# data[].attributes.body
1. Choose a spokesperson

2. All students in the group are to work on all of the following questions/sections included in the Padlet

3. You can use diagrams/images other visual resources to explain each respiratory condition.

4. When we come back to the whole class, the spokesperson will feedback the groups findings

# data[].attributes.body
Excess fluid in the lung due to cardiac, pneumonia, medications, trauma, travelling and exercising in high altitude, exposure to certain toxins (cocaine, heroine)

# data[].attributes.headline
How does the condition effect lung compliance and/or airway resistance?

# data[].attributes.subject
How does the condition effect lung compliance and/or airway resistance?

# data[].attributes.body
Lungs will be poorly compliant due to increased fluid in the alveoli and greater alveoli surface tension making expansion difficult

# data[].attributes.body
low Pa02- poor oxygenation

Low C02- due to hyperventilation initially, then after some time, the patient will fatigue, resulting in hypercapnia and hypoventilation.

High PH- alkalotic

HC03- initially normal then will trend down to compensate

# data[].attributes.headline
What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

# data[].attributes.subject
What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

# data[].attributes.body
Anxiety/ restlessness- due to SOB
Crackles (especially to bases)- popping sound due to air in the alveoli trying to come of the fluid
Frothy sputum- the fluid in the lungs contains red blood cells as a result of small amounts of blood leaking from capillaries, this can then be coughed up causing pink frothy sputum
Increased JVP and CVP as a result of fluid retention
CXR- diffuse opacities across the lung fields
Increased WOB, hypoxaemia
Dullness to percussion over lung bases

# data[].attributes.body
Restrictive - there is no obstruction of airflow, there is a restriction in expanding and collapsing of alveoli, and shunting of deoxygenated blood R-L.

# data[].attributes.headline
How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

# data[].attributes.subject
How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

# data[].attributes.body
O2 content - Nil changes to haemoglobin oxygen affinity, low arterial partial pressure of oxygen.
O2 delivery - As it is a cardiogenic cause, cardiac output is most likely diminished resulting in hypoperfusion of alveoli, and pulmonary hypertension. O2 delivery to the tissues diminished
Alveolar Ventilation - increased surface tension results in poor alveolar ventilation. Stiffness of the alveolocapillary membrane results in slower diffusion of gasses.

# data[].attributes.headline
Explain the role of V/Q mismatch in this condition.

# data[].attributes.subject
Explain the role of V/Q mismatch in this condition.

# data[].attributes.body
Low V/Q, < 0.8
This is a result of shunting as a result of fluid accumulation in the alveoli. Which causes the alveoli to receive perfusion but poor ventilation.

# data[].attributes.headline
What type of respiratory failure would this condition lead to?

# data[].attributes.subject
What type of respiratory failure would this condition lead to?

# data[].attributes.body
This would lead to hypoxic (T1RF), but could eventuate in T2RF with fatigue longer ncvcnvcv