strong>What are the symptoms</strong>?<br> - muscle weakness/exercise intolerance<br> - heart failure<br> - dementia<br> - movement disorders<br> - stroke-like episodes<br> - deafness<br> - blindness<br> - vomiting<br> - Diabetes<br> - Increased risk of infection<br> - Neurological problems, seizures<br>3.<strong>Who discovered mitochondrial disease?</strong><br> -In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondriahave their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4. <strong>What happens when mitochondria is not working?</strong><br>-If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is aproblem and they may become weaker and get tired faster.<br>5.<strong>What are some examples of mitochondrial disease? Can this be cured?</strong><br><br>• Kearns-Sayre Syndrome<br>• MELAS<br>• MERRF<br>• PEO<br>• Pearson Syndrome<br>• Leigh encephalopathy<br>• NARP<br>The Mitochondrial Diseases cannot be cured but it can be prevented.<br><br><strong>INSIGHTS</strong><br> <br>Mitochondria is a very important part for the cell especially in energy production. It is so useful to our body. If It will be damaged It may cause a lot of different Mitochondrial Diseases that cannot be cured but it can be prevented.<br><br><strong>Case Study<br><br></strong>The patient is an 11-year-old boy diagnosed with mitochondrial disease who has been under the care of a specialized genetics clinic since he was one year old, and has since then been taking medications to replace the amino acids his body cannot produce: l-carnitine, thiamine, coenzyme, vitamins C and E, riboflavin and folic acid. He undergoes periodic hearing and vision evaluations, with results within normal standards. He goes to a regular private school and during the study was attending the second and third years of elementary school. The school’s main complaint was about his behavior, especially in regard to his interaction with other students and his aggressiveness, both of which were associated with the patient’s communication difficulties. His communication with his family was based mostly on oral language, with the support of some non-symbolic gestures like pointing12, always associated with vocalizations. His relatives reported having trouble understanding him. The speech-language evaluation found: presence of rare vocalizations or monosyllabic words (as the patient’s phoneme articulation is limited, it is often difficult to understand the word) and use of gestures and body language, with low functionality due to his limited discursive autonomy and communicative intentions, associated with repetitive speech, i.e., he repeats his own speech. All this led to irritability – he would slap his interlocutor or his own head – whenever he was not understood<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 06:59:33 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277856721</guid>
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         <title>Lorania, Chorena Grace G. 8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277858379</link>
         <description><![CDATA[<div>Lorania, Chorena Grace G. 8-ARISTOTLE</div><div><br></div><div>1. What is the mitochondrial disease?</div><div>Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells).</div><div>2. What are the symptoms of a mitochondrial disease?</div><div>Autistic Features</div><div>Dysautonomia</div><div>Diarrhea or constipation</div><div>Renal tubular acidosis or wasting</div><div>Cardiomyopathy</div><div>Hypoglycemia (low blood sugar)</div><div>Visual loss and blindness</div><div>Exocrine Pancreatic Failure</div><div>Failure to gain weight</div><div><br></div><div>3. Who discovered the mitochondrial disease?</div><div>(this answer answers the question how instead of who) </div><div>In 1962, the first patient was diagnosed with a mitochondrialdisorder. In 1963, researchers discovered that mitochondriahave their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.</div><div><br></div><div>4. What happens when the mitochondria stops working?</div><div>When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.</div><div>5. What are the examples of mitochondrial diseases? Can these be cured?</div><div>LHON</div><div>LHON Plus</div><div>LEIGH’S DISEASE</div><div>PYRUVATE DEHYDROGENASE COMPLEX DEFICIENCY (PDCD/PDH)</div><div>AUTOSOMAL DOMINANT OPTIC ATROPHY (ADOA)</div><div>Sadly, these diseases cannot be cured.</div><div><br></div><div>INSIGHTS:</div><div><br></div><div>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.</div><div><br></div><div>Dr. Cohen “Finding A Cure for Mitochondrial Diseases” http://www.circuitblue.com/mito/caseStudies.shtml .</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-09-05 07:07:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277858379</guid>
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      <item>
         <title>Lorania, Chorena Grace G. 8-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277859085</link>
         <description><![CDATA[<div>Lorania, Chorena Grace G. 8-ARISTOTLE</div><div><br></div><div>1. What is the mitochondrial disease?</div><div>Mitochondrial diseases result from failures of the mitochondria, specialized compartments present in every cell of the body (except red blood cells).</div><div>2. What are the symptoms of a mitochondrial disease?</div><div>Autistic Features</div><div>Dysautonomia</div><div>Diarrhea or constipation</div><div>Renal tubular acidosis or wasting</div><div>Cardiomyopathy</div><div>Hypoglycemia (low blood sugar)</div><div>Visual loss and blindness</div><div>Exocrine Pancreatic Failure</div><div>Failure to gain weight</div><div><br></div><div>3. Who discovered the mitochondrial disease?</div><div>(this answer answers the question how instead of who)&nbsp;</div><div>In 1962, the first patient was diagnosed with a mitochondrialdisorder. In 1963, researchers discovered that mitochondriahave their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.</div><div><br></div><div>4. What happens when the mitochondria stops working?</div><div>When the energy supply slumps, cells can become damaged or destroyed. ... Depending on which cells are affected, people with mitochondrial diseases may have muscle weakness and pain, digestive problems, heart disease, seizures and many other symptoms.</div><div>5. What are the examples of mitochondrial diseases? Can these be cured?</div><div>LHON</div><div>LHON Plus</div><div>LEIGH’S DISEASE</div><div>PYRUVATE DEHYDROGENASE COMPLEX DEFICIENCY (PDCD/PDH)</div><div>AUTOSOMAL DOMINANT OPTIC ATROPHY (ADOA)</div><div>Sadly, these diseases cannot be cured.</div><div><br></div><div>INSIGHTS:</div><div><br></div><div>1984 A 7 day old boy was transferred to our hospital for jaundice. He was born at term without problems. He began regular infant feeds and ate will initially. He became jaundice at 3 days of life and was placed under bililights. He became increasingly lethargic and developed diarrhea. He was placed in an incubator for hypothermia, despite bundling him in blankets. He continued to bottle feed, despite his increasing lethargy. His bilirubin was as high as 15 with about 50% conjugated. On arrival the transport team checked his urine for reducing substances (positive) and glucose (negative). A tentative diagnosis of galactosemia was made and subsequently confirmed. He was treated with IV glucose and prophylactic antibiotics for a few days and recovered. He was started on a soy-based formula and did well until 3 weeks of life when he developed e. coli. sepsis. At this time (1999) he is alive and aside from learning problems, doing well on a lactose free diet.</div><div><br></div><div>Dr. Cohen “Finding A Cure for Mitochondrial Diseases” http://www.circuitblue.com/mito/caseStudies.shtml .</div>]]></description>
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         <pubDate>2018-09-05 07:10:19 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277859085</guid>
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         <title>Fajartin, Lrome  Facurib, jhonbert</title>
         <author>lrome_fajartin1</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277869634</link>
         <description><![CDATA[<div><strong>What is mitochondrial disease?<br></strong><br></div><div><strong>Mitochondrial diseases are chronic (long-term), genetic, often inherited disorders that occur when mitochondria fail to produce enough energy for the body to function properly. (Inherited means the disorder was passed on from parents to children.) Mitochondrial diseases can be present at birth, but can also occur at any age.</strong></div><div><strong><br>Mitochondrial diseases can affect almost any part of the body, including the cells of the brain, nerves, muscles, kidneys, heart, liver, eyes, ears or pancreas.<br></strong><br></div><div><strong>&nbsp;<br></strong><br></div><div><strong>What are the symptoms of mitochondrial disease?<br></strong><br></div><div><strong>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are:<br></strong><br></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Poor growth</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Loss of muscle coordination, muscle weakness</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Neurological problems, seizures</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Autism, autistic spectrum, autistic-like features</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Visual and/or hearing problems</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Developmental delays, learning disabilities</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Heart, liver or kidney disease</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Gastrointestinal disorders, severe constipation</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Diabetes</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Increased risk of infection</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Thyroid and/or adrenal dysfunction</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Autonomic dysfunction</strong></div><div>·&nbsp; &nbsp; &nbsp; &nbsp; &nbsp;<strong>Neuropsychological changes characterized by confusion, disorientation, and memory loss.</strong></div><div><strong>&nbsp;<br></strong><br></div><div><strong>Who discovered mitochondrial disorder?<br></strong><br></div><div><strong>when Lars Ernster and Rolf Luft in Stockholm described a patient who ate voraciously yet stayed thin, sweating profusely even in winter.<br></strong><br></div><div><strong>What happens when mitochondrial disease?<br></strong><br></div><div><strong>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is aproblem and they may become weaker and get tired faster.<br></strong><br></div><div><strong>What are some examples of mitochondrial disease? Can these be cured?<br></strong><br></div><div><strong>Alpers Disease<br></strong><br></div><div><strong>Autosomal Dominant Optic Atrophy (ADOA)<br></strong><br></div><div><strong>Barth Syndrome / LIC (Lethal Infantile Cardiomyopathy)<br></strong><br></div><div><strong>Systemic Primary Carnitine Deficiency<br></strong><br></div><div><strong>Long Chain Fatty Acid Transport Deficiency<br></strong><br></div><div><strong>Carnitine Palmitoyl Transferase I (CPT-I) Deficiency<br></strong><br></div><div><strong>Carnitine/Acylcarnitine Translocase Deficiency<br></strong><br></div><div><strong>Carnitine Palmitoyl Transferase II (CPT-II) Deficiency<br></strong><br></div><div><strong>&nbsp; &nbsp; &nbsp; &nbsp; &nbsp; &nbsp; Very Long-Chain Acyl-CoA Dehydrogenase (VLCAD) Deficiency<br></strong><br></div><div><strong>Long-Chain 3-Hydroxyacyl-CoA Dehydrogenase (LCHAD) Deficiency &nbsp;<br></strong><br></div><div><strong>Mitochondrial Trifunctional Protein (TFP) Deficiency<br></strong><br></div><div><strong>Medium-Chain Acyl-CoA Dehydrogenase (MCAD) Deficiency<br></strong><br></div><div><strong>Short-Chain Acyl-CoA Dehydrogenase (SCAD) Deficiency<br></strong><br></div><div><strong>Glutaric Aciduria Type II<br></strong><br></div><div><strong>Short-Chain 3-Hydroxyacyl-CoA Dehydrogenase (SCHAD) Deficiency<br></strong><br></div><div><strong>Short/Medium-Chain 3-Hydroxyacyl-CoA Dehydrogenase (S/MCHAD)<br></strong><br></div><div><strong>Medium-Chain 3-Ketoacyl-CoA Thiolase (MCKAT) Deficiency<br></strong><br></div><div><strong>2,4-Dienoyl-CoA Reductase Deficiency<br></strong><br></div><div><strong>No,&nbsp; but treatment can help reduce symptoms or slow the decline in health.<br></strong><br></div><div><strong>&nbsp;<br></strong><br></div><div><strong>&nbsp;<br>case study:<br></strong>S.K. 57 year old man came from India for evaluation of his cardiac conduction defect. His family history is significant for his mother dying at a young age of a cardiomyopathy, and that his brothers have similar problems to his. He was the president of a successful business in India. As a young man he was diagnosed with calcifying pancreatitis, and had been on replacement digestive enzymes for years. He has had numerous admissions for non-surgical bowel obstruction and has had several exploratory laporatomies, the etiology was never determined. Over the last 15 years he lost 75 pounds, and developed aching in his limbs. In January 1996 he was admitted for a stroke, and a cardiac evaluation determined he was in A-fib. He was placed on Coumadin. He was readmitted for a stroke several months later, and was again found to be in A-fib. His neurologist and cardiologist referred him to CCF Cardiology for placement of a pacemaker. In October 1996, after a 24 hour journey, he visited our cardiology department, and was told that a pacemaker was not indicated. While resting in the waiting room, trying to gather the energy to walk back to the hotel, he collapsed. He spent the next two weeks in a stupor in the NICU, and again, another stroke was detected, along with his A-fib. The patient was found to have an elevated lactate, ammonia and CK (MM). Polarography revealed decreased oxidation of substrates that donate reducing equivulants to complex I and beta oxidation. An evaluation determined that he had a deficiency in carnitine palmitoyl transferase II activity. Treatment was started and included a low fat diet with frequent meals rich in complex carbohydrates, along with levo-carnitine, CoQ10 and other vitamins. He has had no further events since his hospitalization in October 1996.In order for the mitochondria to burn fats, the free fatty acid must first enter the mitochondrial inner membrane. CPT I catalyzes the conversion of the activated free FA (acyl CoA) + carnitine to the acyl-carnitine. A carnitine translocase exchanges the acylcarnitine across the inner membrane for a free carnitine molecule. CPT II catalyzes the conversion of the acyl-carnitine to the acyl-CoA and free carnitine. The acyl-CoA can then enter the beta-oxidation spiral. CPT II deficiency usually results in exercise intolerance, muscle cramping and fatigue in young adults, but is also known to cause an early cardiomyopathy.<br><br>Disease:calcifying pancreatitis<br><br>symptoms:stroke<br><br>doctor action:Polarography&nbsp;<br><br>result of the doctor action:<br>&nbsp;</div><div>He has had no further events since his hospitalization in October 1996&nbsp;</div><div><br><br></div>]]></description>
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         <pubDate>2018-09-05 08:09:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277869634</guid>
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         <title></title>
         <author>lrome_fajartin1</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277874504</link>
         <description><![CDATA[He has had no further events since his hospitalization in October 1996.I]]></description>
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         <pubDate>2018-09-05 08:32:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277874504</guid>
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         <title>Sabelita, Francine P. &amp; Fajardo, Nikko G.</title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277898565</link>
         <description><![CDATA[<div><strong>1. What is mitochondrial disease?</strong><br>• Mitochondrial disease is when the mitochondria fail to function normally that becomes more severe with age.<br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>•The symptoms are blindness, deafness, dementia, movement disorders, weakness, cardiac failure, diabetes, renal dysfunction, and liver disease.<br><strong>3. Who discovered mitochondrial disease or disorders?</strong><br>• The early report that identified a mitochondrial disease was in 1962 by Rolf Luft's group at Karolinska University in Stockholm, Sweden.<br><strong>4. What happens when mitochondria are not working?</strong><br>• When mitochondria are not working and fails to produce energy this may result to affecting high metabolism tissues like the nervous systems, muscles, kidneys, and livers.<br><strong>5. What are examples of mitochondrial diseases? Can these be cured?</strong><br>•Ragged red fibers, Luft's disease, Leber's Heredity Optic Neuropathy (LHON disease) are examples of mitochondrial disease. There are no cures for these diseases but treatment options mostly involves alleviating the symptoms of the disease as opposed to curing the disease itself.<br><br><strong>Case Study</strong>:&nbsp;<br>Mitochondrial diseases in humans result when the small organelles called mitochondria, which exist in all human cells, fail to function normally that becomes more severe with age. The symptoms are blindness, deafness, dementia, movement disorders, weakness, cardiac failure, diabetes, renal dysfunction, and liver disease. When the mitochondria are not working and fails to produce energy this may result to affecting high metabolism tissues like the nervous systems, muscles, kidneys, and livers.<br>Ragged red fibers, Luft's disease, Leber's Heredity Optic Neuropathy (LHON disease) are examples of mitochondrial disease. There are no cures for these diseases but treatment options mostly involves alleviating the symptoms of the disease as opposed to curing the disease itself.</div>]]></description>
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         <pubDate>2018-09-05 10:08:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277898565</guid>
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         <title>Sabelita, Francine P. &amp; Fajardo, Nikko G.</title>
         <author>francinenicolesabelita</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277900713</link>
         <description><![CDATA[<div><strong>1. What is mitochondrial disease?</strong><br>• Mitochondrial disease is when the mitochondria fail to function normally that becomes more severe with age.<br><strong>2. What are the symptoms of mitochondrial disease?</strong><br>•The symptoms are blindness, deafness, dementia, movement disorders, weakness, cardiac failure, diabetes, renal dysfunction, and liver disease.<br><strong>3. Who discovered mitochondrial disease or disorders?</strong><br>• The early report that identified a mitochondrial disease was in 1962 by Rolf Luft's group at Karolinska University in Stockholm, Sweden.<br><strong>4. What happens when mitochondria are not working?</strong><br>• When mitochondria are not working and fails to produce energy this may result to affecting high metabolism tissues like the nervous systems, muscles, kidneys, and livers.<br><strong>5. What are examples of mitochondrial diseases? Can these be cured?</strong><br>•Ragged red fibers, Luft's disease, Leber's Heredity Optic Neuropathy (LHON disease) are examples of mitochondrial disease. There are no cures for these diseases but treatment options mostly involves alleviating the symptoms of the disease as opposed to curing the disease itself.<br><br><strong>Case Study</strong>: <br>Mitochondrial diseases in humans result when the small organelles called mitochondria, which exist in all human cells, fail to function normally that becomes more severe with age. The symptoms are blindness, deafness, dementia, movement disorders, weakness, cardiac failure, diabetes, renal dysfunction, and liver disease. When mitochondria are not working and fails to produce energy this may result to affecting high metabolism tissues like the nervous systems, muscles, kidneys, and livers.<br>Ragged red fibers, Luft's disease, Leber's Heredity Optic Neuropathy (LHON disease) are examples of mitochondrial disease. There are no cures for these diseases but treatment options mostly involves alleviating the symptoms of the disease as opposed to curing the disease itself.<br><br>Source: https://embryo.asu.edu/pages/mitochondrial-diseases-humans</div>]]></description>
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         <pubDate>2018-09-05 10:21:32 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277900713</guid>
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         <title>Arcilla, Hannah M. and  Omawing, Merryl C. Grade 8-Mendel</title>
         <author>janetpelaez87</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277907591</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?<br>Mitochondrial diseases are group of disorders caused by dysfunctional mitochondria,It is an inherited chronic illness that results the cell Fail to produce enough energy for cell production or to function.<br>2. What are the symptoms of mitochondrial disease?<br>The symptoms are:<br>*Autonomic dysfunction<br>*Neurological problems<br>*Autism<br>*Seizures<br>*Kidney disease<br>*Poor eyesight<br>*Diabetes<br>*Heart failures<br>*Increased risk of infection<br>3. Who discovered Mitochondrial disease or disorder?<br>Mitochondrial diseases has been discovered since 1940. The first patient was diagnosed with a mitochondrial disorder in 1962 then the researchers found out that the mitochondria have their own "blueprint " or mtDNA . (1963)<br>4. What happens when mitochondria are not working?<br>If the mitochondria will malfunction the organs will start to fail and you're less able to convert food into ATP.<br>5. What are some examples of mitochondrial diseases?<br>*Alpers disease<br>*Barth syndrome<br>*Fatty Acid Oxidation Disorders<br>*Carnitine Deficiency Syndrome<br>*Creatine Deficiency Syndrome<br>*Chronic progressive External Ophthalmoplegia<br>*Lactic acidosis<br>*Leigh Syndrome<br>Can these be cured ?<br>Some of these disorders are curable. There are treatments that can be used to treat some mitochondrial diseases.<br><br>Case Study :<br>A case study of a mitochondrial disease by Dr. Cohen<br><br>In 1982 DF was born at term and had no problems until she was four months old when it was found out she was anemic during an evaluation for fever . She had multiple courses of antibiotics for UTIs and Otitis media . She developed a picture of overwhelming sepsis and and her lactic acid was elevated at 12mM . She subsequently went on to have have a chronic sideroblastic anemia , pancreatic failure, cardiac dysfunction, hearing loss ,ptosis,myophaty&nbsp;and retinal degeneration. She died at the age of 15 from cardiac failure . Her mtDNA showed a typical deletion seen Kearn Sayre Syndrome.</div>]]></description>
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         <pubDate>2018-09-05 10:59:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277907591</guid>
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         <title>Cutamora, Mizpah Gabrielle S. 8-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277908026</link>
         <description><![CDATA[<div>1. What is mitochondrial disease?<br>- <strong>Mitochondrial disease</strong>, also called <strong>mitochondrial disorder</strong>, any of several hundred hereditary conditions that result from a functional failure of the <a href="https://www.britannica.com/science/mitochondrion">mitochondrion</a>, a type of cellular <a href="https://www.britannica.com/science/organelle">organelle</a>. Mitochondrial diseases can emerge at any age and are enormously <a href="https://www.merriam-webster.com/dictionary/diverse">diverse</a> in their clinical and molecular features. They range in severity from relatively mild <a href="https://www.britannica.com/science/disease">disease</a> that affects just a single <a href="https://www.britannica.com/science/organ-biology">organ</a>to debilitating and sometimes fatal illness that affects multiple organs. The wide spectrum of symptoms poses significant challenges to the <a href="https://www.merriam-webster.com/dictionary/diagnosis">diagnosis</a> of conditions associated with mitochondrial dysfunction. At least 1 in every 5,000 persons worldwide is affected by mitochondrial disease. <br>2. What are the symptoms of a mitochondrial disease?<br>- Signs and symptoms of mitochondrial disease vary, depending on the organ or organ systems involved. Possible indications include developmental delay, reduced growth, <a href="https://www.britannica.com/science/fatigue-physiology">fatigue</a>, <a href="https://www.britannica.com/science/migraine-pathology">migraine</a>, <a href="https://www.britannica.com/science/muscle">muscle</a> weakness, muscle pain, <a href="https://www.britannica.com/science/cardiomyopathy">cardiomyopathy</a>, liver failure, <a href="https://www.britannica.com/science/blindness-medicine">blindness</a>, <a href="https://www.britannica.com/science/optic-atrophy">optic atrophy</a> (degeneration of the optic nerve), <a href="https://www.britannica.com/science/deafness">hearing loss</a>, <a href="https://www.britannica.com/science/diabetes-mellitus">diabetes</a>, and seizures. Clusters of signs and symptoms often emerge, indicating a discrete syndrome. For example, in individuals of age three months to two years, failure to thrive, progressive neurological degeneration (with decreased muscle tone, uncoordinated movement, and involuntary and repetitive muscle contraction), and visual, respiratory, and cardiac problems are characteristic of Leigh syndrome. Kearns-Sayre syndrome, on the other hand, is characterized primarily by progressive weakness or paralysis of the <a href="https://www.britannica.com/science/human-eye">eye</a> muscles and retinopathy (damage to the light-sensing <a href="https://www.britannica.com/science/retina">retina</a> of the eye), which can result in drooping eyelids and a loss of vision; onset typically is before age 20.<br>3. Who discovered mitochondrial diseases?<br>-In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondriahave their own DNA or "blueprint" (mtDNA), which is different than the nuclear DNA (nDNA) found in the cells' nucleus.<br>4. What happens when mitochondria are not working?<br>-For our bodies the conversion from food energy to ATP <strong>happens</strong> in <strong>mitochondria</strong>. If your <strong>mitochondria are not working</strong> properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a<strong>problem</strong> and they may become weaker and get tired faster<br>5. What are some examples of mitochondrial diseases? Can this be cured?<br>-Alper's Disease, Autosomal Dominant Optic Athropy (ADOA), Barth Syndrome, Carnitine Deficiency, Creatine Deficiency Syndromes, Chronic Progressive External Opththalmoplegia (CPEO), Friedreich's Ataxia.<br>-There are no cures for mitochondrial diseases, but treatment can help reduce symptoms, or delay or prevent the progression of the disease.<br>Treatment is individualized for each patient, as doctors specializing in metabolic diseases have found that every child and adult is "biochemically different." That means that no two people will respond to a particular treatment in a specific way, even if they have the same disease.</div>]]></description>
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         <pubDate>2018-09-05 11:02:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277908026</guid>
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         <title>Dejillo, Lanz S.</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277912955</link>
         <description><![CDATA[<div>8-Aristotle<br><br>1)What is mitochondrial disease?<br>-Mitochondrial diseases are disorders caused by the dis-function of mitochondria. It is the organelles that generate energy for the cell.<br>2)What are the symptoms of mitochondrial diseases?<br>-*Weakness<br>*Fainting<br>*Absent reflexes<br>*Developmental delays<br>*Migraines<br>*Seizures<br>*Cramping<br>*Diarrhea or constipation<br>3)Who discovered the mitochondrial disease/disorder?<br>- In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint".<br>4)What happens when mitochondria are/is not working?<br>-Your organs will be weak and your body wont get some energy to be used.<br>5)What are some examples of mitochondrial disease? Can this be cured?<br>- *Mitochondrial myopathy<br>*Diabetes mellitus and deafness<br>*Leber's hereditary optic neuropathy<br>*Leigh syndrome, subacute sclerosing encephalopathy<br>*Neuropathy, ataxia, retinitis pigmentosa, and ptosis<br>*Myoneurogenic gastrointestinal encephalopathy <br>-Treatments could slowdown the symptoms but it cannot be cured.<br><br>Case Study:<br>A 7 month old female child, 2<sup>nd </sup>product of second degree consanguineous marriage, with an uneventful perinatal history presented to our hospital with status epilepticus, delayed developmental milestones and regression of the achieved milestones. On initial examination, she was unconscious (Glassgow Coma Scale-5) and afebrile. Initial management aimed at controlling the seizures with Diazepam (i.v., 0.3 mg/kg stat) and Phenytoin (i.v., 20 mg/kg stat followed by 5 mg/kg per 12 hourly). Following control of seizures the child went into decerebrate posturing. The raised intracranial tension was treated with Mannitol (i.v., 5 mg stat). Her pulse was 154 beats per minute, respiratory rate 36 cycles per minute and blood pressure 84/46 mm Hg. Her weight was 5 kg and height 62 cms. CNS examination showed increased tone in the lower limbs. Deep tendon reflexes were exaggerated with bilateral Babinski sign. Pupils were dilated and sluggishly reacting to light. Fundus examination and visual evoked potentials were normal. After an hour of admission, she became apnoeic and was put on ventilator. The above clinical findings were highly suggestive of a neurodegenerative disorder and the patient was further investigated.<br><br></div><div><br>Routine haemogram revealed haemoglobin 8.8 gm%, packed cell volume 28.6%, total leucocyte count 26,800 cells per mm<sup>3 </sup>with marked neutrophilia (85%) and lymphocyte count 10%. Cerebrospinal fluid examination showed 4 cells, all lymphocytes and normal sugar and protein levels. CSF lactate was significantly raised (8.8 mmol/L). Gram and ZN staining of the CSF showed no organism and pus cells. Serum lactate (6.8 mmol/L) and creatinine kinase (320 U/L) levels were abnormally raised. Liver function test showed mild derangement with AST- 54 IU/L, ALT- 49 IU/L and ALP- 109 IU/L. Renal function test was within normal limits. Arterial blood gas analysis indicated metabolic acidosis. Blood and urine cultures were negative. Magnetic Resonance Imaging was done which showed bilateral, symmetrical abnormal lesions in the basal ganglia, thalami, cerebral peduncles, dorsal medulla and peri aqueductal grey matter which were hyperintense in T2W, FLAIR and DW images (Figure <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2949674/figure/F1/">1A, B</a> &amp;<a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2949674/figure/F1/">1C</a>). There were prominent extracerebral CSF spaces in the fronto-temporo-parietal region on both the sides and showed the similar signal characteristics (Figure <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2949674/figure/F1/">​(Figure1D).1D</a>). Frontal atrophy with myelination normal for age was noticed. Above radiological findings on MRI established the clinical diagnosis of a neurodegenerative disorder as Leigh syndrome.<br><br></div><div><br><br></div>]]></description>
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         <pubDate>2018-09-05 11:30:12 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277912955</guid>
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         <title>8-Mendel</title>
         <author>leibuladaco12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277916541</link>
         <description><![CDATA[<div> Pertimos, Ashley and Buladaco, Lei Marie  (7:50 pm) <br><br><br></div><div><strong>1.</strong>      <strong>What is a Mitocondrial  Disease?</strong> </div><div> </div><div>Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. It could be inherited chronic illness that could present after birth. </div><div> </div><div><strong>2.</strong>      <strong>What are the Symptoms of Mitochondrial Disease?</strong> </div><div>The severity of mitochondrial disease symptoms is different from person to person. The most common symptoms are: </div><div>Poor growth </div><div>Loss of muscle coordination, muscle weakness </div><div>Neurological problems, seizures </div><div>Autism, autistic spectrum, autistic-like features </div><div>Visual and/or hearing problems </div><div>Developmental delays, learning disabilities </div><div>Heart, liver or kidney disease </div><div>Gastrointestinal disorders, severe constipation </div><div>Diabetes </div><div>Increased risk of infection </div><div>Thyroid and/or adrenal dysfunction </div><div>Autonomic dysfunction </div><div>Neuropsychological changes characterized by confusion, disorientation, and memory loss </div><div>. </div><div><strong>3.</strong>      <strong>Who discovered mitochondrial disease or disorders?</strong> </div><div> </div><div>In 1962, the first patient was diagnosed with a mitochondrial disorder. In 1963, researchers discovered that mitochondria have their own DNA or "blueprint" (mDNA), which is different than the nuclear DNA (n\DNA) found in the cells' nucleus. </div><div> </div><div><strong>4.</strong>      <strong>What happens when mitochondria are not working</strong>? </div><div> </div><div>For our bodies the conversion from food energy to ATP happens in mitochondria. If your mitochondria are not working properly then you are less able to convert food into ATP. For cells that require a lot of ATP, for example your muscles, this is a problem and they may become weaker and get tired faster. </div><div> </div><div><strong>5.      What are the examples of Mitocondrial Disease? Can it be cure?</strong> <br><br></div><div>Examples of mitochondrial diseases include: <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Mitochondrial_myopathy"><em>Mitochondrial myopathy</em></a> <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Diabetes_mellitus_and_deafness"><em>Diabetes mellitus and deafness</em></a>(DAD) <br><br></div><div>o    this combination at an early age can be due to mitochondrial disease <br><br></div><div>o    <a href="https://en.wikipedia.org/wiki/Diabetes_mellitus"><em>Diabetes mellitus</em></a> and <a href="https://en.wikipedia.org/wiki/Deafness"><em>deafness</em></a> can be found together for other reasons <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Leber%27s_hereditary_optic_neuropathy"><em>Leber's hereditary optic neuropathy</em></a> (LHON) <br><br></div><div>o    visual loss beginning in young adulthood <br><br></div><div>o    eye disorder characterized by progressive loss of central vision due to degeneration of the optic nerves and retina <br><br></div><div>o    affects 1 in 50,000 people in Finland <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Leigh_syndrome"><em>Leigh syndrome</em></a>, subacute sclerosing encephalopathy <br><br></div><div>o    after normal development the disease usually begins late in the first year of life, although onset may occur in adulthood <br><br></div><div>o    a rapid decline in function occurs and is marked by seizures, altered states of consciousness, dementia, ventilatory failure <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Neuropathy,_ataxia,_retinitis_pigmentosa,_and_ptosis"><em>Neuropathy, ataxia, retinitis pigmentosa, and ptosis</em></a>(<a href="https://en.wikipedia.org/wiki/Neuropathy,_ataxia,_and_retinitis_pigmentosa">NARP</a>) <br><br></div><div>o    progressive symptoms as described in the acronym <br><br></div><div>o    <a href="https://en.wikipedia.org/wiki/Dementia">dementia</a> <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Myoneurogenic_gastrointestinal_encephalopathy"><em>Myoneurogenic gastrointestinal encephalopathy</em></a> (MNGIE) <br><br></div><div>o    gastrointestinal pseudo-obstruction <br><br></div><div>o    <a href="https://en.wikipedia.org/wiki/Neuropathy">neuropathy</a> <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/Myoclonic_Epilepsy_with_Ragged_Red_Fibers"><em>Myoclonic Epilepsy with Ragged Red Fibers</em></a>(MERRF) <br><br></div><div>o    progressive myoclonic epilepsy <br><br></div><div>o    "Ragged Red Fibers" are clumps of diseased mitochondria that accumulate in the subsarcolemman region of the muscle fiber and appear when muscle is stained with modified <a href="https://en.wikipedia.org/wiki/G%C3%B6m%C3%B6ri_trichrome_stain">Gömöri trichrome stain</a> <br><br></div><div>o    short stature <br><br></div><div>o    hearing loss <br><br></div><div>o    lactic acidosis <br><br></div><div>o    exercise intolerance <br><br></div><div>·         <a href="https://en.wikipedia.org/wiki/MELAS"><em>Mitochondrial myopathy, encephalomyopathy, lactic acidosis</em>, stroke-like symptoms</a> (MELAS) <br><br></div><div>·     <br>    <a href="https://en.wikipedia.org/w/index.php?title=MtDNA_depletion&amp;action=edit&amp;redlink=1"><em>mtDNA depletion</em></a><em> </em><br><br></div><div>o    <a href="https://en.wikipedia.org/wiki/Mitochondrial_neurogastrointestinal_encephalomyopathy"><em>mitochondrial neurogastrointestinal encephalomyopathy</em></a> (<a href="https://en.wikipedia.org/wiki/MNGIE">MNGIE</a>) <br><br></div><div> </div><div>Mutations in mitochondrial DNA leave cells without enough energy to develop and<strong> </strong>function properly. Treatments that can ease some of the symptoms or slow the progression of <em>mitochondrial</em><strong> </strong><em>diseases</em> do exist, but as yet, none can<strong> </strong>cure<strong> </strong>them. </div><div> </div><div> </div><div><strong>Case Study </strong></div><div> </div><div>If the organelles of the Mitocondria will not work properly that becomes more severe with age. That exist in human cells will be a Mitocondrial Disease. blindness, deafness, dementia, movement disorders, weakness, cardiac failure, diabetes, renal dysfunction, and liver disease are one of the symptoms of Mitocondrial Disease. DF was born without no problems in terms. Until she is 4 months old she was concluded anemic when evaluation for fever. She subsequentlywent on to have a chronic sideroblastic anemia, pancreatic failure, cardiac dysfunction, hearing loss, ptosis, myopathy and retinal degeneration. She died at the age of 15 from cardiac failure. Her mtDNA showed a typical deletion seen in Kearn Sayre syndrome. <br><br>Source: <a href="http://www.circuitblue.com/mito/caseStudies.shtml">http://www.circuitblue.com/mito/caseStudies.shtml</a></div><div> </div><div> </div><div> </div><div> <br><br></div>]]></description>
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         <pubDate>2018-09-05 11:44:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277916541</guid>
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         <title></title>
         <author>leibuladaco12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277917355</link>
         <description><![CDATA[subsarcolemmal]]></description>
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         <pubDate>2018-09-05 11:47:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277917355</guid>
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         <title>Leana Jan Riz N. Valdez, Allyza Joy Cerado (8-Mendel) (9: 20 P. M. Wednesday )</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/277917720</link>
         <description><![CDATA[<div>Ans. In no 5 is that some of this disease can be treated and some are not. <br><strong>Article/Case study</strong>:<br> This article is all about a 37 year old women who has diagnosed with a disease called adult onset leigh's disease. This symptoms shows abdominal pain,&nbsp; vomiting, brain stem syndrome charcterized by dizziness, headache and double vision. She was treated with a mitochondrial cocktail.<br> Mitochondrial cocktail is a mixture of several vitamins and supplements. It contain a vitamins B1, B2, coenzyme - Q, L - carmitine and L - arginine. <br> The results that she&nbsp;showed is that there was a dramatic improvements and continued to be free of symptoms after 1 year. <br><br><strong>Insights:<br><br></strong>&nbsp;I have learned that some diseases like the disease called onset leigh's disease can be treated with Mitochondrial cocktail (vitamins B1, B2, coenzyme - Q, L - carmitine and L - arginine). <br>&nbsp;If I were the doctor I will do the same. <br><br><strong>Source: https://mitochondrialdiseasenews.com&gt;a...</strong></div>]]></description>
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         <pubDate>2018-09-05 11:48:09 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/277917720</guid>
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         <title>Activity for Aristotle and Mendel</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/287293240</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-09-30 08:30:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/287293240</guid>
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         <title>GROUP 6- ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302422994</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/302843477/da4d09a7f1bcab2f5489b0b3374caf53/Go_Up_and_go_down.docx" />
         <pubDate>2018-11-09 04:20:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302422994</guid>
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         <title>GROUP 5 -</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302426726</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 04:58:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302426726</guid>
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         <title>gr</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302426727</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 04:58:55 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302426727</guid>
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         <title>GROUP 5 -ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302426776</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 04:59:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302426776</guid>
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         <title>GROUP 4- ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302426790</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 05:00:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302426790</guid>
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         <title>GROUP 5 -ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302426946</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296104059/7906ce0c218728424bb24933f0944f0d/SALINITY_EXPERIMENT.docx" />
         <pubDate>2018-11-09 05:02:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302426946</guid>
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         <title>Group 5-</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428375</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 05:20:25 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428375</guid>
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         <title>Group 3-Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428379</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296100250/f5b294656489be460739ddbac0700f96/BIOTECH_EXPERIMENT.docx" />
         <pubDate>2018-11-09 05:20:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428379</guid>
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         <title>group 3 aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428700</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-11-09 05:25:24 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428700</guid>
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         <title>Mendel - Group 6</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428720</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/298129699/61a4741a239b6552669d8e30f30b0944/biotech.docx" />
         <pubDate>2018-11-09 05:25:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428720</guid>
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         <title>Mendel Group - 6</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428784</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-11-09 05:26:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428784</guid>
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         <title>w</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302428865</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-09 05:28:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302428865</guid>
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         <title>GROUP 4-ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302429456</link>
         <description><![CDATA[<div>Members:<br>Cabiles, Zyrreg Zyhller <br>Berame, Bernadette Sue B.<br>Alberto, Achilles Alexis R.<br>Cutamora, Mizpah Gabrielle <br>Natividad, Deniel Dave</div>]]></description>
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         <pubDate>2018-11-09 05:36:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302429456</guid>
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         <title>groipu</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302429762</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-11-09 05:39:53 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302429762</guid>
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         <title>Group-4 Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302430411</link>
         <description><![CDATA[<div>Member<br>Hamajo, Mika<br>Seromines, Zofia Amina<br>Malunes, Jaesar Justin<br>Benjamin, Chrys Jan<br>Deleverio, Alvin Jay<br><br><br></div>]]></description>
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         <pubDate>2018-11-09 05:47:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302430411</guid>
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         <title>Group 2-Mendel</title>
         <author>jeff_bros</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302430526</link>
         <description><![CDATA[<div>members:<br>James Baluso<br>Jefferson Llamera<br>Kristine Palmaera<br>Elias Cadiz<br>Merryl Omawing<br><br></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295892210/aa5f1f70be4af08011d141ab10197eb2/Biotech.docx" />
         <pubDate>2018-11-09 05:48:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302430526</guid>
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         <title>GROUP 1- ARISTOTLE</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302431014</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/332093125/288d768202bfa6228fb913b263c42aed/COME_AND_SIP_ME.docx" />
         <pubDate>2018-11-09 05:54:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302431014</guid>
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         <title>Group 6 Aristot</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302523365</link>
         <description><![CDATA[<div>Ca<br><br><br><br><br><br><br></div>]]></description>
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         <pubDate>2018-11-09 12:43:50 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302523365</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/302525651</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/332181873/80dac9b705a722a278f77bcd2117f960/Biotechnology.mp4" />
         <pubDate>2018-11-09 12:51:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/302525651</guid>
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      <item>
         <title>Group 5 Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303109450</link>
         <description><![CDATA[<div>Group 5<br>Stack it up!<br>I. Introduction<br>      Do you want to stack up colored liquid materials?<br>Density is a measurement that compares the amount of matter on an object has to it's volume.<br>We know that every liquid has its own density, with that liquids are stacked up. In this activity you  can understand the science of stacking up liquids .<br>II. Objective<br>          1. To understand the science of stacking up liquids<br>          2. To successfully stack up liquids with different densities<br>II.  Materials<br>Empty bottle<br>Pipette<br>Beam Balance<br>Graduated Cylinder<br>Dishwashing Liquid<br>Water<br>Vegetable oil<br>Rubbing Alcohol<br>Food Coloring (we used Orange  and Blue)<br>IV. Procedure<br>Prepare the liquid materials to be used.<br>Get an empty bottle and measure its liquid capacity. (600mL)<br>Measure the amount of liquids to be poured. (150 mL each liquid)<br>Pour the dishwashing liquid.<br>Before pouring the next liquid which is the colored water, slightly tilt the bottle.<br>Do it again when pouring the other liquids.<br>V. Questions<br>1. What liquid is the densest?<br>      The dishwashing liquid is the densest.<br>2. What liquid is the least dense?<br>      The colored rubbing alcohol is the least dense.<br>3. Why do you need to know the density of the liquids beforey stacking them?<br>     We need to know the standard densities of the liquids used to be able to arranged them.<br>4.Why do you think the liquid should be arranged from the densest to dense?<br>     We need to arrange the liquids from densest to dense so that the liquids will not mixing each other.<br>5. Why are the liquids not mixing each other?<br>The liquids are not mixing each other because of their stacking arrangement which is from densest to dense.</div>]]></description>
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         <pubDate>2018-11-12 04:57:30 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303109450</guid>
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      <item>
         <title>Mendel Kr</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303109702</link>
         <description><![CDATA[<div>Group 5<br>Stack it up!<br>I. Introduction<br>      Do you want to stack up colored liquid materials?<br>Density is a measurement that compares the amount of matter on an object has to it's volume.<br>We know that every liquid has its own density, with that liquids are stacked up. In this activity you  can understand the science of stacking up liquids .<br>II. Objective<br>          1. To understand the science of stacking up liquids<br>          2. To successfully stack up liquids with different densities<br>II.  Materials<br>Empty bottle<br>Pipette<br>Beam Balance<br>Graduated Cylinder<br>Dishwashing Liquid<br>Water<br>Vegetable oil<br>Rubbing Alcohol<br>Food Coloring (we used Orange  and Blue)<br>IV. Procedure<br>Prepare the liquid materials to be used.<br>Get an empty bottle and measure its liquid capacity. (600mL)<br>Measure the amount of liquids to be poured. (150 mL each liquid)<br>Pour the dishwashing liquid.<br>Before pouring the next liquid which is the colored water, slightly tilt the bottle.<br>Do it again when pouring the other liquids.<br>V. Questions<br>1. What liquid is the densest?<br>      The dishwashing liquid is the densest.<br>2. What liquid is the least dense?<br>      The colored rubbing alcohol is the least dense.<br>3. Why do you need to know the density of the liquids beforey stacking them?<br>     We need to know the standard densities of the liquids used to be able to arranged them.<br>4.Why do you think the liquid should be arranged from the densest to dense?<br>     We need to arrange the liquids from densest to dense so that the liquids will not mixing each other.<br>5. Why are the liquids not mixing each other?<br>The liquids are not mixing each other because of their stacking arrangement which is from densest to dense.</div>]]></description>
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         <pubDate>2018-11-12 04:59:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303109702</guid>
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         <title>Group 2-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303541175</link>
         <description><![CDATA[<div>members:<br>Lei Buladaco<br>Julianne Agorilla<br>Patrick James Lagang<br>Howard Godwin Delos Reyes<br>Lucky Mato<br>Gabrielle Galo</div>]]></description>
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         <pubDate>2018-11-13 01:59:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303541175</guid>
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         <title>Group 2</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303542712</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-13 02:08:00 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303542712</guid>
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         <title>Group 2-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303543249</link>
         <description><![CDATA[<div>members:<br>Lei Buladaco<br>Julianne B. Agorilla<br>Patrick James Lagang<br>Lucky Mato<br>Howard Godwin Delos Reyes<br>Gabrielle Galo</div>]]></description>
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         <pubDate>2018-11-13 02:10:45 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303543249</guid>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303543541</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/333114734/51da994409d8d1d53f71e6e1eedf83d8/Untitled_document.docx" />
         <pubDate>2018-11-13 02:12:20 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303543541</guid>
      </item>
      <item>
         <title>Princess kyla m. E</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303648949</link>
         <description><![CDATA[<div>Princess </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 10:44:26 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303648949</guid>
      </item>
      <item>
         <title>Princess Kyla M. Endaya 8-Mendel Group 3</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303649752</link>
         <description><![CDATA[<div>     MEASURE AND WEIGH ME<br><br>II INTRODUCTION<br>        In this activity you will know how to use beam balance, graduated cylinder and pipette. With the help of the laboratory instrument, we can easily measure the different kinds of liquids like softdrink  and oil. The pipette is use in measuring small amount of liquids. The beam balance is use to weigh different objects. Graduated Cylinder is also used to measure liquid.<br>III MATERIALS <br>            Graduated Cylinder   Beam balance.      salt<br>            Vinegar      water     pipette      beaker       <br>IV PROCEDURE <br> 1.Prepare all the materials nedded.                                                                                        <br>2.put a small amout of vinegar and water in the 2  different graduated cylinder. then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3.Put 10 grams of salt  to the 2 different graduated cylinde<br>4. After 1 minute remove the water and vinegar  to the other container.            .                                                                                                                  5.  Use the pipette to remove vinegar or water if there are small parts of it.                                      <br>6.place the container  with vinegar in the beam balance for it to get the mass.        <br>7.After measuring the container with vinegar  place the container with water in the beam balance .<br>8.record  the results.<br>Results:<br>Vinegar<br>Mass before: 75 grams<br>Mass after: 85 grams<br><br>Water<br>Mass before: 75 grams<br>Mass after: 85 grams<br>Explanation:<br>This experiment is <br><br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 10:47:17 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303649752</guid>
      </item>
      <item>
         <title>Ej Carl U. Banlasan 8 MENDEL Group 3</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303654522</link>
         <description><![CDATA[<div>Measure and Weigh Me<br><br>II.  Introduction<br>              -  In this activity you will know how to use beam balance , graduated cylinder and pipette. With the help of the laboratory instrument, we can easily measure the different kinds of liquids like softdrink  and oil. The pipette is use in measuring small amount of liquids. The beam balance is use to weigh different objects. Graduated Cylinder is also used to measure liquid.<br><br>III.  Materials <br>         -  Graduated Cylinder <br>         -  Beam Balance<br>         -  Salt<br>         -  Vinegar<br>         -  Water<br>         -  Pipette<br>         -  Beaker<br><br>IV.  Procedure<br><br>1.  Prepare all the materials needed.<br>2.  Put a small amount of vinegar and water in the 2  different graduated cylinder. then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3.Put 10 grams of salt  to the 2 different graduated cylinder.<br>4. After 1 minute remove the water and vinegar  to the other container.            .                              5.  Use the pipette to remove vinegar or water if there are small parts of it.                                      <br>6.  Place the container  with vinegar in the beam balance for it to get the mass.        <br>7.After measuring the container with vinegar  place the container with water in the beam balance .<br>8.record  the results.<br><br>V.  Questions<br>1.  What is the mass of vinegar and water before the salt is added ?<br>              -  The mass of vinegar and water before the salt is added is the same. 75 grams.<br>2.  What is the mass of vinegar and water after the salt is added ?<br>              -  The mass of vinegar and water after the salt is added is the increased by 10, so the mass is 85 grams.<br>3. Is law of conservation applied to it ? <br>                -  Yes<br>4.  Is the mass of vinegar and water change after the salt is added ?<br>                - Yes<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 11:02:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303654522</guid>
      </item>
      <item>
         <title>rincess Kyla </title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303655913</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 11:07:34 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303655913</guid>
      </item>
      <item>
         <title>Princess Kyla M. Endaya Group 3-Mendel</title>
         <author>princesskylaendaya</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303656753</link>
         <description><![CDATA[<div>MEASURE AND WEIGH ME<br><br>II INTRODUCTION<br>        In this activity you will know how to use beam balance, graduated cylinder and pipette. With the help of the laboratory instrument, we can easily measure <br>the different kinds of liquids like softdrink  and oil. The pipette is use in measuring small amount of liquids. The beam balance is use to weigh different objects. Graduated Cylinder is also used to measure liquid.<br>III MATERIALS <br>            Graduated Cylinder   Beam balance.      salt<br>            Vinegar      water     pipette      beaker       <br>IV PROCEDURE <br> 1.Prepare all the materials nedded.                                                                                        <br>2.put a small amout of vinegar and water in the 2  different graduated cylinder. then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3.Put 10 grams of salt  to the 2 different graduated cylinde<br>4. After 1 minute remove the water and vinegar  to the other container.            .                                                                                                                  5.  Use the pipette to remove vinegar or water if there are small parts of it.                                      <br>6.place the container  with vinegar in the beam balance for it to get the mass.        <br>7.After measuring the container with vinegar  place the container with water in the beam balance .<br>8.record  the results.<br>Results:<br>Vinegar<br>   Mass before: 75 grams<br>   Mass after: 85 grams<br>Water<br>   Mass before: 75 grams<br>   Mass after: 85 grams<br><br>Explanation:<br>This experiment is connected to the law of conservation of mass because the mass of the vinegar and water in before and after putting the salt is the same.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 11:10:23 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303656753</guid>
      </item>
      <item>
         <title>Aira Shyn Leigh P. Vergara 8-Mendel</title>
         <author>renzabogadil</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303666968</link>
         <description><![CDATA[<div>                      <br>     MEASURE AND WEIGH ME<br><br>II INTRODUCTION<br>        In this activity you will know how to use beam balance, graduated cylinder and pipette. With the help of the laboratory instrument, we can easily measure the different kinds of liquids like softdrink  and oil. The pipette is use in measuring small amount of liquids. The beam balance is use to weigh different objects. Graduated Cylinder is also used to measure liquid.<br>III MATERIALS <br>            Graduated Cylinder   Beam balance.      salt<br>            vinegar              water     pipette      beaker       <br>IV PROCEDURE <br> 1.Prepare all the materials nedded.                                                                                        <br>2.put a small amout of vinegar and water in the 2  different graduated cylinder. then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3.Put 10 grams of salt  to the 2 different graduated cylinde<br>4. After 1 minute remove the water and vinegar  to the other container.            .                                                                                                                  5.  Use the pipette to remove vinegar or water if there are small parts of it.                                      <br>6.place the container  with vinegar in the beam balance for it to get the mass.        <br>7.After measuring the container with vinegar  place the container with water in the beam balance .<br>8.record  the results.<br><br><br> <br>Vinegar<br> 75 - Mass Before<br> 85-Mass After<br><br>Water<br>75- Mass Before<br>85-Mass After<br><br><br>Observations:<br>The volume of 2 liquids did not change the mass after we conducted the activity was added 10 grams it is because of the 10 grams salt we added. Also the salt dissolve faster in water  than in vinegar<br><br><br><br>Questions and Answers<br><br>Q1. what happen when you pour the salt in the 2 graduated cylinder with water and vinegar.<br>          - The salt dissolve in the 2 different graduated cylinder when we stir it<br>Q2. What is the use of beam.balance on your activity?<br>          - The use of beam balance in our activity is to measure the mass begore and after we conducted the activity<br>Q3. What is the volume of the water before adding the salt?<br>       - The volume of the water before we add salt is 75mL<br>Q4. What is the mass of the vinegar after the activity?<br>      - The mass of the vinegar is 85 grams<br>Q5.What is the use of the pipette in your activity?<br>        - The use of pipette is to transfer or get small amout of liquid.<br>Q6. What is the concept of your activity?<br>        -Low of the conservation of mass<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 11:44:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303666968</guid>
      </item>
      <item>
         <title></title>
         <author>mejaresw54</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303672940</link>
         <description><![CDATA[<div><strong>Group 4 Mendel</strong></div>]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/295999563/e48b2d359ca80a03791e8519648bc100/biotech.docx" />
         <pubDate>2018-11-13 12:06:54 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303672940</guid>
      </item>
      <item>
         <title>Tyra Arduo 8-Mendel (Group 3)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303675128</link>
         <description><![CDATA[<div>MEASURE AND WEIGH ME<br><br><strong>I. Introduction</strong><br>In this activity you will know how to use beam balance,graduated cylinder,and pipette. With the help of the laboratory instrument , we can easily measure the different kinds of liquids like vinegar and water. The pipette is used in measuring small amounts of liquids. The beam balance is use to weigh different objects. Graduated cylinder is also used to measure liquid.<br><strong>II. Materials<br></strong>-Graduated cylinder<br>-vinegar<br>-beam balance<br>-water<br>-pipette<br>-salt<br>-beaker<br><strong>III. Procedures<br></strong>1. Prepare all the materials needed.<br>2. Put a small amount of vinegar and water in the 2 different graduated cylinder. Then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3. Put 10 grams of salt to the 2 diff. grad. cylinder.<br>4. After 1 min. remove the water and vinegar to the water to the other container.<br>5. Use the pipette to remove vinegar or water if there are small parts of it.<br>6. Place the container with vinegar in the beam balance for it to get the mass. Same aslo with the water.<br>7. Record the results.<br><br>Vinegar <br>*mass before-75g<br>*mass after-85g<br>Water<br>*mass before -75g<br>*mass after-85g<br><br>Questions and answers:<br>1. What is the purpose of the pipette in this activity?<br>-To remove vinegar or water if there are small parts of it.<br>2. What did you observe on the mass of vinegar and water?<br>-The mass before and after of the vinegar and water is constant.<br>3. What is the mass before of vinegar?<br>-75g<br>4. What is the mass after of vinegar?<br>-85g<br>5. What concept did you relate to this activity?<br>-Law of conservation of mass.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 12:14:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303675128</guid>
      </item>
      <item>
         <title>Jhonbert U. Facurib   Grade 8 - Mendel     Group 3 (individual)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303691703</link>
         <description><![CDATA[<div>MEASURE AND WEIGH ME<br><br>II INTRODUCTION<br>        In this activity you will know how to use beam balance, graduated cylinder and pipette. With the help of the laboratory instrument, we can easily measure the different kinds of liquids like softdrink  and oil. The pipette is use in measuring small amount of liquids. The beam balance is use to weigh different objects. Graduated Cylinder is also used to measure liquid.<br>III MATERIALS <br>            Graduated Cylinder   Beam balance.      salt<br>            vinegar            w ater     pipette      beaker  <br>IV PROCEDURE <br>1.Prepare all the materials needed.                                  2.put a small amout of vinegar and water in the 2  different graduated cylinder. then measure the mass of each material that was placed in the 2 different graduated cylinder.<br>3.Put 10 grams of salt  to the 2 different graduated cylinder<br>4. After 1 minute remove the water and vinegar  to the other container.    <br>5.place the container  with vinegar in the beam balance for it to get the mass.        <br>6.After measuring the container with vinegar  place the container with water in the beam balance .<br>7.record  the results.<br>Vinegar<br> 75 - Mass Before<br> 85-Mass After<br>Water<br>75- Mass Before<br>85-Mass After<br>Observations:<br>The volumes of both water and vinegar increased a little as we put 10 grams of salt each to both the water and the vinegar. The salt also dissolved faster when put into the vinegar than with the water because of the acidic property of the vinegar. Following the Law of the Conservation of Mass, as both of the water and vinegar had the same volume and mass, as we added 10 grams of salt, the Result after we added salt was just the mass of the liquid (solvent: 75 grams) + mass of the solute (10 grams). The result after we added salt each to both liquids is 85 if we would follow the law of Conservation of Mass, just the same as our presented data which is still 85.<br>Questions and Answers:<br><br>Q1. what happens when you put the salt in the 2 graduated cylinder with water and vinegar.<br>          - The salt dissolves in the 2 different graduated cylinder when we put salt in it.<br>Q2. What is the use of beam.balance on your activity?<br>          - The use of beam balance in our activity is to measure the mass begore and after we conducted the activity<br>Q3. What is the mass of the water before adding the salt?<br>       - The volume of the water before we add salt is 75 grams<br>Q4. What is the mass of the vinegar after salt was added ?<br>      - The mass of the vinegar is 85 grams<br>Q5.What is the use of the pipette in your activity?<br>        - The use of pipette is to transfer or get small amout of liquid.<br>Q6. What is the concept of your activity?<br>        -Law of the conservation of mass<br><br>The literature review that would support that the law of Conservation of Mass is our Concept in the statement "the mass of any one element at the beginning of a reaction will equal the mass of that element at the end of the reaction. As the variables' Mass in this activity stayed the same .<br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 12:59:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303691703</guid>
      </item>
      <item>
         <title></title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303704864</link>
         <description><![CDATA[<div>Mercado,Grejie Lou G.           8-Mendel<br>Group 3(Individual)<br>      Measure and Weigh Me<br><br>II. INTRODUCTION<br>            In this activity you will know how to use beam balance,graduated cylinder and pipette.With the help of the laboratory instrument,we can easily measure the different kinds of liquids like softdrink and oil.The pipette is used in measuring small amount of liquids.The beam balance is use to weigh different objects.Graduated cylinder is also used to measure liquid.<br><br>IIi. MATERIALS<br> Graduated cylinder<br>Beam balance <br>Salt<br>Vinegar<br>Water<br>Pipette<br>Beaker<br><br>IV. PROCEDURE<br>Prepare all the materials needed.<br>Measure the mass of the graduated cylinder.<br>Put 75mL of vinegar and water into two different graduated cylinder,then measure the mass of each material that was placed in the two graduated cylinder.<br>After getting the mass,subtract the mass of the 75mL vinegar from the mass of graduated cylinder,then record,do the same thing in getting the final mass of 75mL water.<br>Put 10g of salt into the 75mL vinegar,and do the same thing to the other,wait for 1 MINUTE.(Do not stir)<br>After 1 minute,get the mass of the two separately for the second time.<br>Record the results.<br>   <br><br>mass before adding salt<br>mass after adding salt<br><br>Vinegar<br>               75g<br>                   85g<br><br>Water<br>            75g<br>                   85g   <br><br><br>Variables:1)Increase of mass<br>                 2)Amount of salt<br>Concept:<br>Law of Conservation of mass<br>-The law of conservation of mass or principle of mass conservation states that for any system closed to all transfers of matter and energy, the mass of the system must remain constant over time, as system's mass cannot change, so quantity cannot be added nor removed. Hence, the quantity of mass is conserved over time.<br>Simple Understanding:The mass stays the same<br><br>Questions:<br>1)What is the solute in the activity?<br>Answer:Salt<br>2)What are the solvent in the said activity?<br>Answer:Vinegar and Water<br>3)What is the fourth step in the procedure?<br>Answer:After getting the mass,subtract the mass of the 75mL vinegar from the mass of graduated cylinder,then record,do the same thing in getting the final mass of 75mL water.<br>4)How much is the increase of mass?<br>Answer:10g<br>5)What conclusion can you conclude in the said activity?<br>Answer:The salt and the mass of the vinegar and water are directly proportional,the greater the amount of salt added,the greater the increase in mass.</div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-13 13:28:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303704864</guid>
      </item>
      <item>
         <title></title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303706620</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/296147650/8de3879aa3847786d594f98fb0d5d448/Measure_and_Wei.doc" />
         <pubDate>2018-11-13 13:32:12 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303706620</guid>
      </item>
      <item>
         <title>Mercado,Grejie Lou 8-Mendel</title>
         <author>mercadogrejielou12</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303706730</link>
         <description><![CDATA[]]></description>
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         <pubDate>2018-11-13 13:32:27 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303706730</guid>
      </item>
      <item>
         <title>lrome, fajartin 8- mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/303754859</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/333294587/6631177aef5d17efef2681813b46516b/biotech.docx" />
         <pubDate>2018-11-13 14:45:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/303754859</guid>
      </item>
      <item>
         <title>Mendel and aristotle  good morning: This will be tomorrow November 20,2018</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/305733268</link>
         <description><![CDATA[<div>Download a video on how to use burette and autoclave. You can have one video. Then base on the  video, make your reaction /comments. Indicate your learnings from that video. If you are to conduct an experiment using these apparatus, what changes will you make to suit our set up in school?<br>The output is by pair. Submit a hard copy and the website. <br> For Mendel only:<br>Make a procedure in sterilizing the following using autoclave:<br>20 testtubes<br>30 petri dishes<br>10 beakers<br>10 graduated cylinder<br>15 erlenmeyer flask<br>Make steps on how to sterilize these apparatus. You can  look for your pair.<br>Submit at the end of biotech. <br><br></div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-18 23:56:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/305733268</guid>
      </item>
      <item>
         <title>Coscos, mejares</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/306861381</link>
         <description><![CDATA[<div>PROCEDURES ON STERALIZING THE FOLLOWING USING AUTOCLAVE<br>Test tubes<br>Petri dishes<br>Beakers<br>Graduated cylinder<br>Erlenmeyer flask<br>Procedures<br>Prepare first the dirty apparatus<br>Clean them first using basic sterilizing compounds(alcohol, fuel, acid, etc.) that is highly trusted when killing microorganisms. <br>Dry them up<br>Cover them using a scratch paper with rubber bands tight in it<br>Put it up in autoclave and cover the autoclave<br>set the autoclave temperature to 15 PSI<br>Then wait<br>After that, pull off the cleaned apparatus<br><br>The video<br>It talks about that Burette is a quite hard to use when doing an experiment focusing on that material. Pouring the solution is like pouring a water in perfect place so you must using a small funnel inserted at the top of the Burette to make sure that it doesn’t make any error. Once it filled, put into the Burette clan and record the initial findings using the Burette. When the stop cock is parallel, it is fully open.<br><br>The changes it will take to suit up the school is that the amount of liquid will not be wasted, throwed upon and it can exactly measure the exact volume of liquid because it can pour the liquid slowly into the container. </div>]]></description>
         <enclosure url="" />
         <pubDate>2018-11-21 22:25:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/306861381</guid>
      </item>
      <item>
         <title>Roduard Rynch Magallano and Ej Ca</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/306886166</link>
         <description><![CDATA[]]></description>
         <enclosure url="" />
         <pubDate>2018-11-22 01:51:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/306886166</guid>
      </item>
      <item>
         <title>Activity in protein synthesis for aris and mendel for Jan. 21 -Jan. 22.( Monday to Teusday)</title>
         <author>lalaine65biboso</author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/322471871</link>
         <description><![CDATA[]]></description>
         <enclosure url="https://padlet-uploads.storage.googleapis.com/247484831/0ccaeebde25abeee06aee0605b2be644/Instructions_for_activity_in_protein_synthesis.pptx" />
         <pubDate>2019-01-20 10:50:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/322471871</guid>
      </item>
      <item>
         <title>Christian Ian Charles R. Salas, Kenny Macjewr Sulilap, Deniel Dave Natividad </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/325312582</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-01-29 10:38:56 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/325312582</guid>
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      <item>
         <title>MUTATION </title>
         <author>safwan_kano426</author>
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         <pubDate>2019-02-07 05:58:08 UTC</pubDate>
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      </item>
      <item>
         <title>Elias </title>
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         <pubDate>2019-02-07 12:01:52 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/328695301</link>
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         <pubDate>2019-02-07 12:55:52 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/328695301</guid>
      </item>
      <item>
         <title>Alyanna Shin D. Suarez 8 Aristotle</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/328701705</link>
         <description><![CDATA[<div>MUTATION<br><br></div>]]></description>
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         <pubDate>2019-02-07 13:12:48 UTC</pubDate>
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      </item>
      <item>
         <title>Genetic Disorders</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/328705469</link>
         <description><![CDATA[<div>By: Allyza Joy B. Cerado Grade 8- Men</div>]]></description>
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         <pubDate>2019-02-07 13:21:14 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/328726373</link>
         <description><![CDATA[<div>Roduard Rynch Magallano 8-Mendel</div>]]></description>
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         <pubDate>2019-02-07 13:58:48 UTC</pubDate>
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      </item>
      <item>
         <title>Kristine Joy B. Palmaera 8-Mendel</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/328759333</link>
         <description><![CDATA[<div>MUTATION<br><br><br><br></div>]]></description>
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         <pubDate>2019-02-07 14:48:21 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
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         <pubDate>2019-02-07 15:02:32 UTC</pubDate>
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      <item>
         <title>GROUP 5 (9-MV)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413795884</link>
         <description><![CDATA[<div>Members:<br>Berame<br>Cadiz<br>Figuracion<br>Seromines<br>Urdaneta</div>]]></description>
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         <pubDate>2019-11-20 03:17:45 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413800434</link>
         <description><![CDATA[<div>Group 4-MV<br>Calonia<br>Salas<br>Sarmiento<br>Lagang<br>Hamajo</div>]]></description>
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         <pubDate>2019-11-20 03:39:29 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413800589</link>
         <description><![CDATA[<div>Group 1.  9-Mendeleev<br><br>Benjamin, Chrys Jan <br>Facurib, Jhonbert<br>Natividad, Deniel Dave<br>Belicena, Michael Francis<br>Sangatanan, Shanlee<br>Sulilap, Kenny Macjewr<br><br>When to use mean, median, and mode?<br><br>Mean: The mean is the arithmetic <a href="https://statisticsbyjim.com/glossary/mean/">average</a>, and it is probably the measure of central tendency that you are most familiar. In short, you will only use the mean if you'll get the average of the datas given. Example of this is a teacher computing grades of her students. Different grades are given to the teacher like: 95, 94, 88, 93, 89. The teacher will add it all up and divide the sum by 5 so that the teacher will know the average grade.<br><br>Median: The median is the middle value. It is the value that splits the dataset in half. To find the median, order your data from smallest to largest, and then find the data point that has an equal amount of values above it and below it. The method for locating the median varies slightly depending on whether your dataset has an even or odd number of values. For example, there are numbers given: 7, 8, 3, 9, 6, 2, and 4. In order to get the median, you just have to identify which is the middle of the numbers given which is the number 9.<br><br>Mode: The mode is the value that occurs the most frequently in your data set. On a bar chart, the mode is the highest bar. If the data have multiple values that are tied for occurring the most frequently, you have a multimodal distribution. If no value repeats, the data do not have a mode. Example of this is you are given 5 numbers: 6, 7, 8, 3, 6. In order to identify the mode, choose which numbers shows repeatedly and in that case it is the number 6.<br><br>Interpreting the given:<br>Mode: 600,000<br>Mean: 500,000<br>Median: 300,000<br><br>The given above means that the average income of homeowners in a prestigious village is 300,000. Their common income is 600,000 and the middle income of the homeowners is 500,000.<br><br>Standard Deviation<br>-is a measure of dispersement in statistics. “Dispersement” tells you how much your data is spread out. Specifically, it shows you how much your data is spread out around the <a href="https://www.statisticshowto.datasciencecentral.com/probability-and-statistics/statistics-definitions/mean-median-mode/#mean">mean</a> or <a href="https://www.statisticshowto.datasciencecentral.com/arithmetic-mean/">average</a>. For example, are all your scores close to the average? Or are lots of scores way above (or way below) the average score?<br><br><br></div>]]></description>
         <pubDate>2019-11-20 03:40:12 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413801410</link>
         <description><![CDATA[<div>Group 4<br>Calonia <br>Salas<br>Sarmiento<br>Lagang<br>Hamajo</div>]]></description>
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         <pubDate>2019-11-20 03:44:51 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413802083</link>
         <description><![CDATA[<div>Group 4-MV<br>Calonia<br>Salas<br>Sarmiento<br>Lagang<br>Hamajo</div>]]></description>
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         <pubDate>2019-11-20 03:48:26 UTC</pubDate>
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      <item>
         <title>Group2-Mendeleev</title>
         <author></author>
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         <description><![CDATA[<div>Bantes<br>Brocoy<br>Cutamora (Absent)<br>Lapating<br>Silva<br>Suarez</div>]]></description>
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         <pubDate>2019-11-20 03:48:54 UTC</pubDate>
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      <item>
         <title>Group 3-MV (Part 1)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413803290</link>
         <description><![CDATA[<div>Members:<br>Evangelista<br>Juanico<br>Baluso<br>Llamera<br>Miase (Absent)<br>Lata</div>]]></description>
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         <pubDate>2019-11-20 03:55:00 UTC</pubDate>
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      <item>
         <title>Group 6</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413803626</link>
         <description><![CDATA[<div>Cabiles(Absent)<br>Deleverio<br>Dejillo<br>Lorania(Absent)<br>Palmaera<br>Sangatanan,Shandee<br><br></div>]]></description>
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         <pubDate>2019-11-20 03:56:38 UTC</pubDate>
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      <item>
         <title>Group 3-MV (Part 2)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/413806392</link>
         <description><![CDATA[<div>Members:<br>Evangelista<br>Juanico<br>Baluso<br>Llamera<br>Miase (Absent)<br>Lata</div>]]></description>
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         <pubDate>2019-11-20 04:09:33 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421436760</link>
         <description><![CDATA[<div>Benjamin, Chrys Jan 9-Mendeleev</div>]]></description>
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         <pubDate>2019-12-09 03:45:02 UTC</pubDate>
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         <title></title>
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         <pubDate>2019-12-09 03:45:08 UTC</pubDate>
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      <item>
         <title>Alvin Jay Deleverio</title>
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         <pubDate>2019-12-09 03:45:41 UTC</pubDate>
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      <item>
         <title>Jannah Bantes 9-Mendeleev</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421437532</link>
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         <pubDate>2019-12-09 03:48:41 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421437625</link>
         <description><![CDATA[<div>Benjamin, Chrys Jan 9-Mendeleev Part 2</div>]]></description>
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         <pubDate>2019-12-09 03:49:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421437625</guid>
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      <item>
         <title>Jannah Bantes 9-Mendeleev (Part 2)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421437891</link>
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         <pubDate>2019-12-09 03:50:32 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438372</link>
         <description><![CDATA[<div>Belicena, Michael Francis P. 9-MV </div>]]></description>
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         <pubDate>2019-12-09 03:53:14 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438421</link>
         <description><![CDATA[<div>Francis Albert L. Calonia<br>9-Mendeleev</div>]]></description>
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         <pubDate>2019-12-09 03:53:34 UTC</pubDate>
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         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438477</link>
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         <pubDate>2019-12-09 03:53:53 UTC</pubDate>
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      <item>
         <title>Jannah Bantes 9-Mendeleev (Part 3)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438526</link>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438749</link>
         <description><![CDATA[<div>Francis Albert L. Calonia<br>9-mendeleev</div>]]></description>
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         <pubDate>2019-12-09 03:54:41 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438910</link>
         <description><![CDATA[<div>Belicena, Michael Francis 9-MV<br><br></div>]]></description>
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         <pubDate>2019-12-09 03:55:47 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421438963</link>
         <description><![CDATA[<div>Benjamin, Chrys Jan 9-Mendeleev Part 3</div>]]></description>
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         <pubDate>2019-12-09 03:56:05 UTC</pubDate>
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         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439153</link>
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         <pubDate>2019-12-09 03:57:14 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439263</link>
         <description><![CDATA[<div>Christian Louie evangelista<br>9 mv</div>]]></description>
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         <pubDate>2019-12-09 03:57:53 UTC</pubDate>
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         <title></title>
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         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439411</link>
         <description><![CDATA[<div>DJ Christian A. Juanico g9 MENDELEEV</div>]]></description>
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         <pubDate>2019-12-09 03:58:46 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439435</link>
         <description><![CDATA[<div>Cutamora, Mizpah Gabrielle S. 9-MV<br>STATISTICS </div>]]></description>
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         <pubDate>2019-12-09 03:58:52 UTC</pubDate>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439483</link>
         <description><![CDATA[<div>Patrick James M. Lagang<br>9-MV</div>]]></description>
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         <pubDate>2019-12-09 03:59:07 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439549</link>
         <description><![CDATA[<div>Christian Louie evangelista<br>9 mv</div>]]></description>
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         <pubDate>2019-12-09 03:59:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439549</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439591</link>
         <description><![CDATA[<div>Patrick James M. Lagang<br>9-MV</div>]]></description>
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         <pubDate>2019-12-09 03:59:44 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439591</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439734</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:00:28 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439734</guid>
      </item>
      <item>
         <title>James Baluso 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439815</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:00:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439815</guid>
      </item>
      <item>
         <title>Christian Ian Charles R.Salas</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439831</link>
         <description><![CDATA[<div>9-MV</div>]]></description>
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         <pubDate>2019-12-09 04:01:03 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439831</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421439988</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:01:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421439988</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440011</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:02:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440011</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440023</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:02:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440023</guid>
      </item>
      <item>
         <title>Christian Ian Charles R. Salas</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440149</link>
         <description><![CDATA[<div>9-MV</div>]]></description>
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         <pubDate>2019-12-09 04:02:29 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440149</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440186</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:02:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440186</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440187</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:02:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440187</guid>
      </item>
      <item>
         <title>Jhonbert Facurib </title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440248</link>
         <description><![CDATA[<div>9 - Mendeleev <br>Front page</div>]]></description>
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         <pubDate>2019-12-09 04:02:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440248</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440362</link>
         <description><![CDATA[<div>Cutamora, Mizpah Gabrielle S. 9-MV STATISTICS <br><br></div>]]></description>
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         <pubDate>2019-12-09 04:03:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440362</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440425</link>
         <description><![CDATA[<div>Palmaera, Kristine Joy B. 9-MV<br>STATISTICS</div>]]></description>
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         <pubDate>2019-12-09 04:03:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440425</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440443</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:04:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440443</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440474</link>
         <description><![CDATA[<div>Angelie Mae M. Lapating 9-MV<br>Part 1</div>]]></description>
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         <pubDate>2019-12-09 04:04:16 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440474</guid>
      </item>
      <item>
         <title>James Baluso 9-MV (part 2)</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440599</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:04:57 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440599</guid>
      </item>
      <item>
         <title>Ronan Lata 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440602</link>
         <description><![CDATA[<div>Part 1</div>]]></description>
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         <pubDate>2019-12-09 04:04:58 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440602</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440656</link>
         <description><![CDATA[<div>ZOFIA AMINA SEROMINES 9-MENDELEEV<br>Page 1<br><br></div>]]></description>
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         <pubDate>2019-12-09 04:05:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440656</guid>
      </item>
      <item>
         <title>2</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440770</link>
         <description><![CDATA[<div>Palmaera 9-MV<br>STATISTICS</div>]]></description>
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         <pubDate>2019-12-09 04:05:54 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440770</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440801</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:06:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440801</guid>
      </item>
      <item>
         <title>Jhonbert Facurib</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440802</link>
         <description><![CDATA[<div>9 - Mendeleev<br>Back page</div>]]></description>
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         <pubDate>2019-12-09 04:06:02 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440802</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440884</link>
         <description><![CDATA[<div>Angelie Mae M. Lapating 9-MV<br>Part 2</div>]]></description>
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         <pubDate>2019-12-09 04:06:31 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440884</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421440912</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:06:38 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421440912</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441001</link>
         <description><![CDATA[<div>ZOFIA AMINA SEROMINES 9-MENDELEEV<br>PAGE 2</div>]]></description>
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         <pubDate>2019-12-09 04:07:04 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441001</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441040</link>
         <description><![CDATA[<div>Miase, Bea Clarisse 9 MV</div>]]></description>
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         <pubDate>2019-12-09 04:07:10 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441040</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441128</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:07:40 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441128</guid>
      </item>
      <item>
         <title>1</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441217</link>
         <description><![CDATA[<div>Cadiz, Elias Ma. Seraphina 9-MV<br>Statistics</div>]]></description>
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         <pubDate>2019-12-09 04:08:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441217</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441226</link>
         <description><![CDATA[]]></description>
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         <pubDate>2019-12-09 04:08:15 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441226</guid>
      </item>
      <item>
         <title>Ronan Lata 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441247</link>
         <description><![CDATA[<div>Part 2</div>]]></description>
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         <pubDate>2019-12-09 04:08:22 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441247</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441321</link>
         <description><![CDATA[<div>Chorena Grace Lorania 9-Mendeleev <br>Page 1</div>]]></description>
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         <pubDate>2019-12-09 04:08:48 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441321</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441411</link>
         <description><![CDATA[<div>Miase, Bea Clarisse 9 MV </div>]]></description>
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         <pubDate>2019-12-09 04:09:13 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441411</guid>
      </item>
      <item>
         <title>Ronan Lata 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441483</link>
         <description><![CDATA[<div>Part 3</div>]]></description>
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         <pubDate>2019-12-09 04:09:37 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441483</guid>
      </item>
      <item>
         <title>2</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441494</link>
         <description><![CDATA[<div>Cadiz, Elias 9-MV<br>Statistics</div>]]></description>
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         <pubDate>2019-12-09 04:09:43 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441494</guid>
      </item>
      <item>
         <title>Zyrreg Cabiles     9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441560</link>
         <description><![CDATA[<div>Part 1</div>]]></description>
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         <pubDate>2019-12-09 04:10:11 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441560</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441649</link>
         <description><![CDATA[<div>Angelie Mae M. Lapating 9-MV<br>Part 3</div>]]></description>
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         <pubDate>2019-12-09 04:10:43 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441649</guid>
      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441706</link>
         <description><![CDATA[<div>Brocoy, Angelle Kate 9 MV</div>]]></description>
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         <pubDate>2019-12-09 04:11:06 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441706</guid>
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      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441836</link>
         <description><![CDATA[<div>Chorena Grace Lorania 9-Mendeleev<br>page2 </div>]]></description>
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         <pubDate>2019-12-09 04:11:51 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441836</guid>
      </item>
      <item>
         <title>Jasmin Imelda P. Silva 9-Mendeleev</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441891</link>
         <description><![CDATA[<div><br><br>page 2</div>]]></description>
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         <pubDate>2019-12-09 04:12:10 UTC</pubDate>
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      </item>
      <item>
         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/421441976</link>
         <description><![CDATA[<div>Shanlee S. Sanagatanan 9-MV<br>Part 1</div>]]></description>
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         <pubDate>2019-12-09 04:12:36 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/421441976</guid>
      </item>
      <item>
         <title>Jhonbert Facurib</title>
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         <title>Jhonbert Facurib</title>
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      <item>
         <title>Jannah H. Bantes 9- MV</title>
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         <title></title>
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         <title>Figuracion, Reanne Kathleen B.</title>
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         <title></title>
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      <item>
         <title>Alyanna Shin D. Suarez</title>
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         <title></title>
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         <title>Howard Godwin Delos Reyes</title>
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         <author>mtskmisa</author>
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         <author>safwan_kano426</author>
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         <description><![CDATA[<div>Emata, Simone Anne C. <br><br></div>]]></description>
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         <author>safwan_kano426</author>
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         <title>MIKA C. HAMAJO 9-MV</title>
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         <link>https://padlet.com/lalaine65biboso/biotech/wish/423100184</link>
         <description><![CDATA[<div>Why it was passed late?<br>-I joined the YES-O Leadership Congress</div>]]></description>
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         <title></title>
         <author></author>
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         <description><![CDATA[<div>Pertimos, Ashley Marie</div>]]></description>
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         <title>Llamera, Jefferson 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/424246064</link>
         <description><![CDATA[<div>-joined the National Robotics Competition in Davao last Dec 9-11.</div>]]></description>
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         <title>Llamera, Jefferson 9-MV</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/424257443</link>
         <description><![CDATA[<div>-joined the National Robotics Competition in Davao last Dec. 9-11.</div>]]></description>
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         <title>Berame, Bernadette Sue B. 9-Mendeleev</title>
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         <description><![CDATA[<div>-joined the Robotics competition in Davao last December 9-11,2019.<br>-wasn’t able to put in the MS Word because my sister is using the computer for their research and she don’t want me to disturb her. Hoping for your consideration ma’am.</div>]]></description>
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         <title></title>
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         <pubDate>2019-12-15 13:48:43 UTC</pubDate>
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         <pubDate>2019-12-15 13:50:42 UTC</pubDate>
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         <pubDate>2019-12-15 13:53:46 UTC</pubDate>
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         <title></title>
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         <link>https://padlet.com/lalaine65biboso/biotech/wish/424272206</link>
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         <pubDate>2019-12-15 13:55:53 UTC</pubDate>
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         <title></title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/424272720</link>
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         <pubDate>2019-12-15 14:01:46 UTC</pubDate>
         <guid>https://padlet.com/lalaine65biboso/biotech/wish/424272720</guid>
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      <item>
         <title>hi guys g12 na ta karon hehe</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/2526503624</link>
         <description><![CDATA[]]></description>
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         <pubDate>2023-03-22 03:43:33 UTC</pubDate>
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      <item>
         <title>6/14/23</title>
         <author></author>
         <link>https://padlet.com/lalaine65biboso/biotech/wish/2622912098</link>
         <description><![CDATA[<div>hello guys graduating na us &lt;3 goodluck sa college!</div>]]></description>
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         <pubDate>2023-06-14 02:49:51 UTC</pubDate>
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