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Assignment Brief - Musculoskeletal System

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Assignment Brief - Musculoskeletal System

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Ass Brief HB Muscular and Skeletal System

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How your muscular system works - Emma Bryce

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Introduction to the Muscular system - video

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Introduction to the Muscular system - video

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The Muscular System Explained In 6 Minutes

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Muscle Tissues and Sliding Filament Model - Video

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Muscle Tissues and Sliding Filament Model - Video

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Muscle Tissues and Sliding Filament Model

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Positive and negative feedback homeostasis video

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Positive and negative feedback homeostasis video

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positive and negative feedback homeostasis - Search Videos

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How Common Diseases Affect the Function of the Urinary System Introduction

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How Common Diseases Affect the Function of the Urinary System Introduction

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The urinary system — including the kidneys, ureters, bladder, and urethra — is responsible for removing waste, regulating fluid balance, controlling blood pressure, and maintaining electrolyte levels.

Several common diseases can disrupt these functions, leading to serious complications.

1. High Blood Pressure (Hypertension)

Impact on Urinary System:

Kidney Damage: High blood pressure forces blood through the tiny blood vessels of the kidneys at high pressure, damaging their delicate structures.

Reduced Filtration: Damage to kidney filters (glomeruli) reduces the kidneys' ability to filter waste.

Chronic Kidney Disease (CKD): Prolonged high blood pressure is a leading cause of CKD and eventually kidney failure.

Proteinuria: Damaged kidneys leak protein into the urine (a sign of declining kidney function).

Key Terms:

Nephrosclerosis – Hardening of the kidney arteries due to prolonged hypertension.

2. Diabetes (Type 1 and Type 2)

Impact on Urinary System:

Diabetic Nephropathy: High blood sugar levels damage the glomeruli over time.

Increased Risk of Infection: Sugar in urine promotes bacterial growth, leading to urinary tract infections (UTIs).

Polyuria: Excess glucose in the blood leads to excess urination (frequent need to urinate).

Kidney Failure: Diabetes is the leading cause of end-stage kidney disease globally.

Key Terms:

Glomerular Basement Membrane Thickening – A structural change in the kidneys that reduces their filtering efficiency.

3. Heart Disease

Impact on Urinary System:

Reduced Blood Flow: A weak heart cannot pump blood efficiently, reducing blood supply to the kidneys.

Fluid Retention: The kidneys sense low blood flow and retain salt and water, worsening edema (swelling).

Cardiorenal Syndrome: A condition where heart failure and kidney failure worsen each other.

Risk of Acute Kidney Injury (AKI): Heart disease can cause sudden and severe drops in kidney function, especially after a heart attack.

Key Terms:

Renin-Angiotensin-Aldosterone System (RAAS) Activation – A hormone system that becomes overactive in heart failure, promoting kidney damage.

4. Obesity

Impact on Urinary System:

Increased Kidney Workload: Excess body fat increases metabolic demands and blood volume, straining the kidneys.

Risk of Kidney Stones: Obesity alters the balance of salts and minerals in urine, promoting stone formation.

Glomerulomegaly: Enlargement of kidney filtering units, which eventually leads to scarring (glomerulosclerosis).

Increased Risk for Hypertension and Diabetes: Obesity often leads to high blood pressure and diabetes, both of which severely impact kidney health.

Key Terms:

Obesity-Related Glomerulopathy (ORG) – A kidney disease directly linked to obesity.

Discussion Questions

Why do you think diabetes and hypertension often occur together in kidney disease patients?

How can early detection of urinary system changes help manage these diseases?

What lifestyle changes can protect the kidneys if a person has heart disease or obesity?

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Assignment Brief - Numeracy in a Health Context

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Assignment Brief - Numeracy in a Health Context

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Numeracy in a Health Context Assessment Brief Overall Student Copy

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Share your videos with friends, family, and the world

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How to add, subtract, multiply and divide Negative Numbers - Video

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How to add, subtract, multiply and divide Negative Numbers - Video

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How to Add, Subtract, Multiply & Divide Negative Numbers : Math Questions & Answers

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Fractions, decimals and percentages - Video

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Fractions, decimals and percentages - Video

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What are Proportions? How to Convert Between Fractions, Decimals and Percentages (Proportions 1)

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Cognitoedu.org - Learn and Test Yourself

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Cognitoedu.org - Learn and Test Yourself

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Homeostasis and the Endocrine System Full Unit Assignment Brief 2425

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Homeostasis and the Endocrine System including Diabetes 

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Homeostasis and the Endocrine System including Diabetes 

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Learning Outcomes

Discuss why ATP is necessary for muscle movement

The motion of muscle shortening occurs as myosin heads bind to actin and pull the actin inwards. This action requires energy, which is provided by ATP. Myosin binds to actin at a binding site on the globular actin protein. Myosin has another binding site for ATP at which enzymatic activity hydrolyzes ATP to ADP, releasing an inorganic phosphate molecule and energy.

ATP binding causes myosin to release actin, allowing actin and myosin to detach from each other. After this happens, the newly bound ATP is converted to ADP and inorganic phosphate, P i. The enzyme at the binding site on myosin is called ATPase. The energy released during ATP hydrolysis changes the angle of the myosin head into a "cocked" position. The myosin head is then in a position for further movement, possessing potential energy, but ADP and P i are still attached. If actin binding sites are covered and unavailable, the myosin will remain in the high energy configuration with ATP hydrolyzed, but still attached.

If the actin binding sites are uncovered, a cross-bridge will form; that is, the myosin head spans the distance between the actin and myosin molecules. P i is then released, allowing myosin to expend the stored energy as a conformational change. The myosin head moves toward the M line, pulling the actin along with it. As the actin is pulled, the filaments move approximately 10 nm toward the M line. This movement is called the power stroke, as it is the step at which force is produced. As the actin is pulled toward the M line, the sarcomere shortens and the muscle contracts.

When the myosin head is "cocked," it contains energy and is in a high-energy configuration. This energy is expended as the myosin head moves through the power stroke; at the end of the power stroke, the myosin head is in a low-energy position. After the power stroke, ADP is released; however, the cross-bridge formed is still in place, and actin and myosin are bound together. ATP can then attach to myosin, which allows the cross-bridge cycle to start again and further muscle contraction can occur (Figure 1). The movement of the myosin head back to its original position is called the recovery stroke. Resting muscles store energy from ATP in the myosin heads while they wait for another contraction.

Regulatory Proteins

When a muscle is in a resting state, actin and myosin are separated. To keep actin from binding to the active site on myosin, regulatory proteins block the molecular binding sites. Tropomyosin blocks myosin binding sites on actin molecules, preventing cross-bridge formation and preventing contraction in a muscle without nervous input. Troponin binds to tropomyosin and helps to position it on the actin molecule; it also binds calcium ions.

To enable a muscle contraction, tropomyosin must change conformation, uncovering the myosin-binding site on an actin molecule and allowing cross-bridge formation. This can only happen in the presence of calcium, which is kept at extremely low concentrations in the sarcoplasm. If present, calcium ions bind to troponin, causing conformational changes in troponin that allow tropomyosin to move away from the myosin binding sites on actin. Once the tropomyosin is removed, a cross-bridge can form between actin and myosin, triggering contraction. Cross-bridge cycling continues until Ca ²⁺ ions and ATP are no longer available and tropomyosin again covers the binding sites on actin.

The following video explains how a muscle contraction is signaled:

Practice Question

Which of the following statements about muscle contraction is true?

1. The power stroke occurs when ATP is hydrolyzed to ADP and phosphate.

2. The power stroke occurs when ADP and phosphate dissociate from the myosin head.

3. The power stroke occurs when ADP and phosphate dissociate from the actin active site.

4. The power stroke occurs when Ca²⁺ binds the calcium head.

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Discuss why ATP is necessary for muscle movement The motion of muscle shortening occurs as myosin heads bind to actin and pull the actin inwards. This action requires energy, which is provided by ATP. Myosin binds to actin at a binding site on the globular actin protein.

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Teesside Uni Guide to Harvard Referencing

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Teesside Uni Guide to Harvard Referencing

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Include the pathophysiology of disease linking it to the effects such reduced bone strength, joint swelling, joint damage or loss of flexibility.

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Osteoarthritis

This serious, painful condition is the most common form of arthritis and can affect any joint.

Osteoarthritis is a degenerative joint disease that can affect the many tissues of the joint. It is by far the most common form of arthritis, affecting more than 32.5 million adults in the United States, according to the Centers for Disease Control and Prevention.

Historically, osteoarthritis (OA) was known as a "wear and tear" condition, generally associated with aging. But we know now that it is a disease of the entire joint, including bone, cartilage, ligaments, fat and the tissues lining the joint (the synovium). Osteoarthritis can degrade cartilage, change bone shape and cause inflammation, resulting in pain, stiffness and loss of mobility.

OA can affect any joint, but typically affects hands, knees, hips, lower back and neck. Its signs and symptoms typically show up more often in individuals over age 50, but OA can affect much younger people, too, especially those who have had a prior joint injury, such as a torn ACL or meniscus. It typically develops slowly over time, but after such an injury, it can develop much more rapidly, within just a few years. OA is not an inevitable aging disease; some people never develop it.

There is no cure for OA, but there are ways to manage OA to minimize pain, continue physical activities, maintain a good quality of life and remain mobile.

Causes

Factors that may contribute to the development of OA include

Age. The risk of developing OA increases with age and symptoms generally, but not always, appear in people over 50.

Joint injury. A bone fracture or cartilage or ligament tear can lead to OA, sometimes more quickly than in cases where there is not an obvious injury.

Overuse. Using the same joints over and over in a job or sport can result in OA.

Obesity. Excess weight adds stress and pressure on a joint, plus fats cells promote inflammation.

Musculoskeletal abnormalities. Malalignment of bone or joint structures can contribute to faster development of OA.

Weak muscles. If muscles don't provide adequate joint support, poor alignment can result, which can lead to OA.

Genetics. People with family members who have OA are more likely to develop it.

Gender. Women are more likely to develop OA than men.

Environmental Factors. Modifiable environmental risk factors include things like someone's occupation, level of physical activity, quadriceps strength, presence or absence of prior joint injury, obesity, diet, sex hormones, and bone density.

Symptoms

Symptoms tend to build over time rather than show up suddenly. They include

Pain or aching in a joint during activity, after long activity or at the end of the day.

Joint stiffness usually occurs first thing in the morning or after resting.

Limited range of motion that may go away after movement.

Clicking or popping sound when a joint bends.

Swelling around a joint.

Muscle weakness around the joint.

Joint instability or buckling (as when a knee gives out).

OA may affect different parts of the body in different ways.

Hips. Pain in the groin area or buttocks and sometimes on the inside of the knee or thigh.

Knees. A "grating" or "scraping" feeling when moving the knee.

Fingers. Bony growths (spurs) at the edge of joints can cause fingers to become swollen, tender and red, sometimes with pain at the base of the thumb.

Feet. Pain and tenderness in the big toe, with possible swelling in the ankles or toes.

Potential Consequences

Pain, reduced mobility, side effects from medications and other factors associated with osteoarthritis can lead to health complications that are not caused by the disease itself.
Obesity, Diabetes and Heart Disease
Painful joints, especially in the feet, ankles, knees, hip or back, make it harder to exercise. But physical activity is not only key to managing OA symptoms, it also can help prevent weight gain, which can lead to obesity. Being overweight or obese can lead to the development of high cholesterol, type 2 diabetes, heart disease and high blood pressure.
Falls
Research indicates people with OA experience more falls and risk of fracture than those without OA. Although study results vary, some research shows they may have up to 30% more falls and have a 20% greater risk of fracture. Having OA can decrease function, weaken muscles, affect overall balance, and make falls more likely, especially among those with OA in knees or hips. Side effects from pain medications, such as dizziness, can also contribute to falls.

Support vital advocacy, research and programs for the nearly 60 million people in America living with arthritis.

Give to support the 1 in 4 adults and hundreds of thousands of children in America who live with painful and often debilitating arthritis.

Diagnosis

Medical history, a physical examination and lab tests help to make up the OA diagnosis.

A primary care doctor may be the first person you talk to about joint pain. The doctor will review your medical history, symptoms, how the pain affects activities, as well as your medical problems and medication use. He or she will also look at and move your joints, and may order imaging. These tests help to make the diagnosis:

Joint aspiration. After numbing the area, a needle is inserted into the joint to pull out fluid. This test will look for infection or crystals in the fluid to help rule out other medical conditions or other forms of arthritis.

X-ray. X-rays can show joint or bone damage or changes related to osteoarthritis.

MRI. Magnetic resonance imaging (MRI) gives a better view of cartilage and other parts of the joint.

Treatment

There is no cure for OA, but medication, assistive devices and other therapies that don't involve drugs can help to ease pain. As a last resort, a damaged joint may be surgically fused or replaced with one made of a combination of metal, plastic and/or ceramic.
Medications
Pain and anti-inflammatorymedicines for osteoarthritis are available as pills, syrups, patches, gels, creams or injectables. They include:

Analgesics. These are pain relievers and include acetaminophen and opioids. Acetaminophen is available over the counter (OTC); opioids must be prescribed by a doctor.

Nonsteroidal anti-inflammatory drugs (NSAIDs). These are the most commonly used drugs to ease inflammation and pain. They include aspirin, ibuprofen, naproxen and celecoxib, available either OTC or by prescription. The OTC versions help with pain but not inflammation.

Counterirritants. These OTC products contain ingredients like capsaicin, menthol and lidocaine that irritate nerve endings, so the painful area feels cold, warm or itchy to take focus away from the actual pain.

Corticosteroids. These prescription anti-inflammatory medicines work in a similar way to a hormone called cortisol. The medicine is taken by mouth or injected into the joint at a doctor's office.

Platelet-rich plasma (PRP). Available from a doctor by injection, this product is intended to help ease pain and inflammation. This is not approved by the Food & Drug Administration and evidence is still emerging, so discuss it with your doctor before trying it.

Other drugs. The antidepressant duloxetine (Cymbalta) and the anti-seizure drug pregabalin (Lyrica) are oral medicines that are FDA-approved to treat OA pain.

Nondrug Therapies Exercise
Movement is an essential part of an OA treatment plan. Getting 150 minutes of moderate-to-vigorous exercise per week should be the goal, according to the U.S. Department of Health and Human Services. A good exercise program to fight OA pain and stiffness has four parts:

Strengthening exercises build muscles around painful joints and helps to ease the stress on them.

Range-of-motion exercise or stretchinghelps toreduce stiffness and keep joints moving.

Aerobic or cardio exercises help improve stamina and energy levels and reduce excess weight.

Balance exercises help strengthen small muscles around the knees and ankles and help prevent falls.

Talk to a doctor or physical therapist before starting a new exercise program.
Weight Loss
Excess weight puts additional force and stress on weight-bearing joints, including the hips, knees, ankles, feet and back, and fat cells promote inflammation. Losing extra weight helps reduce pain and slow joint damage. Every pound of weight lost removes four pounds of pressure on lower-body joints.

Physical Therapies and Assistive Devices
Physical therapists, occupational therapists and chiropractors can provide:

Specific exercises to help stabilize your joints and ease pain.

Information about natural treatments and products that can ease pain.

Instruction to make movement easier and to protect joints.

Braces, shoe inserts or other assistive devices.

Surgery
Joint surgery can improve pain and function. Joint replacement surgery replaces damaged joints to restore mobility and relieve pain. Hips and knees are the joints most commonly replaced. An orthopedic surgeon can determine the best procedure based on how badly damaged the joint is.

Self-Care

Practicing these habits can slow down OA, keep you healthier overall and delay surgery as long as possible. It is important to pursue a number of different self-care approaches simultaneously. They are listed below.
Maintain a Healthy Weight
Excess weight worsens OA. Combine healthy eating with regular exercise to maintain a healthy weight.
Control Blood Sugar
Many people have diabetes and OA. Having high glucose levels can make cartilage stiffer and more likely to break down. Having diabetes causes inflammation, which also weakens cartilage.
Maintain Range of Motion
Movement is medicine for joints. Make a habit of putting your joints through their full range of motion, but only up to the point where it doesn't cause more pain. Gentle stretching, raising and lowering legs from a standing or seated position, daily walks and hobbies such as gardening can help. But listen to your body and never push too hard.
Protect Joints
Make sure to warm up and cool down when doing exercise. If you play sports, protects joints with the right gear. Use your largest, strongest joints for lifting, pushing, pulling and carrying. Watch your step to prevent falls. Balance rest and activity throughout the day.
Relax
Find ways to reduce or avoid stress through meditation, listening to music, connecting with friends and family, doing fun activities, and finding ways to relax and recharge.
Choose a Healthy Lifestyle
Eating healthy food, balanced nutrition, not smoking, drinking in moderation and getting good sleep will help you to feel your best.

Diagnosed With Osteoarthritis?

Get the latest news and tips about living with OA in the Living Your Yes! e-newsletter.

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Osteoarthritis: Symptoms, Diagnosis, and Treatment | Arthritis Foundation

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Osteoarthritis is recognized as a disease of the whole joint. Read important information about osteoarthritis, the most common form of arthritis, and its treatment.

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Osteoporosis is a condition where your bones lose strength, making you more likely to break a bone than the average adult.

Living with osteoporosis means you are at higher risk of breaking a bone. But not everyone with osteoporosis goes on to break a bone. It's not inevitable, and we're here to help you live well with osteoporosis.

A broken bone and a fracture are the same thing. As bones lose strength, they can break after a minor bump or fall. You may hear or read these described as fragility fractures.

Become a member today to get all the latest information to help keep your and your family's bones healthy and strong.

Newly diagnosed?

Learn more and find answers to common questions in our video series.

Get support from a Specialist Nurse

Contact our free Helpline for tailored information about osteoporosis and bone health:
nurses@theros.org.uk

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Osteoporosis is a condition where your bones lose strength, making you more likely to break a bone than the average adult. Living with osteoporosis means you are at higher risk of breaking a bone. But not everyone with osteoporosis goes on to break a bone.

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Angles: compass directions Video 27b Practice Questions

Area: quarter-circle Video 47a Practice Questions

Averages: combined mean Video 53a Practice Questions Textbook Exercise

Averages: mode (frequency table) Video 56a

Averages: range (frequency tables) Video 57a

Averages: Quartiles Video 57b Practice Questions

Circles: perimeter - quarter-circle Video 62a Practice Questions

Circle theorems proof: right angle in a semi-circle Video 65a Practice Questions

Circle theorems proof: angles at circumference\centre Video 65b Practice Questions

Circle theorems proof: angles in the same segment Video 65c Practice Questions

Circle theorems proof: cyclic quadrilaterals Video 65d Practice Questions

Circle theorems proof: alternate segment theorem Video 65e Practice Questions

Circle theorems proof: radius and tangent proof Video 65f Practice Questions

Circle theorems proof: Textbook Exercise

Constructions: loci part 3 Video 77 Practice Questions Textbook Exercise

Coordinates: 3D Video 86 Practice Questions

Data handling cycle Video 89 Practice Questions

Dimensional analysis Video 97 Practice Questions

Division: long division Video 98a

Enlargements: ray method Video 109 Practice Questions

Equations: involving fractions Video 111 Practice Questions Textbook Exercise

Equations: x squared Video 116a Practice Questions

Equations: Think of a number Video 116b Practice Questions

Factorisation: splitting the middle Video 119a Textbook Exercise

Factorisation: difference of 2 squares Video 120 Practice Questions Textbook Exercise

Fractions: finding the midpoint Video 141a Practice Questions

Graphs composite bar charts Video 148a

Graphs: dual bar charts Video 148b

Graphs: misleading graphs Video 160a

Graphs: real life linear graphs Video 171a

Linear graphs: real life Video 198a

Money - See Video 400 or click here for collection

Multiplication: grid method Video 199 Practice Questions Textbook 1 Textbook 2

Multiplication: column method Video 200 Practice Questions Textbook 1 Textbook 2

Multiplication: times tables Video 204a Textbook Exercise

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Number: product of primes (squares/cubes) Video 223a Practice Questions Textbook

Percentages: simple interest Video 236a Practice Questions Textbook Exercise

Proportion: unitary method Video 255a Practice Questions Textbook Exercise

Proportion: Graphs Video 255b

Proportion: Time Video 255c

Pythagoras: area of a regular hexagon Video 262 Practice Questions

Quadratic graphs: sketching using key points Video 265 Textbook Exercise

Quadratic graphs: finding turning point Video 265a Practice Questions

Quadratic graphs: line of symmetry Video 265b

Quadratics: solving (factorising) Video 266 Practice Questions Textbook 1 Textbook 2

Quadratics: solving graphically Video 267c Practice Questions Textbook Exercise

Quadratics: solving graphically advanced Video 267d Practice Questions

Questionnaires Video 268 Practice Questions

Ratio: express as fractions or % Video 269a Practice Questions Textbook Exercise

Ratio: given two ratios Video 271a Textbook Exercise

Ratio: difference between Video 271b Textbook Exercise

Ratio: expressing as 1:n Video 271c Textbook Exercise

Ratio: equations/ratios Video 271d

Ratio: solving problems 1 Video 271e Textbook Exercise

Sampling: using samples Video 281a

Sampling: random Video 282 Practice Questions

Scales and maps Video 283 Practice Questions

Sequences: nth term for fractional sequences Video 289 Textbook Exercise

Similar shapes: definition Video 291 Practice Questions

Similar shapes: further Video 294 Practice Questions

Simultaneous equations (substitution, both linear) Video 296 Practice Questions

Simultaneous equations (graphical) Video 297 Textbook Exercise

Surface area: mixture Video 309 Practice Questions

Surface area: other prisms Video 312

Transformations: mixture Practice Questions

Transformations of graphs Video 323 Practice Questions

Transformations of trigonometric graphs Video 324 Practice Questions

Trigonometry: sine rule (ambiguous case) Video 334a Textbook Exercise

Trigonometry: area of a triangle Video 337 Proof Practice Questions Textbook Exercise

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Types of graph: cubics Video 344 Practice Questions

Types of graph: exponential Video 345 Practice Questions

Types of graph: reciprocal Video 346 Practice Questions

Types of graph: mixture

Units: types of metric and imperial unit Video 347 Practice Questions

Units: Converting imperial units for length Video 348a Practice Questions

Units: Converting imperial units for mass Video 348b Practice Questions

Units: Converting imperial units for capacity Video 348c Practice Questions

Units: metric to imperial (length) Video 349d Practice Questions

Units: metric to imperial (mass) Video 349e Practice Questions

Units: metric to imperial (capacity) Video 349f Practice Questions

Vectors Video 353 Practice Questions

Views: front/side elevation and plan view Video 354 Practice Questions

Volume: Frustum Video 360a Textbook Exercise

Additional Topics

Flow Charts Video 364 Practice Questions

Geometric Proof Video 366

Identities: Equating coefficients Video 367 Practice Questions

Identities, Formula, Equations etc Video 367a Practice Questions

Intersecting Chord Theorem Video 368a Practice Questions

Intersecting Secant Theorem Video 368b Practice Questions

Quadratic graph (completing the square) Video 371

Equation of a Tangent to a Circle Video 372 Practice Questions

Iteration Video 373 Practice Questions

Iterative Processes Video 373a

Change of Sign Video 373a Practice Questions

Arithmetic progressions Video 374

Geometric progressions Video 375

Set notation Video 379

Venn Diagrams Video 380 Practice Questions

Shortest distance from point to a line Video 381 Textbook Exercise

Time series Video 382

Population Density Video 384a Practice Questions

Invariant Points Video 392 Textbook Exercise

Simultaneous Equations (3 Unknowns) Video 393 Textbook Exercise

Measuring Lines Video 397 Practice Questions

Drawing Lines Video 397a Practice Questions

Money: Coins Video 400a Practice Questions

Money: Change Video 400b

Money: Buying Video 400c Practice Questions

Money: Regular Payments Video 400d Practice Questions

Money: Bills Video 400e

Money: Discounts Video 400f Practice Questions

Money: VAT Video 400g Practice Questions

Money: Wages Video 400h Practice Questions

Money: Bank Statements Video 400i Practice Questions

Money: Tax Video 400j Practice Questions

Money: Cost per kg Video 400k Practice Questions

Money: Cost per litre Video 400l Practice Questions

Money: Cost per metre Video 400m Practice Questions

Money: Reading meters Video 400n

Money: Profit Video 400p Practice Questions

Money: Greatest/Maximum Video 400q Practice Questions

Money: Estimation Video 400r Practice Questions

Money: Midpoint Video 400s Practice Questions

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Videos, Practice Questions and Textbook Exercises on every Secondary Maths topic

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Functional Skills Maths Level 2 | Pass Functional Skills

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Functional skills maths level 2 revision, resources, past papers and mark schemes. Everything you need to revise level 2 maths.

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Numeracy in a Health Context Assessment Brief Overall Student Copy

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Numeracy in a Health Context Presentation

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https://corbettmaths.com/contents/

https://passfunctionalskills.co.uk/functional-skills-maths-level-2/