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A condition caused by a failure of the left ventricle to contract forcefully enough. Often as the result of damage caused by an infarct or prolonged high BP.
--> the left ventricle is unable to pump blood efficiently.
-> pulmonary vein "backed up"
-> cause increased cap. hydrostatic pressure
-> drawing fluid out of intravascular space into interstitial and alveoli
-> fluid accumulation in lungs

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2. Is it obstructive or restrictive? Why?

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2. Is it obstructive or restrictive? Why?

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its restrictive as fluid prevents alveoli to expand and collapse.

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3. How does the condition effect lung compliance and/or airway resistance?

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3. How does the condition effect lung compliance and/or airway resistance?

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Decreased lung compliance due to increased pulmonary blood volume/ venous congestion
Fluid accumulation
Decreased surfactant - alveoli collapse, poor expansion

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4. Explain the role of V/Q mismatch in this condition.

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4. Explain the role of V/Q mismatch in this condition.

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The alveoli are full of fluid which means they are unable to effectively participate in gas exchange which creates a V/Q mismatch (low V/Q ratio). The person may show symptoms such as SOB, hypertension, pink frothy sputum.

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5. How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

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5. How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

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Alveolar oxygen content may be high as normal O2 ventilation but O2 is unable to move into capillaries

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6. What type of respiratory failure would this condition lead to?

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6. What type of respiratory failure would this condition lead to?

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Initially would present as T1RF (hypoxaemia) but if not treated / reversed in time pt would get tired and would start retaining CO2 and become hypercapnic which would be a T2RF

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1. Choose a spokesperson

2. All students in the group are to work on all of the following questions/sections included in the Padlet

3. You can use diagrams/images other visual resources to explain each respiratory condition.

4. When we come back to the whole class, the spokesperson will feedback the groups findings, with the teams support

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High pH in the beginning
Hypoxic
Low to normal CO2 at start, increases as time goes on
Respiratory alkalosis in the beginning and changes overtime

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8. What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

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8. What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

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Clinical Manifestations:
Short of breath
Tachypneic
Inability to lie down
Oedema in legs
Increase weight
Frothy sputum (can be blood stained)

Patient Assessment Data:
Respiratory assessment including chest auscultation.
SpO2 monitoring
CO2 monitoring
ABGs
Physical appearance; anxious, tripod position
Chest xray

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9. OPTIONAL: Important vocabulary (include list of key vocabulary referenced; if you do not know the term(s), look up and add a definition for the word(s))

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9. OPTIONAL: Important vocabulary (include list of key vocabulary referenced; if you do not know the term(s), look up and add a definition for the word(s))