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7.  The specific drug examples found on your drug list under each drug classification including generic (lowercase) and brand (capitalized in parentheses with the trademark symbol at the end- ®) 

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7.  The specific drug examples found on your drug list under each drug classification including generic (lowercase) and brand (capitalized in parentheses with the trademark symbol at the end- ®) 

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Alpha- 1 adrenergic agonists

ephedrine

Phenylephrine (Neo-Synephrine®)

Adrenergic Agonists

epinephrine (Adrenalin®)

Alpha-1 Adrenergic Blocking Drugs

prazosin (Minipress®)

terazosin (Hytrin®)

doxazosin (Cardura®)

Beta Adrenergic Agonists (Beta-1 adrenergic agonist)

dobutamine

Beta-Adrenergic Agonists (Beta-2 adrenergic agonists

Albuterol (Proventil®)

terbutaline (Bretheine®)

formoterol (Foradil®)

salmeterol (Serevent®)

Beta-Adrenergic Blocking Drugs (Selective Beat-1 Blockers)

atenolol (Ternormin®)

metoprolol (Lopresoor®)

Beta-Adrengergic Blocking Drugs

nadolol (Corgardr®)

propanolol (Inderalr®)

timolol (Blocadrenr®)

Direct-Acting Cholingeric Agonists

acetylcholine

pilocarpine (Pilocar®)

bethanechol (Urecholine®)

Indirect -Acting cholinergic Agonist

neostigmine (Prostigmin®)

Antocholingerics/Parasympatholytics

atropine

dicyclomine (Bentyl®)

Depolarizing Agents

succinylcholine

Non-Depolarizing Agents

rocuronium bromide (Zemuron®)

Direct Acting Agents

dantrolene (Dantrium®)

Mast cell stabilizers

cromolyn sodium (Intal®, Nasalcrom®)

Antihistamines (sedating)

brompheniramine (Dimetapp®)

chlorpheniramine (Chlor-Trimeton®)

clemastine (Tavist®)

diphenhydramine (Benadryl®)

hydroxyzine (Atarax®)

promethazine (Phenergan®)

azelastine (Astelin®, Astepro®)

Antihistamines (non-sedating)

loratadine (Claritin®)

fexofenadine (Allegra®)

cetirizine (Zyrtec®)

Beta-2 adrenergic agonist (sympathomimetics)

albuterol (Proventil®, Ventolin®)

formoterol (Foradil®)

terbutaline (Brethine®)

salmeterol (Serevent®)

Xanthine derivatives

theophylline (Theo-Dur®)

aminophylline (Paladron®)

Antocholinergics

ipratropium bromide (Atrovent®)

Combo bronchodilators

ipratropium bromide + albuterol sulfate (Combivent®)

Cysteinyl leukotriene receptor inhibitor

montelukast (Singulair®)

Corticosteroids

budesonide (Rhinocort®, Pulmicort®)

mometasone (Nasonex®, Asmanex®)

fluticasone (Flonase®, Flovent®)

flunisolide (Nasalide®)

triamcinolone (Nasacort®, Azmacort®)

Expectorants

guaifenesin (Robitussin®, Mucinex®, Organidin®)

Antitussives

hydrocodone (Hycodan®)

codeine/guaifenesin (Robitussin AC®)

Decongestants

oxymetazoline (Afrin®)

pheniramine (Dristan®)

phenylephrine (Neo-Synephrine®)

pseudoephedrine (Sudafed®)

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 6.  The drug classifications for each division of the ANS. Under each classification, identify which receptors are interacted with, then include the action that occurs. 

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 6.  The drug classifications for each division of the ANS. Under each classification, identify which receptors are interacted with, then include the action that occurs. 

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6.  The drug classifications for each division of the ANS. Under each classification, identify which receptors are interacted with, then include the action that occurs.

Alpha- 1 adrenergic agonists

Alpha 1 receptor is stimulated

Contraction of smooth muscle, B.V constrict, ^ B.P

Vasoconstrict nasal passages which leads to decongestant effect

Adrenergic Agonists

Stimulates all alpha and beta receptors

Vasopressor-vasoconstriction, ^B.P

Cardiac stimulation

Bronchodilator

Alpha-1 Adrenergic Blocking Drugs

Binds to alpha-1 and blocks them.

Relaxes smooth muscle, ureters

^ Urine flow

Vasodilation of B.V, v B.P

Beta Adrenergic Agonists (Beta-1 adrenergic agonist)

Low dose: stimulates dopaminergic receptors in kidneys which leads to vasodilation and ^ renal blood flow.

Moderate dose- stimulates beta 1 receptors, heart. ^ force of contraction and cardiac output.

High dose- Stimulates alpha-1 receptors causing vasoconstriction to ^ B.P

Beta-Adrenergic Agonists (Beta-2 adrenergic agonists)

Stimulates Beta 2 receptors

Relaxation of smooth muscle in the lungs (bronchodilation)

Relaxation of uterus

Beta-Adrenergic Blocking Drugs (Selective Beat-1 Blockers)

Beta-1 receptros blocked (effect EPI and NE)

v force of contraction (negative inotropic)

v HR (negative chronotropic)

v conduction (negative dromotropic)

v cardiac output, v BP

Beta-Adrengergic Blocking Drugs

Block beta-1 and beta-2

v force of contraction, v cardiac output

v HR

v conduction

v cardiac output, v BP

Can cause bronchoconstuction

Direct -Acting cholinergic Agonists

Direct stimulate to muscarinin receptors in Parasympathetic div

Miimcs ACH

Indirect -Acting Cholinergic Agonists

Inhibit acetycholinesterase, enzyme that destroys ACH in synapse

Increase ACH

Anticholinergics/parasmpatholytics

Binds to cholnergic receptor

Acts as a blocking drug

Mast cells stabilizers

Inhibits mast cell release of histamine-decrease histamine, prophylaxis

Antihistamines

Block histamines from H-1 recep

Beta -2 adrenergic agonist (sympathomimetics

Stimulate beat-2 adrenergic recepetors

Xanthine deratives
-increase cyclic AMP, bronchodilation,
Anticholinergics

Blocks muscarianic receptors, block action of ACH

Cysteinly leukotriene receptor inhibitor

Anit-infammatory, formsation of prostaglandin derivatives

inahled/nasal steroids

Inhibit inflammatory response

Expectorants

Increase respiritory secretions, lubricate and liquefy mucus

Mucolytics

Liquefy bronchial secretions

Antitussives

Prevent coughs center brain from being stimulated

Decongestants

Vasoconstriction, stimulate alpha . decrease mucus swell, alleviate nasal stuffiness

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5.  The end organ responses that occur when each transmitter is stimulated. List the specific organ and state what response is generated in that organ when stimulated by each ANS division.

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5.  The end organ responses that occur when each transmitter is stimulated. List the specific organ and state what response is generated in that organ when stimulated by each ANS division.

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Sympathetic Adrenal medulla: vasoconstriction, ^H.R, ^AV conduction, ^ contractility, decreased GI, dilation, bronchodilation, bronchodilation, relaxation, contraction.

Parasympathetic Adrenal medulla: Arteries not supplied by parasympathetic nerves, decrease HR and AV, and increase GI, Constriction, bronchoconstriction, contraction, and relaxation.

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4.  The receptors of each division with main locations of each receptor.

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4.  The receptors of each division with main locations of each receptor.

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Acetylcholine (ACH)- at ganglia from presynaptic nerve endings

Norepinephrine (NE)- released at postganglionic nerve endings

Epinephrine (EPI)- adrenergic receptors

Dopamine (DA)- sympathetic and CNS neurotransmitters

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3.  The locations where each NT is released in each division of the ANS.

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3.  The locations where each NT is released in each division of the ANS.

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Acetylcholine (ACH)- Released at ganglia presynaptic nerve ending (both divisions)

Norepinephrine (NE)- Adrenal Medulla and neurons

Epinephrine (EPI)- Adrenal Medulla

Dopamine (DA)- Sympathetic and CNS neurotransmitters

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2.  The neurotransmitters for each division

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2.  The neurotransmitters for each division

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Acetylcholine (ACH)- cholinergic

Norepinephrine (NE)- adrenergic

Epinephrine (EPI)- adrenergic

Dopamine (DA)

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1.  The two divisions of the ANS and alternate names associated with each

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1.  The two divisions of the ANS and alternate names associated with each

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divisions of the ANS would be the sympathetic nervous system and the parasympathetic nervous system.

Sympathetic nervous system = Adrenergic

Parasympathetic nervous system = Cholinergic