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1. https://www.britannica.com/science/Chagas-disease

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The Memory B-cells are in charge of remembering what antibody was used to defeat this foreign invader. For Chaga's disease the specific antibody is called IgA or even an IgM has been tested to work. Once the B-cell successfully has memory of the foreign invader and the antibody needed, they rest until another infection occurs,

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These cells are in charge of regulating the immune systems response and essentially tell the immune system to stop once the disease is successfully defeated. This cell would come into play once it realizes that there are no longer any more of the bacteria that causes the Chaga's Disease.

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These cells are alerted by the Helper T- Cells. They then multiply and destroy the foreign invaders and in some cases even the macrophages that engulfed the invaders. The Killer T- cells would kill and eat the deadly Chaga bacteria.

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B- cells are in charge of making antibodies that lock onto foreign antigens to help make it easier for the other immune cells to defeat it. Each antibody has two binding sites with a specific pattern that can only match one specific disease. So the B-cells would have to create an antibody that will be able to lock onto the pathogen of the Chaga's Disease.

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The Helper T-cell's job is to spot the foreign antigen, in this case the bacteria from the Chaga's Disease, on the macrophage and then begin to multiply rapidly. They then would alert other white blood cells to help with the bodies defense to defeat the Chaga's disease.

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If the macrophages were unsuccessful at destroying all of the antigen's there is a last line of defense to fully get rid of the Chaga's Disease. It starts with the Helper T- cell, then the B-cells, then the Killer T- cells, then the Suppressor T- cells, then finally the Memory B- cells.

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Once the infection reaches inside the body the next defense mechanism is the macrophage. The macrophages engulf the foreign body and then displays the surface of the antigen (the foreign body) . This helps stimulate the immune system to begin making anti-bodies.

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With transmission through the skin, the skin is the first obvious line of defense. With an insect bite the skin acts as a barrier, but more often than not when it is bit the skin cannot add much protection because the reduviid bugs break the skin. Or once the feces is on the skin, itching the area can also cause breakages in the skin that allows transmission. Through the transmission of the eye and mouth, the eyes have tear ducts that release tears to try and wash away any bacteria. The mouth has saliva to do that of the same thing. The bacteria is able to get past the membrane and diffuse into the body

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Symptoms will occur about a week after. Symptoms include edema, lymph node enlargements, possible fevers and prostration for a few weeks. The disease can become chronic and can cause heart problems such as failure and in severe cases can eventually lead to death.

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The disease is passed on from the feces of contagious and infected insects. The inoculation is commonly created from itching an insect bite where the feces is there and comes into contact. Or it can be transmitted through the mucous membranes found in the eyes and the mouth.

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This is an infection casued by the flagellate protozoan called Trypanosoma Cruzi. It is passed on by reduviid bugs which are blood suckers who live in rural areas of South America.