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1. Choose a spokesperson

2. All students in the group are to work on all of the following questions/sections included in the Padlet

3. You can use diagrams/images other visual resources to explain each respiratory condition.

4. When we come back to the whole class, the spokesperson will feedback the groups findings, with the teams support

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Acute pulmonary oedema (APO) refers to the rapid buildup of   fluid in the alveoli and lung interstitium that has extravasated out of the pulmonary circulation. As the fluid accumulates, it impairs gas exchange and decreases lung compliance, producing dyspnoea.

Cardiogenic APO is caused by a failure of the left ventricle to contract enough bloods to the lungs. As a result, the left ventricle is unable to pump blood efficiently and leads to build up back pressure into the left atria and the lungs.

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2. Is it obstructive or restrictive? Why?

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2. Is it obstructive or restrictive? Why?

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APO is restrictive as it reduces compliance of the lungs

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5. How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

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5. How is oxygenation and ventilation affected? Eg. consider O2 content, O2 delivery, alveolar ventilation 

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Increase in A-a gradient, impaired diffusion of O2 across the alveolocapillary membrane. Low O2 content. Low Pao2

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3. How does the condition affect the lung compliance or airway resistance? 

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3. How does the condition affect the lung compliance or airway resistance? 

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Lung compliance is reduced due to fluid filled in alveolar, difficult for the lung to expand and recoil.

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6. What type of respiratory failure would this condition lead to?

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6. What type of respiratory failure would this condition lead to?

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Type 1, could be type 2 if they start to retain Co2

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7. What are the typical ABGs and whyPaO2?

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7. What are the typical ABGs and whyPaO2?

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Type 1=hypoxemia
Normal or high pH
Low PaO2

If patient gets tired (hypoventilation),
can lead to Type 2 resp failure
High CO2, low pH, low PaO2, high bicarb

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8. What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

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8. What are the typical clinical manifestations/ patient assessment data? (relate these to pathophysiology concepts already described)

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Pallor, oedema, SOB, increase in WOB, poor peripheral perfusion, high mucus secretion (thick), crackles, tachypnea, dyspnea,

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9. OPTIONAL: Important vocabulary (include list of key vocabulary referenced; if you do not know the term(s), look up and add a definition for the word(s)): 

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9. OPTIONAL: Important vocabulary (include list of key vocabulary referenced; if you do not know the term(s), look up and add a definition for the word(s)): 

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1. Hypoxaemia
2. V/Q mismatch
3. Edema
4. Restrictive lung condition
5. Low lung compliance
6. impaired gas exchange
7. dyspnoea
8. Cardiac causes :
Acute coronary syndrome (ACS)
Cardiac arrhythmia e.g. atrial fibrillation (AF)

Valvular heart disease

Hypertension

Cardiomyopathy

Cardiac tamponade

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4. Explain the role of V/Q mismatch in this condition.

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4. Explain the role of V/Q mismatch in this condition.

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Low ventilation and high perfusion
=low V/Q ratio
shunt