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bind to cholingeric receptors and act as a blocking drug (competitive antagonism)
Drug examples =
ipratropium bromide
(Atrovent™)
dicyclomine (Bentyl™)

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inhibit acetylchotinesterase, increases ACH. Drug example = neostigmine (Prostigmin™)

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directly stimulate the muscarinic receptors in the parasympathetic system, mimics ACH.
Drug example = bethanechol (Urecholine™)

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block beta 1 and 2 receptors, decreases force of contraction/HR/conduction, decrease in cardiac output which causes a decrease in BP, can cause bronchoconstriction
Drug example = propanolol (Inderal™)

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Selective Beta adrenergic blockers (only beta 1)

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Selective Beta adrenergic blockers (only beta 1)

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blocks beta 1 receptors, causes a sympatholytic effect, decreases force of contraction/HR/conduction, decrease in cardiac output which causes a decrease in BP.
Drug example = atenolol (Tenormin™)

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Selective Beta adrenergic agonists (only beta 2)

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Selective Beta adrenergic agonists (only beta 2)

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stimulates only the beta 2 receptors. causes relaxation of the smooth muscle of the lungs (bronchodilation), relaxation of the smooth muscle of the ureters and uterus in some drugs.
Drug examples = albuterol (Proventil™, Ventolin™)

terbutaline (Brethine™)

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Selective Beta adrenergic agonists (only beta 1)

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Selective Beta adrenergic agonists (only beta 1)

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low dose - stimulates dopaminergic receptors in kidneys causing vasodilation and increased renal blood flow
moderate dose - stimulates beta 1 receptors on the heart, increases the force of contraction and cardiac output
high does - stimulates alpha 1 receptors causing vasoconstriction, increasing BP
Drug example = dobutamine (Dobutamine™)

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Binds to alpha 1 receptors and blocks them, relaxant of smooth muscle, vasodilation of blood vessels, decreases BP, relaxes smooth muscle of the ureters and increase urine output. Drug example = doxazosin ( Cardura™)

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stimulate all alpha and beta receptors. cause vasoconstriction, increases BP, cardiac stimulant, and bronchodilator. Drug example = epinephrine (Adrenalin™)

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stimulates alpha 1 receptors, cause contraction of the smooth muscle, blood vessels constrict (vasoconstriction), increases BP, has a decongestant effect. Drug example = phenylephrine (Neo-Synephrine™)

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relaxation

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vasoconstriction, increases heart rate and blood pressure

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bronchodilation

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dilation (mydriasis)

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vasoconstriction

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contraction

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Relaxation

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bronchoconstriction

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constriction (miosis)

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increased activity and secretions

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decreases heart rate and conduction, slightly decreases contractility

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somatic nerves found at the neuromuscular junction of the skeletal muscle

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located on the ganglia of parasympathetic and sympathetic nerves

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located on cardiac, smooth muscle, and glands

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released at the preganglionic nerve ending and the postganglionic nerve ending

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located in adrenergic nerve endings

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located on smooth muscle, lungs, uterus, skeletal muscle arteries, and coronary arteries

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located on the heart

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found on glands, smooth muscle, eyes, and most arteries/veins

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precursor to Norepinephrine, stimulates many receptors (alpha, beta, dopaminergic)

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produced by the adrenal medulla and acts on adrenergic receptors

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released at postganglionic nerve

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released at preganglionic synapse, acts on postganglionic neuron

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also known as the Cholinergic system

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also known as the Adrenergic system