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Women = 2 X
Men = X Y

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Clotting cascade --> fibrin --> clots the injury

The two pathways of the coagulation (blood turns into solid) phase:
1. Extrinsic:
- activated by damaged cells in tissue; clotting factor VII; release of lipoprotein (tissue factor)
2. Intrinsic:
-activated by platelets exposed to collagen of damaged blood vessels; multiple clotting factors (speeds up reaction of the pathway)

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(Homostasis)

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a medical condition in which the ability of the blood to clot is severely reduced, causing the person to bleed severely from even a slight injury such as a papercut

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Helps form blood clots to reduce risk of excess bleeding.

Examples:
Desmopressin: t can treat diabetes insipidus. It can also treat bed wetting problems and certain bleeding disorders.
Aminocaproic acid: It can treat conditions that cause excessive bleeding. It can also control bleeding during or after surgery.

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Main gender affected are males with a high range of more than 50%

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Infancy to late 40s
Rates lower from ages 40+

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Hematologist

Focuses on diagnosing and treating blood disorders.

Orthopedic surgeon

Performs surgery for conditions affecting bones and muscles.

Physical therapist

Restores muscle strength and function through exercise.

Pediatrician

Provides medical care for infants, children, and teenagers.

Medical geneticist

Diagnoses and manages hereditary disorders.

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-injections of a clotting factor

-Desmopressin (DDAVP). In mild hemophilia, this hormone can stimulate your body to release more clotting factor. It can be injected slowly into a vein or provided as a nasal spray.
-Clot-preserving medications (anti-fibrinolytics. These medications help prevent clots from breaking down.

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Hemophilia acquired the name the royal disease due to the high number of descendants of Queen Victoria afflicted by it. The first instance of hemophilia in the British Royal family occurred on the birth of Prince Leopold on 7th April, 1853, Leopold was the fourth son and eighth child of Queen Victoria and Prince Albert of Saxe-Coburg-Gotha. No earlier occurrence of the disease in the Royal family had been known, it is assumed that a mutation occurred in the sperm of the Queen's father, Edward Augustus, Duke of Kent.

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-Large deep bruises
-Joint pain or swelling
-unexplained bleeding
-Blood in urine or stool
-internal bleeding
-nosebleeds