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Então, foi muita coisa vivida e fico muito feliz de ver o progresso de vocês, ver crescer o amor pela odontologia, o amor pela odontopediatria, vencer os medos, olhar com ternura e firmeza, olhar para dentro de si. Direi que foi lindo, intenso, necessário e também conflitante.

Também precisamos avaliar, sugerir, afinal não acontecem só coisas boas.

Vocês podem fazer uma postagem, citando pontos positivos dessa jornada e também o que pode melhorar.

Podem fazer individualmente ou podem elencar tudo e pedir para a representante da turma postar. Sugiro fazer o check-in do início e também o final.

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Encerro as clínicas de COP I com inúmeras surpresas positivas, pois, aos poucos fui me permitindo vivenciar momentos felizes e desafiadores, compreendendo que o novo traz medo, mas, é enriquecedor e construtivo. E como refleti no inicio da disciplina, que o encanto infantil estimule nossa caminhada. Obrigada COP I!

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Sala de espera - A importância da higiene oral  

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Sala de espera - A importância da higiene oral  

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A sala de espera é um momento oportuno para o compartilhamento de informações acerca da odontologia, bem como, da saúde sistémica. Nesse momento, utilizamos de histórias infantis, uma linguagem ''simples'' e/ou acessível, manequins ilustrativos e dinamismo para conversarmos sobre a cárie dentária, a escovação dentária e o uso do fio dental. As crianças foram interagindo aos poucos e no final estavam todos conhecendo o famoso Tomé. Amei a experiência!

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Técnica de manejo da criança com idade inferior a 3
anos, com o auxilio do responsável. Nessa clínica conseguimos identificar as adequações necessárias para a realização do exame radiográfico, com o intuito de ser eficiente e ágil.

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Os nossos sorrisos demostram como o momento foi especial, alegre, divertido e significativo. Até breve, abrigo!

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As Clínicas são regadas de muitas mandalas, ambiente aromatizado e músicas. Tudo isso, é marca registrada do Professor Nilton, trazendo/ Contribuindo para a leveza do ambiente.

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Momento único que jamais esqueceremos!

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Na clínica do dia 16.03.23
Nessa clínica, fui solicitada a marcar Mizael para podermos fotografar a sua oclusão, para que posteriormente, pudéssemos avaliar e traçar o melhor plano de tratamento, visto que será necessário o uso de aparelho ortodôntico. (É valido ressaltar que é de grande valia, tratar o paciente integralmente e fazer os acompanhamentos periódicos).
O Caso derradeiro, foi o fato fatal de estar desparamentada, realmente me passei, mas isso não vem ao caso, o que realmente importa é o que foi proposto a realizar, no qual foi feito com êxito com a ajuda da minha dupla, Felipe. No mais, acompanharei o meu paciente com a finalidade de devolver saúde oral e função.

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Dia 02.03.23 foi o meu último atendimento com o meu querido paciente, Mizael. Nesse dia, fizemos a uma exodontia da unidade 46, sendo uma cirurgia tranquila, na qual procedeu como planejado. Ao final do atendimento, pelo bom comportamento e compreensão, presenteamos Mizael com um controle de jogos, já que ele relatava gostar bastante. Só gratidão por tudo que essas clínicas me proporcionaram, mostrando como é importante estramos firmes e preparados para executar os procedimentos, passando SEMPRE confiança aos pacientes, e a todo momento explicar com tranquilidade e firmeza o que será realizado. Dessa forma, chegaremos ao êxito. E claro, não poderia de expressar a minha gratidão aos professores de modo geral, pela paciência, confiança e pelo conhecimento compartilhado.

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Nesse dia, falamos da importância de uma alimentação saudável associada a uma boa higienização bucal, que são o melhor remédio para o combate á cárie. E depois tivemos uma mensagem em homenagem ao dia das mulheres!

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Eu e minha dupla na última clínica de COP I. Gratificante todos os momentos vividos no decorrer desse semestre!

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Nesse dia conversamos muito sobre a cárie e como ela adoece os nossos dentinhos. Falamos também da importância de ter hábitos de higiene bucal na prevenção da cárie, associados a uma alimentação saudável. Dia muito proveitoso!! Paciente super colaborativo, participativo e muito conversador!

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Descobrindo habilidades: fazer delineado rosa e pulserinhas as meninas das COP I.

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Encerrei a clínica com as extrações da Ana, esse doce de menina 💕

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A COP me deu esse pequeno de presente! Sempre colaborativo em todos os procedimentos 🤍
Se o check-in do início da COP foi “Esperando o melhor, mas pronta pro pior” o check-out será “Pronta pra COP II”

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Aromas, música da Sandy e anestesia bem feita = Completamente relaxado na cadeira da dentista, rs. 💙

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HMI (Hipomineralização de Molares e Incisivos)

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HMI (Hipomineralização de Molares e Incisivos)

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Fazer o diagnóstico diferencial entre os defeitos de esmalte é fundamental para o diagnóstico e o planejamento terapêutico.
Então vamos conhecer um pouco mais sobre HMI, pois ela acomete ambas as dentições.
Segue uma revisão sistemática sobre a prevalência nos segundos molares decíduos.
Boa leitura!!!

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Int J Paed Dentistry - 2021 - McCarra - Prevalence of hypomineralised second primary molars  HSPM   A systematic review and

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Bom dia turma! Segue uma revisão sistemática sobre a negligência odontológica em crianças e adolescentes. Precisamos entender que negligenciar a saúde odontológica também compromete a saúde geral do paciente

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Int J Paed Dentistry - 2021 - Khalid - Prevalence of dental neglect and associated risk factors in children and adolescents

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Por que às vezes é preciso usar antibióticos em associação?

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Antimicrobianos são produtos elaborados durante o metabolismo microbiano capazes de inibir parcial ou totalmente a multiplicação e crescimento de microrganismos. Estas substâncias são classificadas como antibióticos (substâncias químicas produzidas por microrganismos) e como quimioterápicos (substâncias sintetizadas ou produtos microbianos modificados estruturalmente em laboratório). O uso dessas substâncias químicas revolucionou a abordagem das infecções e o seu sucesso gerou grande otimismo em relação à prevenção e ao tratamento dos processos infecciosos. Entretanto, a prescrição nem sempre criteriosa ou racional desses antimicrobianos, rapidamente gerou dificuldades para seu uso, devido à progressiva resistência bacteriana a essas drogas (Montelli & Sadatsune, 2001).

A resistência a agentes antimicrobianos não é um fenômeno recente. A detecção das β-lactamases, tanto em bactérias Gram-positivas quanto em Gram-negativas, remonta-se aos inícios dos anos 40, antes do uso generalizado da penicilina no mundo todo (Abraam & Chain, 1940; Kirby, 1944). A produção de enzimas β-lactamases, tem sido relatada como um importante mecanismo de resistência a antibióticos β-lactâmicos, hidrolisando o anel beta-lactâmico pela quebra da ligação amida, perdendo assim, a capacidade de inibir a síntese da parede celular bacteriana (Williams, 1999). As β-lactamases podem ser detectadas em Enterobacteriaceae, Haemophilus influenza, Neisseria gonorrhoeae, Vibrio cholerae, Pseudomonas aeruginosa, Moraxella spp., (GARAU, 1994) e anaeróbios, tais como as espécies do grupo Bacteroides fragilis, cepas de Prevotella que produzem pigmento, Porphyromonas spp., Bilophila wadsworthia, Fusobacterium spp. e Clostridium spp. (Summanen et al., 1993; Könönen et al., 1998).

A produção dessa enzima por um microrganismo explica o porquê da sua sobrevivência em um foco infeccioso, apesar do uso de um antibiótico β-lactâmico. Além disso, a presença desses microrganismos produtores de β-lactamase pode favorecer a sobrevivência de outros microrganismos sensíveis ao antibiótico em um processo infeccioso (Tavares, 2001).

O aparecimento e a disseminação de microrganismos com múltipla resistência às drogas estão ocorrendo tanto nos hospitais quanto nas comunidades. Isto pode ser o resultado da interação de vários fatores, sendo talvez o mais importante, a pressão seletiva exercida pelas drogas sobre os microrganismos, dificultando assim, a sua sobrevivência na presença de agentes antimicrobianos (McGowan & Tenover, 1997). Por outro lado, não pode ser esquecida a troca de informações genéticas entre microrganismos, com transferência de genes para novos hospedeiros, e as mutações que podem ocorrer nos genes de resistência ampliando seu espectro de resistência (DeFlaun & Levy, 1989; Tenover & Hughes, 1996).

Mutações nos genes de resistência aumentam o espectro de atividade das β-lactamases de amplo espectro ou espectro extendido (ESBL - Extended Spectrum β-lactamases) (Philippon et al., 1989). As enzimas ESBL são capazes de hidrolisar as cefalosporinas de amplo espectro, como a cefotaxima e ceftazidima, e os monobactâmicos, como o aztreonam (Jacoby & Medeiros, 1991). Estudos têm sugerido que a pressão seletiva causada pelo uso constante de cefalosporinas nos centros de saúde contribui para o aparecimento e disseminação desses microrganismos resistentes (Rice et al., 1990; Coundron et al., 1997). Monnet et al. (1997) observaram que a resistência bacteriana causada pela produção de ESBL pode aumentar num período de 2 anos em até 57% em um único hospital. A presença de microrganismos que expressam novas enzimas capazes de hidrolisar carbapenêmicos, como imipenem e meropenem, vem cada vez mais sendo observada (Rasmussen & Bush, 1997).

A hidrólise do anel beta-lactâmico do núcleo estrutural das penicilinas - o ácido 6-aminopenicilâmico - provoca a formação do ácido penicilóico desprovido de atividade antimicrobiana. Consequentemente a quebra deste anel nas diversas penicilinas forma derivados do ácido penicilóico, igualmente inativos. De forma similar, ocorre com as cefalosporinas e carbapenêmicos (Tavares, 2001). Os antibióticos beta-lactâmicos são classificados pela semelhança estrutural adicionados de radicais que fornecem a estabilidade dessa droga (Figura 1).

Figura 1. Estrutura dos antibióticos beta-lactâmicos (Williams, 1999).

As β-lactamases podem ser encontradas extracelularmente em bactérias Gram-positivas, ou no espaço periplasmático em bactérias Gram-negativas (Bush, 1988). Os genes que codificam a produção dessas enzimas podem estar localizados no cromossomo bacteriano ou em plasmídeos. As β-lactamases de origem cromossômica são universais em algumas espécies bacterianas, enquanto que, aquelas de origem plasmídial são variáveis, permitindo que esses elementos sejam transferidos entre espécies. A mobilidade genética pode ser ampliada por meio de transposons, os quais transportam os genes da β-lactamase desde os plasmídeos até os cromossomos. Esta mobilidade é importante, pois permite que os genes resistentes se disseminem através das diversas comunidades bacterianas (Williams, 1999).

Embora, todas as β-lactamases catalisem a mesma reação, determinados tipos dessas enzimas têm sido isoladas e caracterizadas. Assim, essas enzimas foram classificadas segundo a estrutura primária (classe A à D), e as características funcionais e bioquímicas (grupo I à IV). Enzimas classificadas como classe A ou grupo II, hidrolisam penicilinas e cefalosporinas; classe B ou grupo III, carbapenêmicos; classe C ou grupo I, cefalosporinas; classe D, penicilinas e cloxacilina; e grupo IV (penicilinases de ), penicilina (Ambler, 1980; Bush et al., 1995). Pseudomonas cepacia

Os efeitos da enzima variam segundo a distribuição nas diferentes espécies bacterianas, a prevalência de produção da β-lactamase dentro de cada espécie e a prevalência em espécies patogênicas. Considerando-se então, esses três fatores, as enzimas da classe A mediadas por plasmídeos são consideradas as mais importantes por seus efeitos na prática clínica, incluindo-se as ESBL (Nicolas-Chanoine, 1996).

As cepas bacterianas que produzem as enzimas da classe A: TEM-1, SHV-1, ROB-1 ou PC-1 são resistentes à amoxicilina e à ticarcilina e apresentam susceptibilidade reduzida a piperacilina (Jacoby & Sutton, 1985; Livermore & Yang, 1987). Essas enzimas são inibidas por outros beta-lactâmicos denominados de inibidores de β-lactamase como sulbactam, tazobactam e ácido clavulânico, assim as cepas que as produzem são sensíveis às associações de antibióticos β-lactâmicos - inibidores de b-lactamase (Livermore, 1995).

Entre as enzimas da classe A mediadas por plasmídeos, as penicilinases destacam-se porque ocorrem numa grande variedade de patógenos, incluindo estafilococos, enterococos, Moraxella catarrhalis, Neisseria, Haemophilus, Bacteroides, Pseudomonas, Acinetobacter, e membros da família Enterobacteriaceae (Nicolas-Chanoine, 1996). A maioria (90%) das cepas de Staphylococcus aureus produz penicilinases (Lacey, 1984), e como consequência disso houve o desenvolvimento de novas penicilinas resistentes às beta-lactamases direcionadas ao S. aureus, tais como oxacilina e meticilina.

Penicilinases também são produzidas por 9-30% de H. influenzae que causam otite média (Doern et al., 1988) e em 60-80% de M. catarrhalis envolvida em broncopneumonia (Calder et al., 1986). As espécies do grupo Bacteroides fragilis também estão associadas a uma elevada (> 80%) produção dessa enzima (Nakano & Avila-Campos, 2004). Infecções envolvendo estes microrganismos geralmente são tratadas com uma associação de penicilina + inibidor de β-lactamase ou metronidazol, embora a cefoxitina ou carbapenêmicos também possam ser usados (Aldridge et al., 2001; Wexler et al., 2001).

Entre as enterobactérias, Escherichia coli é a espécie mais frequentemente encontrada tanto nas infecções adquiridas na comunidade como nas hospitalares. Cerca de 40-50% das E. coli, particularmente aqueles isolados de infecções do trato urinário (ITU) produzem penicilinases (Henquell et al., 1995). As espécies de Klebsiella pneumoniae podem produzir β-lactamases codificadas por cromossomo ou por plasmídeo (Liu et al., 1992). Entre os outros membros da família Enterobacteriaceae 10% de Enterobacter cloacae produzem penicilinases. Em Pseudomonas aeruginosa que é responsável por 9-12% das infecções hospitalares e nas UTI a prevalência de cepas resistentes à tricarcilina é 10% a 30 % (Vicent et al., 1995; Chen et al., 1995).

Já as β-lactamases da classe C são produzidas por vários membros da família Enterobacteriaceae, incluindo as espécies Enterobacter e Serratia, Morganella morganii, Citrobacter freundii e Acinetobacter baumannii, assim como P. aeruginosa e E. coli produz cefalosporinase, mas essa enzima manifesta-se de maneira constitutiva, em níveis baixos e não apresenta ação contra os antibióticos β-lactâmicos. De modo inverso, a produção da cefalosporinase em outras espécies, pode ser induzida e regulada por proteínas ativadoras ou repressoras (Lindberg & Normark, 1986; Honoré et al., 1989). (Lindberg & Normark, 1986; Jarvis & Martine, 1991). Entretanto, o impacto dessas enzimas na terapia antibacteriana varia de acordo com seu modo de expressão. Por exemplo,

As cefalosporinas de amplo espectro com uma cadeia lateral oximino, tais como cefotaxima, ceftazidima e ceftriaxona, e o monobactâmico aztreonam, foram lançadas nos anos 60 visando-se a ação contra os patógenos Gram-negativos, porque essas drogas não eram hidrolisadas pelas β-lactamases de origem plasmidial. Entretanto, em pouco tempo foi relatada a presença de isolados clínicos resistentes a um ou mais desses antibióticos, que poderiam propagar esta resistência através de plasmídeos transmissíveis e que eram capazes de hidrolisar oximino-β-lactâmicos. As enzimas envolvidas foram chamadas de β-lactamases de espectro estendido (ESBLs) derivadas de enzimas mediadas por plasmídeos (Medeiros, 1997).

Várias β-lactamases de origem plasmidial são produzidas por patógenos Gram-negativos, porém a enzima mais comum é a TEM-1. A β-lactamase TEM-1 tem sido considerada uma enzima totalmente eficiente (Bush et al., 1995). Hidrolisa a penicilina e os derivados de penicilina, tais como ampicilina, carbenicilina e piperacilina. É também bastante ativa contra a cefalotina e a cefuroxima, porém apresenta atividade insignificante contra cefotaxima, ceftazidima, ceftriaxona ou aztreonam, e não possui nenhuma ação contra as cefamicinas (cefoxitina ou cefotetan). O TEM-1 possui um isotipo menos comum, chamado de TEM-2, uma enzima com propriedades cinéticas idênticas, porém com a diferença de possuir um único aminoácido, que muda o ponto isoelétrico (pI) da proteína de 5,4 a 5,6. Na espécie K. pneumoniae a enzima chamada SHV-1 é o tipo mais comum encontrado nos plasmídeos e é também determinada por genes cromossômicos (Leung et al., 1997). A enzima SHV-1 possui 68% de aminoácidos idênticos aos do TEM-1 e um pI de 7,6. O grupo OXA contém uma série menos comum de β-lactamases codificados por plasmídeos, caracterizadas por hidrolisar oxacilina, floxacilina e meticilina, mas que não apresentam qualquer atividade contra os substratos oximino, TEM-1, TEM-2 e SHV-1. As enzimas do grupo OXA, são rapidamente inibidas pelo ácido clavulânico, sulbactam e tazobactam (Jacoby, 2000). Atualmente, são conhecidas 61 ESBL na família TEM-1, 12 na família SHV e 6 na família OXA. Elas são tão numerosas que foi criado uma página na Internet para documentar as suas propriedades ( www.lahey.org/studies/webt.htm).

As enzimas ESBL foram observadas inicialmente em bacilos Gram-negativos isolados em pacientes da Europa e foram logo em seguida encontrados nos Estados Unidos (Quinn et al., 1989). Atualmente, essas enzimas são descritas em nível mundial (Philippon et al., 1994). Este tipo de resistência é comum, principalmente em K. pneumoniae e, em menor grau, em E. coli. Porém, as ESBLs também são encontradas em Citrobacter diversus, C. freundii, E. cloacae, Enterobacter aerogenes, Proteus mirabilis, P. aeruginosa, K. oxytoca, S. marcescens e Salmonella spp. (Jacoby, 2000).

Duas estratégias bacterianas têm sido utilizadas para superar a resistência dos beta-lactâmicos à β-lactamase. A primeira é a modificação da estrutura do antibiótico de forma que não haja mais substrato para a enzima hidrolisar, e a segunda pela inibição da enzima por um composto que seja estruturalmente relacionado ao substrato beta-lactâmico (Moosdeen, 1996).

Os inibidores de β-lactamase são estruturalmente semelhantes às penicilinas, retendo a ligação amida do grupo beta-lactâmico, mas possuem uma cadeia lateral modificada (Figura 2). Tais aspectos estruturais permitem aos inibidores ligar-se irreversivelmente às β-lactamases como substratos suicidas, mantendo-as inativas. Atualmente, três inibidores de β-lactamase são frequentemente usadas na clínica médica, sulbactam, tazobactam e ácido clavulânico (Williams, 1999).

Figura 2. Estrutura dos inibidores de β-lactamase (WILLIAMS, 1999).

A combinação de um agente β-lactâmico e um inibidor de β-lactamase tem mostrado ser uma boa opção de tratamento, já que o inibidor inativará a ação da enzima permitindo que as penicilinas, as quais são eficientes e bem toleradas continuem a tratar as mais variadas infecções (Williams, 1999).

A incidência crescente da produção de β-lactamases por patógenos presentes na comunidade como em hospitais, tem exercido um impacto considerável na maneira de prescrever os antimicrobianos. No tratamento de infecções comuns adquiridas pela comunidade tais como infecções do trato urinário (ITU), otite média ou broncopneumonia, o uso de penicilinas tem diminuído, aumentando assim, o uso de associações de penicilinas ou cefalosporinas de segunda e terceira geração com algum inibidor de b-lactamase (Nicolas-Chanoine, 1996).

Os microrganismos produtores de ESBL ocorrem com frequência em surtos e causam um dilema terapêutico devido ao padrão de resistência a múltiplas drogas. Esses microrganismos também causam uma preocupação com o controle da infecção devido à disseminação de cepas de paciente para paciente, e pelo aparecimento de cepas policlonais devido à transmissão de elementos genéticos entre cepas e gêneros distintos de bacilos Gram-negativos (Patterson, 2000).

As características epidemiológicas dos surtos de ESBL têm sido estudadas por vários pesquisadores. Dentre os fatores de risco associados à aquisição de microrganismos que produzem ESBL, podemos citar o uso de cateteres (arterial, venoso central e urinário), colonização intestinal, duração da estada na UTI ou mesmo no hospital, administração anterior de ceftazidima ou aztreonam, cirurgia abdominal de emergência e assistência ventilatória (DeChamps et al., 1991; Schiappa et al., 1996; Lucet et al., 1996; Piroth et al., 1998).

Medidas para controlar surtos foram relatadas incluindo a restrição ao uso de oximino-beta-lactâmico (especialmente ceftazidima) e um controle rígido das infecções com ênfase nos cuidados de barreira e lavagem das mãos. O trato intestinal é um reservatório de microrganismos que produzem ESBL, assim, a descontaminação intestinal tem sido também usada para o controle de surtos nas UTI (Brun-Buisson et al., 1989; Peña et al., 1998).

A avaliação dos tratamentos para o combate às infecções devido aos microrganismos que produzem ESBL é complicada pela diversidade dos tipos dessa enzima e pela variedade de fatores que podem modificar sua expressão. Os resultados podem ser influenciados pelo tipo de infecção e dependendo se o paciente está efetivamente infectado ou apenas colonizado. Frequentemente os pacientes estão infectados com vários patógenos necessitando de múltiplos agentes antimicrobianos. Também, é importante mencionar que, trocas significativas de uso de antibióticos também exercem uma pressão seletiva para novos microrganismos resistentes (Jacoby, 2000).

Na era dos antibióticos, as β-lactamases mostraram ser extremamente adaptáveis. Apareceram em novos hospedeiros, mudaram sua expressão para níveis mais altos, alteraram sua susceptibilidade aos inibidores e aumentaram o leque de seus substratos. Os principais tipos moleculares das β-lactamases nos patógenos Gram-negativos são codificados por genes localizados em plasmídeos conjugáveis ou transposons, facilitando assim, a disseminação da resistência aos β-lactâmicos, mas também promovendo a co-seleção em relação a outros antibióticos. As bactérias são capazes de evolução rápida em resposta à pressão seletiva exercida pelo uso generalizado de antibióticos, e assim, as mudanças continuadas observadas nas características das β-lactamases não devem ser surpresas.

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Autores: Viviane Nakano (Jovem Pesquisador - Professor Colaborador) Sheila A. Nishiyama (Doutoranda em Microbiologia) Mario J. Avila-Campos (Responsável pelo Laboratório de Anaeróbios)

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Beta-lactamases: Sua importância na resistência bacteriana

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Antimicrobianos são produtos elaborados durante o metabolismo microbiano capazes de inibir parcial ou totalmente a multiplicação e crescimento de microrganismos. Estas substâncias são classificadas como antibióticos (substâncias químicas produzidas por microrganismos) e como quimioterápicos (substâncias sintetizadas ou produtos microbianos modificados estruturalmente em laboratório).

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 Como agir frente ao risco de endocardite devido ao atendimento odontológico ?

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 Como agir frente ao risco de endocardite devido ao atendimento odontológico ?

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O melhor medicamento é amor e cuidado, mas umas gotinhas ajudam, neh?, sendo controladinhos ou não.

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Procedimentos dentários e endocardite infecciosa

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Procedimentos dentários e endocardite infecciosa

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Infective endocarditis (IE), also called bacterial endocarditis, is an infection caused by bacteria that enter the bloodstream and settle in the heart lining, a heart valve or a blood vessel. IE is uncommon, but people with some heart conditions have a greater risk of developing it.

Risk factors for developing IE include:

View an illustration of endocarditis.

Dental Procedures and Infective Endocarditis

People with the highest risk for poor outcomes from IE may be prescribed antibiotics (IE prophylaxis) prior to certain dental procedures to reduce their risk of developing it. These include procedures that involve manipulation of gingival (gum) tissue or the periapical region (area around the roots) of teeth, or perforation of the oral mucosa.

Antibiotic prophylaxis is reasonable before the above-mentioned dental procedures for people with heart valve disease who have any of the following:

1. Prosthetic cardiac valves, including transcatheter-implanted prostheses and homografts.

2. Prosthetic material used for heart valve repair, such as annuloplasty rings, chords or clips.

3. Previous IE.

4. Unrepaired cyanotic congenital heart defect (birth defects with oxygen levels lower than normal) or repaired congenital heart defect, with residual shunts or valvular regurgitation at the site adjacent to the site of a prosthetic patch or prosthetic device.

5. Cardiac transplant with valve regurgitation due to a structurally abnormal valve.

Except for the conditions listed above, antibiotic prophylaxis before dental procedures is not recommended for any other types of congenital heart disease.

In addition, antibiotic prophylaxis is not recommended for patients with valvular heart disease who are at high risk of IE for nondental procedures (e.g., TEE, esophagogastroduodenoscopy, colonoscopy, or cystoscopy) in the absence of active infection.

You can reduce the risk of IE by maintaining good oral health through regular professional dental care and the use of dental products such as manual, powered and ultrasonic toothbrushes; dental floss; and other plaque-removal devices.

If you meet the requirements for antibiotic prophylaxis for dental treatment or oral surgery, your cardiologist or other health care professional may give you an American Heart Association wallet card (PDF). Show this card to your dentist, pediatrician, family doctor or other health care professional. It recommends the type of antibiotic and dose for IE prophylaxis. For smaller children, the dose will vary according to the child's weight. Always remind your dentist or doctor if you (or your child) are allergic to any antibiotics or other medications.

Your health care team can provide you more information and answer your questions about preventing IE.

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Infective endocarditis (IE), or bacterial endocarditis, is an infection caused by bacteria that enter the bloodstream and settle in the heart lining, a heart valve or a blood vessel. IE is uncommon, but people with some heart conditions have a greater risk of developing it.

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E sobre essas regras práticas para prescrição?

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E sobre essas regras práticas para prescrição?

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O que acham? Será que podemos confiar? Amanhã falaremos mais sobre.

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Sabemos que o índice ICDAS é importantíssimo para a odontopediatria. Por conta disso, fiz um resumo ilustrativo para melhor dinâmica nos atendimentos. Para a clínica do dia 02/03 teríamos paciente novo, estávamos animadas, porém, novamente ele faltou. A professora Lívia nos passou então duas pacientes, e QUASE que eu faria minha primeira cirurgia em COP, mas a pró achou melhor encaminhar as duas pacientes para COP III para serem acompanhadas de início ao fim na disciplina. Semana que vem tem mais, estamos ansiosas para as surpresas de COP I. 👧🏻🧒🏽

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Escola de Samba UNIDOS DOS SEM PACIENTES 🥳🍾

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Escola de Samba UNIDOS DOS SEM PACIENTES 🥳🍾

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A clínica após o Carnaval (23/02) não teve muitos pacientes. Infelizmente neste dia, o meu paciente (novo) faltou e seria um dia atípico, pois estaria acompanhada por Igão. 😭💔

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Nossa primeira clínica na manhã do dia 02.02.2022! Muito bom estar de volta... nosso paciente faltou, mas quase no finalzinho da clínica apareceu um rapazinho precisando de atendimento. Tratava de uma grande lesão coronária de cárie. No exame radiográfico ficou constato que não há necessidade de endo, a dor se dava por conta do contato íntimo e exposição da dentina. Então, foi feita uma limpeza do tecido dentinário com cureta, sem necessidade de anestesia. Foi feita também a desinfecção do tecido com clorexidina a 0,2%, seguida da deposição de coltosol (por falta do material de primeira escolha CIV). Como o coltosol não tem uma boa retenção e nossa próxima clínica vai demorar um pouquinho o prof° Nilton resolveu terminar o caso do paciente na mesma semana, para que ele tivesse mais segurança e estivesse livre de dor.
Nossa clínica da semana foi assim... Ainda teve essa mesa linda, cheia de brinde para os pacientes!

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Na clínica do dia 02.03 atendemos uma paciente, de 10 anos, bem legal, estilosa, cabelo verde, gente boa, adoramos kkk
Ela estava com mobilidade nos caninos superiores. Fizemos a panorâmica, chamamos a professora Lívia para análise ortodôntica, essa paciente será encaminhada para a COP II...
Enfim, a mãe da criança nos relatou que a menina tinha medo e que chorava quando precisava extrair algum dentinho.
De início tentamos a extração apenas com o anestésico tópico, seguindo as orientações da profa Carol... Mas não rolou. Então, fizemos anestesia infiltrativa interpapilar e foi mole! Rapidinho, sem choro, sem estresse apareceram duas janelinhas no sorriso da nossa paciente!
Pensei q dessa vez ia ter choro e uma situação delicada, só que mais uma vez foi tão leve e bom! A COP me surpreende todo dia, é a matéria que está nos dando alegria na clínica. Aleluias!!!!! :D <3

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Ampliando o conhecimento sobre crianças com condições cardíacas diversas

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Ampliando o conhecimento sobre crianças com condições cardíacas diversas

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Question:

My 4-year-old daughter was recently diagnosed with a hole in her heart. It is called an atrial septal defect. We were told that it should be closed. I heard that there is a new way to close the hole without having surgery. Can this be done any way other than with surgery? My older son had a patent ductus arteriosus surgically repaired 10 years ago when he was 1 year old. Could his patent ductus arteriosus have been closed by this new method that does not require surgery?

Answer:

An atrial septal defect is a hole in the wall that separates the two upper chambers of the heart known as the right atrium and left atrium. The hole can be located in various locations in the wall (the septum). There are new methods of closing this hole without open-heart surgery, but it depends on the location of the hole. Presently, the only holes that can be closed are known as secundum atrial septal defect and patent foramen ovale (PFO). Both of these types of holes have an adequate rim around the hole onto which the device is attached. The non-surgical method of closure is by implanting a device, which resembles a dumbbell into the hole to "plug it up" from within the heart. This is accomplished by passing a small tube (catheter) from a vein located in the groin and up into the heart. The procedure is performed in the cardiac catheterization laboratory and does not require surgery. Several devices are available and the results have been comparable to surgical results. The risks, which are rare, are much the same as for any cardiac catheterization. They include clot formation, bleeding, infection, perforation of the heart, and arrhythmias (heart rhythm disturbances). For device implantation, device embolization is an additional risk (the device could break free and travel to another part of the heart or blood vessels), which may require surgical retrieval. This is an extremely rare risk because most of the devices are designed for safe repositioning and retrieval in the cath lab if necessary. Should embolization occur, the surgeon would simply repair the defect at the time of the retrieval. Another risk is a residual leak across the device, but usually the leak is sealed in two to six months as the body grows new tissue over the device. The cath procedure would also require transesophageal echocardiography, which is an echocardiogram performed with the probe passed down the throat to measure the size of the defect and assist in determining proper position of the device. The entire procedure is performed under general anesthesia. Keep in mind that you will need to be on aspirin for six months after the procedure. The aspirin is used to keep blood clots from forming on the device while the body heals over it. The risks of surgical closure would include most of the cath lab risks such as bleeding, infection, blood clots, arrhythmia and residual leaks. Other inherent risks involving open-chest and open-heart surgery include the need for blood transfusions, prolonged bed rest, lung collapse and lung infections, and fluid forming within the sac of the heart. Overall, both procedures are effective and have low risk. While surgery will be more painful and require longer recovery time and will result in a scar on the chest, it has a proven track record over many decades. The cath lab method is newer and the long-term results are not yet known but appear to be as good as surgery.

The patent ductus arteriosus can also be closed without surgery. It is similar to the ASD closure but different devices are available. The most commonly used device is called a coil, which has FDA approval for use in various unwanted blood vessels in the body. The coil has been in use for nearly 30 years and has proven to be quite safe and effective for the smaller PDA's. Other devices are available for larger PDA's, some of which are still investigational. Except for the very small infant, or the patient with a very large ductus, the preferred method of PDA closure in most pediatric cardiology centers is by cardiac catheterization.

Question:

My 11-year-old son was recently diagnosed with a bicuspid aortic valve. What is this? Can he participate in competitive sports? Will he ever need surgery or medical treatment for this?

Answer:

The term bicuspid aortic valve refers to the leaflets of the aortic valve. This is the valve that leads out of the heart to the aorta, the big blood vessel that delivers oxygen-rich blood to the body. Normally, the aortic valve has three leaflets. A bicuspid valve has only two leaflets.

Because of this, patients with a bicuspid valve may develop a narrowing or leakage of the aortic valve. Narrowing or leakage of aortic valves do not necessarily develop in childhood, but may occur in adulthood. On rare occasions these problems require repair, either with a balloon (cardiac catheterization) or surgery. To determine whether narrowing or leakage is present and related to the bicuspid aortic valve, the cardiologist follows the patient with non-invasive tests that may include: electrocardiograms, echocardiograms and stress tests. If narrowing or leakage of the valve becomes severe enough to require intervention, your doctor will explain this to you in more detail. In many cases, bicuspid aortic valves do not require any intervention in childhood and children with them may not need activity restriction. A pediatric cardiologist will make specific recommendations for activity restrictions for each patient based on the severity of the valve narrowing or leakage. Restrictions may be put in place to assure that a normal amount of oxygen gets to the patient's heart muscle at all times.

Question:

What is "cardiac asthma"? I thought asthma was caused by allergies. My son has seasonal asthma and takes an inhaler. Does he have a heart problem also?

Answer:

Asthma is a spasm of small air tubes (bronchioles) in the lungs caused by many factors that affect how air passes through the lungs. These include allergies, pollens, pollutants, exercise, stress and lung disease. Patients usually have difficulty breathing associated with wheezing and coughing. Treatment is directed at opening up the small air tubes with inhalers or other oral medications. Most children with asthma have this type of problem. "Cardiac asthma" mimics lung asthma but is much less common. Patients with heart failure or heart valves that don't open properly can also experience wheezing and coughing that is known as cardiac asthma. However, the bronchial spasm in cardiac asthma is caused by back pressure from the left side of the heart to the lungs. Symptoms usually occur with exercise or at night after going to bed. If a child has a congenital heart defect, coughing and wheezing may be symptoms of heart failure and require evaluation by a congenital heart specialist. Special tests such as an echocardiogram or chest x-ray may be required to determine if a child has a congenital heart defect. Treatment is directed at improving the pump function of the heart with medications. If the asthma is caused by a heart valve that is not working properly, or a hole between the heart chambers, surgery or other procedures may be required.

Question:

I had a child with a congenital heart defect; why did this happen? What is the likelihood of my having another child with the same defect or a different defect?

Answer:

Heart defects, especially those occurring as isolated problems in otherwise healthy infants, do not always have readily identifiable genetic or environmental causes. As the circulatory system is one of the first organ systems to begin functioning, blood flow problems occurring in early fetal life can be compounded as the fetus develops.

Slightly less than 1 percent of infants born in the United States (about eight per 1,000 live births) have a congenital heart defect. Depending on the defect, the chances of a sibling also being born with the same defect or other heart defect are about 2 percent to 3 percent. While this is higher than the general population rate of 0.8 percent, it is still a relatively small chance. Some genetic defects such as Down Syndrome are associated with heart defects. Several genetic syndromes have heart defects as well as defects in other organ systems. About 25 percent of children with a heart defect have one or more additional abnormalities in another organ. These facts all point to a genetic component.

Numerous environmental exposures are also associated with congenital heart defects. These include using some medications for seizures, acne or bipolar psychiatric disorder, or having some diseases such as rubella (German measles), other viral diseases, diabetes or lupus early in pregnancy. Alcohol and street drugs may increase the risk also, as can exposure to chemicals such as certain pesticides and solvents.

Most congenital defects are not that common and not likely to occur in future children. Consultation with a genetics counselor to find out more about your particular risk for future pregnancies is recommended. Remaining healthy and avoiding certain exposures is helpful. Fetal echocardiograms (ultrasounds) may help detect heart problems as early as 12-14 weeks of pregnancy.

Question:

My 12-year-old son complains of chest pain when he plays soccer. Is this serious? Do I need to take him to a heart specialist? What is causing his chest pain?

Answer:

We're aware that heart problems can cause chest pain. It is natural to worry when a child or adolescent complains of chest pain that their heart is the cause. Many parents may even wonder if their child might be having a heart attack.

It's important to realize problems that result in heart disease in adults are rare in children. And, though chest pain is pretty common in youngsters, it is very rare that this pain is related in any way to their heart. Most chest pain in children is not serious. Many times the source of the pain cannot even be determined.

Other causes of chest pain common in children include pains from pulled muscles or sports-related injury, pain related to asthma or cough, and pain due to infections like pneumonia and pleurisy. Usually, if the pain is caused by anything serious, there will be other signs. For example, infections usually cause fever, too.

Your child's primary care doctor is the best person to speak with if you have concerns about your child's pain. If your child's chest pain was associated with a fainting episode, this may be a sign of a serious problem, and you should seek medical attention right away; meanwhile, your child should refrain from sports.

Question:

My granddaughter was born with coarctation. What is coarctation and how is it treated?

Answer:

Coarctation of the aorta is a narrowing of the aorta, the large blood vessel coming out of the heart that provides oxygen-rich blood to the body. Coarctation of the aorta causes the heart to work harder to generate enough pressure to push the blood through the narrowing. This causes high pressure in the left side of the heart, and in the arms, while the legs will have lower blood pressure. In babies with coarctation of the aorta, the heart may fail to generate the high pressure needed, and the child may show signs of heart failure and need urgent intervention. Overall, the long-term outlook for a child born with this defect is excellent, as long as the defect is repaired in a timely manner.

Coarctation of the aorta can be fixed with an operation or an interventional catheterization. There are a few different ways in which surgery can be done. The surgeon can cut out the area of the narrowing and sew the two ends back together or use extra tissue (patch) to make the area bigger. Depending on your granddaughter's age and anatomic details it may be possible to fix the coarctation with a catheterization using a balloon and possibly a stent. The long-term problems to watch for after surgery or catheterization include the possibility that the narrowing would come back in the area of the operation, usually related to scar tissue, requiring another intervention. In most cases, if the narrowing comes back the narrowing can be stretched open with a balloon via cardiac catheterization with a high success rate. A more rare possibility is that the area of the aorta where surgery was done could weaken over time, resulting in dilation (enlargement) and aneurysm (weakening) of the vessel walls. If this happens, she may also need another operation.

Your granddaughter's doctors will be checking her after the operation as she grows, to make sure those problems are not present. Your granddaughter could have high blood pressure in the future. However, when coarctation of the aorta is fixed early in life, this is less likely to become a problem. There is a small risk of endocarditis (heart infection) after repair of coarctation. Some girls with coarctation may have a chromosome problem called Turner Syndrome and these children may need to have medical evaluation for other possible non-heart problems.

As long as there is no significant narrowing left in the aorta and her blood pressure is normal, she should be able to participate in sports without limitations and have a normal life.

Question:

My baby turns "blue" when she cries. Does she have a heart problem?

Answer:

It's very common for babies to develop a "blueness" around the mouth when they are crying or if they seem to be holding their breath. It's also common for babies to get blue hands and feet when they're cold. This is a very common observation made by parents and describes a normal condition termed "acrocyanosis." It's a normal response in babies and adults to reflect cool fingers and toes. Even normal room temperature may seem cold to an unclothed baby.

A physician should be consulted for persistent blueness (even at rest), blueness involving lips or gums, or blueness associated with difficulty breathing or lethargy and poor feeding.

Enlarged Heart

Question:

What is the cause of an enlarged heart in an 11-year-old child? His chief complaints are chest pain and severe coughing and sometimes vomiting.

Answer:

The finding of an enlarged heart in a child or an adolescent who complains of chest pain, cough and some vomiting should prompt an immediate investigation to determine the precise nature of the problem. Several heart conditions can lead to these symptoms in a child who has been previously in good health. Examples include inflammation of the membrane or sac that surrounds the heart (a condition called pericarditis), which is often associated with accumulation of fluid in that sac (called pericardial effusion), and inflammation of the heart muscle itself (a condition called myocarditis that may be caused by a virus). Less common conditions that can manifest with these symptoms and an enlarged heart include weakness of the heart muscle (a condition called dilated cardiomyopathy), rheumatic fever (infrequent in the United States), infection of the heart valves (called endocarditis), and a previously unknown congenital abnormality of the heart or blood vessels. Heart attacks, which can occur in adults, are extremely rare in children and most likely not the cause of chest pain and enlarged heart.

Most frequently, the enlarged heart is detected on a chest x-ray, a test that only gives a general idea about the overall size of the heart and blood vessels. The pediatric cardiologist will conduct a thorough investigation of your child, including detailed history, careful physical examination, and an electrocardiogram (ECG). An echocardiogram (cardiac ultrasound) is a key test in evaluating an enlarged heart. This test will provide detailed information about the anatomy, size and function of the cardiac chambers. Sometimes, additional tests, such as cardiac catheterization and heart muscle biopsy, may be required to complete the investigation.

Fainting Spells in Children

Question:

For the past few months, my daughter has been passing out at school. I have been told that there is nothing to worry about and all I should do is put more salt into her diet. Although she eats lots of pretzels and potato chips, she still keeps passing out. I am afraid! What could this be and can she be treated?

Answer:

Fainting spells are a common problem in children. Most of the time fainting is not a life-threatening problem, but there are some serious causes of fainting that must be ruled out by a specialist in pediatric heart conditions. Assuming that these serious causes of fainting have been ruled out, it sounds like your daughter has simple fainting spells, which has the scientific name of "neurocardiogenic syncope." The important thing to remember is that this condition is not life threatening and it can be treated in many ways. The treatment that you described of increasing salt intake, and/or fluid intake, is usually the first step for treating neurocardiogenic syncope or "fainting spells." If that doesn't work, you can use several other things. At this time, the best thing for you to do is to call your doctor back and let him know that the current treatment is not working so that other treatments can be considered or other tests can be performed.

Fetal Echocardiogram

Question:

I am 20 weeks pregnant and thought everything was going fine. My obstetrician said she noticed a "bright spot" in my unborn baby's heart and told me to go see a pediatric cardiologist for a fetal echocardiogram. My daughter has a heart murmur, but I was told it did not mean anything. What is a fetal echocardiogram and why do obstetricians refer patients for them? Is a "bright spot" on the heart serious? Does my daughter's heart murmur make my unborn baby more likely to have a heart problem? Does the heart of my unborn baby work the same way before and after the baby is born?

Answer:

Your obstetrician may want to explore the possibility of a heart defect in more detail. Heart problems are the most common birth defect, occurring in about 1 percent of children. A fetal echocardiogram is a specialized ultrasound performed by a pediatric cardiologist that focuses on the fetus's heart. The machine looks exactly the same as a regular ultrasound machine but images only the heart. It has specialized probes and hardware that allow clear images of the fetal heart. There is no risk to the fetus and you do not have to have a full bladder for the test. If a heart problem is found, the pediatric cardiologist can explain the diagnosis and what it would mean for the baby after birth.

An obstetrician may refer someone to a pediatric cardiologist for a fetal echocardiogram for several reasons. Some examples are

suspicion of a heart defect on routine ultrasound

heart rhythm abnormality

family history of congenital heart disease

a problem in another organ of the fetus

certain maternal illnesses or medication exposures

In most cases this test is just precautionary and is done to reassure you that everything is normal. This is almost always the case when the reason for referral is a "bright spot." They almost always go away by the time the baby is born. If you or one of your children has a structural congenital heart defect, then future babies are at slightly increased risk. However, most heart murmurs are innocent and in no way mean that future children are at increased risk.

It is important to know that the fetal circulatory pattern is different than after the baby is born. That's because the placenta, not the lungs, provide oxygen to the baby. There is an extra vessel and hole in the heart that allow blood to bypass the lungs. The fetal circulatory pattern may protect the fetus from getting sick, even if there is a heart problem. This circulatory pattern also makes it impossible for the pediatric cardiologist to rule out small holes in the heart or a blockage in the blood vessel that leads to the lower half of the body. All normal fetal echocardiogram studies have this limitation.

Fetal echocardiography is usually very accurate but can be limited if the baby is not in a good position or there is low amniotic fluid. Sometimes, it is recommended to come back for a second fetal echocardiogram or an echocardiogram on the baby after birth, if a complete study can't be obtained.

Your pediatric cardiologist will communicate with your obstetrician to make sure the results of your fetal echocardiogram are included in your record. Pediatric cardiologists also work closely with pediatricians and neonatologists (newborn specialists) to make sure that all the doctors that may be involved in the care of your baby before and after birth are aware of any issues related to the heart.

Follow-Up Care

Question:

My 15-year-old son had heart surgery when he was 14 months old. He saw his cardiologist once a year until he was 7, and we were given a good report. We moved to a new city seven years ago and have not found a new cardiologist. Other than the scar on the middle of his chest, he seems just like any other kid, but our pediatrician recommended we make an appointment with a local cardiologist. Is this necessary? Does it have to be a pediatric cardiologist?

Answer:

Almost all children who have had heart surgery need regular follow-up by a cardiologist with special expertise in caring for patients with congenital heart defects. For children, this specialist would be a pediatric cardiologist and for adults it would be an adult or pediatric cardiologist who has had extra training in caring for adults with congenital heart defects. The regular follow-up is necessary because even though heart surgery was performed, problems can happen with the heart as the child gets older. These problems can include trouble with the rhythm of the heart, narrowing or leaking of the heart valves, weakness in the heart muscle and other more rare complications.

It is important to know your son's diagnosis. If possible, contact your son's cardiologist from when he was small and request a copy of his medical records from the time of surgery and all subsequent cardiology visits. This will be very helpful when you go to a new doctor, especially in a new city. If you don't have these old records, you or your new doctor may be able to call the cardiology department where your son had surgery to get a copy of the records.

For most patients who have had congenital heart surgery, follow-up is routine and often recommended yearly. The cardiologist will examine your son looking for sites of old scars and listening to his heart. Electrocardiography (EKG), echocardiography (ultrasound of the heart), 24-hour Holter monitoring, stress testing, and magnetic resonance imaging (MRI) or CT scanning are some of the tests your son's cardiologist may recommend. The cardiologist will tell you if your child is at risk for needing future procedures (surgery or heart catheterizations) and also will describe any limitations to exercise and other strenuous activity that should be followed. You will also be told whether your child needs to take antibiotics prior to going to the dentist.

Given all the different types of congenital heart defects that exist, it is very important that a cardiologist who is knowledgeable about congenital heart disease be involved in your child's care indefinitely. Specialized centers that provide care to adults with congenital heart defects are available in some places. These centers usually have a team of physicians, nurses and other professionals who have expertise in caring for adult patients who have had heart surgery. Your pediatric cardiologist can guide you on follow-up as your son enters adulthood.

Fontan Procedure

Question:

When my boy was born, he was called a "blue baby." The doctor said his heart has only one ventricle instead of two and no blood is flowing from the heart to the lungs. He said they will have to do three operations. The last one is called a Fontan Procedure. What does he mean? Can you explain why my baby needs all these surgeries?

Answer:

Your baby was born with a very rare heart defect called pulmonary atresia. He has only one usable pumping chamber, or ventricle. The baby does have one normal main artery, called the aorta, which gives oxygenated blood to his body and two normal main veins (called the vena cavae) that bring unoxygenated blood back to the heart. To direct enough blood flow to the lungs, the two large veins that bring unoxygenated blood from the body must eventually be connected directly to the arteries that go to the lungs (the pulmonary arteries). Because the blood pressure in a newborn baby's pulmonary arteries is high, this vein-to-pulmonary-artery connection can't be done immediately. Therefore, the baby's surgery will need to be done in three stages. The first operation is a shunt or connection from the main artery, or aorta, to the pulmonary artery. The second operation is a shunt from one large vein to the pulmonary artery (the first shunt is removed at the second operation). In the third operation, the second main vein is connected to the pulmonary artery. This third stage is called a Fontan Procedure. Your baby needs these three operations in separate stages because this helps him adjust gradually to the changes in the direction of blood flow. Doing the Fontan Procedure right away would put too much strain on his heart.

Getting a Second Opinion

Question:

How do I get a second opinion regarding my child's heart problem?

Answer:

Obtaining more information about your child's heart problem is important. Never be afraid to ask your doctor questions and make sure you understand the answers. Sometimes parents feel that they would like a second opinion regarding their child's problem. Here are some helpful hints on how to get the information you need to make sure that your child is getting the best care possible.

First, talk to your child's cardiologist. Heart problems in children can be confusing, the working of the heart complex and the treatment options difficult to explain. It takes medical students many hours of classroom instruction to understand these difficult problems, so don't be embarrassed if you didn't understand or remember it all after your first or second session.

Make a list of questions. These can be emotional and difficult discussions, so it helps to have a written list of questions.

Ask for diagrams. Again, because of the heart's complexities, pictures can go a long way towards helping you understand the problem and treatment.

Sometimes children's heart problems can be emergencies that require immediate attention and might not allow time for second opinions. However, when time permits, if you still want more information or another point of view, consider the following:

Ask your child's cardiologist who to go to for the second opinion. Although this may seem awkward or embarrassing, your doctor should understand and should help you find another pediatric cardiologist in the field relating to your child's problem. If this is not possible, you may be able get a referral from your child's pediatrician. Medical care in the United States has become very subspecialized, especially for some of the complex heart problems of children. In certain circumstances, it might be even more appropriate to speak with a congenital heart surgeon or a pediatric cardiologist who specializes in areas such as cardiac catheterization, arrhythmia (irregular heart rhythm) treatment, heart transplantation, etc.

Get the pertinent records from your child's cardiologist. To make the most of your second opinion, it will greatly help the consulting specialist if he or she can have detailed records to review. Such records might include notes of office visits, catheterization reports, operative reports, videotape, CD, or DVD copies of echocardiograms or even copies of pictures obtained during cardiac catheterizations.

If possible, contact the consulting physician before the visit. The consulting physician may want to review your child's records before the visit or may ask you to bring them with you. In either case, being prepared will make it more likely that the visit will provide you with the information you are seeking. Remember to bring your list!

Complete AV Block "Heart Block"

Question:

My baby was born with a slow heart rate. We were told that she has " heart block." What is this? Her doctors also told us she needs a pacemaker, but when we hold her and feed her, she seems fine. We're scared. What caused her heart block? I feel guilty. Did I do something that caused my baby to be sick? Will she ever lead a normal life if she has a pacemaker? Will she be able to go swimming and play with other children? Will she be able to have children when she grows up? I'm sorry I have so many questions.

Answer:

The heart is a pump with a built-in electrical system. Normally, electricity starts in the upper chamber and spreads to the lower pumping chambers through special heart tissue called the conduction system or AV node. This system is like a nerve running through the middle of the heart. It permits the upper and lower chambers to contract one after the other, which permits the best pumping function.

Heart block occurs when the spread of electricity from the upper chambers (atria) to the lower chambers (ventricles) is interrupted. This interruption can be partial or complete. If partial, not all electrical signals travel from the upper to lower chambers. The heart rate may be irregular, but this may not cause problems. At other times, however, the interruption is complete and the upper and lower chambers have no electrical connection. The lower chambers pump independently of the upper chambers. This can cause problems if the heart rate of the lower chambers is too slow. Symptoms of tiredness, dizziness, headache and fainting can occur. If the rate is very slow, the heart pump function may not supply enough blood to the body. Then the heart becomes an inefficient pump and congestive heart failure can result.

When heart block is detected in a newborn, its exact cause may not be known, as in most types of congenital heart defects. Heart block usually occurs in hearts that are otherwise normal. Surgery on the heart can't fix the problem. However, if the infant's heart rate is too slow and symptoms develop, a pacemaker may be needed. Pacemakers are small battery-powered electronic devices, a little bigger than the size of two silver dollars, that remind the heart to beat at an appropriate rate to prevent symptoms of the very slow heart rate. These devices are surgically implanted under the skin in the upper abdomen or chest. A wire connects the pacemaker generator to the heart itself. The pacemaker will usually be necessary throughout a person's life and will need to be surgically replaced, usually every five to 10 years as the battery wears down. Infants with pacemakers can develop normally and lead normal lives. Later in life, they'll need to avoid some types of contact sports like football and wrestling to prevent damage to the pacemaker system.

Some children do not need pacemakers until they are teenagers. These pacemakers have their wires placed through veins leading to the heart and connected to the pacemaker in the upper chest under the collarbone.

At times, a mother with an infant who has heart block may unknowingly have a condition that causes problems with some of her own body's tissues, like skin and joints. An example of one of the types of this condition is called lupus erythematosus. Her condition can affect the developing fetus by interfering with the infant heart's conduction system. The infants themselves don't develop the skin and joint problems. Since mothers having a newborn infant with heart block may develop skin and joint problems themselves later in life, they should notify their physicians and consider having special testing.

Heart Murmur, Innocent

Question:

I was recently told that my 6-year-old daughter has a murmur but that it is an " innocent murmur" and I have nothing to worry about. My sister was born with a hole in her heart and had open heart surgery and now is on medicines because her heart is weak. I am scared that my daughter has something serious. Is there such a thing as an innocent murmur? If so, what is it? Should I be more worried about my daughter because my sister has congenital heart disease?

Answer:

Innocent heart murmurs are sounds made by the blood circulating through the heart's chambers and valves or through blood vessels near the heart. They're sometimes called other names such as "functional" or "physiologic" murmurs.

Innocent murmurs, also called functional or normal murmurs, are very common during infancy and childhood. In fact, it is estimated that at some time in their lives most children will have an innocent heart murmur. There are a few types of innocent murmurs, but all represent the normal sound of blood as it flows from the heart into the main blood vessels of the body or lungs. Normal murmurs are usually diagnosed by the typical sound heard with a stethoscope during a physical examination. Some murmurs require additional tests, such as an electrocardiogram (ECG) or echocardiogram (Echo) to distinguish them from much less common abnormal murmurs.

Heart abnormalities can on rare occasions run in the family. However, if you do not have congenital heart disease and your sister is the only family member to be born with a heart problem, then your daughter is not at increased risk of having such a problem.

Sometimes, when a doctor first hears the murmur through a stethoscope, he or she may want to have other tests done to be sure the murmur is innocent. After that, there's no need for a cardiac reevaluation unless the patient or doctor has more questions. The child doesn't need medication, won't have cardiac symptoms, and doesn't have a heart problem or heart disease. A parent doesn't need to pamper the child or restrict his or her diet or activities. The child can be as active as any other normal, healthy child.

Heart Rate

Question:

When my son is sleeping, his heart rate frequently varies at 80, 60 and 40 with a few seconds pause; no color change is noted. Is this an indication of any potentially serious problems?

Answer:

Although you did not mention your son's age, I am assuming he may be about 7 or 8 years old. If so, a heart rate of 80 beats per minute would be within the realm of normal. The heart rate will go down during sleep and it will vary from time to time, most often with his breathing pattern. However, if you are concerned about this, you should discuss this with your child's pediatrician. Because he or she will be familiar with your son's medical history and the results of any further evaluation he may wish to do, the doctor will be able to answer your question and, equally as important, give you peace of mind.

Heart Transplantation

Question:

My sister was told her daughter needs a heart transplant. Can you provide me with information about this?

Answer:

Heart transplantation involves replacing a failing heart with a new one (donor heart). Children undergo heart transplantation for two primary reasons -- a congenital heart defect or severe malfunction of the heart muscle. Heart transplantation is considered when there are no other medical or surgical options. The hearts are donated from someone who has suffered an injury resulting in death of his or her brain, but not the other vital organs and whose family has agreed to make the heart and other organs available for donation. Many children who receive heart transplants live years longer than they otherwise would have and have an excellent quality of life.

Children who need heart transplants are placed on a waiting list administered by a national agency called United Network of Organ Sharing (UNOS). The waiting time for a heart can range from days to months (even more than a year) and depends on multiple factors including size, blood type, and whether the patient is hospitalized on intravenous heart medications.

Patients who receive a heart transplant need to be on medications to prevent their own body from attacking the heart (rejection). This is because the body's defense system against infection (immune system) recognizes the heart as a foreign body and tries to damage it. Unfortunately, these medicines have many side effects, including making the body more susceptible to a dangerous infections, damaging the kidneys, and even cancer. Frequent visits (at least every three months) to the transplant cardiology team are required for close monitoring.

Hypertrophic Cardiomyopathy

Question:

Please tell me about hypertrophic cardiomyopathy in children. My 8-year-old daughter was just diagnosed with ventricular hypertrophy. When she was 2, she was diagnosed with a heart murmur and I was told that she would probably outgrow it. Can you shed any light on how this could have happened?

Answer:

The murmur your child had at age 2 likely had nothing to do with her current diagnosis. Murmurs, or "extra heart sounds," are very common in children and are usually "benign" or "innocent" murmurs. These extra sounds may arise from the thin chest wall and rapid growth seen in early childhood.

You have been told that your daughter has "ventricular hypertrophy," which is a thickening of the heart muscle. This diagnosis can be suspected by an electrocardiogram, but needs to be confirmed by an echocardiogram read by a pediatric cardiologist. Ventricular hypertrophy can occur from stenotic (blocked) heart valves, high blood pressure, or can be the result of a condition called hypertrophic cardiomyopathy. Hypertrophic cardiomyopathy is caused by several genetic defects resulting in an abnormally thick heart muscle. This disease can run in families or be the result of a new genetic mutation. It can result in poor heart function, heart failure, heart rhythm problems and in some severe cases, can cause passing out or cardiovascular collapse. If ventricular hypertrophy is confirmed by further testing, it is important that the cause be determined and the appropriate treatment and medical checkups be followed.

Hypoplastic Left Heart Syndrome

Question:

My son was born with Hypoplastic Left Heart Syndrome. He has had two operations and we have been told that he will need another one. Although his doctor has explained what he has to me, I still don't understand what is wrong with him and why he is blue and can't keep up with other children his age. Will he always be like this?

Answer:

The heart normally has two lower pumping chambers. The right ventricle normally pumps blood to the lungs, while the left ventricle normally does all the work to pump the blood to the rest of the body. In your son's case, the primary problem is that the left ventricle did not develop properly during the pregnancy and the right ventricle has to do all the work. The first two operations result in the oxygen poor blood from the upper half of the body going directly to the lungs and the rest of the blood mixing together and going to the body. The oxygen poor blood returning from the body mixes with the oxygen rich blood. The mixed blood is then pumped to supply the body, causing him to appear blue. Your son's next operation, known as the Fontan procedure, may result in much improved oxygen levels because all the oxygen poor blood will go to the lungs. This may allow him to better keep up when participating in physical activities.

Infective (Bacterial) Endocarditis

Question:

Our 4-year-old daughter was born with a small hole between the upper two chambers of her heart (it's called an isolated ASD). Her cardiologist said it would not need to be repaired. Recently, I was talking with some parents in a local support group. They said kids with congenital heart defects need to take antibiotics before going to the dentist to keep them from getting endocarditis. We've never been told that our daughter should get antibiotics, and she is about to go for her first dental visit. I called her doctor, and she said that our daughter did not need antibiotics. We are still concerned, because we don't understand why most of the kids need the antibiotics. Should I tell her dentist that she needs antibiotics? What are the antibiotics for? What is endocarditis?

Answer:

Most children with congenital heart defects are at an increased risk for developing bacterial endocarditis, an infection of the lining of the inside of the heart or the heart valves (this lining is called the endocardium). However, children with the condition called isolated secundum atrial septal defect (ASD) are not at increased risk, so that's why your cardiologist did not recommend antibiotics for your daughter.

Bacterial endocarditis (also called infective endocarditis) occurs when bacteria in the bloodstream (bacteremia) lodge on abnormal heart valves or most structural abnormalities of the heart. Certain bacteria normally live on parts of the body, such as the mouth and upper respiratory system, the intestinal and urinary tracts, and the skin. Some surgical and dental procedures cause a brief bacteremia. Although bacteremia is common following many invasive procedures, only certain bacteria commonly cause endocarditis.

Most cases of endocarditis can't be prevented, because we don't always know when a bacteremia occurs. For many years the American Heart Association recommended that children and adults at increased risk for endocarditis take antibiotics before certain dental and surgical procedures. This was done because it was thought that when someone was having one of the dental or surgical procedures that can cause bacteremia, antibiotics could be given to prevent the bacteria from surviving in the bloodstream. This is called "endocarditis prophylaxis". However, the AHA made major changes to their guidelines in 2007. After they reviewed all of the scientific evidence on the causes and prevention of endocarditis, they concluded that the prophylactic dose of antibiotics given before dental and surgical procedures prevent very few, if any, cases of endocarditis. There is also some risk involved with taking antibiotics. Therefore, the new guidelines say that antibiotic prophylaxis should be reserved only for those children and adults who have the greatest risk for a bad outcome if they get endocarditis. Patients with congenital heart disease who are at the greatest danger of bad outcomes from IE and for whom preventive antibiotics prior to a dental procedure are worth the risks include those with:

Unrepaired or incompletely repaired cyanotic congenital heart disease, including those with palliative shunts and conduits

A completely repaired congenital heart defect with prosthetic material or device, whether placed by surgery or by catheter intervention, during the first six months after the procedure

Any repaired congenital heart defect with residual defect at the site or adjacent to the site of a prosthetic patch or a prosthetic device

Except for the conditions listed above, antibiotic prophylaxis is no longer recommended for any other form of congenital heart disease.

Other people who are still recommended to take antibiotics before most dental procedures include those who have a prosthetic heart valve, those who have previously had a case of endocarditis, and a cardiac transplant patient whose heart develops a problem in a heart valve. Most of these situations would be in adults, but some could be children.

So, while your daughter doesn't need antibiotics before these procedures, children with any type of congenital heart defects listed above do need them.

Furthermore, the AHA emphasizes that for anyone at increased risk for endocarditis, good oral health and hygiene by regular tooth brushing, etc. is more protective against getting endocarditis than taking the prophylactic antibiotics.

Interrupted Aortic Arch, Double Outlet Right Ventricle, and DiGeorge Syndrome

Question:

My daughter was born with an interrupted aortic arch and a double outlet right ventricle. Please explain these heart defects to me. What are my chances of having another child with the same condition?

Answer:

Your daughter was born with several complicated heart defects that involve problems with the heart pump as well as both main blood vessels that supply the body and lungs. In this condition, the main blood vessel to the body (aorta) is interrupted in the area of the blood vessels that supply the upper body. In addition, instead of the lung artery (pulmonary artery) and aorta coming out of separate chambers, both arise from the right-sided ventricle. There is also an associated hole (ventricular septal defect or VSD) between the pumping chambers. This heart defect requires surgery during infancy. After surgery, your child will need to be seen by a congenital heart disease specialist for the rest of her life.

This combination of heart defects may be associated with a genetic syndrome called "DiGeorge Syndrome," which may also include other defects such as abnormal function of the immune system as well as the gland that controls calcium in the bloodstream. This syndrome is caused by a genetic defect on one of the chromosomes, (chromosome 22q11). The heart defects may also occur separately and can also be associated with a similar defect on the same gene. This gene defect can be detected by a specialized genetic test (FISH or fluorescent in situ hybridization). This test can determine if one of the parents also has this genetic defect or if this is a new defect (gene mutation). The results of this testing will help determine the risk of having another child with the same or a related heart defect. If neither parent has this gene defect, the risk of having another child with congenital heart disease is 4 percent to 6 percent. But if one parent has the gene defect then the odds of having a child with a similar gene defect may be 50 percent. These issues should be discussed with your cardiologist or genetics specialist.

Irregular Heart Rhythm

Question:

My obstetrician detected an irregular heart rhythm during a regular visit today. I am 24 weeks pregnant and everything else has been fine. What is an irregular heartbeat and what does it mean for my unborn baby? Will this problem go away before my baby is born? Do I need to take medication?

Answer:

An irregular heart rhythm is usually benign, but your obstetrician may refer you to a pediatric cardiologist for a fetal echocardiogram to better define the heart rhythm.

The normal fetal heart rate is between 120 and 160 beats per minute. The heartbeat is usually regular with only a little variation in the heart rate. Each normal heartbeat begins from the top part of the heart (atrium), followed by the bottom part of the heart (ventricle). The bottom part of the heart is what causes blood to leave the heart and make a sound on the heart monitor.

The most common cause of an irregular heart rhythm is when the top part of the heart beats too early (premature atrial contraction, or PAC). Sometimes this premature beat is transmitted to the bottom part of the heart; other times the communication between the top and bottom part of the heart is blocked after a premature beat.

An early beat that is transmitted or blocked will cause an irregularity in the heart's rhythm. These premature atrial contractions are very common in the second half of pregnancy and usually don't cause any problems. They may persist after the baby is born, but usually go away by one month of life. Medication is typically not necessary.

Irregular heart rhythms can cause problems in a small percentage of cases if multiple consecutive premature beats occur. When this happens for a long enough period, it's called tachycardia (fast heart rate). If the heart rate stays above 200 beats per minute for a long period of time (hours or days), it could damage the baby's heart and other organs. If tachycardia occurs, your pediatric cardiologist and obstetrician will recommend medicine for you to take to help regulate your fetus's heart rhythm. If there are any signs of damage to the fetal heart, it may be recommended that you be hospitalized while beginning the medicine. If you are put on medicine, it is likely the baby will need to take a similar heart rhythm medicine after birth.

Another cause of slow fetal heart rate is a condition called heart block, or atrioventricular (AV) block. This is more commonly seen in mothers with lupus and other related autoimmune conditions. Heart block may also be seen in fetuses with structural congenital heart disease, and may lead to excessive fluid buildup around the heart and the fetus in general - a condition called "hydrops."

Most fetuses with tachycardia have a good prognosis if the heart is structurally normal. Some fetuses have a pattern of frequent blocked premature contractions resulting in a low heart rate (bradycardia). This does not usually cause any problems to the baby as long as the heart rate remains above 60-70. Some fetuses with premature atrial contractions can have bradycardia on one visit and develop tachycardia later on. An irregular heart rhythm is not usually worrisome in the fetus, but should be followed closely by your obstetrician and pediatric cardiologist.

Kawasaki Disease

Question:

I was told that my daughter has Kawasaki Disease. What is this? Is it serious? Will she get better and lead a normal life?

Answer:

Kawasaki Disease (KD) is an inflammatory disease of unknown cause. Some evidence suggests it may be caused by an infection. KD usually occurs in children, and is very rare in adults. Children with KD have high fevers lasting at least five days, along with a variety of other symptoms. These may include conjunctivitis (inflammation of the inner eyelid and whites of the eyes), rash, swollen lips and tongue, enlarged lymph nodes, and reddened, peeling hands and feet. There is no specific test to prove that a child has KD. The diagnosis is made based on a characteristic illness pattern, and the exclusion of other diseases, such as strep throat. Most children with KD are very irritable.

Children with uncomplicated KD generally recover without treatment. However, serious complications can develop if the disease involves the coronary arteries. The coronary arteries, which supply the heart muscle with blood, can become enlarged, forming "coronary artery aneurysms." Mild degrees of enlargement may not cause problems, but significant enlargement, known as "giant aneurysms," can result in coronary occlusion (blockage). If the enlarged coronaries become blocked from blood clots, then the heart muscle supplied by the coronary artery suffers from a lack of oxygen. This can cause chest pain (known as angina) or a heart attack. Some children require medicine to dissolve or prevent blood clots. Some require urgent coronary artery bypass surgery. The dilation of the coronary arteries and the risk for blockage can be permanent.

Fortunately, a child's risk for coronary artery aneurysms can be reduced. Treatment with an intravenous medicine known as gamma globulin can help reduce this risk. Children with known or suspected KD are generally admitted to the hospital and treated with intravenous gamma globulin. They are also usually given high doses of aspirin until their fever goes away. Afterward, a low dose of aspirin is continued to prevent blood clots. An echocardiogram is performed to find out if there is any involvement of the coronary arteries. Children who remain with ongoing fevers, or who have coronary involvement, are treated more aggressively. These children may receive additional doses of gamma globulin or be treated with steroids or other medicines.

Whether your daughter will get better and lead a normal life depends partly on how well she responds to treatment. Most importantly, in depends on whether she develops coronary artery aneurysms. Most children who are treated with gamma globulin within 10 days of illness recover completely. Those without giant aneurysms usually have normal health after they recover. However, children with giant aneurysms usually require ongoing treatment to prevent blood clots. They often have significant physical limitations. These children require close medical follow-up and may require surgery.

Learn more about Kawasaki Disease.

Marfan Syndrome

Question:

My pediatrician suspects that my son has Marfan syndrome and has referred him to a pediatric cardiologist for evaluation. What exactly is Marfan and how does it affect the heart?

Answer:

Marfan syndrome is a rare disorder; it affects one in every 5,000-10,000 people. It usually "runs in the family," but your son could also be the first in your family with Marfan syndrome. Almost all body parts and organs function normally, and the brain is not affected. Many historians believe that Abraham Lincoln had Marfan syndrome. People with Marfan syndrome are tall and thin, have long arms, legs and fingers, and tend to be double-jointed. They also are likely to have curvature of the spine and an unusually shaped breastbone. Patients with Marfan syndrome tend to be near-sighted, and some can develop more severe vision problems due to shifting in the position of the lens of the eye.

Your son was referred to a pediatric cardiologist because 60 percent to 80 percent of people with Marfan syndrome develop enlargement of the first portion of the aorta -- the main artery that takes blood from the heart and distributes it to all parts of the body. For this reason, an echocardiogram helps to confirm the diagnosis of Marfan syndrome.

Problems from enlargement of the aorta in children or teenagers are very rare. Later in life, many people with Marfan syndrome develop so much enlargement of the aorta that open-heart surgery may be needed to replace the first portion of the aorta before it ruptures. Therefore, if your son is diagnosed as having Marfan syndrome, echocardiograms will probably be needed every year to check on the growth of his aorta.

Some doctors will prescribe a medication called a beta-blocker for patients with Marfan syndrome. By relaxing the heart and slowing the heart rate, beta-blockers may slow down the rate at which the aorta dilates. A new drug treatment for Marfan syndrome is currently being tested in children and adolescents with Marfan syndrome and dilated aorta. This study is sponsored by the National Institutes of Health and information about it can be found through the Pediatric Heart Network website(link opens in new window) and at www.clinicaltrials.gov(link opens in new window) .

You can find a lot more information at the National Marfan Foundation website(link opens in new window) .

Mitral Valve Prolapse (MVP)

Question:

My 10-year-old daughter has been complaining of a headache and dizziness for a week. The pediatric cardiologist diagnosed it as mitral valve prolapse (MVP) and explained there was not much to be concerned about. Please tell me what this condition is. What should my daughter avoid?

Answer:

MVP is usually a very benign condition that occurs in children and adults. The mitral valve prevents blood from going backwards by closing when the main pumping chamber of the heart (left ventricle) is squeezing or contracting. Some times the leaflets of the mitral valve moves backwards a little during the contraction of the left ventricle. In most patients this causes a minor and insignificant amount of leakage and does not affect the function of the heart.

Some patients with MVP may have a variety of symptoms such as palpitations, headaches, dizziness, or even fainting. This aspect of MVP is not related to the severity of the valve leakage; in fact, it is most common in patients with minimal valve leakage. This condition is rarely dangerous but can be uncomfortable. Rare disorders such as thyroid disease, inflammatory diseases, and defects in the strength of heart vessels and valves may all be associated with MVP and can usually be easily diagnosed when they are present, because of other signs and symptoms.

Reduction or elimination of caffeine containing food and drinks (coffee, soda, chocolate) is often helpful in making the headaches, dizziness and palpitations go away. Many patients improve with an increase in their daily fluid and/or salt intake. Some patients may require a medication to offset the effect of the adrenalin. These medicines, called beta-blockers, will lower the heart rate and blood pressure, and help to reduce or eliminate symptoms. Almost all patients with MVP can participate in sports without restrictions.

Although many physicians may suspect MVP when listening to the heart, echocardiography (ultrasound of the heart) is the only way to definitively diagnose MVP. It is also important to know that MVP is the most over-diagnosed heart condition and should only be made and treated by an experienced pediatric cardiologist.

Pulmonary Hypertension

Question:

I have been told that my 6-year-old daughter has pulmonary hypertension. What does this mean? Is it the same as the hypertension that has been diagnosed in my mother?

Answer:

Pulmonary hypertension is a condition in which pressure in the blood vessels in the lungs is higher than normal. This is different from systemic hypertension where pressure in the blood vessels to the body is higher than normal. Systemic hypertension is a common condition especially in people as they age. It is diagnosed by taking a blood pressure in the arm. Systemic hypertension is usually successfully treated by oral medications. In contrast, pulmonary hypertension is a rare condition in both children and adults. Patients usually present to the doctor with symptoms of fatigue, chest pain, difficulty breathing with activity and episodes of fainting or nearly fainting. As you can imagine, these symptoms are difficult to get a handle on, especially in infants. Thus, pulmonary hypertension is often diagnosed late, after the patient has had vague symptoms for some time. Pulmonary hypertension can be diagnosed by echocardiography (cardiac ultrasound), although cardiac catheterization is often recommended to confirm the diagnosis and as a way to better understand the cause.

There are two broad classes of pulmonary hypertension: those without any obvious cause (termed idiopathic) and those caused by or occurring with other diseases. Diseases that can cause pulmonary hypertension include:

lung disease (such as immature lungs in premature babies or airway obstruction)

some congenital heart diseases with excessive blood flow to the lungs or blockage of blood flow from the lungs back to the heart

diseases of the clotting system of the body

diseases of inflammation (such as systemic lupus erythematosus, SLE)

some drugs and toxins

HIV infection

certain blockages in the drainage of the liver (portal hypertension)

When possible, the best treatment for pulmonary hypertension is treating the underlying disease. When this is possible, the pulmonary hypertension may resolve or at least improve. In situations where treatment of the underlying disease does not improve the pulmonary hypertension and in situations of idiopathic pulmonary hypertension, medications are available. In the past, these medications were only intravenous and often involved long-term intravenous therapy. Inhaled and oral medications have more recently become available. Although rare, lung transplantation has been used successfully in severe cases.

For more information on pulmonary hypertension, visit the Pulmonary Hypertension Association's(link opens in new window) website.

Pulmonary Valve Stenosis

Question:

We were told that our child has pulmonary valve stenosis. His heart valve doesn't open completely. What can be done to fix the valve?

Answer:

The pulmonary valve is like a one-way swinging door on the right side of the heart that prevents back flow of blood from the lungs to the heart. Sometimes, during heart development, the valve does not form properly. If the valve is tight (stenosis), the right side of the heart needs to work harder to push blood through the valve to the lungs. Depending on how tight the valve is, it may or may not need to be fixed. Your congenital heart specialist can discuss this with you. If the valve is only mildly tight, it can be left alone and may get better as your child gets older. However, if the valve is tight and causes the heart to work harder than it should, it may need to be opened with a small balloon using a special tube (catheter) placed into the heart from a blood vein as part of an interventional catheterization procedure. If the valve is very tight or malformed, it may need to be surgically opened or replaced.

Single Ventricle

Question:

My baby was just diagnosed with a single ventricle. What does this mean?

Answer:

The normal heart has two pumping chambers or ventricles. " Single ventricle" refers to congenital heart defects where one of these chambers is absent or very small, or defects where the partition between the two pumping chambers is missing. These defects are often called hypoplastic left heart or hypoplastic right heart. Hypoplastic left heart includes both aortic and/or mitral atresia (absence of the valves on the left side of the heart). Hypoplastic right heart includes a wide range of defects with a small or absent tricuspid valve or pulmonary valve (the valves on the right side of the heart). In addition, a single ventricle occurs if there is no partition between the two pumping chambers. These defects can result in severe heart failure (not enough blood to the body) or severe cyanosis (not enough blood to the lungs). All of these defects require a common treatment designed to ensure blood flow to both the lungs and the body. Sometimes this requires limiting blood flow to the lungs and creating a new blood vessel to provide blood flow to the body. Other times, operations are required to ensure blood flow goes to the lung arteries. Ultimately, most babies with single ventricle need two or three operations that result in the channeling of oxygen poor blood to the lungs and oxygen rich blood to the body. There are good surgical options for most babies with single ventricle and many children with this condition can do most normal childhood activities. Your pediatric cardiologist can give you the best information on the specifics of your child's condition.

Sports Participation

Question:

My 10-year-old daughter had a hole in her heart repaired when she was 1. She has been healthy since then. Will she be able to participate in sports?

Answer:

Questions about athletic participation in patients with heart problems are frequent. There are a wide variety of cardiac defects, surgical repairs, and sports. It is best for you to discuss with your pediatric cardiologist as to whether your daughter needs any restrictions from sports. This is particularly true if she wants to eventually participate at the high school or college level. Most children who have relatively simple cardiac defects, and who have had a good result after catheter or surgical repair can participate in at least some, if not all, athletic activities. Other children may have heart conditions that warrant some restrictions. Patients with pacemakers or implantable cardioverter/defibrillators (ICDs) may have restrictions not required for other patients with heart defects. Patients that are taking anticoagulants (drugs that decrease blood clotting) should also use caution and check with their physician about sports participation

Transposition of the Great Arteries (DTGA) and Genetics

Question:

We have a 1-year-old son who is doing well after surgery for transposition of the great arteries (DTGA). We'd like to have another child. Will our second child also have congenital heart disease?

Answer:

Congenital heart disease is the most common birth defect. About eight out of every 1,000 babies (or 0.8 percent) will be born with some form of heart problem. Most of these problems, however, are milder than your son's. For a couple who's had one child with DTGA and has no other family members with transposition, the chance of having another child with DTGA increases slightly to about 18 in 1,000 or 1.8 percent. This is also about the same chance of this baby being born with a form of congenital heart disease other than DTGA. This gives a greater than 98 percent chance that the baby will be born with a normal heart.

If, however, other family members (first-degree relatives) have DTGA, other forms of transposition, or other forms of congenital heart disease, the chance that the baby will be born with DTGA may be higher. Transposition rarely runs in the family.

We don't know why congenital heart defects like DTGA occur. The reason is presumed to be genetic, although a specific gene for DTGA hasn't been identified yet. With regard to DTGA, some evidence exists that multiple genes as well as other factors may be involved.

Because you've had one child with a congenital heart disease, it may be helpful for you to have a fetal echocardiogram between 18 and 20 weeks gestation during your next pregnancy. This specialized ultrasound is done by a pediatric cardiologist and shows the developing heart. It should be able to exclude most forms of significant heart disease. Most often the fetal ultrasound is a reassurance that the baby's heart is developing normally.

Tricuspid Valve Abnormalities

Question:

My 4-year-old son was evaluated for a heart murmur today and underwent an echocardiogram. The doctor told me he had mild tricuspid valve regurgitation. What does this mean? Should I be worried?

Answer:

The tricuspid valve is one of four heart valves in the heart. This valve prevents blood from leaking backwards from the right ventricle (the chamber of the heart that pumps blood to the lungs) when this chamber squeezes.

Almost all children undergoing echocardiography (ultrasound of the heart) have a little bit of tricuspid valve leakage or regurgitation. This is often detected because newer echocardiography machines have technology sensitive enough to pick up a wide variety of normal signals from blood moving through the heart. Mild tricuspid valve leakage, in the absence of any other heart problems, is considered to be a normal finding and nothing to worry about.

Ventricular Septal Defect (VSD)

Question:

My 5-day-old son has been diagnosed with ventricular septal defect (VSD). The size of the hole is about 2 mm. Is this considered large or small, and what are long-term and short-term effects of VSD?

Answer:

A ventricular septal defect (VSD) 2 mm in diameter is considered small. Because it is small, one would not expect your baby to have any problems. This VSD will likely close completely as your baby grows, depending somewhat on the precise location of the hole. This could occur quite soon (within months) or take a few years. In general, a VSD that has not closed by about age 5 will not close spontaneously. VSDs that are very small do not become big as babies grow.

Two complications from small VSDs are worth knowing about, both relatively rare. First, there is a small risk that a child or adult with a small VSD will develop an infection in the heart (endocarditis). These infections are bacterial and can occur after events when bacteria are in the blood.

The other complication from a small VSD depends on the precise location of the hole. People with a VSD immediately below the aortic valve (the valve at the exit from the left side of the heart to the body) have a small chance that one of the leaflets of that valve may start to sag towards the hole over time. This rarely happens, but when it occurs, can lead to leakage of the valve. For the rare occasion that it does, your physician might recommend surgical closure of the small VSD to prevent further problems with the aortic valve.

Wandering Pacemaker

Question:

My 8-year-old son complained of chest pain and rapid heartbeat while taking a shower. This happened twice. He was on Flovent and Singulair for asthma. His doctor assures me that the medication had nothing to do with the chest pain. My son had an EKG and the pediatric cardiologist said it showed a wandering pacemaker and no further evaluation is necessary. Should I get a second opinion?

Answer:

The diagnosis of wandering pacemaker refers to a type of heart rhythm with normal variations in heart rate. The pacemaker is an area of the heart responsible for initiating the heartbeat. It's normally located in the upper part of the right atrium (the upper right chamber of the heart). Sometimes the wandering pacemaker can move from the high right atrium to the low right atrium.

Chest pain can be associated with an asthmatic condition. So, in your son's case, chest pain alone would not be a cause for concern about his heart. Some asthma medications can also cause a transient increase in heart rate, but the medicines your son is taking generally don't do this. Rarely, patients may experience a heart rhythm problem called tachycardia (an abnormally fast heart rate).

If your son's perceived fast heart rate persists, your doctor may want to consider more testing. A transtelephonic EKG monitor is a device that can be used when symptoms occur. The patient presses a button on the recorder and the heartbeat tracing is saved in the monitor's memory. This tracing can then be sent over the telephone to a cardiologist. Another test is called a Holter monitor. It records an EKG continuously for 24 to 48 hours, while the child is at home, both active and asleep. Either of these two tests can then be analyzed to detect any abnormal heartbeats and determine whether further testing or treatment is needed.

Your son's symptoms are most likely related to his asthma, but if these continue your doctor may want to consider more testing, including the two tests just described. In addition, if your son experiences any dizziness or fainting, your doctor should be notified at once.

It's important that you're comfortable with the diagnosis of the first pediatric cardiologist who examined your child. If you aren't, then seek another opinion

Wolff-Parkinson-White Syndrome

Question:

My son was diagnosed with WPW. What is this? Will he need treatment? Will he need surgery or a pacemaker?

Answer:

Wolff-Parkinson-White Syndrome is a heart rhythm problem that can usually be detected on an electrocardiogram (ECG or EKG).

Drs. Wolff, Parkinson and White published the first article about this type of rhythm in 1930. Wolff-Parkinson-White (WPW) means that there is an extra electrical connection from the top chambers of the heart (atria) to the bottom chambers (ventricles).

Sometimes, an abnormal rhythm occurs when heart electrical signals travel in the wrong direction, from the ventricles to the atria, across the extra electrical connection. This heart rhythm causes the heart to beat too fast (tachycardia). Sometimes patients may feel dizzy or nauseous during tachycardia, and fainting may occasionally occur. Medication may be used to slow the heart rate. In rare patients, the condition can be life-threatening.

If medication fails to control the tachycardia, or in patients at higher risk of life-threatening events, or if patients are averse to medical therapy, a procedure called catheter ablation may be used. It's done in the cardiac catheterization laboratory under sedation or anesthesia. It involves inserting several specialized catheters into the blood vessels in the groin. These catheters can locate the origin of the abnormal heartbeats in the atrium. They abolish the extra electrical connection using radiofrequency waves (heating) or cryoablation (freezing). Since the 1980s, the procedure has been performed in tens of thousands of children safely and effectively, and provides a permanent cure more than 90 percent of the time. Some patients and physicians prefer ablation over medications as first-line therapy for WPW.

It's important for each patient to discuss questions and concerns directly with their cardiologist to understand how these recommendations apply to their specific circumstance.

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Commonly Asked Questions About Children and Heart Disease

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My 4-year-old daughter was recently diagnosed with a hole in her heart. It is called an atrial septal defect. We were told that it should be closed. I heard that there is a new way to close the hole without having surgery. Can this be done any way other than with surgery?

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A minha primeira experiência com crianças antes da COP. Uma rica experiência diga-se de passagem e muito gratificante. ❤️❤️

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O jacaré e o ukulele invadiram o nosso box 🐊🪕

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O jacaré e o ukulele invadiram o nosso box 🐊🪕

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Os preparativos para a manhã do dia 02/02/2023 começaram bem antes de chegarmos ao módulo: planejamento, ideias, instrumentos, música, lápis de cor e pintura foram tramados para a manhã desse dia. 👶🏻🎉
Foi dia de encontrarmos o nosso paciente e continuar com o seu tratamento, entretanto foi um dia atípico: pela primeira vez nosso paciente chorou, mas com o apoio da mãe e com o auxílio da minha dupla Beatriz, nós conseguimos realizar todas as etapas propostas. 🏆🙏🏻
Iniciamos nosso primeiro contato de forma descontraída, vocês conhecem a musiquinha do jacaré? 🤔🎶
O JACARÉ FO PASSEAR LÁ NA LAGOA (2X)
FOI POR AQUI 👏🏻👏🏻👏🏻
FOI POR ALI 👏🏻👏🏻👏🏻
AÍ ELE PAROU, OLHOU, VIU UM PATINHO, ABRIU O SEU BOCÃO E NHACK 🐊🦆 e falamos com ele que seria o dia dele abrir o bocão de jacaré para deixarmos o sorriso dele lindão.
Também demos para ele um desenho do jacaré nadando na lagoa e dessa maneira começamos o atendimento. 🦷👶🏻
Foi uma manhã de muito aprendizado, às vezes, as coisas não saem como no script, entretanto, com o auxílio das aulas teóricas de como nos comportamos em determinadas situações, conseguimos realizar as 2 restaurações e no final, recebemos o sorriso que sempre recebemos nos atendimentos, o beijo e o abraço de urso do nosso pequeno. 💗

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A manhã do dia 02/02/2023 começou em grande estilo: com clínica de COP 1 👶🏻🍼🎉. Foi o momento de reencontrar os amigos/incríveis colegas e os professores que têm nos ensinando tanto nesse período que estamos juntos. E para começar com chave de ouro, depois de uma manhã repleta de aprendizados, nós registramos este soflagrante, quero deixar aqui a nossa foto! 📸😍

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A tarde do dia 02.02.22 foi extremamente gratificante! Dia em que o meu querido paciente retornou para dar continuidade ao tratamento proposto. Foi bastante colaborativo e participativo em todo o processo até a realização da exodontia da unidade 36, o qual se encontrava com lesão periapical. A cirurgia foi realizada com muito sucesso, além do que, o paciente vem tendo uma grande evolução nos cuidados de higiene oral, tendo uma melhora significativa na saúde gengival, fruto de muita conversa e instruções corretas de higienização.  Lembrando que tivemos participação especial do meu querido LUIZINHO, me auxiliando nessa tarefa. Obrigada, Lu.  PS; Felipe estava cuidando da saúde oral de Saulinho.

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Vamos aprender mais sobre os dentes decíduos?
Cuidado!!! Tem muita gente que ainda confunde dente permanente com decíduo, às vezes o dente decíduo tem comprimento MD maior que o permanente.

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Finalizando os atendimentos de 2022 com esse rapazinho muito gente boa e dorminhoco! Bom demais ter um paciente assim, não é mesmo Evy?! Até pra exodontia a criança dorme. Excelente!
Pro próximo ano desejo mais pacientes bonzinhos como esse hahaha.

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Dia 15/12 foi a última clínica do ano, regada de muita alegria e contagiada pelo espírito natalino. Nessa clínica atendi um paciente especial, que requer um olhar cuidadoso e atenção, como qualquer outro paciente. Foi feita toda a parte inicial do seu tratamento, para quando retornamos, dar continuidade.

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No dia 01 de dezembro foi realizado a sala de acolhimento com a finalidade de acolher e também, de transmitir conhecimento para todos que se faziam presentes. Nesse dia, eu abordei a importância da vacinação, tanto em relação as crianças, mas também de um modo geral. Frisando a pandemia que vivenciamos e as suas consequências. Foi um momento bastante proveitoso, onde compartilhamos conhecimentos e experiências. No mais, não poderia de deixar de registar, a participação especial do nosso querido professor Carlos, que ao me ver apresentando, correu pra participar da "plateia" kkkkkk. # Completamente Complexada.

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É coisa, viu?

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65K likes, 2,705 comments - lizandra.macedo.79 on November 10, 2022

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A post shared by Lizandra Macedo (@lizandra.macedo.79)

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Esse é um registro do fim da primeira clínica. Olha esse olhinho brilhando por ter atendido um doce 🤌🏽🥰

Mas o mais importante foi o suporte que  Rhanna me deu. Sem seu apoio não iria fluir da mesma maneira. Obrigada, dupla! 🤝

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O dia em que chegamos às 6 horas da manhã para arrasar e só atendemos às 10. 😂😂😂😂 No fim deu tudo certo, graças à minha dupla maravilhosa @vinicius

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Experiência e inovadora poder levar alegria para os pequeninos do abrigo através de jogos e socialização.

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No segundo dia de atendimento eu e a minha dupla Beatriz tivemos mais uma experiência: um novo paciente. -A, de apenas 3 anos (apesar da pouca idade) me ensinou muitas coisas nesse dia. A paciência e cooperação dele durante o atendimento foi crucial para termos êxito e a ajuda da mãe A.G também foi fundamental para estabelecermos uma boa relação naquele momento. Minhas expectativas aumentaram após esse encontro, confesso que saí da clínica com um mix de sensações, mas acima de tudo: FELICIDADE. 👶🏻💕🥳

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Esse baby foi um menino ótimo e muito colaborativo  com apenas 2 aninhos de idade, foi o nosso primeiro paciente na odontopediatria e também seu primeiro atendimento em consultório odontológico! A experiência foi desafiadora e  bem diferente do que estamos acostumados, porém o atendimento transcorreu em perfeita harmonia, dando tudo certo no final!
E ainda colaborou conosco para fazermos a posição de joelho a joelho que é indicado para criança nesta faixa etária! 🥳🤗😚

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S. é possui Trissomia do cromossomo 21, e desde início eu me apaixonei! É um paciente inteligente, esperto e muito especial, que requer uma maior atenção e cuidado. É paciente de Felipe, mas ganhou o meu coração! Com os cuidados certos, a saúde oral vai melhorar 100%.

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Não sou de chegar atrasada, mas vim me apresentar com atraso, juros e multas... Quase uma nômade, mas não por opção, nascida em SP, com um pezinho soteropolitano e outro barrestivense, aqui estou eu, em Jequié atrás de um sonho, (ser dentista) o que não está sendo uma tarefa tão fácil, mas creio que iremos todos finalizar essa etapa de nossas vidas em breve.
A pediatria desde o início me trazia pesadelos, mas há um bom tempo busco novas percepções sobre a odontopediatria, para que todos os bloqueios com crianças sejam quebrados e as experiências as melhores possíveis.

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A minha primeira experiência como operadora foi relativamente desafiadora! Fico até me perguntando de como irei relatar os acontecimentos em relção ao atendimento, mas enfim! O meu paciente é um rapaizinho de 11 anos de idade que precisava extrair os dois dentinhos( 73 e 83), pois ambos apresentavam rizólise completa/ retenção prolongada do 33 e 43. Entretanto, ele tinha uma experiência / lembrança não muito boa em procedimentos anteriores, fato esse, que acarretou a episódios de medo, choro e insegurança. Entretanto, mesmo com os desafios encontrados durante o atendimento, pude perceber o quanto a odontopediatria é um local de descobertas, de aprendizagem, de acolhimento, mas  também de transmitir segurança, ser firme no seu posicionamento quanto profissional e saber conduzir a situação compreendendo a individualidade. ( Mas também se não colaborar o professor Nilton manda ir pra Narnia, ficando a critério do paciente, kakaka). Contudo, a experiência está sendo enriquicedora e fico feliz e grata de poder estar vivenciando tudo isso!

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Nosso primeiro paciente foi o P, um menino tão colaborativo e amoroso, de verdade eu estou encantada por ele. Ele mesmo nos escolheu, chegou no box com mais um coleguinha e ele decidiu que iria ficar por lá mesmo. Já me encheu de perguntas, uma atrás da outra, queria saber de tudo!!!
A COP está sendo ótima, esse primeiro momento está sendo muito bom mesmo e pessoalmente é um momento confortante, ainda mais com esse paciente que já é super especial pra mim!
Sabemos que cada paciente reage de uma forma, mas nos esforçaremos para lidar da maneira adequada com todos.

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A primeira clínica foi um misto de sensações aliado ao desafio de conquistar o coraçãozinho do Pyetro. No fim todas as coisas dão certo, com muita conversa, músicas e histórias. Rs

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A criançada atenta e muito esperta!

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A  primeira clínica foi muito enriquecedora, mesmo ainda aprendendo a trabalhar com as crianças o primeiro contato foi tranquilo e eu amei a experiência, interagir com as crianças é muito gratificante podendo sentir o amor e a inocência que elas emanam. Espero que as próximas sejam ainda melhores!!! 😍😍😍
OBS: Estou apaixonada pelo meu pequeno paciente K. 🤩🤩🤩

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Ao término da primeira clínica de COP I um misto de sentimentos e expectativas se fazem presentes, à medida que o contato com os pequeninos (uma criança de 2 anos e outra 7 anos) é capaz de estimular o olhar atento as necessidades, mas também, as particularidades de cada atendimento. Cada criança costuma comportar-se de maneira variada e desde o início (eu e minha dupla) buscamos conduzir os acolhimentos e procedimentos com tranquilidade e paciência, sempre com o auxilio do professor Nilton.
Sendo assim, tratou-se de um momento com inúmeros aprendizados e adaptações profissionais, a começar pelo correto preenchimento das informações dos pacientes até a agilidade da conduta clínica. Já iniciamos os planejamentos individuais, que serão realizados nas próximas semanas. Fiquei bem feliz com a colaboração das crianças!

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A primeira clínica foi mais tranquila do que esperava. A paciente que iriamos atender não compareceu, aguardamos para ver se chegava mais pacientes. No fim, atendemos um pequeno que estava na clínica com a mãe para a consulta do seu irmão. Ocorreu tudo da melhor maneira possível!! O paciente muito colaborativo! Apaixonei <3

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Olá! Me chamo Carla Machado, tenho 23 anos e sou natural da cidade de Livramento de Nossa Senhora-Ba. Em relação as expectativas sobre Odontopediatria são bastante positivas! Espero que seja um momento de aprendizado e de superação do novo. Que possamos ao fim sair com uma bagagem cheias de novas experiências e gratos por todo o processo!

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Me chamo Vitória, tenho 23 anos e sou da cidade de Brumado. Cursar odontologia sempre foi um sonho, e me sinto realizada em poder viver esse sonho. Tenho boas expectativas para a odontopediatria, mas também carrego comigo anseio e medos diante dessa nova fase. Mas estou aqui de coração aberto pra viver essa fase, e espero que seja muito proveitosa e de bastante aprendizado!!

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Ou você prefere partir de moto sem destino certo?

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Esse é Heitor Ravi, primo e afilhado. Uma "bença"!

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Primeira experiência na clínica foi incrível!! Ainda melhor do que eu esperava 😍 Obrigada ao Professor Nilton, a monitora Evelin (minha xará) e a minha duplinha Maislla pelo suporte, ocorreu tudo da melhor maneira possível 🥰

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Primeira experiência na clínica de Odontopediatria

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Primeira experiência na clínica de Odontopediatria

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Foi extraordinário. Gente, eu não sei nem como passar o quão emocionante foi esse primeiro contato. Com as orientações do professor Nilton, seu apoio e sempre ali solicito, tudo ficou mais fácil e amistoso. Não imaginava que fosse me apaixonar tanto por estes pequenos 😍 Aiii meu Deus, e agora? (Kkkk). Fica aqui os meus agradecimentos ao preceptor Nilton, que esteve ao nosso lado e à minha duplinha Érika. Estou muito feliz e com grandes expectativas da clínica. AMEI!!!🌹❤️

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Nosso primeiro contato com os pequenos logo pela manhã, na recepção. Olhares atentos, apreensivos e curiosos. Para o primeiro contato, elaboramos e contamos uma historinha e após isso, falamos sobre a importância da escovação. Logo depois, veio o tão esperado mundo mágico da Odontopediatria.

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Bom, eu me surpreendi. Enquanto eu organizava o box eu pensava: "Meu Deus, tu sabe de todas as coisas e o que eu menos quero é criança birrenta", porém minha paciente tinha 12 anos... Não foi como eu imagina, que eu iria perder a minha paciência por atender uma criança assustada, mas foi legal, mesmo diante da timidez da minha paciente a gente conseguiu conversar um pouco, e ela não me deu trabalho nenhum.

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Que rufem os tambores a COP I começou! 🥁🥁

Definitivamente, ontem foi um dia de experiências únicas e gratificantes. O momento de acolhimento com a histórinha da cárie de Tomé e a participação dos pequenos foi maravilhoso. Todos aqueles olhinhos vidrados e focados em nós me fez sentir um mix de sentimentos. Alegria por poder estar ali com eles, mas confesso que também estava com um pouco de receio em como proceder e como seria atender uma criança. Afinal, seria algo novo, a minha primeira vez com os pequenos pacientes. MAS NO FINAL DEU TUDO CERTO. Atendemos K., e em meios aos sorrisos, bocão de jacaré e cócegas (que a escovinha fazia no dente) pudemos completar a atividade proposta para o dia.
(Na foto acima, eu e minha dupla Beatriz atendendo K.)

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Agora foi, não tem mais como retroceder.

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Agora foi, não tem mais como retroceder.

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É caminhando que se aprende a caminhar. Partilhe como foi a sua primeira experiência de atendimento na Clínica Odontopediátrica!

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Me chamo Leila, mas poderiam facilmente me chamar de Maria APARECIDA! Sou natural de Vitória da Conquista - Ba e tenho 25 anos.
O curso me trouxe gratas surpresas, grandes encontros e experiências que me transformam diariamente. Por não querer ouvir a vivência de outros alunos sobre disciplinas, professores e afins, vou lidar com a odontopediatria da minha forma, aprendendo tudo o que posso. Não espero "que seja lindo, que seja leve", espero desenvolver a capacidade de gerenciar problemas, de ter uma postura profissional e de levar saúde aos pequenos.

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Meu nome é Maislla Mayara, logo se vê que é "fi de pobre", porque pobre que gosta de inventar arte nos nomes dos filhos. Mas estou há 24 anos lidando com esse nome e às vezes até eu estranho kkk
Sou de Maracás, cidade das flores!

Encaro a odontopediatria realmente de coração aberto, disposta a aprender tudo o que for possível, dando sempre o meu melhor e me esforçando pra servir, ser útil, saber ouvir os que precisam e ajudá-los da melhor maneira que eu conseguir. Sempre reconhecendo minhas dificuldades e buscando ajuda.

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Olá, professores! Certa vez eu li a seguinte frase "Se enxerguei mais longe, foi por estar sobre o ombro de gigantes". Essa afirmação de Isaac Newton me faz ressignificar diversas ações em minha vida, principalmente com relação as minhas escolhas. Nesses últimos meses tenho experimentado de coisas inovadoras em minha jornada pessoal e também acadêmica. As oportunidades que têm surgido são como os gigantes que me colocam sobre os ombros e me permitem enxergar um futuro encantador. Cada experiência com disciplinas novas me faz encarar a odontologia como algo sólido para minha carreira, possibilitando maior firmeza na minha escolha. Eu não sou apaixonado pelo curso, como achei que eu seria, muito menos pela pediatria, mas eu encaro tudo isso como um desafio, que independente do resultado me fornecerá uma experiência pessoal podendo ser boa ou ruim. Mas espero que essa nova experiência seja legal, divertida e que eu consiga experimentar de momentos marcantes com cada um dos meus pacientes. Bom, e eu tenho 23 anos e me chamo Luís Victor.

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Eu sou o Vinícius, nascido em Jequié, mas criado em Barra da Estiva-BA. Numa fazendinha bem legal. Sou do interior e para conquistar o tão sonhado objetivo de estar na faculdade, tivemos, meus pais e eu, que vencer alguns obstáculos nesse meio curso… Mas graças a Deus e aos nossos esforços, cá estou eu, na realização do SONHO! 🙏🏼🥰

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Olá! 🙋🏽‍♀️ Meu nome é Evylin Leal, sou natural de Gandu-Ba e a área da saúde sempre foi um sonho/meta. Hoje cursando odontologia posso ver esse sonho tomando forma e a cada passo me sinto mais feliz e realizada. Espero que esse próximo passo da caminhada (denominado de Odontopediatria) seja leve e recheado de boas experiências e aprendizados, estou entusiasmada e confiante que dará tudo certo! 😍 Que venham as clínicas.

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Olá! 🙋🏽‍♀️ Meu nome é Evylin Leal, sou natural de Gandu-Ba e a área da saúde sempre foi um sonho/meta. Hoje cursando odontologia posso ver esse sonho tomando forma e a cada passo me sinto mais feliz e realizada. Espero que esse próximo passo da caminhada (denominado de Odontopediatria) seja leve e recheado de boas experiências e aprendizados, estou entusiasmada e confiante que dará tudo certo! 😍 Que venham as clínicas.

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Eu sou Beatriz Brandão, tenho 23 anos sou de Itacaré, amo praia e natureza, nesses lugares me sinto completa e cai de paraquedas no curso de odontologia tem dias bons e ruins, mas os dias bons prevalecem e a experiência do curso está me fazendo cresce muito.
Gosto muito de crianças tenho expectativas positivas sobre a clínica, mas tenho  um pouquinho de medo da nova  experiência.

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Nordestina, Baiana, família simples, primeira pessoa da cidade do
São Félix do Coribe a ingressar na UESB. Nomeada como Raíza e
acariciada como Raí! Desde criança que sonho em me formar, já quis
ser , dançaria, professora e até arquiteta sempre muito inquieta por
que meu sonho mesmo sempre foi voar! A partir de então, formei
em uma profissão que estava disposta aprender a amar , técnica em
enfermagem, quem diria de passagem que um dia iria me apaixonar,
e foi por essa paixão ao zelar que decidi me ingressar na arte do
cuidar, “AAAAAA odontologia”, profissão de muitos lampiões, mas
marcada mesmo por Joaquim Xavier, conhecido como Tiradentes,
que sempre esteve presente nos livros de historiar. Assim, encerro

meu cordel agradecendo a Deus do céu por sempre me abençoar!

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Assim eu me apresento na Clínica Odontopediátrica

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Assim eu me apresento na Clínica Odontopediátrica

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Elabore um vídeo de apresentação que deve ser enviado previamente ao seu paciente ou exibido antes do atendimento.

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A post shared by Marcio Lobo (@marcioplobo)

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Conte-me um pouco sobre você, suas expectativas, sua vida e o que você espera da Odontopediatria.

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Olá, eu sou Anny! Tudo bem?

Natural de Caculé, município do interior do sudoeste da Bahia, onde ''todo mundo conhece todo mundo''. Tenho 22 anos e sou a filha mais nova de uma família pequena, núcleo marcado pelo apoio através do cuidado e incentivo diante dos detalhes rotineiros. A odontologia surgiu como mecanismo de promoção do cuidado com os outros, ou seja, durante a graduação venho tentado colocar este princípio em prática.

A disciplina de odontopediatria está estimulando a criação de expectativas positivas pelo aprendizado, mas também, a apreensão perante o novo e o público alvo dos atendimentos. Espero que as clínicas sejam caracterizadas pela alegria e/ou leveza das crianças, o entusiasmo da infância e o companheirismo da turma 2018.2.
Seja bem vinda, odontopediatria! Segura na mão de Deus e vamos!

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A post shared by 🦋 Ellen Cristina 🦋 (@ae_cristina18)

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Como não amar essa energia, pureza, amor e liberdade?

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Bem vindxs ao maravilhoso mundo da Odontopediatria!

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Bem vindxs ao maravilhoso mundo da Odontopediatria!